Download Назва наукового напрямку (модуля): Семестр: 10 Ішемічна

Survey
yes no Was this document useful for you?
   Thank you for your participation!

* Your assessment is very important for improving the workof artificial intelligence, which forms the content of this project

Document related concepts

Heart failure wikipedia , lookup

Remote ischemic conditioning wikipedia , lookup

Coronary artery disease wikipedia , lookup

Hypertrophic cardiomyopathy wikipedia , lookup

Cardiac contractility modulation wikipedia , lookup

Mitral insufficiency wikipedia , lookup

Lutembacher's syndrome wikipedia , lookup

Jatene procedure wikipedia , lookup

Cardiac surgery wikipedia , lookup

Myocardial infarction wikipedia , lookup

Management of acute coronary syndrome wikipedia , lookup

Electrocardiography wikipedia , lookup

Arrhythmogenic right ventricular dysplasia wikipedia , lookup

Quantium Medical Cardiac Output wikipedia , lookup

Atrial fibrillation wikipedia , lookup

Ventricular fibrillation wikipedia , lookup

Heart arrhythmia wikipedia , lookup

Transcript
Назва наукового напрямку (модуля):
Семестр: 10
Ішемічна хвороба серця
Опис:
Перелік питань:
1.
A. *
B.
C.
D.
E.
2.
A. *
B.
C.
D.
E.
3.
A. *
B.
C.
D.
E.
4.
A. *
B.
C.
D.
E.
5.
A. *
B.
C.
D.
E.
6.
A. *
B.
C.
D.
E.
7.
A. *
B.
C.
D.
E.
Atrial pacing is shown at:
Synoatrialniy blockade
Second degree atrioventricular block
Third degree atrioventricular block
All of the states
None of these states
The absolute indications for permanent electrostimulation are:
Third degree atrioventricular block with attacks Morgan-Adams-Stokes
Sinus bradicardic action
Sinus arrhythmia
Atrial fibrillation
All of the states
Bicameral stimulation is most effective when:
The Full atrioventricular block
Paroxysmal supraventricular tachycardia
Atrial fibrillation
All of the states
None of these states
Temporary eleсtroсardiostimulation shown patients with:
Postinfarct acute atrioventricular block
Sinus arrhythmia
Congenital complete atrioventricular block
All the states listed
None of the above states
The absolute contraindication for implantation of artificial drivers are:
Sepsis
Complete atrioventricular block
Sinus syndrome weakness
Age older than 1990
All listed
At the turn of the endocardial electrode stimulator pulse frequency:
No
Increased
Decreases
Increases
Significantly reduced
What is happening in the ventricle, when an effect Venkebaha:
The frequency of ventricular rate remains at upper limit
Upovilnyayetsya ventricular rate cuts
Cavity is no longer declining
None of the listed
The frequency of ventricular rate doubles
8.
A. *
B.
C.
D.
E.
9.
A. *
B.
C.
D.
E.
10.
A. *
B.
C.
D.
E.
11.
A. *
B.
C.
D.
E.
12.
A. *
B.
C.
D.
E.
13.
A. *
B.
C.
D.
E.
14.
A. *
B.
C.
D.
E.
15.
A. *
When threshold stimulation reaches the highest value after stimulator implantation?
Between two and six weeks after surgery
At 9-10 months after surgery
In a few hours after surgery
A year after surgery
After 5 years after surgery
Form stimulating wave QRS:
A similar blockade of the left bundle branch block legs
Such a normal QRS
A similar blockade of the right bundle branch block legs
Similar regulations
Similar to symptoms of acute coronary syndrome
When endocardial electrode dislocation:
Burst stimulator is realized in part, or all
Accelerated heart rhythm
The rhythm of the heart does not change
I get pain syndrome
Endangered pain syndrome
Availability of ECG symptoms characteristic of peredzbudzhennya ventricle in patients with mitral
defect, requires osobolyvoyi attention due to:
Great features of atrial fibrillation and risk associated with this high frequency of ventricular rhythm
Violations result of abnormal hemodynamics in order of ventricular rate
