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Slide 1 - AccessCardiology
Slide 1 - AccessCardiology

Prognostic value of atrial fibrillation in heart failure with preserved
Prognostic value of atrial fibrillation in heart failure with preserved

... Aim: To assess the influence of atrial fibrillation on mortality in heart failure with preserved ejection fraction (HFPEF) in a prospective study compared to heart failure with reduced ejection fraction (HFREF). We have hypothesized that atrial fibrillation decreases survival in HFPEF. Patients and ...
ECG Analysis Electrocardiography (ECG) is an
ECG Analysis Electrocardiography (ECG) is an

... Electrocardiography (ECG) is an interpretation of the electrical activity of the heart over time as collected from a patient during the procedural test. Modern ECG devices use sophisticated techniques like amplification, filtering, and signal analysis to accurately and conveniently measure, display, ...
chapter-5-hf-lecture
chapter-5-hf-lecture

... 4-Calcium blockers are better avoided because of their negative inotropic effect and activation of the sympathetic nervous system 5- Inotrpics (Digoxin): Symptomatic improvement in patients with systolic dysfunction - Indication: LV systolic dysfunction that remains symptomatic after treatment ACE i ...
EP show 2
EP show 2

... assigned to either ICD or antiarrhythmic-drug therapy. Over three years, statistically significant relative reductions in mortality from 27-39% were seen in the ICD group. ...
Interventional Cardiology for Structural Heart Disease
Interventional Cardiology for Structural Heart Disease

... number of patients who are being treated by interventional cardiology is growing exponentially year by year. A number of reasons have contributed to this: the patients’ demand for less invasive procedures; the development of new devices and technologies; the publication of favourable results in rand ...
Heart Rate notes
Heart Rate notes

... 4. Help maintain body __________. ...
AV Nodal Reentrant Tachycardia (AVNRT)
AV Nodal Reentrant Tachycardia (AVNRT)

... AV Nodal Reentrant Tachycardia (AVNRT) BACKGROUND INFORMATION ...
Intravenous Nicardipine Quick Reference Cardene
Intravenous Nicardipine Quick Reference Cardene

... If a peripheral vein is used, the infusion site should be changed every 12 hours. Initiate therapy at 5 mg/hour as a continuous IV infusion. The initial infusion rate of 5mg/hr IV may be increased by 2.5 mg/hr every 5 minutes to a maximum of 15 mg/hr, to maintain a SBP less than 220 mm Hg and DBP <1 ...
Template for BMJ Cases - ELSO 2016
Template for BMJ Cases - ELSO 2016

... After reviewing the international literature it was found that ECMO could be a good alternative as a rescue therapy in patients with acute right ventricular failure in the immediate postoperative of heart transplant and other high complexity cardiac surgeries (1)(2). A detailed analysis on our exper ...
Cardiovascular System
Cardiovascular System

... – Depolarizes every .8 sec. At rest – Depolarizes due to change in permeability – Pacemaker ...
Cardiac Arrest Induced by Anti-Bradycardia and Anti
Cardiac Arrest Induced by Anti-Bradycardia and Anti

... programming reducing lower rate to 30 bpm and antitachycardia pacing with inactivation of ATP. We decided not to change the position of defibrillation lead within the right ventricle and we decided not to perform a defibrillation test before hospital discharge for the poor hemodynamic stability of t ...
The Cardiac Cycle Cardiac conduction system Cardiac Muscle
The Cardiac Cycle Cardiac conduction system Cardiac Muscle

... This system, composed of specialized cardiac muscle tissue, initiates and conducts depolarization waves though the myocardium. Impulses from the S-A node pass slowly to the A-V node; impulses travel rapidly along the A-V bundle and Purkinje fibers. ...
Lymphatic System and Immunity Review
Lymphatic System and Immunity Review

... 5. What is the difference between the visceral pericardium and the parietal pericardium? 6. What is the purpose of chordae tendinae and papillary muscles? Where are they located? 7. What three distinct layers comprise the tissues of the heart wall? 8. What are the seven important functions of fibrou ...
Diseases of The Myocardium
Diseases of The Myocardium

... Beta-blocker and rate lowering cacium channel blocker can help to relieve angina and some times prevent syncope but not the prognosis. Arrythmias are common and often respond to amiodarone. Dual-chamber pacing and surgery are useful in selected group of patient to relieve outflow obstruction. ICD fo ...
Inpatient Management of Heart Failure
Inpatient Management of Heart Failure

