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GRACE Personality factors Insomnia Primary Insomnia Secondary Insomnia RESEARCH –Primary Insomnia RESEARCH – Secondary insomnia Apnoea Dispositional traits affecting normal sleep patterns. A long term problem initiating and/or maintaining sleep. Insomnia not attributable to an underlying cause. Subtypes include: Psychophysiological insomnia, idiopathic insomnia and sleep state misperception. Insomnia attributable to an underlying cause. (e.g hormonal changes in women, medical illness, decreased melatonin production, psychiatric disorders, stimulants, lifestyle factors.) Dement (1999) reported several cases of sleep state misperception. One case involved a patient who complained of severe insomnia being asked to sleep for 10 consecutive nights in the sleep laboratory. Each morning he completed a questionnaire estimating how long it took to fall asleep at night. Participant said 90mins on average. In reality it took an average of 15mins. Dauvilliers et al (2005) asked 256 primary insomniacs to complete a clinical interview, psychometric questionnaires and a questionnaire on the family history of insomnia, and to undergo detailed psychological sleep measurements. A control group of non-insomniacs were also tested. Of the primary insomniacs, 72.7% reported a family history of insomnia compared with 24% of noninsomniacs, suggesting a familial link to primary insomnia. Monti (2004) found many cases of insomnia could be treated successfully by treating the underlying cause, such as a medical or psychiatric condition, which suggests that some forms of insomnia are secondary – the result of other illnesses – and are not an illness themselves like primary insomnia is. Katz et al (2002) studied insomniacs with chronic medical conditions, such as diabetes, depression, hypertension and heart conditions. 50% were found to suffer insomnia, 34% were found to suffer a mild form and 16% were found to suffer a severe form. Suffereres have persistent pauses in their breathing that lasts for minutes, as well as occasional loud snorts as breathing recommences. Obstructive sleep apnoea (OSA) is often found in overweight middle aged men when air is prevented from entering the lungs as a result of lack of muscle tone in the upper airway causing it to collapse. It is considered severe if breathing ceases for more than 10 seconds and more than 10 seconds every night. Chest (2001) found a significant positive correlation between insomnia and OSA, suggesting a relationship between the two conditions. Stickgold (2009) believes a range of mental disorders, including depression and attention deficit disorder, are caused by sleep apnoea and insomnia. Apnoeac insomniacs had twice the incidence of depression than the normal population, suggesting the best way to treat mental disorders is to GRACE Sleepwalking Narcolepsy alleviate these. Activities occurring unconsciously when someone is asleep that normally occur when awake. Sleepwalkers are unaware of their activity and engage in automatic behaviour. Usually occurs in stage three or four at early points in the night. Environmental factors that induce sleepwalking include stress, alcohol intoxication, sleep deprivation, chaotic sleep schedule & drugs such as sedatives and antihistamines. There is thought to be a genetic link as family some family histories demonstrate. It also occurs more frequently in identical twins. Broughton (1968) found that the disorder is heritable, with sufferers 10 times more likely than the general population to have a close relative with the disorder, suggesting a genetic factor. Hublin et al (1997) found the disorder to be more common among children, with up to 20% being affected. In adults, the prevalence rate is about 2%. This indicates that the condition is linked to development and maturation. A disorder characterised by disruption to the sleep/wake cycle, whereby sufferers suddenly fall asleep at unexpected times, often in the middle of activites. These sporadic micro-sleeps are common, with the sufferer awaking without realising that he or she has been asleep. Type and severity of symptoms vary between individuals and can improve or worsen over time. It appears to be a neurological condition associated with a fault in brain mechanisms controlling wakefulness and sleep. Another common symptom is cataplexy, where muscular control is lost, usually as a result of being aroused, for example when excited. Cataplexy takes the form of attacks of muscle weakness or near total paralysis occurring suddenly for a few seconds or minutes and then subsiding. Dement (1999) reported that a sleep research team in Texas found that mice who could not produce hypocretin in their brains developed symptoms of narcolepsy, demonstrating the importance of the neurotransmitter. Daniels et al (2001) gave questionnaires to 500 patients of the Narcolepsy Association and found they had lower energy levels, reduced social functioning, and lessened physical activity, with 57% of them depressed. The condition put limitations on their educational, work, home and social life, matching results from other countries and demonstrating the extensive impact of the disorder on health related quality of life.