More serious attack tachycardia
All listed
Nothing listed
When a temporary stimulation stimulation threshold necessary to check:
Every day
Every 6 hours
Once in 3-4 days
Once a week
No need
Permissible limit of stimulus amplitude at a temporary stimulation:
6 Volt
2 Volta
15 Volt
20 Volt
50 volts
Indications for permanent atrial stimulation:
Sinus syndrome weakness with reduced heart rate to 40 beats per minute or loss of consciousness
Frederick Syndrome
Atrial extrasystoles
All of the listed
Nothing listed
Indications for permanent ventricular stimulation:
Complete AV-block with attacks Morhanyi-Adams-Stokes
B.
AV-block II degree
C.
D.
E.
16.
A. *
B.
C.
D.
E.
17.
A. *
B.
C.
Sinus syndrome weakness of attacks Morhanyi Adomsa-Stokes
Ventricular hemodynamics in violation ekstasystoliya
All of the listed
Paroxysmal atrial fibrillation differs from SUPRAVENTRICULAR tachycardia:
Distance between the tips of irregular ventricular complexes
Higher heart rate
Wide complex ventricular
All the features listed
None of these signs
To slow the rate in the treatment of atrial fibrillation paroxysms apply:
Verapamil
Novokainamid
Digoxin
Rytmalen
Any of these drugs
To treat a form of permanent atrial fibrillation patient weighing 70 kg is prescribed:
Hinidyn of 600-800 mg 4 times a day
Finoptin to 80 mg 4 times a day
Hinidyn 300 mg 3 times daily
Hinidyn 1200 mg 4 times a day
Finoptin of 800 mg 3 times daily
Indications for surgical treatment of patients with tachycardia are:
Go near paroxysmal tachycardia accompanied with loss of consciousness
The presence of ECG signs of ventricular overexcited
The presence of ECG signs of ventricular overexcited BEATS
Go near tachicardia 2-3 times per year
Go near tachicardia 1-2 times a month
In the treatment of paroxysmal tachycardia undesirable combination:
Finoptin and obzidan
Novokainamid and Digoxin
Hinidin and Digoxin
Novokainamid and finoptin
There are no restrictions on the combinations of these drugs
The most effective method for termination of atrial flutter include:
Esophageal pacing
Vahusni tests (nadavlennya of the eyes, etc.)
Countershock terpiya
Retrograde pacing
None of these methods are not effective
Left ventricular function to evaluate by:
Indexed (listed on 1 square meter area of ??the body) end-diastolic value of volume, end-systolic
volume, stroke volume, minute volume of the heart
The absolute value of end-diastolic volume, stroke volume, minute volume of the heart for each age
group
D.
E.
18.
A. *
B.
C.
D.
E.
19.
A. *
B.
C.
D.
E.
20.
A. *
B.
C.
D.
E.
21.
A. *
B.
C.
D.
E.
22.
A. *
B.
C.
D.
E.
23.
A. *
B.
C.
D.
E.
24.
A.
B.
C. *
D.
E.
25.
A. *
B.
C.
D.
E.
26.
A. *
B.
C.
D.
E.
27.
A. *
B.
C.
D.
E.
28.
A. *
B.
C.
D.
E.
29.
A. *
B.
C.
D.
E.
The absolute value of end-diastolic volume, end-systolic volume, stroke volume
Size end-diastolic volume and heart rate
Any of these methods
Which of the following parameters characterizing skorochuvannist miokaroda:
Ejection fraction
Fraction Reduction
Speed ??circulation reducing fibers
The thickness of the back of the interventricular membrane
All listed
What is for use of digitalis:
Second degree atrioventricular block
All listed
Atrial fibrillation in mitral heart gaps
Zdavlyuyuchyy pericarditis with heart failure
Tachycardia at tyreotoksyozi
Sick prymav digitalis in the case of paroxysmal tachycardia in it you need:
Stop receiving digitalis
Increase dose of digitalis
Zmeneshyty double dose
Advanced assign other medicines
None of the listed
Beat this:
An extraordinary reduction in relation to the main heart rhythm
Upovilnyuvane reduction
The reduction is not a source of basic rate
All listed
None of the listed
