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O2-1 Significance of Premature Restriction or Closure of Foramen
O2-1 Significance of Premature Restriction or Closure of Foramen

... cavity imitating hypoplastic left heart, partial obstruction of left ventricular inflow, and premature atrial contractions were other additional findings. One fetus who was born prematurely at 26 weeks died after birth, two foetuses had to be delivered early at 37 weeks of gestation due to severe re ...
Biochemistry - U
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Emergency Department use of Esmolol in Refractory Ventricular
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... difficult to treat and carries a high mortality rate • Treatment with epinephrine: – Activates α-1 receptors: this is probably helpful – Activates β-1 and β-2 receptors: this is probably harmful ...
Left Ventricular Structure and Function in Aortic Stenosis: The Inner
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Eur J Heart Fail
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After atrial excitation, impulse travels through the AV node

... volume,  and  ventricular  systole  includes  both  isovolumetric  contraction  and  ventricular   ejection.  The  ventricle  does  not  empty  completely  during  ejection,  which  is  normally  half   diastole  blood  volume  is  pumped  out. ...
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Ventricular Assist Devices: - Vanderbilt University Medical Center
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Arrhythmogenic Right Ventricular Cardiomyopathy (ARVC)
Arrhythmogenic Right Ventricular Cardiomyopathy (ARVC)

... They may predispose to bacterial endocarditis. ...
Valve Disease – From Bench to Bedside
Valve Disease – From Bench to Bedside

... Aspirin 75 mg to 100 mg per day is reasonable in all patients with a bioprosthetic aortic or mitral valve Anticoagulation with a VKA is reasonable for the first 3 months after bioprosthetic MVR or repair to achieve an INR of 2.5 ...
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Hypertrophic cardiomyopathy



Hypertrophic cardiomyopathy (HCM) is a primary disease of the myocardium (the muscle of the heart) in which a portion of the myocardium is hypertrophied (thickened) without any obvious cause, creating functional impairment of the cardiac muscle. It is a leading cause of sudden cardiac death in young athletes.The occurrence of hypertrophic cardiomyopathy is a significant cause of sudden unexpected cardiac death in any age group and as a cause of disabling cardiac symptoms. Younger people are likely to have a more severe form of hypertrophic cardiomyopathy.HCM is frequently asymptomatic until sudden cardiac death, and for this reason some suggest routinely screening certain populations for this disease.A cardiomyopathy is a disease that affects the muscle of the heart. With HCM, the myocytes (cardiac contractile cells) in the heart increase in size, which results in the thickening of the heart muscle. In addition, the normal alignment of muscle cells is disrupted, a phenomenon known as myocardial disarray. HCM also causes disruptions of the electrical functions of the heart. HCM is most commonly due to a mutation in one of nine sarcomeric genes that results in a mutated protein in the sarcomere, the primary component of the myocyte (the muscle cell of the heart). These are predominantly single-point missense mutations in the genes for beta-myosin heavy chain (MHC), myosin-binding protein C, cardiac troponinT, or tropomyosin. These mutations cause myofibril and myocyte structural abnormalities and possible deficiencies in force generation. Not to be confused with dilated cardiomyopathy or any other cardiomyopathy.While most literature so far focuses on European, American, and Japanese populations, HCM appears in all ethnic groups. The prevalence of HCM is about 0.2% to 0.5% of the general population.
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