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Azacitidine - Aplastic Anemia and MDS International Foundation
Azacitidine - Aplastic Anemia and MDS International Foundation

... 2011 – Oral AZA given 7 days out of 28 is safe and appears effective 2012 – Treating for 14 or 21 days enhances biologic activity and is effective – 34% ORR and 40% transfusion independent 2013 – Phase III Clinical Trial of Lower Risk Transfusion Dependence - should lead to FDA approval PROS Oral dr ...
Invest in ME – London, June 1st 2012
Invest in ME – London, June 1st 2012

... In Norway, Mella and Fluge are investigating anti-CD20 intervention using the drug Rituximab. Results suggest that ME/CFS may be an autoimmune condition. NK cytotoxic activity, NK phenotypes and NK proteins remain as the most consistent immunological markers. Emerging areas include: CD8 lysis, micro ...
PPT - Med Study Group
PPT - Med Study Group

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INFLADASE FORTE

... Serratiopeptidase binds to α2-macroglobulin in the blood in a 1:1 ratio. This helps to mask its antigenicity but retains its enzymatic activity. Levels of serratiopeptidase are slowly transferred to the exudates at the site of infection and inflammation and gradually blood levels decline. The mechan ...
by Laura Crawford St. Luke`s to install MRI scanner, build a building
by Laura Crawford St. Luke`s to install MRI scanner, build a building

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PowerPoint 演示文稿
PowerPoint 演示文稿

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POSTERIOR URETHRA VALVES AS A REASONS OF THE
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Triple-Negative First-Line Study: Neoadjuvant Trial of nab
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European Respiratory Society Annual Congress 2013
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clinical features of adult irgn
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Dermatophilosis - Rain Rot - Brandon Equine Medical Center

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SLE (systemic lupus erythematosus)
SLE (systemic lupus erythematosus)

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Pathophysiology of multiple sclerosis

Multiple sclerosis (MS) is a condition where the CNS of a person present a special kind of distributed glial scars (sclerosis) which are a remaining of a previous inflammatory demyelination. MS pathophysiology is complex and still under investigation and there is no agreement about its scope. Some authors consider it a pathological entity, while others consider it a clinical entity. From a pathological point of view, the demyelinating lesions can be classified as encephalomyelitis, and sometimes the disease is known as encephalomyelitis disseminata.There are two phases for how an unknown underlying condition may cause damage in MS: First some MRI-abnormal areas with hidden damage appear in the brain and spine (NAWM, NAGM, DAWM). Second, there are leaks in the blood–brain barrier where immune cells infiltrate causing the known demyelination and axon destruction. Some clusters of activated microglia, transection of axons and myelin degeneration is present before the BBB breaks down and the immune attack beginsPathophysiology is a convergence of pathology with physiology. Pathology is the medical discipline that describes conditions typically observed during a disease state; whereas physiology is the biological discipline that describes processes or mechanisms operating within an organism. Referring to MS, the physiology refers to the different processes that lead to the development of the lesions and the pathology refers to the condition associated with the lesions.
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