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_j L 91159 ,. . 1111111111111111111 NZ~ SUPERVISOR'S USE ONLY NEW ZEALAND QUALIFICATIONS AUTHORITY MANA TOHU MATAURANGA 0 AOTEAROA Level 2 Biology, 2013 91159 Demonstrate understanding of gene expression 9.30am Friday 22 November 2013 Credits: Four Achievement Demonstrate understanding of gene expression. Achievement with Merit Demonstrate in-depth understanding of gene expression. Achievement with Excellence Demonstrate comprehensive understanding of gene expression. Check that the National Student Number (NSN) on your admission slip is the same as the number at the top of this page. You should attempt ALL the questions in this booklet. If you need more space for any answer, use the page(s) provided at the back of this booklet and clearly number the question. Check that this booklet has pages 2-9 in the correct order and that none of these pages is blank. YOU MUST HAND THIS BOOKLET TO THE SUPERVISOR AT THE END OF THE EXAMINATION. Merit TOTAL 16 ASSESSOR' S USE ONLY ©New Zealand Qualifications Authority, 2013. All rights reserved. No part of this publication may be reproduced by any means without the prior permission of the New Zealand Qualifications Authority. 2 . y~~ are advised to spend 60 minutes answering the questions in this booklet. QUESTION ONE: EFFECT OF ENVIRONMENT One way to examine the role of the environment in variation among organisms is to compare the phenotypes _of various traits in genetically identical organisms. Armadillos are ideal animals to use in such research, because they are born as quadmplets derived from a single fertilised egg. This means that all four armadillo pups share the same genetic sequence. In a number of experiments carried out by scientists in the 1960s, genetically identical armadillos were found to show significant phenotypic differences when exposed to a range of environmental factors. For copyright reasons, this resource cannot be reproduced here. Image Source: bttp://www.nature.com/scitablt/natedlcontoot/5884/~our_armadillos_83-72_mid_l.jpg Discuss how genetically identical annadillo.s could be used to show the relationship between environmental factors and phenotype. In your answer: describe what is meant by 'phenotype' describe.what is meant by 'mutagen', and explain, using appropriate examples, why not all environmental factors are mutagens AS~SSO!t'3 USEOHlY 3 mLAfc.OJn CalAf& 60VV1ffhiVVJ_ ~ -::;ow ~>-P-os~ J.n iYtc s~b ro01lk i" L {')<CAVY\ ~ r_L __tW.J sl.Ao~ ~-s l}v ru~-s O-f 0\ CCALAsiv;iiJ 06~oC'L___I-o_~/ ,sk,v-c ceocu. ttD\Ne.\Jir VIO~ oJ\ ei\\JiVbhmef\J.rAl -Rtc /-v-0 Clr-c-- ~ ~ IIV\LA0~4\S~_5_()Y'V'e avt. lfYI-erd~ ~lclf of L ~\J1rOV\VV\l0+. 