arrhythmias in adult congenital heart disease
... There are now more adults living with congenital heart disease (CHD) than children with CHD, due to the remarkable improvements in medical, interventional, and surgical care of these complex patients. However, despite increased survival and longevity, the longterm hemodynamic abnormalities and sutur ...
... There are now more adults living with congenital heart disease (CHD) than children with CHD, due to the remarkable improvements in medical, interventional, and surgical care of these complex patients. However, despite increased survival and longevity, the longterm hemodynamic abnormalities and sutur ...
physio unit 4 Ch22 Ch 23
... What’s the major cause of death after an MI?: rupture, fibrillation, low output, or edema? And by that I don’t mean “sudden cardiac death” (Pathology), which is most often caused by lethal arrhythmias. Decreased cardiac output is the major cause of death after MI ...
... What’s the major cause of death after an MI?: rupture, fibrillation, low output, or edema? And by that I don’t mean “sudden cardiac death” (Pathology), which is most often caused by lethal arrhythmias. Decreased cardiac output is the major cause of death after MI ...
SCA CPB abstract 98-R - Society of Cardiovascular Anesthesiologists
... interatrial, interventricular and atrio-ventricular interaction, impedes reparative remodeling after myocardial infarction, and exacerbates symptoms and mortality of ESHD. Cardiac resynchronization therapy (CRT) involves placement of atrial, right ventricular (RV) and left ventricular (LV) leads, wi ...
... interatrial, interventricular and atrio-ventricular interaction, impedes reparative remodeling after myocardial infarction, and exacerbates symptoms and mortality of ESHD. Cardiac resynchronization therapy (CRT) involves placement of atrial, right ventricular (RV) and left ventricular (LV) leads, wi ...
Left Ventricular Regional Contraction Abnormalities by
... Maastricht University Medical Center, Department of Cardiology, Maastricht, The Netherlands Duke University Medical Center, Department of Cardiology, Durham, North Carolina, USA* ...
... Maastricht University Medical Center, Department of Cardiology, Maastricht, The Netherlands Duke University Medical Center, Department of Cardiology, Durham, North Carolina, USA* ...
Laboratory Exercise 13: Cardiac Physiology
... The work of the heart is performed by cardiac muscle. It is unique in its ability to depolarize itself (start an electric current) and initiate its own contraction. To coordinate the electrical impulses, the heart evolved a conduction system. The conduction system’s purpose is to ensure that the con ...
... The work of the heart is performed by cardiac muscle. It is unique in its ability to depolarize itself (start an electric current) and initiate its own contraction. To coordinate the electrical impulses, the heart evolved a conduction system. The conduction system’s purpose is to ensure that the con ...
Cardiac Dysfunction - UBC Critical Care Medicine, Vancouver BC
... You intubate her (grade 2 airway) but her sats are still 90% on FiO2 1.0 and her BP is 84/37. Question 3. What physiological effect does mechanical ventilation and PEEP have on the R ventricle? ...
... You intubate her (grade 2 airway) but her sats are still 90% on FiO2 1.0 and her BP is 84/37. Question 3. What physiological effect does mechanical ventilation and PEEP have on the R ventricle? ...
Cover - Circulation: Arrhythmia and Electrophysiology
... Copyright © 2017 American Heart Association, Inc. All rights reserved. Print ISSN: 1941-3149. Online ISSN: 1941-3084 ...
... Copyright © 2017 American Heart Association, Inc. All rights reserved. Print ISSN: 1941-3149. Online ISSN: 1941-3084 ...
CARDIAC FAILURE-Medical surgical nursing ppt
... function, liver function tests, thyroid function tests, complete blood count, C-reactive protein ...
... function, liver function tests, thyroid function tests, complete blood count, C-reactive protein ...
MADIT II - Boston Scientific
... Read the product labeling thoroughly before implanting the pulse generator to avoid damage to the ICD system. Such damage can result in patient injury or death. Program the pulse generator ventricular Tachy Mode to Off during implant, explant or post-mortem procedures to avoid inadvertent high volta ...
... Read the product labeling thoroughly before implanting the pulse generator to avoid damage to the ICD system. Such damage can result in patient injury or death. Program the pulse generator ventricular Tachy Mode to Off during implant, explant or post-mortem procedures to avoid inadvertent high volta ...
the chemokine receptor cxcr7 functions in endocardial
... from great vessels, and allow unidirectional blood flow through the heart. They are derived from specific sets of endocardial cells in the outflow tract and atrioventricular canal. Cardiac valvulogenesis is a highly ordered process and small perturbations in any of signaling pathways involved can re ...