... – Mechanism: Acute reduction in preload and afterload – Administration: Orally or IV. Escalate dose as BP tolerates. – Comments: Continue/start even in mild to moderate renal failure without hyperkalemia as renal failure will likely resolve with increased perfusion. ...
Cardiovascular Disorders/homeostatic Imbalances
Cardiovascular Disorders/homeostatic Imbalances

... • One or both cusps of mitral valve stretches and bulges into left atrium during ventricular contraction • Blood can regurgitate into the left atrium • Palpitations, fatigue, anxiety, chest pains • associated with arrhythmias (atrial fibrillation) that may progress ...
Heart 4: Fibrous Skeleton of the Heart
Heart 4: Fibrous Skeleton of the Heart

... proceed to Heart 5: Blood Supply of the Heart ...
abiocor® frequently asked questions
abiocor® frequently asked questions

... this study some patients were able to resume their normal activities such as exercising, going to the movies, or out to dinner. One patient survived for 512 days with the device and was able to be there for the birth of his great-granddaughter. What is different about the AbioCor compared to artific ...
Acute management of myocardial infarction
Acute management of myocardial infarction

... • Beta blockers (eg. Atenolol, propranolol, metoprolol) • Decreases mortality and ventricular arrythmias • Start within 24 hours • Contraindications • Acute heart failure • Heart block • Asthma • Hypotension ...
Size: 2 MB - diastolic dysfunction mgmc
Size: 2 MB - diastolic dysfunction mgmc

... of active myocardial relaxation and passive ventricular filling. • Condition that includes classic CHF findings and abnormal diastolic and normal systolic function at rest ...
Cardiovascular Disease Prevention and Treatment
Cardiovascular Disease Prevention and Treatment

... Treatment of Advanced Heart Failure Part 6  Implantable defibrillators reduce the risk of sudden death in patients with CHF, with and without prolonged QRS duration  Patients with Class II-III benefit more than Class IV patients ...
Heart Practice Quiz
Heart Practice Quiz

... 5. Movement of what ion, in what direction, causes repolarization of autorhythmic cells? Cardiac muscle cells? 6. Movement of what ions, in what directions, causes the plateau phase of cardiac muscle action potentials? 7. What is the function of the AV node? How long is the average delay? 8. Name th ...
chapter_7 - Elsevier
chapter_7 - Elsevier

... Figure 7.4 Heart regeneration in the zebrafish. (A) Longitudinal section through an intact heart. ba, bulbus arteriosus. (B) Heart after amputation of 20% of ventricle. (C) Higher magnification of unamputated ventricular apex, showing the level of amputation. (D) One day post-amputation, showing pla ...
What is Heart Failure?
What is Heart Failure?

... Sinus rhythm NYHA Functional Class III or Ambulatory Class IV symptoms ...
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Cardiac contractility modulation



Cardiac contractility modulation (CCM) is a treatment for patients with moderate to severe left ventricular systolic heart failure (NYHA class II–IV). The short- and long-term use of this therapy enhances both the strength of ventricular contraction and the heart’s pumping capacity. The CCM mechanism is based on stimulation of the cardiac muscle by non-excitatory electrical signals (NES). CCM treatment is delivered by a pacemaker-like device that applies the NES, adjusted to and synchronized with the electrical action in the cardiac cycle.In CCM therapy, electrical stimulation is applied to the cardiac muscle during the absolute refractory period. In this phase of the cardiac cycle, electrical signals cannot trigger new cardiac muscle contractions, hence this type of stimulation is known as a non-excitatory stimulation. However, the electrical CCM signals increase the influx of calcium ions into the cardiac muscle cells (cardiomyocytes). In contrast to other electrical stimulation treatments for heart failure, such as pacemaker therapy or implantable cardioverter defibrillators (ICD), CCM does not affect the cardiac rhythm directly. Rather, the aim is to enhance the heart’s natural contraction (the native cardiac contractility) sustainably over long periods of time. Furthermore, unlike most interventions that increase cardiac contractility, CCM is not associated with an unfavorable increase in oxygen demand by the heart (measured in terms of Myocardial Oxygen Consumption or MVO2). This may be explained by the beneficial effect CCM has in improving cardiac efficiency. A meta-analysis in 2014 and an overview of device-based treatment options in heart failure in 2013 concluded that CCM treatment is safe, that it is generally beneficial to patients and that CCM treatment increases the exercise tolerance (ET) and quality of life (QoL) of patients. Furthermore, preliminary long-term survival data shows that CCM is associated with lower long-term mortality in heart failure patients when compared with expected rates among similar patients not treated with CCM.
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