5~ fo1 t!.fiCiVhf2k.. D\ Jrc.-e- wCiJ -(6fOW'w-:_~ (;y\ck- C\n orlrt> " so Cot.-tl?1 A6j_?£J- ff\{)~~ JrqV~L. __ (Q~iZ\[;W)tiJ,_iL_§t-cW Jf\ a dif-k.nrd 6~ ~1-o"_~J Jol/fc~. -~ ·hA; otv- !r-te._l-olv kq~lTh0 s OW\ u a~'le_ of 1 ~tfi'C tlf\VJ/y__J ./ho lbtA/d bL shown ih nfri!-io-, 1-o K +kV'/1 !Vt c~,·--....-~ USEONLY c?, c;~g htlk,L 11.-G {r\\J1VtV\\f\'\tfl1 hc.s oo{ 0o o1{e d-oh~ J2kolf;~ a{ W\ Ot!JO! n;s- ~~wl n o_/!Vj_~9_uy_) """0''' ., , oe··. ~£f:t;f /).1--{Ve 1 ·eJ?vlro rrer:r··. - 4 QUESTION TWO: PROTEIN SYNTHESIS ASSESSOR'S USE ONLY The DNA sequence determines the structure of a protein and how that protein is produced. Table of mRNA CODONS (a) SECOND CODON ELEMENT u '•, ~ c el ~ \1\h)i\.. A 5 A otvA \ A 0 lA A b G G u f-< ~ c, G u c PRE PRE LEU LEU SER SER SER SER LEU .LEU LEU LEU ILE ILE ILE MET VAL VAL VAL VAL A G ;~ CYS CYS u STOP STOP STOP A G PRO PRO PRO PRO HIS HIS GLU GLU ARG ARG ARG ARG u THR THR THR SER SER ARG ARG u THR ASPN ASPN LYS LYS ALA ALA ALA ALA ASP ASP GLU GLU GLY GLY GLY GLY u c TRP c I A 0 G 8 c A G c §l i:Ij ~ ~ A G Using the information provided above, complete the table below. Note: In the table below, yon need only to give one possible codon for eachmRNA. DNA ~lAC mRNA !\LAG Amino Acids GTT CP\1\ MET GLU ATA Ud\LA TYR ACT. LAG/\ --STOP Explain with an example from the table why there is more than one possible codon for the same amino acid. Tn{.K Qr( OV\\~ c<-rtCI;{\ V\lAVV\\o-e-- a! ClVV\i/1\0 0Ci05 -flt\0'1- COn bt -b?:clieG\ b:;:1 irv\QJ\JA, ccxAolf\::,, 01\ong tNd\1 Kv ';)iQ(:! WDV\. ~1l--c.rc OvL ot lot VV\ov-c.- COrnbl/\oh'Gn.J of baSt.J ~bSSitJe- [nOn }hen: Ore OV'hiVIo acid;, ();\() k-~~ 5 h/&OY\ tlu-c- {fl--L i"\(Jr<-- fhon {he~V\ Oir-c /Q6J.\ifde (cPoA fir (Ju~ i.S Cod~~ r.e.Nh·c~d.l'\,~w..c£9. !(;,. f><GMplL 1k- iAVV'I,V\1> C\cid D i~ro51nt1nc_,) t.S roch;( fer two c\\~i (V\ RNA codoVIs ,__ldljlA CY\d IAAC. -% ()VV'if\o o.c,'rJi.T\ri-S 5 (b) Discuss the major stages of protein synthesis. ASSESSOR'S USE ONLY In your answer, include each of the following: • the role of the DNA template and coding strands • RNA structure and function • the relationship between codons AND anticodons • the role of start AND stop codons • the purpose of the ribosome. Proti tV\ Sldd\rlt:s\S o~ CA\(\ . i~ Vi~o\ \V'\ O~" ·o.n16YV\. Pro~,{/\ . _Qrt>~ 0\s w~tCh__ Q>\IOW [Jf\ Orf.) . s ') ·IV\ ('OV\~ilr\GiVJ3 su_v-vivoL lf\·\\0€':)1~ \~ lirt.. CtTaliolf'\ . o+ ;:Fe<:= 1-L) LIVICLOn {tort\~- ~\~kc refJ.e._ 'Oiov-rd- i"" 1~<- DNA \l:i) ~ chn:'lf'AaSoVY\eG. Thz_ oro CfJS o4 prdi 0"\ ~jV\ 1he_sis bqgiV'ij i 1'1 1Lc DAJA fAOLA'ok \r-c.\ix. 1k ~ +eVYiplak. S~IW\d of ft._ DIVA ,-.) rtoA b~ ltv .if\Z~ I<Ni' pe.