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Lecture 33 - University of Arizona
Lecture 33 - University of Arizona

... cAMP triggers two types of phosphorylation circuits in muscle cells; one that stimulates glycogen degradation and a second that inhibits glycogen synthesis. ...
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... internal water filled cavity. Due to this protein specially found in mature enterocytes, I-FABP was thought to be crucial in fatty acids trafficking, and targeting ligands to specific organelle for metabolic process. Even so, the specific function of I-FABP in animal intestine remains elusive. LCFAs ...
Lipotoxicity in steatohepatitis occurs despite an increase in
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... Loss of electrons from one substance = oxidation. Addition of electrons to a substance = reduction. Oxidizing agent - accepts electrons. Reducing agent - gives up electrons. ...
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Slide 1
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... and FADH2 molecules  With the help of CoA, the acetyl (two-carbon) compound enters the citric acid cycle – At this point, the acetyl group associates with a fourcarbon molecule forming a six-carbon molecule – The six-carbon molecule then passes through a series of redox reactions that regenerate th ...
appendix a
appendix a

The Syndrome of Carnitine Deficiency: Morphological and Metabolic
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... for fatty acid synthesis is in the form of acetyl-coenzyme A (acetyl-CoA) which cannot cross the plastidic membrane (Weaire and Kekwick, 1975; Roughan et al., 1979). Precursors for acetylCoA synthesis must thus be generated in the plastid or imported from the cytosol. In heterotrophic oilseeds, lipi ...
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electron transport chain.
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... and fatty acids (used in generating acetyl CoA). • Fatty acids are broken down by beta oxidation and yield acetyl CoA. • An oxidized gram of fat produces more than twice as much ATP as an oxidized gram of carbohydrate. ...
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... ketone body metabolism during a classically ketogenic period, the transition to birth, and in a classically ‘non-ketogenic’ state, overnutrition, using novel genetic mouse models, high-resolution measures of dynamic metabolism using ...
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... help women with PKU gain metabolic control before or during a pregnancy by providing social support and promoting a positive attitude about treatment through home visitation. The Resource Mothers are women who have children with PKU, and thus understand the diet and hardships associated with the dis ...
Universal Functional and Model Consistency Testing
Universal Functional and Model Consistency Testing

... aminotransferase reaction and the mitochondrial alanine transporter ALAtm were both removed, based on the evidence that the mitochondrial alanine transaminase is present only on gluconeogenic tissues [DeRosa and Swick, J Biol Chem (1975)] and that there were no other alanine reactions in mitochondri ...
BSC 2010 - Exam I Lectures and Text Pages Citric Acid Cycle • Citric
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... • Glycolysis and the citric acid cycle connect to many other metabolic pathways • Our bodies generally use many sources of energy in respiration (fig 9.19)  regulated by feedback inhibition (fig 9.20) • Carbohydrates  simple sugars, enter glycolysis • Proteins  amino acids (used to build new prot ...
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Ketosis



Ketosis /kɨˈtoʊsɨs/ is a metabolic state where most of the body's energy supply comes from ketone bodies in the blood, in contrast to a state of glycolysis where blood glucose provides most of the energy. It is characterised by serum concentrations of ketone bodies over 0.5 millimolar, with low and stable levels of insulin and blood glucose. It is almost always generalized with hyperketonemia, that is, an elevated level of ketone bodies in the blood throughout the body. Ketone bodies are formed by ketogenesis when liver glycogen stores are depleted (or from metabolising medium-chain triglycerides). The main ketone bodies used for energy are acetoacetate and β-hydroxybutyrate, and the levels of ketone bodies are regulated mainly by insulin and glucagon. Most cells in the body can use both glucose and ketone bodies for fuel, and during ketosis, free fatty acids and glucose synthesis (gluconeogenesis) fuel the remainder.Longer-term ketosis may result from fasting or staying on a low-carbohydrate diet, and deliberately induced ketosis serves as a medical intervention for intractable epilepsy. In glycolysis, higher levels of insulin promote storage of body fat and block release of fat from adipose tissues, while in ketosis, fat reserves are readily released and consumed. For this reason, ketosis is sometimes referred to as the body's ""fat burning"" mode.
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