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Slide ()
Slide ()

... Two types of ion channels regulated by receptors and drugs. A. Diagram of a voltage-activated Na+ channel with the pore in the open and closed state. The pore-forming P loops are shown in blue, angled into the pore to form the selectivity filter. The S4 helices forming the voltage sensor are shown i ...
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Lecture 5 Transmitters and receptors lecture 2015
Lecture 5 Transmitters and receptors lecture 2015

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Ca 2+
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the limbic system
the limbic system

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Guillain-Barré Syndrome
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Neurons, Synapses, and Signaling
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Neurons Notes
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chapter29_Sections 6
chapter29_Sections 6

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MS Word Version
MS Word Version

... Page 8. Acetyl Choline and its Receptors • There are multiple receptors for each neurotransmitter. • Each such receptor activates a different ion channel, causing a different effect in the postsynaptic cell. • There are two groups of receptors, called cholinergic receptors, which bind acetylcholine. ...
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“The Physiology of Excitable Cells”

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... Chapter 2 (The Brain) Study Guide 1. What is a neuron? What are the three basic types of neurons? What is the difference between a neuron with myelin compared to a neuron that is not myelinated? 2. What is stimulus threshold? All-or-none principle? (domino example in class) 3. What is a synapse? 4. ...
48 - Groupfusion.net
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... 2)It opens voltage-gated calcium channels in the membrane, triggering an influx of Ca^2+ 3)The elevated Ca^2+ concentration in the terminal causes synaptic vesicles to fuse with the presynaptic membrane ...
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... (representing a stronger initial stimulus), more neurotransmitter is released into the synaptic cleft and more impulses per second are sent. When the neurotransmitter has done its work, it is removed from the synaptic cleft by an enzyme that breaks down the molecules. The transmission of the impulse ...
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Nerve Impulse Transmission

... carry it toward the cell body, which contains the nucleus. • The axon carries the impulse from the cell body toward the synaptic knobs where it will be transferred to other neurons. ...
KS4_nervous_models_Pupil_Sheets
KS4_nervous_models_Pupil_Sheets

... An electrical impulse cannot travel across a gap so another mechanism needs to be used. When the impulse reaches the end of the neuron chemicals called neurotransmitters are released into the gap. These diffuse across and bind to receptors in the next neuron which sets off a new impulse. ...
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Neuromuscular junction



A neuromuscular junction (sometimes called a myoneural junction) is a junction between nerve and muscle; it is a chemical synapse formed by the contact between the presynaptic terminal of a motor neuron and the postsynaptic membrane of a muscle fiber. It is at the neuromuscular junction that a motor neuron is able to transmit a signal to the muscle fiber, causing muscle contraction.Muscles require innervation to function—and even just to maintain muscle tone, avoiding atrophy. Synaptic transmission at the neuromuscular junction begins when an action potential reaches the presynaptic terminal of a motor neuron, which activates voltage-dependent calcium channels to allow calcium ions to enter the neuron. Calcium ions bind to sensor proteins (synaptotagmin) on synaptic vesicles, triggering vesicle fusion with the cell membrane and subsequent neurotransmitter release from the motor neuron into the synaptic cleft. In vertebrates, motor neurons release acetylcholine (ACh), a small molecule neurotransmitter, which diffuses across the synaptic cleft and binds to nicotinic acetylcholine receptors (nAChRs) on the cell membrane of the muscle fiber, also known as the sarcolemma. nAChRs are ionotropic receptors, meaning they serve as ligand-gated ion channels. The binding of ACh to the receptor can depolarize the muscle fiber, causing a cascade that eventually results in muscle contraction.Neuromuscular junction diseases can be of genetic and autoimmune origin. Genetic disorders, such as Duchenne muscular dystrophy, can arise from mutated structural proteins that comprise the neuromuscular junction, whereas autoimmune diseases, such as myasthenia gravis, occur when antibodies are produced against nicotinic acetylcholine receptors on the sarcolemma.
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