\~~"'~0JX'l a rH o~ 1/V\r;?_f\l:A o f-6muJ. tAi~ ~ p!viJJq_ -~ LAra.ci( iJ v5§1 ,"ls~ 6F ~f/li\rnc.1hr sW o/ ~Q;JA o [f?../\. fk:sPof(ecA t\1\ /-o ~ fJ&-fup&tJ~ ~ rj ,j r(D.O COrfOI/"'- ~~ C10vf; Cot}o-~ C/tA- ri~6~. /L Onk1 (fb/1/f r1 .fq,/ A L,~l ----~-ow.~C/7 '(7d-1 ---ee ·~Lo:~e. There is more space for your answer to Question Two (b) on the following page. 3 7 ASSESSOR'S USE ONLY fibrosis is caused by a mutation in the gene CFTR (cystic fibrosis transmembrane inductance regulator). The most common mutation is a deletion of three nucleotides that in a loss of an amino acid at the 508th position on the protein. This mutation accounts for bbr<JX!lllately 66-70% of cystic fibrosis cases worldwide. It is an inheritable recessive condition. CFTR Sequence: Nucleotide A T C AT C T T Amino Acid lie lie Phe I GGT GTT Gly Val I 506 "---r50=8 510 I Deleted In AF508 .6.F508 CFTR Sequence: Nucleotide AT C AT T G G T G T T Amino Acid lie lie Gly Val I 506 Image Source: https://www.boundless.com/physiology/the-respiratory-system/ respiratory-system-disorders-and-clinical-cases/cystic-fibrosis/ Define the term mutation. A_~i.;'c\Q.~OV\ i6 D1 P-(f/fV\O.r,cv·"\!) ({.Alf\dOirh, chat}-c YLc tux. Se Q\lA -LV'\Cf0 o-f rA OJ\JA. I.l i·l cxc \AI'S i"\ L t''J ~be (bo~~__le_lisj_l~M (U.vllf'lJf ~~Wa, blkl ;{ . Yl _(i_(tLW~~ 1& .(lpi0tlek.S .1+ (qn oc. ·~~ (b) Identify the differences between substitution, insertion and deletion mutations, and explain which type of mutation has the most significant effect on an organism's phenotype. _Suos~iLA.~;oY\ m'-'~~CA._\-1D0S ore w~Aa·l is lel'ohiV\ av C\ eornf _tv\lA~CA~OV\ ~ L\ \) [N\--lfl CJV'L- be- X- ~~ SLAbEhfG ko Por er..drus ~ M A~ rtp!C!Ci"d Lvi/vt o, b. lh) ul6eo no.f o.IWCltcL h01J- C'\ \~c Cl~cf 6'" CIA orgaYir~ OS-~ if ofoc-s V\crl ~ rc:::uH i10:.o. rra.dfvw rro.vv-.e.~Jt- C~ !1vu k::u~5 rhu1 Clr{ rz:ad f6 @t~~ tO fov fV\ OfVli/\ o Cic,"dS.L (}r{A bfCuu)V- o{~ fYIOK 1~ oVL ./.r-io/.d o? bvsd;·eW1ika/f) Jp{)...t.;· ff),.... /L Jlt ~ Vh; V'6 0 Ci 0;) V'v-<~'C-1V\ i0' C( {' /.lv~e. IVJ ~v ScgtL~/]c~_ wV1l~~.~ tf\C)/1;-.;~ cd UJI/. &(jbs·ft·IU~f7o· rorexo""~' AICGTIGCCA I I G Qo~u,.o ex:£iouY(?d bvw .A TCGIA G~A IT G on the ron, -~ IVl. 8 (c) With reference to cystic fibrosis and another named disease, compare .and contraslinherited mutations with mutations that occur during the organisin'S lifes-pan. ~ s\t:iA ('anc .er ASSESSOR'S USE ONLY In your answer include: a description ofboth types of mutation an explanation of possible causes of these mutations and why they are different in terms of their effect on the organism a discussion of each type of mutation, in relation to the two diseases, and whether the diseases are inherited. [OI"' i V~lrle.ri\ec.