Download • - Paramedic EMS Zone

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The primary components of the respiratory system are like an inverted tree, with
the trachea representing the tree’s trunk and the alveoli resembling the tree’s
leaves.
The mouth and oropharynx are very vascular and are covered by a mucous
blanket. Swelling can be profound and potentially dangerous.
The larynx and glottis are typically considered the dividing line between the upper
airway and the lower airway. The thyroid cartilage is the most obvious external
landmark of the larynx. The glottis and vocal cords are found in the middle of the
thyroid cartilage.
The cricoid cartilage can be palpated just below the thyroid cartilage in the neck.
It forms a complete ring and maintains the trachea in an open position. Applying
cricoid pressure or Sellick manoeuvre may be helpful with intubation.
The small space between the thyroid and cricoid cartilages is the cricothyroid
membrane. It is a good choice for inserting a large IV cannula or a small
breathing tube.
Cilia line the larger airways and help move foreign material out of the
tracheobronchial tree. If a patient is dehydrated or has taken medications that dry
secretions (such as antihistamines), the cilia will not be able to effectively move
secretions. The same is true if the patient is overhydrated.
Pulmonary circulation begins at the right ventricle, where the pulmonary artery
branches into increasingly smaller vessels until it reaches the pulmonary capillary
bed, which surrounds the alveoli and terminal bronchioles.
Patients who have traumatic brain injuries may exhibit bizarre respiratory
patterns, including agonal gasps; apneustic and ataxic patterns; and Biot, CheyneStokes, and Kussmaul respirations; as well as central neurogenic hyperventilation,
bradypnoea, hyperpnoea, hypopnoea, and tachypnoea.
The respiratory system delivers oxygen to the body and removes the primary
waste product of metabolism, carbon dioxide. If the lungs are not functioning
appropriately, both of these vital functions may be impaired. Hypoxia, cell death,
and acidosis can then occur.
The brain is very sensitive to reduced levels of oxygen. It requires a regular
supply of both oxygen and glucose to function, but can store neither. Alteration in
level of consciousness could represent respiratory compromise.
A patient who has respiratory disease may not be able to talk because he or she is
having difficulty breathing. You may have to obtain the history from a family
member or from only a few clues.
It is critical to evaluate how hard your patient is working to breathe. Patients in
respiratory distress may be able to compensate at first, but will eventually become
sleepy, have a decreased respiratory rate and depth, and then, decompensate.
Patients who have chronic respiratory disease are often knowledgeable about their
disease and may have tried several potential treatment options already. Ask them
about these efforts and what results, if any, they produced.
Onset and duration of distress are important considerations in determining the
underlying cause. Find out if the problem happened suddenly or gradually
worsened over time.
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Assessing the patient’s position of comfort and difficulty speaking may help you
gauge the patient’s degree of distress. A patient sitting completely upright and
speaking only in two- or three-word statements is probably in considerable
distress.
Patients in respiratory distress tend to seek the tripod position. The condition of a
patient in respiratory distress who is willing to lie flat may be quickly
deteriorating. Head bobbing is also an ominous sign.
Find out if the patient’s condition is a recurrence of a past condition. If so,
compare the current situation with other episodes. _ Note any audible abnormal
respiratory noises. Noisy breathing is obstructed breathing.
Snoring indicates partial obstruction of the upper airway by the tongue. Stridor
indicates narrowing of the upper airway, usually as a result of swelling (laryngeal
oedema).
Assessing the level of consciousness is enormously important in dyspnoeic
patients.
Assess the patient’s mucous membranes for cyanosis (a bluish or dusky colour),
pallor, and moisture.
Look for jugular venous distention in the neck, with the patient in a semi-sitting
position. Distended neck veins may be caused by cardiac failure.
Feel the chest for vibrations as the patient breathes. Check for oedema of the
ankles or lower back. Check for peripheral cyanosis. Check the pulse and note the
patient’s skin temperature. Attach any available monitors.
Auscultate the lungs whenever possible. Adventitious breath sounds are the extra
noises that you may hear; they include wheezing or crackles.
Crackles are any discontinuous noises heard on auscultation of the lungs. They
are caused by the popping open of air spaces and are usually associated with
increased fluid in the lungs.
Wheezes are high-pitched, whistling sounds made by air being forced through
narrowed airways, which makes them vibrate. Wheezing may be diffuse in
conditions such as asthma and congestive heart failure or localised when caused
by a foreign body
obstructing a bronchus.
Silence means danger! If you don’t hear anything with your stethoscope, the
patient is not moving enough air to ventilate the lungs.
Note whether the patient is coughing up colourful sputum, if the colour or amount
of sputum has changed, and if it contains blood or pus.
A pulse oximeter indicates what percentage of the patient’s haemoglobin has
oxygen attached to it. An oxygen saturation of 95% or more is considered normal.
The peak flow is the maximum flow rate at which the patient can expel air from
the lungs. Normal peak flows run from about 300/650 l/min. A peak flow of less
than 150 l/min is very low and signals significant distress.
A variety of infections can cause swelling in the upper airway. Croup is one of the
most common, though it usually occurs only in small children.
Pulmonary aspiration of stomach contents is very dangerous. Avoid causing
gastric distention when bagging the patient, and monitor the patient’s ability to
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protect his or her airway. If you determine that the patient can’t protect his or her
airway, one of your primary jobs is to protect it for them.
