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POLIOMYELITIES Copyright 2005 Lippincott Williams & Wilkins POLIOMYELITIES Definition: Acute viral infection of motor nuclei of CNS mainly motor nuclei of anterior horn cell of spinal cord and motor nuclei of the cranial nerve leading to lower motor neuron lesion (LMNL) of flaccid paralysis with normal sensation. Poliomyelitis, often called polio or infantile paralysis, is an acute viral infectious disease spread from person to person, primarily via the fecal-oral route.The term derives from the Greek poliós (πολιός), meaning "grey", myelós (µυελός), referring to the "spinal cord", and the suffix -itis, which denotes inflammation. Types of Polio virus: 1- Brunhilde 2-Lansing 3- Lean infection of one virus give immunity to this type only Copyright 2005 Lippincott Williams & Wilkins Types of Vaccination: 1- Sabine: Attenuated live polio virus Anticipated intramuscular Dose------- 2,4,6 month poster dose 18 month preschool age 4 years Advantage : Complete immunity with in 3 days 2- Salk: • • • • Killed virus attenuated oral or not supported by intestinal tract Advantages: effective, safe Dose: 2,4,6 month, 4 years Disadvantages: need repeated injection, delay in immunity takes 3 weeks to response & incomplete immunity Stages: 1- Acute Stage; The first 2 Weeks • Viral infection (vomiting, headache at the end of acute stage then paralysis appear in respiratory muscle and extremity) Copyright 2005 Lippincott Williams & Wilkins 2- Recovery Stage; From 2 weeks to 2 Years • Called stage of functional recovery because: • 1- Subside to edema • 2- Improved neural plasticity • 3- Hypertrophy of non affected muscle Character paralysis: • 1- LMNL 2- Flaccid • 3- massive (affected more than one group) • 4- Asymmetrical lesion • 5- Patchy distribution After 2 years recovery cannot occur because: • 1- New formation of new motor end plate • 2- Orientation of the axon is parallel to muscle Chronic Stage: After 3 years a- Muscle skletal deformity : Muscle weakness------contracture……Deformity b- Respiratory affection: Affection in respiratory center in medulla lead to irregular breathing Affection in Spinal cord or nerves of respiration ------- respiratory muscles become weak Copyright 2005 Lippincott Williams & Wilkins Approximately 90% of polio infections cause no symptoms at all, affected individuals can exhibit a range of symptoms if the virus enters the blood stream. 1% of cases the virus enters the central nervous system, and destroying motor neurons, leading to muscle weakness and acute flaccid paralysis. Different types of paralysis may occur, depending on the nerves involved. Spinal polio is the most common form, characterized by asymmetric paralysis that most often involves the legs. Bulbar polio leads to weakness of muscles innervated by cranial nerves. Bulbospinal polio is a combination of bulbar and spinal paralysis Copyright 2005 Lippincott Williams & Wilkins Classification of poliomyelitis: • The term poliomyelitis is used to identify the disease caused by any of the three serotypes of poliovirus. Two basic patterns of polio infection are described: • Minor illness which does not involve the central nervous system (CNS), sometimes called abortive poliomyelitis. • Major illness involving the CNS, which may be paralytic or nonparalytic • In most people with a normal immune system, a poliovirus infection is asymptomatic. Rarely the infection produces minor symptoms; these may include upper respiratory tract infection (sore throat and fever), gastrointestinal disturbances (nausea, vomiting, abdominal pain, constipation or, rarely, diarrhea), and influenza-like illness. Copyright 2005 Lippincott Williams & Wilkins • The virus enters the central nervous system in about 3% of infections. Most patients with CNS involvement develop non-paralytic aseptic meningitis, with symptoms of headache, neck, back, abdominal and extremity pain, fever, vomiting and irritability. Approximately 1 in 1000 cases progress to paralytic disease, in which the muscles become weak, floppy and poorly controlled, and finally completely paralyzed; this condition is known as acute flaccid paralysis Depending on the site of paralysis, paralytic poliomyelitis is classified as spinal, bulbar, or bulbospinal • Transmission • Poliomyelitis is highly contagious via the oral-oral (oropharyngeal source) and fecal-oral (intestinal source) routes . It is seasonal in temperate climates, with peak transmission occurring in summer and autumn. • • Risk Factors of polio include: immune deficiency, malnutrition, tonsillectomy, physical activity immediately following the onset of paralysis, skeletal muscle injury due to injection of vaccines or therapeutic agents, and pregnancy. Although the virus can cross the placenta during pregnancy, the fetus does not appear to be affected by either maternal infection or polio vaccination.