\ \'Yil;t\o\io\'1 ~ occ_cw- , {\A.-e~ mv.s-1-hO E{!~ iV\ h ~C(Vh e ks , C15 J""i,S i0 wh-ere t'he;o5c::, 8CUv-s ~J. ~ (IV'J Ove. ~;:,sed Of\. IV\h.-rikd CotAio be cau:t.d ~ o {IJiry)o~ ChM'}L nIt_ DNA, a) ~~~·fv, 0 bro;rs. Kowc..w-. tMultA.f;Git\1 1h~ occw- duv1v;::g CV\ C~sk iltaiAV'Ii~VVJS liCe hVI-C qr-c off4_ I~ /{351Aii 0~ v'VIG~W (i Ch5 .exrz(){,(/'('b for oomple.-- d- ~~_~JO~A skiY) /o6 . 11\1\wch ~fQ6Sld"- /.o LAV ra~S tfotlh. L 5W"- ~Ol.A !Wl::'J· boLL 0\ VVll/l i6t0\A\ VJkUV" ~ v-c~!ls ln VJr'€1onoVVW( _ StYY?f:J;fiC,. Qr____S[u-Y\ carc.rr, . Sb, CIA~C<J i,7 Vlo_t thhanl-ct!::k-, 03 d o--d /r:r.;~r Ctc w i V\ rv S6nw li c cei!J Ol-t1 1-,:; lev· trC:M IJ of ~ . iJ!; '-feVfl~ . f'\U~)X'i<V,Ud ":J /"' trxJv, Vloi a yv,~h<h {h;J~~ [(COl~~ 1"' /u @tir11ck0 . t\OIAXvr VVIu~vhtiv1 /t.cAf r:p ~1J~ C\ft tvdr-uA{A ((YI. Ql,so bG~ CC1CA ~ VVl4W¥".--2.,. fl)jl/ te1f soT o VVllAtO§ff'l . 1 )wlt\ g~ !A~~ oraV\~-e...COuui~_pVI!:Jsial Ct;ifNl~t\ol VV\e()kll rcP:o..ratAIWY\ i"' 04\teo.,/-ior,s on 10 Ute-ftriQrn. l~5tlc fb-oS,s D (OvXel ~~ C\ Ol-d6h'OV\ o.r a /-ride-1-JOV'\d C(,Vh bv i1\kn kd I () J I r 8{f[Jrs ('\ ~ 9tAVlLtJ.cs rfJ r.) 0Y5Uv1 tV\ oII rei!~ 1/11 q pef.56N 06Jij J 1"rt fb-c:r D'\Jfi1 0.5 (5 (2 ~J1) fo "'1 () i)UJ'1£- s-Lzl' 'J Oc_> Ra-"' Cedlc,e,11\JVVIIt A\&!..j ~ etc~ ~ ac..v 0-Wt, /V\0cniol \MVIi-vlhOY)'j iMltsr~ 1'\ hJ rO'~f¢ ~ ~ Ullolc_ kJ~ Yf!:Q~rY 01.J wluut_s V\llvi?t 1M 'TM.cA1 OL&- ,V] 01 DYsfM'Jh Lk l.·vu.. -. tf\A,~ ~~ ().P{~ct rk:{l OJCv-,DI/VI._ /1:;-- fv 1[~.'11\cLe; Biology 91159, zo13 (}( fUr f I•?-f · 7 9 Extra paper if required. USE ONLY Write the question number(s) if applicable. QUESTION NUMBER ;crif~b. ASSESSOR'S ~OIL\ doL-, \-\oWer roVe C\ VV\uch Wt rb_oVIJ ~1-U!_,_,_,f!_hlxs e1ft-er ·IV1Serhon VY\Woh"OJI\5 ct'Jih biCJ~<rOfRc~ OV\. Cit-\ O~C/v\i<;t-v,J ------j--~~~ldk"' rf.vv.()l£.('.} C\nd bux<-- rs g_~ct. o0 tee{JI a Clfv---~''(\-L._[_ '1· 0 /rt"0lA JJo D\. brt13 IV\, 'dvwv, I Or r ~pI ~ (ukJSih8 ftt ch }n QlJ 0 11-rr /'vr.J NLUI{ ------~-DNA Ore. (2l & Cfi/) 11) be ~LI'v:C12_~ {}v_ /'() o lriDOirtJ [tJPJ:J...~-Ekd-1-~ . ATC GT:~ CCI-\ TAG _M,-"'<'C'----------------+------'-'~·~ \G ~~h~ c.G(~A) ISJ A 6 c. ---'h-CAdo'l Mo{\ o[ CY\ A c~~.s ft({)t, C6l~owr03 tri oi.eA +o b_ Olf~- ~--- ~-~----------------------------------- --1------------------------------{----------- --------- --1------------------------- _j L 91159 ,. . 