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Presentation Outline: TBI Nutritional Treatment Strategies for the
Presentation Outline: TBI Nutritional Treatment Strategies for the

... neuroinflammation, cytotoxic and chemotoxic reactions along with the destruction of neural tissues and local supporting structures. It is important to quench the inflammatory process and modulate the immune reactions as quickly and efficiently as possible following a TBI. The immune response of TH1, ...
Multiple Sclerosis
Multiple Sclerosis

... Sclerosis: Etiology, Diagnosis, and New Treatment Strategies. Totowa (NJ): Humana Press Inc.; p. 103112. ...
MS Treatment - Keely Perry
MS Treatment - Keely Perry

... • Drugs that help with symptoms – Wide variety from anti-depressants (depression) to laxatives (bowel dysfunction) to anti-convulsants (pain/altered sensations) ...
Promising Future Treatments for Multiple Sclerosis
Promising Future Treatments for Multiple Sclerosis

... years, and post-transplant gadolinium-enhanced lesions on MRI occurred in only 8% of cases. However, this study showed relatively significant toxicity, with 15% of patients having serious infections, allergic events, or severe related bone pain. ...
Multiple Sclerosis - faculty at Chemeketa
Multiple Sclerosis - faculty at Chemeketa

... its T Cells. The myelin is unrecognizable and begins to be destroyed. • Studies: myelin basic protein (component of myelin) when injected into lab animals, can develop experimental allergic encephalomyelitis (EAE), a brain and spinal cord disease similar to MS. • Studies: BBB, possible immune system ...
DEMYELINATING DISEASE MULTIPLE SCLEROSIS
DEMYELINATING DISEASE MULTIPLE SCLEROSIS

... complications of chronic disease ...
Southern Methodist University
Southern Methodist University

... inflammation and, through their release of cytokines, to stimulate B cells to secrete antibodies that cause demyelination. Cytokines have proved to be toxic to neurons and oligodendrocytes if secreted in high concentrations over sustained period of time. ...
here - Boston University Medical Campus
here - Boston University Medical Campus

... •Inflammatory cytokines (eg, IL-2, IL-15, interferon-gamma) and cytokinesecreting cells are seen in the serum at low, but higher than normal, levels IL-1, tumor necrosis factor-alpha, IL-6, and IL-15 are present in the CSF. Messenger ribonucleic acid for inflammatory cytokines is elevated in white b ...
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Pathophysiology of multiple sclerosis

Multiple sclerosis (MS) is a condition where the CNS of a person present a special kind of distributed glial scars (sclerosis) which are a remaining of a previous inflammatory demyelination. MS pathophysiology is complex and still under investigation and there is no agreement about its scope. Some authors consider it a pathological entity, while others consider it a clinical entity. From a pathological point of view, the demyelinating lesions can be classified as encephalomyelitis, and sometimes the disease is known as encephalomyelitis disseminata.There are two phases for how an unknown underlying condition may cause damage in MS: First some MRI-abnormal areas with hidden damage appear in the brain and spine (NAWM, NAGM, DAWM). Second, there are leaks in the blood–brain barrier where immune cells infiltrate causing the known demyelination and axon destruction. Some clusters of activated microglia, transection of axons and myelin degeneration is present before the BBB breaks down and the immune attack beginsPathophysiology is a convergence of pathology with physiology. Pathology is the medical discipline that describes conditions typically observed during a disease state; whereas physiology is the biological discipline that describes processes or mechanisms operating within an organism. Referring to MS, the physiology refers to the different processes that lead to the development of the lesions and the pathology refers to the condition associated with the lesions.
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