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Prof Andrew Leisewitz Companion Animal Clinical Studies [email protected] Key Publications Goddard A, Leisewitz AL. Canine parvovirus. The Veterinary clinics of North America Small animal practice 2010;40:1041-1053. Matjila PT, Carcy B, Leisewitz AL, et al. Preliminary evaluation of the BrEMA1 gene as a tool for associating babesia rossi genotypes and clinical manifestation of canine Babesiosis. Journal of clinical microbiology 2009;47:3586-3592. Leisewitz AL, Rockett K, Kwiatkowski D. BCG-malaria co-Infection has paradoxical effects on C57BL/6 and A/J mouse strains. Parasite immunology 2008;30:1-12. Leisewitz AL, Rockett KA, Gumede B, et al. Response of the splenic dendritic cell population to malaria infection. Infection and immunity 2004;72:4233-4239. Andrew Leisewitz graduated with his BVSc degree in 1987. This was followed by an Hons in 1991, an MMedVet(Med) in 1995 and a PhD in 2006. Andrew was appointed as a senior lecturer in the then Departemnt of Medicine at Onderstepoort in June 1990. He is now full professor of Companion Animal Clinical Studies and is a diplomat of the European College of Veterinary Internal Medicine – Companion Animals. He has worked as a specialist clinician in the Onderstepoort Veterinary Academic Hospital for over 20 years. He is an active teacher of undergraduate and post graduate veterinary students, leads the section of Small Animal Medicine within the Department and is a frequently invited speaker and presenter of continuing education for the profession in the sub-region. Research His interest is in disease mechanisms of infectious diseases. Particular interests include the use of animal models of human disease to advance global health. His PhD examined the immune response to blood stage malaria infection in a murine model system and was completed at the Weatherall Institute for Molecular Medicine, Oxford. Canine babesiosis now forms the basis of the majority of his work. This is a malaria like disease and in many ways offers unique modelling opportunities and comparative pathologies for human malaria. In addition to this, he has an interest in the pathogenesis of central nervous system inflammation and demyelination as caused by canine distemper virus infection (which acts as a model for human multiple sclerosis). He has also worked on canine parvo virus induced enteritis where he has examined the acid base and gut wall permeability pathology. 14