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CHAPTER 13- The Spinal Cord and Spinal Nerves
CHAPTER 13- The Spinal Cord and Spinal Nerves

... B) It allows an individual to maintain balance when withdrawing from harm’s way. C) It is contralateral. D) It involves only one spinal segment. E) It is polysynaptic. 43) Which of the following is NOT a reflex typically used for diagnosing neurological disorders? A) patellar reflex B) Babinski sign ...
Nervous Systems
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Slide 1
Slide 1

... 28.5 The action potential propagates itself along the axon  The frequency of action potentials (but not their strength) changes with the strength of the stimulus. ...
powerpoint version - University of Arizona
powerpoint version - University of Arizona

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No Slide Title

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... – Action potentials travels directly from the presynaptic to the postsynaptic cells via gap junctions. – Invertebrate giant axons – Present in vertebrate brain in stereotype behavior like a fish flapping its tail to escape a predator. – Are fast connections. Copyright © 2002 Pearson Education, Inc., ...
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nervous system text b - powerpoint presentation

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... devoid of synaptic endings (Bullock and Horridge, 1965). The neuropile, which can be distinguished by its finer and more tangled texture, thus becomes the most important region, because this is the only known place of neuronal contacts. Hence it has acquired functional significance as the primary pl ...
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... from wholemounts of hatchlings labeled with markers for serotonin and tyrosinated tubulin. Our data show that the central nervous system of a juvenile S. roscoffensis consists of an anterior compact brain, formed by a dense, bilobed mass of neuronal cell bodies surrounding a central neuropile. The n ...
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Neuromuscular junction



A neuromuscular junction (sometimes called a myoneural junction) is a junction between nerve and muscle; it is a chemical synapse formed by the contact between the presynaptic terminal of a motor neuron and the postsynaptic membrane of a muscle fiber. It is at the neuromuscular junction that a motor neuron is able to transmit a signal to the muscle fiber, causing muscle contraction.Muscles require innervation to function—and even just to maintain muscle tone, avoiding atrophy. Synaptic transmission at the neuromuscular junction begins when an action potential reaches the presynaptic terminal of a motor neuron, which activates voltage-dependent calcium channels to allow calcium ions to enter the neuron. Calcium ions bind to sensor proteins (synaptotagmin) on synaptic vesicles, triggering vesicle fusion with the cell membrane and subsequent neurotransmitter release from the motor neuron into the synaptic cleft. In vertebrates, motor neurons release acetylcholine (ACh), a small molecule neurotransmitter, which diffuses across the synaptic cleft and binds to nicotinic acetylcholine receptors (nAChRs) on the cell membrane of the muscle fiber, also known as the sarcolemma. nAChRs are ionotropic receptors, meaning they serve as ligand-gated ion channels. The binding of ACh to the receptor can depolarize the muscle fiber, causing a cascade that eventually results in muscle contraction.Neuromuscular junction diseases can be of genetic and autoimmune origin. Genetic disorders, such as Duchenne muscular dystrophy, can arise from mutated structural proteins that comprise the neuromuscular junction, whereas autoimmune diseases, such as myasthenia gravis, occur when antibodies are produced against nicotinic acetylcholine receptors on the sarcolemma.
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