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Types II and III: Antibody-Mediated and Antigen
Types II and III: Antibody-Mediated and Antigen

... may affect the skin, joints, kidneys, brain, and other organs. Causes, incidence, and risk factors Systemic lupus erythematosus (SLE) is an autoimmune disease, which means the body's immune system mistakenly attacks healthy tissue. This leads to long-term (chronic) inflammation. The underlying cause ...
Ch. 16.5 Viruses
Ch. 16.5 Viruses

... reproduce itself. These host cells are eventually destroyed, weakening the patient's immune system. ...
Immunity
Immunity

... microbes and are particularly vulnerable to infection. At birth, human babies have high levels of antibodies, with the same range of antigens as their mother. This is passive immunity because the fetus does not actually make any memory cells or antibodies, it only borrows them. Short-term passive im ...
When a person breaks a bone, suffers infection organ damage or
When a person breaks a bone, suffers infection organ damage or

... population against a specific epitope, and then systemically reintroducing the T-cells into the patient. "The T-cells hone in on the site of the injury, because they have been sensitized, and attach to the surviving cells or axons to prevent them from dying," says Harel. In studies published in the ...
What causes an immune response and increase of
What causes an immune response and increase of

... 3What is the main difference between lytic and Lysogenic cycles in viruses? • Lytic is the shorter cycle that ends in cell destruction/lysed. Lysogenic is longer and leads into the lytic cycle steps. ...
Virulence Factors of Pathogens Toxin: Botulism toxin Botulism: case
Virulence Factors of Pathogens Toxin: Botulism toxin Botulism: case

... Virulence Factors of Pathogens Virulence Factors of Pathogens • Substances generated by pathogens that enhance their ability to cause disease • Four categories of virulence factors correspond to the different mechanisms pathogens used to cause disease ...
Describe how white blood cells defend the body against infection
Describe how white blood cells defend the body against infection

... → phagocytose foreign material (bacteria, fungi), i.e. ingest + destroy them with lipase, proteolytic enzymes and reactive oxygen species - interaction with antibody and complement here, as neutrophils and monocytes have Fc and C3 receptors, allowing to recognise ‘opsonised’ foreign material - Eosin ...
Jian Xie is from Hunan province, People`s Republic of China
Jian Xie is from Hunan province, People`s Republic of China

... as PTP1B might induce Type II diabetes. Yersinia PTPase was found to be the important virulent determinant in the Black Death, or the Bubonic plague. Another interesting example is PTP1B-deficient mice showed increased insulin sensitivity and obesity resistance. All these studies suggest that inhibi ...
Types of microbes
Types of microbes

... threads called hyphae. ...
self and non
self and non

... recognized by CMI are cancer cells and those cells inflicted by organisms that live in the host cell ...
Mediators of Immediate Hypersensitivity
Mediators of Immediate Hypersensitivity

... Hypersensitivity refers to excessive undesirable (damaging, discomfort producing and sometimes fatal) reactions produced by the normal immune system. Hypersensitivity reactions require a pre-sensitized (immune) state of the host. Hypersensitivity reactions can be elicited by exogenous environmental ...
Types of microbes
Types of microbes

Nertila_Ujkaj:Littin_Kandoth_Sandra
Nertila_Ujkaj:Littin_Kandoth_Sandra

... Meresse, B., et al., Coordinated induction by IL15 of a TCR-independent NKG2D signaling pathway converts CTL into lymphokine-activated killer cells in celiac disease. Immunity, 2004. 21(3): p. 357-66. ...
TIRP Antibody
TIRP Antibody

... TIRP is a member of the Toll/interleukin-1 receptor (TIR) family, a group of proteins that include the Toll-like receptors (TLRs) (1-3). TLRs are signaling molecules that recognize different pathogen-associated molecular patterns (PAMPs) and serve as an important link between the innate and adaptive ...
Fig 1.1
Fig 1.1

... cells responsible for innate immunity and lymphocytes responsible for specific immunity. The guardian cells of the innate immune system form the first line of defence against infection and can digest pathogens or vaccine particles and use these to activate lymphocytes. In addition they produce chemi ...
week six summary - fundamentals of immunology
week six summary - fundamentals of immunology

