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Biosc_48_Chapter_5_lecture
Biosc_48_Chapter_5_lecture

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... minimizing the flux to biomass or to other fermentation byproducts such as glycerol. Since formation of highly reduced fermentation products such as glycerol is driven by accumulation of cytosolic NADH, S. cerevisiae have been engineered to maintain lower level of cytosolic NADH by various genetic m ...
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... synthesis of lipid or possibly an increased lipid during the 'molting' cycle. These ear- rate of lipid catabolism. These data sugly observations were extended by Renaud g-est that a factor (s) in the eyestalks influ(1949) who demonstrated a rise in hepa- ences lipid metabolism. To test this hypothes ...
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... phosphorylation at the 1-position to form fructose 1-P, followed by conversion to intermediates of the glycolytic pathway. The major products of its metabolism in liver are, therefore, the same as glucose (including lactate, blood glucose, and glycogen). Essential fructosuria (fructokinase deficienc ...
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Glyceroneogenesis



Glyceroneogenesis is a metabolic pathway which synthesizes glycerol 3-phosphate or triglyceride from precursors other than glucose. Usually glycerol 3-phosphate is generated from glucose by glycolysis, but when glucose concentration drops in the cytosol, it is generated by another pathway called glyceroneogenesis. Glyceroneogenesis uses pyruvate, alanine, glutamine or any substances from the TCA cycle as precursors for glycerol 3-phophate. Phosphoenolpyruvate carboxykinase (PEPC-K), which is an enzyme that catalyses the decarboxylation of oxaloacetate to phosphoenolpyruvate is the main regulator for this pathway. Glyceroneogenesis can be observed in adipose tissue and also liver. It is a significant biochemical pathway which regulates cytosolic lipid levels. Intense suppression of glyceroneogenesis may lead to metabolic disorder such as type 2 diabetes.
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