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Classical demonstration of atrial flutter with slow ventricular rate
Classical demonstration of atrial flutter with slow ventricular rate

... Atrial flutter is a macro-re-entrant tachycardia predisposing to atrial thrombus formation often seen in patients with structural heart disease.1 2 Atrial flutter with atrioventricular node blockade is a potentially life-threatening cause of bradycardia and decompensation of heart failure usually seen ...
12chuyendao_ECG_2 - maritime advance life
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... MIs can affect a single heart wall or more than one area ST-segment elevations appear in the leads monitoring all of the involved areas Areas involved are reflected by the MI descriptive name ...
Imaging Essentials Before VAD Placement
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... aortic regurgitation has a negative impact on forward flow provided by an LVAD due to regurgitation of VAD flow back into the left ventricular cavity. It is generally recommended that moderate and greater levels of severity of aortic regurgitation should be corrected at the time of VAD insertion (13 ...
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... Medications Used to Manage Feline HCM he most important things an owner can do for a cat diagnosed with HCM are to monitor them with regular checkups and follow his veterinarian’s advice closely. Though there is no known cure for HCM, veterinarians often prescribe medications to treat various signs ...
Cardiac - PBworks
Cardiac - PBworks

... increases the obstruction Right ventricle must work harder to eject blood into the pulmonary artery. ...
l-Transposition of the Great Arteries
l-Transposition of the Great Arteries

... corrected TGA.” Some children may also have ventricular septal defects or obstruction to flow into the pulmonary artery. What causes it? The cause is unknown, but genetic factors may contribute to it. How does it affect the heart? In this condition, the blood is normally routed but the right ventric ...
bio310 test2 with answers
bio310 test2 with answers

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H5 THE TRANSPORT SYSTEM

...  The left ventricle quickly fills with blood and then ventricular systole starts. The pressure in the ventricle increases in 0.1s. The pressure remains high for the next 0.2s and the volume of the ventricle goes from 100% to almost 0%. During diastole, the ventricle also fills up with blood so its ...
Lecture #1 - Jewish Hospital Cardiothoracic Surgical Research
Lecture #1 - Jewish Hospital Cardiothoracic Surgical Research

... (c) Phase III (EJ - Ejection) - Pressure in ventricle now higher than aorta/pulmonary artery, opening aortic/pulmonic valve and blood flow to arterial system occurs. Characteristics: mitral/tricuspid valve closed and aortic/pulmonic valve open, low pressure changes, high volume changes. (d) Phase IV ...
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Paulo Pinho António Gaspar, Jorge Almeida, Benjamim Marinho

... tissue were resected, and 2 bioprosthetic valves were implanted. AMVT is a rare congenital cardiac anomaly, in particular in adults, that was first described by Mclean et al.1 AMVT presents mostly with left ventricular outflow tract obstruction, usually being diagnosed in the first or second decade ...
Bio211 Lecture 1
Bio211 Lecture 1

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BME lecture 6 - pv loops (Sept 14, 2004)
BME lecture 6 - pv loops (Sept 14, 2004)

... LV volume (ml) Figure 6-6. Comparison of pressure-volume loops in calves with normal (top) and failing ventricles (below) during continuous flow (CF) ventricular assist device (VAD) support at 75% and 100% bypass. Notice that in normal ventricle model, the only effect is a leftward shift of PV loop. ...
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Pre op Assessment - Iowa Society of PeriAnesthesia Nurses

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... 2. Patients with symptomatic aortic stenosis (moderate to severe stenosis) even with normal cardiac output at rest. 3. Patients who exhibit LV dysfunction even they are asymptomatic. ...
NUR 4206 By Linda Self - Arkansas Tech University
NUR 4206 By Linda Self - Arkansas Tech University

...  Myoglobin—heme protein. Released from myocardial tissue within 1-3 hours after injury. Less specific as may be elevated in renal and musculoskeletal disease  Troponin T and I—proteins found only in cardiac muscle, detected within 3-4 hours, peak in 4-24 and remain elevated for 1-3 weeks ...
Cardiac - LifeLabs Genetics
Cardiac - LifeLabs Genetics

... Cardiac arrhythmias are heart rhythm disorders that can lead to sudden cardiac death. Inherited arrhythmias are caused by abnormal electrical activity of the heart that results in irregular heart beats or an abnormal heart rhythm. As many as 1 in 500 people may have an inherited arrhythmia. Long QT ...
Basic Hemodynamics for the Cath Lab and ICU
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... As demand goes up, inotropic response of the heart increases and the coronary vasculature adapts by vasodilating to provide an adequate oxygen supply. If, due to atherosclerotic disease, the coronary arteries are unable to sustain the myocardium, ischemia ensues and the flow is predicted by: ...
RAD 204 PATHOLOGY
RAD 204 PATHOLOGY

... INTERventricular septum: membranous (fibrous) portion & muscular portion size & site of defect determines extent of shunt presents as cardiac failure in infants or murmur in older children/adults signs: pansystolic murmur {flow from high pressure left ventricle to low pressure right ventricle during ...
Congenital Heart Disease
Congenital Heart Disease

... *Assessment . (4 to 8 week of age ) fatigue…murmur…thrill may be palpable.. Echo .ECG, MRI ,(RT ventricle hypertrophy ) Treatment … cardiac catheterization .. Surgery ...
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... D) In older patients, paroxysmal atrial fibrillation is a contraindication to sport practice ...
Pathophysiology of hypertrophic cardiomyopathy determines its
Pathophysiology of hypertrophic cardiomyopathy determines its

... Pathophysiology of HCM All HCM patients typically have left ventricular (LV) diastolic dysfunction due to increased chamber stiffness and impaired relaxation, which prevents increase in exercise stroke volume and cardiac output (4). This, along with increased LV diastolic filling pressures correlate ...
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Hypertrophic cardiomyopathy



Hypertrophic cardiomyopathy (HCM) is a primary disease of the myocardium (the muscle of the heart) in which a portion of the myocardium is hypertrophied (thickened) without any obvious cause, creating functional impairment of the cardiac muscle. It is a leading cause of sudden cardiac death in young athletes.The occurrence of hypertrophic cardiomyopathy is a significant cause of sudden unexpected cardiac death in any age group and as a cause of disabling cardiac symptoms. Younger people are likely to have a more severe form of hypertrophic cardiomyopathy.HCM is frequently asymptomatic until sudden cardiac death, and for this reason some suggest routinely screening certain populations for this disease.A cardiomyopathy is a disease that affects the muscle of the heart. With HCM, the myocytes (cardiac contractile cells) in the heart increase in size, which results in the thickening of the heart muscle. In addition, the normal alignment of muscle cells is disrupted, a phenomenon known as myocardial disarray. HCM also causes disruptions of the electrical functions of the heart. HCM is most commonly due to a mutation in one of nine sarcomeric genes that results in a mutated protein in the sarcomere, the primary component of the myocyte (the muscle cell of the heart). These are predominantly single-point missense mutations in the genes for beta-myosin heavy chain (MHC), myosin-binding protein C, cardiac troponinT, or tropomyosin. These mutations cause myofibril and myocyte structural abnormalities and possible deficiencies in force generation. Not to be confused with dilated cardiomyopathy or any other cardiomyopathy.While most literature so far focuses on European, American, and Japanese populations, HCM appears in all ethnic groups. The prevalence of HCM is about 0.2% to 0.5% of the general population.
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