Chapter 36 - Immune System
... with antibodies that bind with the invader’s antigen are stimulated to reproduce rapidly. • B-cells differentiate into either plasma cells or memory B-cells. Plasma cells rapidly produce antibodies. Memory cells retain the “memory” of the invader and remain ready to divide rapidly if an invasion occ ...
... with antibodies that bind with the invader’s antigen are stimulated to reproduce rapidly. • B-cells differentiate into either plasma cells or memory B-cells. Plasma cells rapidly produce antibodies. Memory cells retain the “memory” of the invader and remain ready to divide rapidly if an invasion occ ...
Lecture Outline 7
... 1. monomer with the lowest molecular mass 2. only trace amounts are found in serum 3. surface Ig on blood lymphocytes 4. may have lymphocyte activation and suppression activity e. IgE 1. slightly larger than IgG and IgA 2. involved in allergy 3. large share of antibodies formed during primary allerg ...
... 1. monomer with the lowest molecular mass 2. only trace amounts are found in serum 3. surface Ig on blood lymphocytes 4. may have lymphocyte activation and suppression activity e. IgE 1. slightly larger than IgG and IgA 2. involved in allergy 3. large share of antibodies formed during primary allerg ...
Lecture #14 Bio3124 - University of Ottawa
... – Lethal factor cleaves protein kinases • Blocks immune system from attacking ...
... – Lethal factor cleaves protein kinases • Blocks immune system from attacking ...
blood grouping
... Naturally occurring antibodies occur in the plasma of individuals who lack the corresponding antigen and who have not been pregnant or transfused. The most important are anti-A and anti-B. These are usually IgM antibodies. Immune antibodies develop in response to the introduction of red cells posses ...
... Naturally occurring antibodies occur in the plasma of individuals who lack the corresponding antigen and who have not been pregnant or transfused. The most important are anti-A and anti-B. These are usually IgM antibodies. Immune antibodies develop in response to the introduction of red cells posses ...
VL 08lecture2008
... Recognizes antigens (molecular signatures) specific for each pathogen Effective against both intra- and extracellular pathogens Two main components: Humoral immunity - Relies on Antibodies produced by B-lymphocytes - Fights pathogens outside of cells ...
... Recognizes antigens (molecular signatures) specific for each pathogen Effective against both intra- and extracellular pathogens Two main components: Humoral immunity - Relies on Antibodies produced by B-lymphocytes - Fights pathogens outside of cells ...
The Immune Response - hrsbstaff.ednet.ns.ca
... Killer T cells puncture the cell membranes of intruders. If killer T cells find a viral coat attached to a cell membrane, the T cell attacks the infected cells. ...
... Killer T cells puncture the cell membranes of intruders. If killer T cells find a viral coat attached to a cell membrane, the T cell attacks the infected cells. ...
The Immune Response - hrsbstaff.ednet.ns.ca
... Killer T cells puncture the cell membranes of intruders. If killer T cells find a viral coat attached to a cell membrane, the T cell attacks the infected cells. ...
... Killer T cells puncture the cell membranes of intruders. If killer T cells find a viral coat attached to a cell membrane, the T cell attacks the infected cells. ...
Self tolerance
... Antibodies may also contribute to the disease. About 80% of patients have serum immunoglobulin M (IgM) (and, less frequently, IgA) autoantibodies that bind to the Fc portions of their own (self ) IgG.. ...
... Antibodies may also contribute to the disease. About 80% of patients have serum immunoglobulin M (IgM) (and, less frequently, IgA) autoantibodies that bind to the Fc portions of their own (self ) IgG.. ...
notes
... ligand binds to the extracellular portion of the receptors, phosphorylate one or more SMAD proteins in the cytosol SMAD proteins move into the nucleus form dimers with another SMAD protein designated SMAD4 dimers bind to a DNA sequence (CAGAC) in the promoters of target genes enhance, or repress ...
... ligand binds to the extracellular portion of the receptors, phosphorylate one or more SMAD proteins in the cytosol SMAD proteins move into the nucleus form dimers with another SMAD protein designated SMAD4 dimers bind to a DNA sequence (CAGAC) in the promoters of target genes enhance, or repress ...
Immunity
... • Antigen: substance that triggers immune response – On outer surface of invading cell or virus – MHC (major histocompatibility complex) proteins • Self-antigens on human cell surfaces • Enable immune system to distinguish “self” from “nonself” ...
... • Antigen: substance that triggers immune response – On outer surface of invading cell or virus – MHC (major histocompatibility complex) proteins • Self-antigens on human cell surfaces • Enable immune system to distinguish “self” from “nonself” ...
File
... Antigen Presenting Cell • After a phagocyte destroys an invading pathogen it presents fragments of its antigens on its cell surface • The phagocyte is now referred to as an antigen presenting cell • A specific helper T cell then binds to the antigen on the antigen presenting cell • The helper T cel ...