As often occurs beat in patients with acute myocardial infarction?
Virtually all patients
Rarely parasystoliya
Only in patients with anterior myocardial infarction
Only in patients with posterior myocardial infarction
This dysrhythmia is not characteristic of myocardial
Can register on electrocardiogram extrasystoles in healthy people?
When monitorirovanni on Holter in 50-60%:
Extrasystoles not registered
Only in acute inflammatory diseases
Only at the Oncological Pathology
Only in severe injuries
What is the most frequent mechanism underlying the occurrence of beats?
Turning excitation
Ectopic automatism
Morning trace potentials
Late potentials trace
Asynchronous myocardial cell membrane repolarization
30.
A. *
B.
C.
D.
E.
31.
A. *
B.
C.
D.
E.
32.
A. *
B.
C.
D.
E.
33.
A. *
B.
C.
D.
E.
34.
A. *
B.
C.
D.
E.
35.
A. *
B.
C.
D.
E.
36.
A. *
B.
C.
D.
E.
What changes in hemodynamics observed beats?
Changes in hemodynamics and endure depends on the frequency of extrasystoles, degrees of
peredchastnosti, localization and functional state of cardiac muscle
Changes in hemodynamics and endure depends on concomitant cardiac pathology
Changes in hemodynamics and endure depends on the age of the patient
Pronounced changes
Violation of hemodynamics in beats are not observed
What compensated hemodynamics of beats?
Postekstrasystolichnym potentiation forces beat
Basic rhythm
Pryskorynnyam rhythm after extrasystoles
By stimulating the vagus nerve receptors
Hemodynamic compensation is not required
Is Extrasystolic complexes informative in terms of diagnosis hidden in ordinary complexes
vohneschevosti?
Registration is informative Q wave in complex Extrasystolic
Informative be registered in the S wave Extrasystolic complex
Extrasystolic complexes are informative only for acute myocardial infarction
Extrasystolic complexes do not contain additional information
Informing a member of a complex type QS
How common regulatory dysfunction synoatrial node in patients with clinical and
electrocardiographic signs of his weakness?
In half of patients with this symptomatology
In the small number of patients
In all patients with this symptomatology
Only in young patients
Only in elderly and senile patients
Which of the following antiarrhythmic drugs affects synoatrialnyy knot?
Propranolol
Lidokain
Digoxin
Etmozyn
All of the
Among whom the most common weakness sinus syndrome:
Among women
Among men
Among children
Among the Elderly
In all equally
Bradytahikardiyi syndrome occurs more frequently:
In elderly patients
In younger patients
In patients with drug-node dysfunction synoatrialnoho
In patients with dysfunction of the regulatory unit synoatrialnoho
In children
37.
A. *
B.
C.
D.
E.
38.
A. *
B.
C.
D.
E.
39.
A. *
B.
C.
D.
E.
40.
A. *
B.
C.
D.
E.
41.
A. *
B.
C.
D.
E.
42.
A. *
B.
C.
D.
E.
43.
A. *
B.
C.
D.
E.
44.
When are adults, related to weakness sinus syndrome, often sposterihaetsya formation
tromboemboliv?
When
syndrome bradytahikardiyi
In case of sinus node
When synoatrialniy blockade that occasionally repeated
When expressed bradycardia (less than 50 in 1 min.)
With all the same
What time of day the most unfavorable for patients with sinus syndrome of weakness?
From midnight to dawn
From morning to afternoon
In the afternoon
Evening
Throughout the day the same
Which pathogenetic form Prystupa Morhani-Adams-Stokes equations is most common in patients
with sick sinus syndrome weakness?
Bradykardychna
Tachicardic
Bradytahikardychna
Tahibradykardychna
Mixed
At what podovzhenosti asystole appears clear symptoms Prystupa Morhani-Adams-Stokes equations:
When you pause 9.5 sec
When you pause 3.4 sec
At the 2 minute pause
If you pause more than 5 minutes
When asystole any duration
