Survey
* Your assessment is very important for improving the workof artificial intelligence, which forms the content of this project
* Your assessment is very important for improving the workof artificial intelligence, which forms the content of this project
Running head: BULIMIA NERVOSA 1 Bulimia Nervosa Kelsey Maw DSU BULIMIA NERVOSA 2 Bulimia Nervosa Introduction A 45 year old female patient presented to her primary care provider’s office for a checkup. She reports that she has been feeling more tired than usual and experiences dizzy spells. Upon review of health history the nurse discovers the woman has been having irregular menstrual periods and having difficulty sleeping. The nurse begins her physical assessment. Upon inspection of the oral cavity the nurse notes deterioration of enamel of the patient’s teeth. Mucous membranes and skin of the face and hands are dry. The parotid gland appears enlarged. The patient’s blood pressure is 95/55 with a resting heart rate of 58. The nurse also notes abdominal bloating and some tenderness upon palpation. She also has bilateral lower extremity edema. Blood work reveals hypokalemia and increased BUN. The nurse suspects the patient is suffering from an eating disorder. After reviewing findings with the patient, she reluctantly reports that since her divorce 9 months ago she has been binge-eating and vomiting. Nursing diagnoses and implications for this patient include disturbed thought processes related to poor body image, indicators of depression and insomnia; imbalanced nutrition related to inadequate nutrition and self-induced vomiting; fluid volume deficit related to electrolyte imbalances caused by self-induced vomiting; and impaired skin integrity as evidenced by dry skin and mucous membranes; and lower extremity edema. History Bulimia nervosa (BN) is considered a relatively new disease. It was recognized in 1979 by Gerald Russell. Until recently there was not much research of information available about BULIMIA NERVOSA 3 bulimia. Authors Van den Eynde & Schmidt (2008) attribute “celebrity cases” as well as increasing prevalence of this disorder on the research that has been done. Diagnosis According to the Diagnostic and Statistical Manual of Mental Disorders (DSM 2000) there must be behavior and cognitive symptoms present for a diagnosis of bulimia nervosa to accurately be made. Additional criteria includes experiencing recurrent episodes of eating large amounts of food, or binge eating twice a week for three consecutive months followed by behaviors that reduce or prevent weight gain such as: excessive exercise, vomiting, fasting, abusing medications, laxatives, diuretics, or enemas. Cognitive symptoms must also be present and are described as having “undue influence of body weight or shape on self-evaluation” (Williams, Watts, & Wade, 2012) which is generally related to self-worth . Labs & Imaging In most cases a “complete blood cell count, erythrocyte sedimentation rate, urinalysis, and measurements of liver function, electrolytes, and thyroid-stimulating hormone” tests should be ordered (Williams & Goodie, 2007, p. 19). In addition, if a patient presents with bradycardia or electrolyte abnormalities they should have an electrocardiogram (Williams & Goodie, 2007). BULIMIA NERVOSA 4 Treatment Options The exact cause of bulimia nervosa is still unclear, which is one of the reasons it is so difficult to predict and treat. Despite these reasons, there are some options available that have shown to have good outcomes. Psychological treatment is one option. There are different types of therapy such as cognitive behavioral therapy or interpersonal therapy that may be helpful. Cognitive behavior therapy is provided by a trained mental health professional, which can often be an impracticable option for some patients, “with a recommended course of treatment of twenty 50-minute sessions over 4 to 5 months.” Self-help and guided self- help are another option discussed by Van den Eynde & Schmidt (2008). The last treatment option is pharmacological. Anti-depressants, specifically SSRI’s and have been used in cases of bulimia nervosa. There is still a lot of research to be completed in this area, especially in regards to the combination of treatment options and determining success. Pharmacological Treatment of Bulimia Nervosa Antidepressants are the main class of medications prescribed in the treatment of bulimia nervosa. “The rationale for this was high comorbidity between BN and affective disorders, and findings of serotonin system