... from great vessels, and allow unidirectional blood flow through the heart. They are derived from specific sets of endocardial cells in the outflow tract and atrioventricular canal. Cardiac valvulogenesis is a highly ordered process and small perturbations in any of signaling pathways involved can re ...
• Muscle Tissues- highly cellular, well
... Voluntary muscle- contraction; conscious control (skeletal muscle) Involuntary muscle- no control over contraction (Cardiac & Smooth) Intercalated Discs ...
... Voluntary muscle- contraction; conscious control (skeletal muscle) Involuntary muscle- no control over contraction (Cardiac & Smooth) Intercalated Discs ...
Cardio Vascular Division Overview_SMIT
... [1] Oz. M., Annetine, C., Miller L. et al., Left Ventricular Assist Devices as permanent Heart Failure Therapy: The price of progress, Annals of Surgery. 238(4): 577-83, October 2003. ...
... [1] Oz. M., Annetine, C., Miller L. et al., Left Ventricular Assist Devices as permanent Heart Failure Therapy: The price of progress, Annals of Surgery. 238(4): 577-83, October 2003. ...
Slide 1
... • Risk factors: family hx, male, increased cholesterol, hypertension, smoking. • Nitroglycerin – submucosal for angina only. ...
... • Risk factors: family hx, male, increased cholesterol, hypertension, smoking. • Nitroglycerin – submucosal for angina only. ...
Heart Failure
... Non-specific symptoms of heart failure include nocturia, anorexia, abdominal bloating and discomfort, constipation, and cerebral symptoms such as confusion, dizziness and memory impairment Signs and symptoms do not correlate to the severity of heart damage ...
... Non-specific symptoms of heart failure include nocturia, anorexia, abdominal bloating and discomfort, constipation, and cerebral symptoms such as confusion, dizziness and memory impairment Signs and symptoms do not correlate to the severity of heart damage ...
Heart failure
... New York heart failure classification I = Cardiac disease but NO SYMPTOMS in ordinary physical activity II = MILD SYMPTOMS and slight limitation during ordinary physical activity III = Marked limitation in activity due to symptoms during LESS-THAN-ORDINARY ACTIVITY IV = Severe limitations. Experien ...
... New York heart failure classification I = Cardiac disease but NO SYMPTOMS in ordinary physical activity II = MILD SYMPTOMS and slight limitation during ordinary physical activity III = Marked limitation in activity due to symptoms during LESS-THAN-ORDINARY ACTIVITY IV = Severe limitations. Experien ...
Pharmacology Objectives 11
... Triggered automaticy – is characterized by after depolarizations, depolarizations that occur before or after full repolarization of the cell. After depolarizations can be early (EAD) or delayed (DAD). Re-entry – occurs when there is slower conduction down one branch of fibers than on a neighboring b ...
... Triggered automaticy – is characterized by after depolarizations, depolarizations that occur before or after full repolarization of the cell. After depolarizations can be early (EAD) or delayed (DAD). Re-entry – occurs when there is slower conduction down one branch of fibers than on a neighboring b ...
Name_____________________________________ Per_____
... Explain how impulses travel through each of the following areas of the heart. 1) Sinoatrial node ...
... Explain how impulses travel through each of the following areas of the heart. 1) Sinoatrial node ...
C Cardiac Resynchronization Therapy
... control station known as the atrioventricular or AV node. After a split-second delay, the signal spreads to the ventricles by way of specialized routes called the left and right bundle branches. The bundle branches fan out in the ventricles, thereby enabling the electrical signal to stimulate both v ...
... control station known as the atrioventricular or AV node. After a split-second delay, the signal spreads to the ventricles by way of specialized routes called the left and right bundle branches. The bundle branches fan out in the ventricles, thereby enabling the electrical signal to stimulate both v ...
Primary ptca in resuscitated cardiac arrest complicating acute
... Resuscitated Cardiac Arrest (CA) victims are an high risk subgroup of Acute Myocardial Infarction (AMI), that could be excluded from lytic therapy, because of prolonged cardiopulmonary resuscitation and could evolve to cardiogenic shock. Primary PTCA is potentially useful, but logistics and outcomes ...