[Maternal antibodies also cross the placenta, providing passive immunity that protects the infant from polio infection during the first few months of life. • Copyright 2005 Lippincott Williams & Wilkins Physical Therapy Evaluation: 1- History: Personal History Past History Present History History of vaccination 2- Informal evaluation; A- Look to the child while entering the treatment room (if mother carrying him or not, walk alone, gait and type of brace B- On the plinth, general conditions of the child(anemic , healthy) C- Observe the chest from the anterior view(rate, pattern of breathing: shallow, irregular D- Positions of the limb and leg length discrepancy E- Skin condition(ulcer due to brace , scar due to tendon release F-Spontaneous motility G- Deformities:# Acute Stage : Muscle imbalance , prolonged bed rest, inadequate P.T, Shortening, Contracture Weight bearing on weak joints Copyright 2005 Lippincott Williams & Wilkins Chronic Stage: Deformities like: Lower Limb : Hip---- Flexion, abduction, external rotation Knee: Flexion deformity, genu valgus, genu varum, Genu Recurvatum due to Ms imbalance between quadriceps and hamstring Ms Ankle: equines deformity(plantar flexion), calcenous deformity(dorsiflexion) Varus deformity(inversion), valgus deformity(eversion) Talipus equino varus, Talipus equino valgus Talipus calcenus varus, Talipus calcenus valgus Quick Test : Crock lying position to detect leg discrepancy: If end of femur is forward --------- shortening of tibia If end of femur upward------------ shortening of femur Postural Fixation ; raise limb upward let it drop Sudden drop limb ---------- hypotonia Copyright 2005 Lippincott Williams & Wilkins 3- Formal Evaluation; Muscle Tone Flexibility test Muscle Test ROM Measurement Postural assessment Gait Analysis A- Muscle tone: By Observation: Supine----- Flaccid paralysis of limb due to hypotonia---------Frog like position(Abd, Ext Rotation hip, Plantar flex ankle). Passive Movement Don’t forget to support distal part Postural fixation raise limb then sudden drop : polio ----sudden Drop B- Flexibility Test: To test tightness of tendoachilis : Supine lying with hip and knee flexion Grasp : Thumb on sole of foot , Index on shaft of tibia, 4 fingers cubing on the heel Copyright 2005 Lippincott Williams & Wilkins From flexion knee make dorsiflexion ankle---------tightness of soleus From extension knee make dorsiflexion ankle- Tightness in Gastrocniemus Muscle Test: Above 3 Years------------------ MMs Test Below 3 years----------------Functional Ms test Zero---------- No Contraction Sub functional-----------Not complete ROM Functional ------------------- complete ROM Methods: a- Unfavorable position: Prone lying will facilitate neck trunk extension and by this way you can test neck and back Ms Half prone for testing hip and knee flexors b- Reflexes according to age : Positive supporting to test extensor of lower limb and plantar flexors C- Toys put toy in direction that can facilitate movement d- spontaneous motility: uncontrolled movements e- Tactile stimulation( Scratching, Squeezing, tapping) Hip:Tactile Stimulation of hip flexors (iliopsoas): supine lying Copyright 2005 Lippincott Williams & Wilkins Grasp: Thumb on the sole of the foot, Index on shaft of tibia - 4 fingers cubing heel of the foot Scratch: on inguinal ligament by followed passive movement until you feel that child can perform it actively Gluteus Maximus: Half Prone lying with untested limb between my leg, Tested limb kept in flexion to isolate hamstring Scratch on Gluteus Maximus followed by passive hip extension, or painful stimulus on ant aspect of thigh above just knee Hip Adductors: Supine lying: Grasp One hand cubing heel of the foot other hand Scratch medial aspect of thigh followed by passive hip adduction by the hand which cup heel or painful stimulus on the greater trochanter Knee: Tactile Stimulation: Knee flexors Position : prone lying Knee slight flexion(10-15) to isolate gastrocnemius Scratch by the hand which support pelvis on the back of the knee Other hand which cup heel make passive knee flexion Painful Stimuli on the ant aspect of distal part of tibia To isolate biceps femoris put tested knee slight flexion with Ext Rotation To isolate Semitendinosus put tested knee slight flexion with Med Rotation Copyright 2005 Lippincott Williams & Wilkins Knee extensors (Quadriceps): Position : Supine lying tested limb in hip and Knee slight flexion Grasp : thumb above knee , index on lateral aspect of thigh, middle two fingers below knee from back for supporting, little finger to initate movement Scratch by the hand which support pelvis on the Pelly of the muscle Other hand which cup heel make passive knee flexion Painful Stimuli on the post aspect of calcaneus Ankle: Tibilais anterior Tibilais posterior Pronei(longus, brevis, tertus) in these 3 muscles must put knee in slight flexion(10-15) : 1-To isolate gastrocnemius 2- to prevent damage of collateral ligaments of knee 3- To prevent compression of post aspect of knee Tactile stimulation to Tibilais anterior: Position: supine lying with flexion knee , Thumb flex toes to isolate flexor hallicus, and digitorium , Index on ant aspect and medial of foot to maintain full inversion and plantar flexion, Middle finger palpat tendon of tibialis anterior, Ring and little finger on heel to perform passive dorsi flexion, Painful stimulus : On the sole of the foot Copyright 2005 Lippincott Williams & Wilkins • Tibialis Posterior: Mainly inversion & assist in • • • • • planter flexion Grasp: one hand catch lower end of tibia and other hand hold foot from ankle Scratch: on medial malleoli by little finger Peroneus longus---------eversion +plantar flexion Tendon Below Lat Malleoli Peroneus brevis--------eversion +mid position of ankle Tendon Below Lat Malleoli Peroneus Tartius -------eversion +dorsi flexion Tendon above Lat Malleoli Painful stimulus on the medial aspect of foot Copyright 2005 Lippincott Williams & Wilkins