1111111111111111111 NZ~ SUPERVISOR'S USE ONLY NEW ZEALAND QUALIFICATIONS AUTHORITY MANA TOHU MATAURANGA 0 AOTEAROA Level 2 Biology, 2013 91159 Demonstrate understanding of gene expression 9.30am Friday 22 November 2013 Credits: Four Achievement Demonstrate understanding of gene expression. Achievement with Merit Demonstrate in-depth understanding of gene expression. Achievement with Excellence Demonstrate comprehensive understanding of gene expression. Check that the National Student Number (NSN) on your admission slip is the same as the number at the top of this page. You should attempt ALL the questions in this booklet. If you need more space for any answer, use the page(s) provided at the back of this booklet and clearly number the question. Check that this booklet has pages 2-9 in the correct order and that none of these pages is blank. YOU MUST HAND THIS BOOKLET TO THE SUPERVISOR AT THE END OF THE EXAMINATION. High Merit TOTAL 18 ASSESSOR' S USE ONLY ©New Zealand Qualifications Authority, 2013. All rights reserved. No part of this publication may be reproduced by any means without the prior permission of the New Zealand Qualifications Authority. 2 You are advised to spend 60 minutes answering the questions in this booklet. QUESTION ONE: EFFECT OF ENVIRONMENT · One way to examine the role of the environment in variation among organisms is to compare the phenotypes of various traits in g~netically identical.organisms. Annadillos are ideal animals to use in such research, because they are born as quadruplets derived from a single fertilised egg. This means that all four·armadillo pups share the same genetic sequence. In a number of expeliments canied out by scientists in the 1960s, ~:~~!.~~~~armadillos were found to show significant phenotypic differences when exposed to a range of environmental factors. For copyright reasons, this resource cannot be reproduced here. Image Source: http:l/www.nature.com/scitablelnatedlcontent/5884/four_armadillos_83-72_mid_l.jpg \ Discuss how genetically identical armadillos could be used to show the relationship between environmental factors and phenotype. In your answer: • describe what is meant by 'phenotype' • describe what is meant by 'mutagen', and explain, using appropriate examples, why not all environmental factors are mutagens • evaluate how studies on the rumadillos could show that 'non-mutagenic' environmental factors may change phenotype without changing genotype. . ~~CJI 0d or!Jat:LJi~_j~j~!Je~~e i.s e~~~ f1~!f=~G¥f2rG%On £&~() 0e \' · .iLtt:ud~ t5 ta_b_Q_~~caf~. _I a: Cbf1t1t1c'atl ~6b t t~i Q/u4re.J:.. tnukM4 hj eitAR..r .ruk'ti~" ~J. .£vLdf.'-f!?._{;xa.JL~!d&uR.. co .f aliA 7 e~rt26 · · ~ ~S.e~ 'fh;c· /eev;(J lo /1~/z;<.;o $J~Uf?4c.:rf?_M::J d.~~ =--'--- ~,-at~~"L~J ~~ f-4.tct.t.Mizh-~~~lti5Mf.._.!JJrft c.?/!