... Resuscitated Cardiac Arrest (CA) victims are an high risk subgroup of Acute Myocardial Infarction (AMI), that could be excluded from lytic therapy, because of prolonged cardiopulmonary resuscitation and could evolve to cardiogenic shock. Primary PTCA is potentially useful, but logistics and outcomes ...
What is ARVC? Arrhythmogenic Right Ventricular Cardiomyopathy
... How does ARVC affect the heart? The genetic defect results in a change in the heart muscle. In people with a normal heart, the muscle is held together by proteins: in people with ARVC these proteins are abnormal and not well developed. This causes a fragmentation of the heart muscle: some part of th ...
... How does ARVC affect the heart? The genetic defect results in a change in the heart muscle. In people with a normal heart, the muscle is held together by proteins: in people with ARVC these proteins are abnormal and not well developed. This causes a fragmentation of the heart muscle: some part of th ...
CRT Overview - February 2004
... Etiology of Heart Failure What causes heart failure? • The loss of a critical quantity of functioning myocardial cells after injury to the heart due to: – Ischemic Heart Disease ...
... Etiology of Heart Failure What causes heart failure? • The loss of a critical quantity of functioning myocardial cells after injury to the heart due to: – Ischemic Heart Disease ...
SHSST Cyclodextrin Complex Prevents the Fibrosis Effect on CCl4
... effects between Rheum, Scutellaria and Coptis chinensis are due to the same bioactive compounds, baicalein and other flavonoids [24–28]. Both SHSST and silymarin are potential liver protection drugs, but both present poor water solubility and poor bioavailability [17]. A formulation approach is nece ...
... effects between Rheum, Scutellaria and Coptis chinensis are due to the same bioactive compounds, baicalein and other flavonoids [24–28]. Both SHSST and silymarin are potential liver protection drugs, but both present poor water solubility and poor bioavailability [17]. A formulation approach is nece ...
module #25 congestive heart failure
... with HFrEF and NYHA class III-IV results in reduction in mortality, readmission for HF, and improvement in the quality of life (A-HeFT 2004). --Digoxin is another option. It reduces symptoms in patients with HFrEF and also decreases rates of hospitalization (DIG 1997). -- For summary, see Table 3 in ...
... with HFrEF and NYHA class III-IV results in reduction in mortality, readmission for HF, and improvement in the quality of life (A-HeFT 2004). --Digoxin is another option. It reduces symptoms in patients with HFrEF and also decreases rates of hospitalization (DIG 1997). -- For summary, see Table 3 in ...
Cardiac contractility modulation
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Cardiac contractility modulation (CCM) is a treatment for patients with moderate to severe left ventricular systolic heart failure (NYHA class II–IV). The short- and long-term use of this therapy enhances both the strength of ventricular contraction and the heart’s pumping capacity. The CCM mechanism is based on stimulation of the cardiac muscle by non-excitatory electrical signals (NES). CCM treatment is delivered by a pacemaker-like device that applies the NES, adjusted to and synchronized with the electrical action in the cardiac cycle.In CCM therapy, electrical stimulation is applied to the cardiac muscle during the absolute refractory period. In this phase of the cardiac cycle, electrical signals cannot trigger new cardiac muscle contractions, hence this type of stimulation is known as a non-excitatory stimulation. However, the electrical CCM signals increase the influx of calcium ions into the cardiac muscle cells (cardiomyocytes). In contrast to other electrical stimulation treatments for heart failure, such as pacemaker therapy or implantable cardioverter defibrillators (ICD), CCM does not affect the cardiac rhythm directly. Rather, the aim is to enhance the heart’s natural contraction (the native cardiac contractility) sustainably over long periods of time. Furthermore, unlike most interventions that increase cardiac contractility, CCM is not associated with an unfavorable increase in oxygen demand by the heart (measured in terms of Myocardial Oxygen Consumption or MVO2). This may be explained by the beneficial effect CCM has in improving cardiac efficiency. A meta-analysis in 2014 and an overview of device-based treatment options in heart failure in 2013 concluded that CCM treatment is safe, that it is generally beneficial to patients and that CCM treatment increases the exercise tolerance (ET) and quality of life (QoL) of patients. Furthermore, preliminary long-term survival data shows that CCM is associated with lower long-term mortality in heart failure patients when compared with expected rates among similar patients not treated with CCM.