Common obstructive airway diseases include emphysema, chronic bronchitis, and
asthma. Emphysema and chronic bronchitis are collectively classified as COPD,
along with some cases of chronic asthma.
Asthma is caused by allergens or irritants and is characterised by widespread,
reversible narrowing of the airways (bronchospasm), oedema of the airways, and
increased mucous production. It can cause significant airway obstruction.
Primary treatment of bronchospasm is bronchodilator medication. Primary
treatment of bronchial oedema is corticosteroids, which may be administered in
the prehospital setting. Status asthmaticus is a severe, prolonged asthmatic attack
that cannot be broken with conventional treatment. It is a dire medical emergency.
Any person with asthma who feels sick enough to call an ambulance is in status
asthmaticus until proved otherwise.
When a patient has recurring asthma attacks, his or her inhaler could be empty or
the medication could no longer be effective. Try administering a new
bronchodilator.
Emphysema is a chronic weakening and destruction of the walls of the terminal
bronchioles and alveoli. A patient with emphysema classically has a barrel chest,
muscle wasting, and pursed-lip breathing. Such patients are often tachypnoeic.
Chronic bronchitis is characterised by excessive mucous production in the
bronchial tree, nearly always accompanied by a chronic or recurrent productive
cough. A patient with chronic bronchitis tends to be sedentary and obese, sleep in
an upright position, use
many tissues, have copious secretions, and be cyanotic.
In assessing patients who have COPD, search for what pushed them over the
edge. Look for signs of infection, peripheral oedema, jugular venous distention
with or without a positive abdominojugular test, and crackles. Find out if the
onset of dyspnoea was sudden or gradual.
Not everyone should be ventilated the same way. Allow each breath to come back
out before the delivery of the next breath. If you don’t, pressures in the thorax will
rise, eventually causing pneumothorax or cardiac arrest. Patients with severe
obstructive diseases should be ventilated as little as 4 to 6 breaths/min.
Noncompliance could trigger an asthma attack. Ask what the patient was doing
when the asthma attack began. Ask if the patient took his or her medications
today. Ask if movement worsens the dyspnoea.
Pneumonia may be caused by a variety of bacterial, viral, and fungal agents. The
patient with pneumonia usually reports weakness, productive cough, fever, and
sometimes chest pain that worsens with coughing. Supportive care includes
oxygenation, suctioning, and transport to an appropriate facility.
Pulmonary oedema occurs when fluid migrates into the lungs. The patient
expectorating pink and foamy secretions probably has severe pulmonary oedema.
When a patient has a pneumothorax, air collects between the visceral pleura and
the parietal pleura. Administer high concentration oxygen and monitor the
patient’s respiratory status closely.
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Pleural effusion will make the patient dyspnoeic. Supportive care, including
proper positioning and aggressive oxygen administration, should be given.
A pulmonary embolism occurs when a blood clot breaks off in the circulation and
travels to the lungs, blocking blood flow and nutrient exchange. Bedridden
patients, those with cancer, and people who have had major surgery are at risk of
pulmonary embolism. The hallmark of pulmonary embolus is cyanosis that does
not resolve with oxygen therapy.
Respiratory failure, or insufficient ventilation, can occur from a multitude of
pathological conditions, from injuries to the lungs, heart, and neurological system
to overdoses. Care includes providing supplemental oxygen.
In hyperventilation syndrome, ventilation is excessive. If it continues, the patient
may experience chest pain and carpopedal spasm. Psychological support
techniques such as counting breaths, and distraction work best and help calm the
patient.
In managing the condition of a patient who is in respiratory distress, begin by
ensuring that there is an open and maintainable airway. Suction if necessary and
keep the airway optimally positioned. Remove constricting clothing. Reduce the
patient’s effort to breathe.
Patients in respiratory failure may ultimately need to be intubated. There are
major drawbacks and risks to intubating in the prehospital environment, but it can
also be lifesaving. Weigh these issues along with guidelines, medical direction,
and the patient’s wishes.
Metered-dose inhalers deliver medication as a nebuliser treatment. They are
usually the delivery method of choice for both bronchodilators and corticosteroids
in the home setting. Improper technique may result in little or no medication
actually getting into the lungs.
Nebulisers deliver liquid medications in the form of a fine mist to the respiratory
tract. Weigh the potential benefits of nebuliser therapy against the lower FiO2
delivered during the treatment.
Drug administration methods that require the patient to inhale the medication may
become unreliable or ineffective when the patient’s airways are severely
compromised. Some cases may warrant delivering medications intramuscularly or
subcutaneously instead.
Medications can be instilled directly into the tracheobronchial tree when patients
are intubated or have a tracheostomy.
Continuous positive airway pressure (CPAP) is used as therapy for acute
cardiogenic pulmonary oedema. Within several minutes of application, the
patient’s oxygen saturation should increase, and the respiratory rate should
decrease.
Bilevel positive airway pressure (BiPAP) is CPAP that delivers one pressure
during inspiration and a different pressure during exhalation. It is more like
normal breathing and is often more comfortable for the patient.
Automated transport ventilators are essentially flow-restricted oxygen-powered
breathing devices with timers on them. They are particularly good choices for
filling the role of the bag-valve-mask ventilator when the intubated patient is in
cardiac or respiratory arrest, but are not intended to ventilate patients without
direct observation and attention from a skilled practitioner.