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Bovine Respiratory Disease
Bovine Respiratory Disease

... Some of the viral agents produce only mild clinical signs by themselves, but they may cause severe signs and death when combined with other viral or bacterial agents and stress. Many normal cattle carry one or more of the bacterial and viral agents in their upper respiratory system with no ill effec ...
How was bovine tuberculosis detected in Kentucky
How was bovine tuberculosis detected in Kentucky

... How is bovine TB transmitted? Bovine TB is spread primarily through the exchange of respiratory secretions between infected and uninfected animals. This transmission usually happens when animals are in close contact with each other. Over 95% of bovine TB cases are the result of direct contact with i ...
Infection and Disease II
Infection and Disease II

... How they get in Tissue specificity (of the pathogen) is a serious barrier to the entry of most microorganisms (more on this later) ...
Our Body`s Defense
Our Body`s Defense

... multiply when they encounter a pathogen • Killer cells-stop the spread of disease • Helper cells-aide in the activation of B-cells and Killer T-cells and control the body’s immune system ...
Emerging viruses (Current Opinion in Virology)
Emerging viruses (Current Opinion in Virology)

... vectors of emerging viruses’ Mackenzie and Jeggo provide a comprehensive review of the role of different host species and vectors as drivers of infectious disease emergence. Their discussion of programs focused on proactively identifying potential risks to human health dovetails with Holmes’ contrib ...
Newsletter
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Foot and Mouth Disease United Kingdom 2001
Foot and Mouth Disease United Kingdom 2001

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molecular mimicry - Institute of Pathophysiology

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Biosecurity in the Suckler Herd

... between groups of animals within the same herd. Biosecurity is the cheapest and most effective method of disease control. To protect the health of your herd it is important to draw up a biosecurity plan with the help of your vet. The risks associated with particular diseases entering a herd will var ...
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... parts of the world including north and south america. it is found in the soil and growth is enhanced by the presence of bird and bat excreta. environments containing such material are often implicated as sources of human infection. the lungs are the main site of infection but dissemination to the li ...
(34.6 Æ 0.2 P 0.05), CD8 (22.6 Æ 0.4 P 0.05), CD4/CD8 (1.62 Æ
(34.6 Æ 0.2 P 0.05), CD8 (22.6 Æ 0.4 P 0.05), CD4/CD8 (1.62 Æ

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old medical terms

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on intestinal worms. - Belle Plaine Animal Hospital

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Livestock diseases following floods

... wounds or being ingested. This disease may not occur immediately and it may be some time before symptoms of the disease show. Erosion and movement from floods provide favourable survival conditions for blackleg spores. Common signs include fever, severe depression, gassy swelling under the skin or i ...
Course programme “Infectious disease epidemiology“
Course programme “Infectious disease epidemiology“

... 12 Jan 2016 ...
No Slide Title
No Slide Title

... hay may very well allow escape from heaves. Overpopulations of horses that turn pastures to dust are to be avoided of course. Reduce dust. The proper care of buildings and reasonable sanitation end the story. A sprinkler truck for the race track is a nice touch. ...
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African trypanosomiasis



African trypanosomiasis or sleeping sickness is a parasitic disease of humans and other animals. It is caused by protozoa of the species Trypanosoma brucei. There are two types that infect humans, Trypanosoma brucei gambiense (T.b.g) and Trypanosoma brucei rhodesiense (T.b.r.). T.b.g causes over 98% of reported cases. Both are usually transmitted by the bite of an infected tsetse fly and are most common in rural areas.Initially, in the first stage of the disease, there are fevers, headaches, itchiness, and joint pains. This begins one to three weeks after the bite. Weeks to months later the second stage begins with confusion, poor coordination, numbness and trouble sleeping. Diagnosis is via finding the parasite in a blood smear or in the fluid of a lymph node. A lumbar puncture is often needed to tell the difference between first and second stage disease.Prevention of severe disease involves screening the population at risk with blood tests for T.b.g. Treatment is easier when the disease is detected early and before neurological symptoms occur. Treatment of the first stage is with the medications pentamidine or suramin. Treatment of the second stage involves: eflornithine or a combination of nifurtimox and eflornithine for T.b.g. While melarsoprol works for both it is typically only used for T.b.r. due to serious side effects.The disease occurs regularly in some regions of sub-Saharan Africa with the population at risk being about 70 million in 36 countries. As of 2010 it caused around 9,000 deaths per year, down from 34,000 in 1990. An estimated 30,000 people are currently infected with 7000 new infections in 2012. More than 80% of these cases are in the Democratic Republic of the Congo. Three major outbreaks have occurred in recent history: one from 1896 to 1906 primarily in Uganda and the Congo Basin and two in 1920 and 1970 in several African countries. Other animals, such as cows, may carry the disease and become infected.
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