dysfunctions in BN. According to some authors antidepressants have antibulimic properties regardless of the presence of mood symptoms, but according to others it remains unclear whether this effect is direct or indirect by lowering depressive symptoms” (Eynde & Schmidt, 2008). BULIMIA NERVOSA 5 SSRI’s are typically the first choice in the treatment of BN. Fluoxetine is approved by the FDA for treatment of bulimia nervosa and has been the most effective up to this point. In general, a higher dose of 60 mg would be prescribed for a patient diagnosed with bulimia nervosa. Other medications, including mood stabilizers and medications such as ondansetron have had positive results in some studies. A major factor with some psychotropics and feasibility of treatment is the fact that they require administration more than once a day and can be very expensive. Outcomes Bulimia nervosa can be a difficult disorder to treat and there are many variables that will affect the outcome. Authors Williams, Watts, & Wade (2012) use the terms remission, recovery and relapse as the best indicators of how effective treatment has been. In many cases a person never fully overcomes bulimia nervosa but simply displays fewer behaviors or has less frequent episodes. Because each person will exhibit different symptoms, treatment can be a long process and it’s difficult to achieve true remission. A problem that has been identified is when focus is only placed on the absence of behavior symptoms when underlying cognitive aspects are still present (Williams, Watts, & Wade, 2012). Plan of Care The most likely course of care for this patient would be to provide a referral for outpatient cognitive behavioral therapy in addition to the administration of fluoxetine. In addition to the physician, a therapist and dietician should be involved in planning care of this patient. The initial goal would be focused on establishing and maintaining a healthy weight. BULIMIA NERVOSA 6 Williams & Goodie (2007) suggest focusing on educating the patient about healthy eating habits and consequences of binging and purging. They also discuss the need to establish “selfmonitoring of eating, reducing overeating and eliminating dieting” (Williams & Goodie, 2007, p. 19). This patient would most likely benefit from support about self-perception and interpersonal therapy (Williams & Goodie, 2007). Conclusion Eating disorders have become more common, affecting both women and men. Environmental, psychological and genetic factors can all contribute to the development of an eating disorder like bulimia nervosa. Nurses and health care workers play important roles in helping identify eating disorders. We should work to build trusting relationships with our patients and be alert to signs and symptoms of bulimia nervosa and diagnostic criteria such as recurrent binge eating, compensatory behaviors, and the way the body shape and weight is viewed by the patient. Treatment can be a very long process that often involves behavior and cognitive therapies that may be combined with pharmacological interventions. Patients often experience many relapses and nurses can play a large supporting role. Nurses can not only work to treat physical symptoms, but can help the patient develop a positive body image and improve self-esteem. BULIMIA NERVOSA 7 References Sedghizadeh, P. P. (2013). Bulimia Nervosa. The New England Journal of Medicine, 368(13), 1238. Retrieved from http://search.proquest.com.libproxy.dixie.edu/nursing/docview/1321934798/abstract/ 81106D510F8245D9PQ/1?accountid=27045 Van den Eynde, F., & Schmidt, U. (2008). Treatment of bulimia nervosa and binge eating disorder. Psychiatry, 7(4), 161-166. http://dx.doi.org/doi:10.1016/j.mppsy.2008.02.001 Walsh, T. B., Fairburn, C. G., Mickley, D., Sysko, R., & Parides, M. K. (2004). Treatment of Bulimia Nervosa in a Primary Care Setting. The American Journal of Psychiatry, 161(3), 556-561. Retrieved from http://search.proquest.com.libproxy.dixie.edu/nursing/docview/220476870/AE98D861 F4F946E3PQ/27?accountid=27045 Williams, P. M., & Goodie, J. (2007). Identifying and treating eating disorders. Family Practice Recertification, 29(8), 16-23. Retrieved from http://web.b.ebscohost.com.libproxy.dixie.edu/ehost/pdfviewer/pdfviewer?sid=6af9a4 ce-650b-4131-9783-1001236c689a%40sessionmgr115&vid=0&hid=128 Willimas, S. E., Watts, T. K., & Wade, T. D. (2012). A review of the definitions of outcome used in the treatment of bulimia nervosa. Clinical Psychology Review, 32(), 292-300. Retrieved from doi:10.1016/j.cpr.2012.01.006