ECG makes it possible to assess:
Electrical activity of the heart
Skorotlyvist infarction
Cardiac
Sonic performance of the heart
All listed
RQ range of evidence:
The timing of pulses from sinus to skorochuvanoho infarction
Time atrial excitation
Time pulse in left bundle branch block nozhtsi
Time of momentum in the right bundle branch block nozhtsi
None of the listed
Which of the following methods to evaluate the localization of disturbances in the heart conduction
system?
Electrophysiological study
Echocardiography
Vektorokardiohrafiya
Fonokardiohrafiya
None of the above
When SR prong R always positive in the following assignments:
A. *
B.
C.
D.
E.
45.
A. *
B.
C.
D.
E.
46.
A. *
B.
C.
D.
E.
47.
A. *
B.
C.
D.
E.
48.
A. *
B.
C.
D.
E.
49.
A. *
B.
C.
D.
E.
50.
A. *
B.
C.
D.
E.
51.
A. *
B.
II
II, III, aVL
I, II, III, aVL
I, II, aVL
Always positive in all assignments
Under normal electrocardiographic transitional zone is located in the following assignments:
V3 V4
V1 V2
V5 V6
V3 V6
No transition zone
In normal adult electrocardiogram in the highest R prong in assignments:
V4
V1
V3
V6
In all assignments the same
The center of automatism of the first order are:
Synoatrialnyy node
Atrioventricular node
A bunch branch block
Purkinje fibers
All of the
Centers automatism third order are:
Bundle, fiber purkinje
Synoatrialnyy node
Cardiomyocytes
Atrioventricular node
All of the
Which of the following identification of electrocardiographic signs of left ventricular hypertrophy
indisputable?
Increasing the amplitude of R wave in aVL 11 mm more
Electrical axis deviation to the left heart
Increased R wave amplitude in the third over 15 mm
Transition zone to V5
All of the
To which of these defects is typical right ventricular hypertrophy?
Membrane defect interatrial
Aortic stenosis mouth
Aortic valve insufficiency
Mitral stenosis hole stage II
Aortic valve stenosis
What is an automatism for the center of the first order?
60-90 in 1 minute
40-60 in 1 minute
C.
D.
E.
52.
A. *
B.
C.
D.
E.
53.
A. *
B.
C.
D.
E.
54.
A. *
B.
C.
D.
E.
55.
A. *
B.
C.
D.
E.
56.
A. *
B.
C.
D.
E.
57.
A. *
B.
C.
D.
E.
58.
A. *
B.
C.
D.
E.
100-115 for 1 minute
30-45 in 1 minute
More than 90 per minute
What is an automatism for the center of the second order?
40-60 in 1 minute
60-90 in 1 minute
100-120 for 1 minute
20-35 in 1 minute
Over 120 per minute
What is an automatism for the third-order center?
20-35 in 1 minute
60-90 in 1 minute
40-60 in 1 minute
90-110 for 1 minute
Over 110 per minute
How viddyferentsiyuvaty complete atrioventricular dissociation from complete atrioventricular
block?
As a result of sample atropinovoyi
By podovzhenistyu Q R interval
Under the form of ventricular QRS complexes
Unable to differentiate
According ehokardioskopiyi
How is the capture of incomplete atrioventricular dissociation from extrasystoles?
When excited no compensatory pause
After extrasystoles no compensatory pause
Hobby premature reduction
No differences
All listed
Lead system includes the heart of everything nyzhchepererahovanoho except:
Coronary sinus
Synoatrialnoho Site
Atrioventricular node
Bundle branch block legs
Purkinje fibers
Mechanism of action of izadrina incomplete atrioventricular block:
Improves atrioventricular conduction
Improves pump function of heart
Improves automatism
None of the listed
All listed
When SLS syndrome:
Shorter interval R Q
Extended Q R interval
Shortened QRS complex
Prolonged QRS complex
None of the listed
59.
A. *
B.
C.
D.
E.
When SLS syndrome characteristic electrocardiographic feature is:
Reducing R interval Q
The presence of incomplete blockade of the right bundle
The presence of incomplete blockade of left bundle branch block
The presence of delta waves
All listed