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Nerve Tissue
Nerve Tissue

...  tactile corpuscles of Meissner – glabrous skin  end bulbs (of Krause) – responds to pressure, genital corpuscles Pacinian (Vater-Pacini) corpuscles – vibration Golgi-Mazzoni corpuscles – in the fingertips Ruffini endings – responds to pressure neurotendinous organs (Golgi tendon organs) neuromusc ...
13-1 CHAPTER 13 SYNAPSES The nervous system consists of
13-1 CHAPTER 13 SYNAPSES The nervous system consists of

... in the electron micrograph of Figure 13-1B. It is believed that transmission from one cell to another at a synapse like the one just described (a chemical synapse, as opposed to an electrical synapse) is accomplished by release of a substance, the transmitter substance, from the synaptic vesicles in ...
Scientific Explanation of Kinesio® Tex Tape
Scientific Explanation of Kinesio® Tex Tape

... increases the tensile strength of the fascia, it can unfortunately restrict the very structure it aims to protect. This may result in a mild decrease in joint range of motion to fascial binding of muscles, nerves and blood vessels (e.g. Compartment syndromes). If this fascial contraction can be inte ...
Neurons and Networks. An Introduction to Behavioral Neuroscience, Second Edition Brochure
Neurons and Networks. An Introduction to Behavioral Neuroscience, Second Edition Brochure

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The Nervous System (ppt).
The Nervous System (ppt).

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PowerPoint version

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Answers to What Did You Learn questions

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Babinski reflex and corticospinal tract lesion
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Nervous System - Serrano High School AP Biology
Nervous System - Serrano High School AP Biology

... membrane, the vesicles move to the membrane that faces the cleft. Once depolarized Ca2+ moves into the cell through voltage channels. The sudden increase of calcium ions stimulates the synaptic vesicles to fuse with the presynaptic membrane and spill the neurotransmitter into the cleft by exocytosis ...
CNS consists of brain and spinal cord PNS consists of nerves CNS
CNS consists of brain and spinal cord PNS consists of nerves CNS

... All stretch reflexes are monosynaptic and ipsilateral Reciprocal inhibition also occurs—IIa fibers synapse with interneurons that inhibit  motor neurons of antagonistic muscles Example: In patellar reflex, stretched muscle (quadriceps) contracts and antagonists (hamstrings) relax ...
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MOTOR NEURON DISEASE
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... multifocal with nerve conduction features reflecting demyelination. to GBS The major differences between the two conditions are in the time course and their response to corticosteroids Prednisone, plasmapheresis, and IVIg Multifocal Motor Neuropathy with Conduction Block more common in men Progressi ...
Handout 1 - Porterville College Home
Handout 1 - Porterville College Home

... a. Carry impulses ________________ _____ the cell body 3. __________________________ a. Carry impulses _____________________ the cell body Course Objective #10: Describe the role played by neurotransmitters in nerve impulse transmission within the synapse. A. Neurons _______________________ to each ...
Examination of Physiology Class_____ Name_____________
Examination of Physiology Class_____ Name_____________

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Neuromuscular junction



A neuromuscular junction (sometimes called a myoneural junction) is a junction between nerve and muscle; it is a chemical synapse formed by the contact between the presynaptic terminal of a motor neuron and the postsynaptic membrane of a muscle fiber. It is at the neuromuscular junction that a motor neuron is able to transmit a signal to the muscle fiber, causing muscle contraction.Muscles require innervation to function—and even just to maintain muscle tone, avoiding atrophy. Synaptic transmission at the neuromuscular junction begins when an action potential reaches the presynaptic terminal of a motor neuron, which activates voltage-dependent calcium channels to allow calcium ions to enter the neuron. Calcium ions bind to sensor proteins (synaptotagmin) on synaptic vesicles, triggering vesicle fusion with the cell membrane and subsequent neurotransmitter release from the motor neuron into the synaptic cleft. In vertebrates, motor neurons release acetylcholine (ACh), a small molecule neurotransmitter, which diffuses across the synaptic cleft and binds to nicotinic acetylcholine receptors (nAChRs) on the cell membrane of the muscle fiber, also known as the sarcolemma. nAChRs are ionotropic receptors, meaning they serve as ligand-gated ion channels. The binding of ACh to the receptor can depolarize the muscle fiber, causing a cascade that eventually results in muscle contraction.Neuromuscular junction diseases can be of genetic and autoimmune origin. Genetic disorders, such as Duchenne muscular dystrophy, can arise from mutated structural proteins that comprise the neuromuscular junction, whereas autoimmune diseases, such as myasthenia gravis, occur when antibodies are produced against nicotinic acetylcholine receptors on the sarcolemma.
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