... TYPES OF IMMUNE RESPONSES TO TUMOR-SPECIFIC ANTIGENS • May involve B cell and CD4 T cell responses • Antibodies and complement bind to antigens on the surface of cancer cells and kill them • CD8 T cells may recognize tumor antigen peptides on MHCI molecules and kill targets • Macrophages and Natu ...
Figure 1.1 The human immune system All blood cells originally
Figure 1.1 The human immune system All blood cells originally

... guardian cells responsible for innate immunity and lymphocytes responsible for specific immunity. The guardian cells of the innate immune system form the first line of defence against infection and can digest pathogens or vaccine particles and use these to activate lymphocytes. In addition they prod ...
Disease as a Failure of Homeostasis
Disease as a Failure of Homeostasis

... Malaria is a mosquito-borne infectious disease of humans and other animals caused by protists (a type of microorganism) of the genus Plasmodium. It begins with a bite from an infected female mosquito, which introduces the protists via its saliva into the circulatory system, and ultimately to the liv ...
The Body`s Lines of Defense
The Body`s Lines of Defense

...  HIV is the virus that causes AIDS (Acquired Immune Deficiency Syndrome) .  HIV has an 8- 10 year incubation period before symptoms arise.  The immune system becomes weakened. A decrease in the function of CNS followed by bodyweight loss occurs.  Eventually death by a secondary infection such as ...
APCH43REV
APCH43REV

Apoptosis – Programmed Cell Death
Apoptosis – Programmed Cell Death

... Superantigens are molecules which short-circuit the immune system, resulting in massive activation of T-cells rather than the usual, carefully controlled response to foreign antigens. The over-response of the immune system produced results in autoimmunity, as rare clones of T-cells which recognize ...
DEFINITIONS - Microbiology Book
DEFINITIONS - Microbiology Book

... Epitope or Antigenic Determinant Antibody (Ab) ...
Nonspecific Defenses Against Infection
Nonspecific Defenses Against Infection

... b. humoral and cell mediated immunity 3) Explain how the physical barrier of skin is reinforced by chemical defenses. 4) Define phagocytosis. Name four types of phagocytic leukocytes. 5) Explain how interferon limits cell-to-cell spread of viruses. 6) Describe the inflammation response, including ho ...
Supplementary Figure Legends (doc 28K)
Supplementary Figure Legends (doc 28K)

... standard deviations. Significant P-values are indicated by asterisks (* P=0.03; ** P=0.003; *** P<0.001). (B) HLA-A2 molecule expression on the surfaces of MCF-7 and MDA-MB-231 cell lines. Immunofluorescence staining was performed using MA2.1 monoclonal antibody and analysed on BD LSR Flow Cytometer ...
Humoral Immunity
Humoral Immunity

... • Subsequent encountering of the same antigen by memory B cells produces antibodies rapidly and in a larger quantity (titer) • The type of antibody is IgG (class switching has occurred) • Indicates the protective immunity • May last a longer period ...
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Molecular mimicry

Molecular mimicry is defined as the theoretical possibility that sequence similarities between foreign and self-peptides are sufficient to result in the cross-activation of autoreactive T or B cells by pathogen-derived peptides. Despite the promiscuity of several peptide sequences which can be both foreign and self in nature, a single antibody or TCR (T cell receptor) can be activated by even a few crucial residues which stresses the importance of structural homology in the theory of molecular mimicry. Upon the activation of B or T cells, it is believed that these ""peptide mimic"" specific T or B cells can cross-react with self-epitopes, thus leading to tissue pathology (autoimmunity). Molecular mimicry is a phenomenon that has been just recently discovered as one of several ways in which autoimmunity can be evoked. A molecular mimicking event is, however, more than an epiphenomenon despite its low statistical probability of occurring and these events have serious implications in the onset of many human autoimmune disorders. In the past decade the study of autoimmunity, the failure to recognize self antigens as ""self,"" has grown immensely. Autoimmunity is a result of a loss of immunological tolerance, the ability for an individual to discriminate between self and non-self. Growth in the field of autoimmunity has resulted in more and more frequent diagnosis of autoimmune diseases. Consequently, recent data show that autoimmune diseases affect approximately 1 in 31 people within the general population. Growth has also led to a greater characterization of what autoimmunity is and how it can be studied and treated. With an increased amount of research, there has been tremendous growth in the study of the several different ways in which autoimmunity can occur, one of which is molecular mimicry. The mechanism by which pathogens have evolved, or obtained by chance, similar amino acid sequences or the homologous three-dimensional crystal structure of immunodominant epitopes remains a mystery.
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