... Antigen Presenting Cell • After a phagocyte destroys an invading pathogen it presents fragments of its antigens on its cell surface • The phagocyte is now referred to as an antigen presenting cell • A specific helper T cell then binds to the antigen on the antigen presenting cell • The helper T cel ...
T cell receptor
... ● CD8 co-receptors confer preference for class I MHC-peptide complexes (cytotoxic T-cell) ● CD4 co-receptors confer preference for class II MHC-peptide complexes (T-helper cells) ● T-cells undergo V(D)J recombination to create a diverse number of TCRs, similar to Immunoglobulins but they do not unde ...
... ● CD8 co-receptors confer preference for class I MHC-peptide complexes (cytotoxic T-cell) ● CD4 co-receptors confer preference for class II MHC-peptide complexes (T-helper cells) ● T-cells undergo V(D)J recombination to create a diverse number of TCRs, similar to Immunoglobulins but they do not unde ...
Presentazione standard di PowerPoint
... Increased plasma or serum IL-1α, IL-2, IL-6, IL-8 and TNF have been found in ARF and RHD patients Serum TNF increases during an acute episode of ARF and is elevated in RHD patients with heart failure, suggesting that TNF could play roles at the onset of ARF and during the development of cardiac dise ...
... Increased plasma or serum IL-1α, IL-2, IL-6, IL-8 and TNF have been found in ARF and RHD patients Serum TNF increases during an acute episode of ARF and is elevated in RHD patients with heart failure, suggesting that TNF could play roles at the onset of ARF and during the development of cardiac dise ...
A41-Immune Response
... pathogen, you retain those antibodies for awhile, explaining why some diseases are only contracted once in a lifetime ...
... pathogen, you retain those antibodies for awhile, explaining why some diseases are only contracted once in a lifetime ...
The Immune System
... trap and kill most pathogens. 2. Inflammatory Response - fluid and some WBC’s leak from blood vessels into tissues to fight pathogens. The WBC’s are called phagocytes- they engulf and destroy the pathogens. Inflammatory responses include a red, swollen, warm area and sometimes fever. 3. Immune R ...
... trap and kill most pathogens. 2. Inflammatory Response - fluid and some WBC’s leak from blood vessels into tissues to fight pathogens. The WBC’s are called phagocytes- they engulf and destroy the pathogens. Inflammatory responses include a red, swollen, warm area and sometimes fever. 3. Immune R ...
File
... trap and kill most pathogens. 2. Inflammatory Response - fluid and some WBC’s leak from blood vessels into tissues to fight pathogens. The WBC’s are called phagocytes- they engulf and destroy the pathogens. Inflammatory responses include a red, swollen, warm area and sometimes fever. 3. Immune R ...
... trap and kill most pathogens. 2. Inflammatory Response - fluid and some WBC’s leak from blood vessels into tissues to fight pathogens. The WBC’s are called phagocytes- they engulf and destroy the pathogens. Inflammatory responses include a red, swollen, warm area and sometimes fever. 3. Immune R ...
Document
... IVIG is extensively treated (solvents, detergents) to inactivate potential pathogens. Also tends to suppress autoimmune disorders??! -- role of carbohydrates? ...
... IVIG is extensively treated (solvents, detergents) to inactivate potential pathogens. Also tends to suppress autoimmune disorders??! -- role of carbohydrates? ...
HP_Tipaje Linfocitario_24 07 13
... Assessment of the immune system The immune system consists of a series of organs, tissues and cells widely distributed throughout the entire body. These components are interconnected by blood and lymph vessels, constituting a single well-communicated system. The response mechanisms of the immune sys ...
... Assessment of the immune system The immune system consists of a series of organs, tissues and cells widely distributed throughout the entire body. These components are interconnected by blood and lymph vessels, constituting a single well-communicated system. The response mechanisms of the immune sys ...
Complement system
The complement system is a part of the immune system that helps or complements the ability of antibodies and phagocytic cells to clear pathogens from an organism. It is part of the innate immune system, which is not adaptable and does not change over the course of an individual's lifetime. However, it can be recruited and brought into action by the adaptive immune system.The complement system consists of a number of small proteins found in the blood, in general synthesized by the liver, and normally circulating as inactive precursors (pro-proteins). When stimulated by one of several triggers, proteases in the system cleave specific proteins to release cytokines and initiate an amplifying cascade of further cleavages. The end-result of this activation cascade is massive amplification of the response and activation of the cell-killing membrane attack complex. Over 30 proteins and protein fragments make up the complement system, including serum proteins, serosal proteins, and cell membrane receptors. They account for about 5% of the globulin fraction of blood serum and can serve as opsonins.Three biochemical pathways activate the complement system: the classical complement pathway, the alternative complement pathway, and the lectin pathway.