_ ef)VIr(Jtl~~~ ASSI USI 4 ASSE QUESTION TWO: PROTEIN SYNTHESIS "" The DNA sequence determines the structure of a protein and how that protein it Table of mRNA CODONS (a) SECOND CODON ELEMENT u E-< ! 5t:1 c A 0 u c A G PHE PHE LEU LEU SER SER SER SER TYR TYR STOP STOP CYS CYS STOP LEU LEU LEU LEU PRO PRO PRO ·PRO HIS HIS GLU GLU ARG ARG ARG ARG u ILE ILE ASPN ASPN LYS LYS SER SER ARG ARG u MET THR THR THR THR YAL VAL VAL VAL ALA ALA ALA ALA ASP ASP GLU GLU GLY GLY GLY GLY u !LE E-< (@ p.. u G TRP u c A G c A G c A G c ~ () § ~ i A G Using the information provided above, complete the table below.. Note: In the table below, you need only to give one possible codon for each r DNA mRNA II\<.. A.\), ra;- Amino Acids . (.,1\ ATA Ac_,'l C:rA A u~I.A v.GrA MET GLU TYR STOP Explain with an example from the table why there is more than one possible codon for the same amino acid. (A'Qo ,0\~ \A\P If\. q,~c}._ U\l G ~ _j_{,~ "h ~ l~ 6 \1-o\ c}_(~\. ~ \\- 5 (b) Discuss the major stages of protein synthesis. . ASSESSOR'S USEON.LY In yonr answer, include each of the following: c(d>'oC\/ the role of the DNA template and coding strands 'i. RNA structure and function A c.> "~\.e the relationship between co dons AND anticodons the role of start AND stop codons the purpose of the ribosome. Bioloov 91159, 2013 (. ''A:-r c_ G. • .. ASSESSOr USE ON!: Bs:M O--c-~7 w~ fu '~ ~~ U5t::)Ov---5 of~ '\'~ ~" ~~V"UZS Ovd fb ~~\G'Q_~~~. Gi' ~ (0~~ Qv1;ls o.-w\1 oo \AQ\'«> ~· o+ ~ ~ ll)\v<S~ &vUs, . 6_-t_~\f\IW_ ~\ c>'r~QJ of ~ d-e~ \Y, {vr.rJ;r:>nl----- y\R ffif_~ \'J \f\0 \~ )"QQ~o--~e--d~\J__~-__1? \O<KJ~ "1 \Y\.Ivl ~ ~ i4. role ~ £)(\\\4 &o,~ 0 {'D c«H for .~-rk>..:c.__-c- ..M n rl'{)\fQmM' v."'\1'- ~~ . 6g\ l A(M(Q l f pC .______/ -~--~~ _llf'&~~ rroi-'ft'_,r.5 . I ~· .~ ~ ., 1 1 1 11 , .r h;l(.,W.</y) ·. ~·"""'"'/· \ / -\-=r&x'''~"~. ~. ·~ f.-t-~V'> , ' r> u_u\e.~;V, . '-.._____ (pN-<Y\ oc~J... r 0b~<'~ f:=.a"'':>~· ~ 7 QUESTION THREE: MUTATIONS ASSESSOR'S USE ONLY Cystic fibrosis is caused by a mutation iu the gene CFTR (cystic fibrosis transmembrane conductance regulator). The most common mutation is a deletion of three nncleotides that results in a loss of an amino acid at the 50 8th position on the protein. This mutation accounts for approximately 66--70% of cystic fibrosis cases worldwide. It is an inheritable recessive condition. CFTR Sequence: Nucleotide ATC ATC TT Amino Acid lie II I GGTGTT Gly Phe I 506 508 Val 510 Deleted in liF508 AF508 CFTR Sequence: Nucleotide ATC ATT GGT GTT Amino Acid lie lie Gly Val I 506 Image Source: https://www.boundless.com/physiology/the-respiratory-system/ respiratory-system-disorders-and-clinical-cases/cystic-fibrosis/ (a) (b) Define the term mutation. Identify the differences between substitution, insertion and deletion mutations, and explain · which type of mutation has the most significant effect on an organism's phenotype. Sul?iritwtio'" 1111!1turfiohf CU'f wl~ne vhe J;.cue /h !IH '-'-'-- 5eqJ1fVI(~--~i!:L_wlfh___fljJ::l() f}tur. T//flf }11fahr __ th.~r !he t¥J~fi/IJ £o~~td .et1cl 1Aj2 tqcl/nq for o drff·mh t ~/JUi 11 o culd ~"' /~t{ elf\ . [ o111 ol /9 e /1cw !~i(fll f/'lf! tv J!!&CL!1iS111 ffecaufr ctf, redw,..dou~c't ile r;~~. fj!.h-dh rode hMever~ 1/o£. JhbfrrfrAhoh /1'1(.1~ }/lt'f j/\.(IVfJ Oh .e/fecf fl1P fe1UPI'lrt (o[!l(d Jl-i/1 (ode for f[!lf ]d,/IIAQ {A.J!Mt}io iJC)ct /Jil JerHo h 01 V\ d d. t I.f. fr'o;, f!/IJ tAt orftOhf l'lC1Vf 01 t!JI!OYP Jif;.biV(Civrl cffed oh tli.t1 uva C111isr~V~f __fjJj2. .() h;,P { Pt( Cl (;( U fh Pf f I f~jJ. H Question Three continues CIJ of {1/1\/), f-cd-i (J)rlJ (Or/Ali C1 HO 0 {l:c!d}. J ontbefollowingpage. ---~---·-·- ·-·. 8 (c) With reference to cystic fibrosis and another named disease, compare and contrast inherited mutations with mutations that occur during the organism's lifespan. ASSESSOR>: USE ONLY In your answer iuclude: a description of both types of mutation an explanation of possible causes of these mutations and why they are different in te1ms of their effect on the qrganism a discussion of each type of mutation, iu relation to the two diseases, and w diseases are · erited , 1 ,_;,/" _ _; //7 /'B-;' If:::(/( /hhevifecl VVII!l/-Ctfr'oh ir ohe 'fh7(T--.__, (J~Vt/er::h'cv! 11 Ct r 0a 11tJrd i 11 tf~ e CA /111€ f<[ Od11 c( :f r!:/}fu /rY:Jo{ f:JOfJe d Clj/1 {/1, e Jil.eYf 7 {IH VO fr r/l/1 ,....-'--;?;-' g -, I J[;tC/1 0.( {Ct./!IIU/ f/rtcd /(. (Ct{)}ed /JJ__ _:._____ f/11 {A fo f1Rt1 f C(l,1C/ If J.1,.of f}af'Jecf 0 h fo f!/1.J' /IJ€Yf @~1{/~CA/lOh. (~Jfr'c 'f;btoJtl IJ {if fvl_LA/Ofr'oh ff;tclf If I h hf /'I fed [ 0 jJ_aVC/Idf Of {A 11 lhdWt'dlt Ct/ M {lltirfT(-cuN/f Ctt1tJ af) Jf 011 tor /1 fo pe f}_f!<)et1I O/%.t hJ '{_.f. cau(if du-e fo radt'()frc'h If pt11'ft11), I , 9 Extra paper if required. Write t he question num ber(s) 1"f app Iicabl e. QUESTION NUMBER 3b ASSESSOR'S USE ONlY J! rodi111 q trCf /111! f fh 1h. /11 0(1 i 11Je tdloh or del £fr'ot/j tiJtJ lA f a1t'oh ill bt1ft //1 f/1 p 5(1 (:/_ [!l.f /"7 ( (' ff t!lhe /! t2_ ~1 f . ov I Ct//tcf atr !//If Th1r ho.;c; {lc{f<iF --!//) ti flo ;'r/) t !?! l/1 d C/II a Jll ci e fA e 1/ti/J/Jih u cJ cid L tl/l rJtf ·fh f Cf { 0d r ] /-or b( {r1 l4f-t J . .e_vfra {)/1 or n 111 tJf.!ed hm /1-1. ([!J /1 J +1/letf fh {' LJI] t~l/1 tf /111 () /iN/. . vi 12./.e If jJ(J}Me/. I ((I tiVlf I /IJ" o11 e fr ·W d) /ll~ (N!c Jih e f h iff) /h ()/rif' t:/; t a /t'oh · f/iyrreJ' %fl;0 ·io· I'm Qf!YI!f?O 5k1 -· .. , ?