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Incremental Prognostic Value of Estimated LV€End
Incremental Prognostic Value of Estimated LV€End

... (Table 1). Using these measures, LVEDVI Estimated was calculated and compared with “true” CTCA-measured LVEDV. The correlation between measured LVEDVI and LVEDVIEstimated was very good (r ¼ 0.910, 95% confidence interval [95% CI]: 0.882 to 0.931; p < 0.0001). LVEDVI Estimated was significantly larger ...
Successful closure of ventricular septal defect in a patient with
Successful closure of ventricular septal defect in a patient with

... the complaint of heart failure. Noncompaction primarily affects the left ventricle, but the right ventricle may also be involved.[4,8,9] Asymptomatic cases have normal systolic function and increased left ventricular enddiastolic pressure, mimicking restrictive cardiomyopathy. Most patients are init ...
Arrhythmogenic Right Ventricular Dysplasia
Arrhythmogenic Right Ventricular Dysplasia

... Arrhythmogenic right ventricular dysplasia (ARVD) is a rare type of cardiomyopathy. It occurs if the muscle tissue in the right ventricle dies and is replaced with scar tissue. This disrupts the heart's electrical signals and causes arrhythmias. Symptoms include palpitations and fainting after physi ...
Syndrome of Left Ventricular-Right Atrial
Syndrome of Left Ventricular-Right Atrial

... mechanism reverted to a sinus one within 18 hours. The patient was discharged to return at a later date for surgical repair of his defect. He was admitted for the third time on Jan. 5, 1954. He had been well between admissions and physical examination was essentially unchanged. The heart was greatly ...
Cardiology QOD Review
Cardiology QOD Review

... ring of tissue, or collarlike with length forming a segment of aortic hypoplasia. Lessthan-normal blood flow through the aortic arch during fetal life may result in hypoplasia of the arch and promote the likelihood of coarctation developing, which forms the basis for the association between aortic s ...
Double Aortic Arch
Double Aortic Arch

... Transparency applied to the left atrium and left ventricle, revealing the reconnecting aortic branches ...
INTERACTIVE CASE 4 CARDIAC B
INTERACTIVE CASE 4 CARDIAC B

... (beginning on the right,then left).  Actuarial survival for untreated tetralogy of Fallot is approximately 75% after the first year of life, 60% by four years, 30% by ten years, and 5% by forty years. ...
Dr.Yoused Aljeesh Dr. Motasem Salah The Heartbeat
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Internal Medicine Board Review (from MKSAP 13)
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... – most have had complete intracardiac repair, occasionally only aortopulmonary shunt. – PI leading to right heart dilation is common – Yearly mortality increases after 25 years due to sudden death, QRS >180ms best predictor, apparent interaction between long QRS and right heart dilation, general agr ...
Interventional Heart Failure - Cardiac Interventions Today
Interventional Heart Failure - Cardiac Interventions Today

... them in various clinical scenarios, including high-risk coronary intervention, AMI complicated by cardiogenic shock, acutely decompensated heart failure, and right heart failure. One of the major issues in the field of temporary circulatory support is the apparent disconnect observed between hemodyn ...
Bolest na hrudi nebo v zádech
Bolest na hrudi nebo v zádech

... mm (norma up to 37 mm), aortic arch cannot be evaluated. Left ventricular wall thickness 10 mm (norma up to 12mm). Normal wall motion of the left ventricle, ejection fraction 65% (norma over 55%). Small circular pericardial effusion. Aortic valve sclerosis with mild insufficiency. ECG recorded 30 mi ...
PATHOPHYSIOLOGY OF HEART FAILURE
PATHOPHYSIOLOGY OF HEART FAILURE

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Congenital third-degree AV block in the infant with a ventricular rate
Congenital third-degree AV block in the infant with a ventricular rate

... in lead and device technology allow pacemaker system implantation in children and even in neonates ...
What is the mitral valve? What causes mitral valve disease? What
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... heart requires considerable knowledge and experience and should always be performed by experts. Your vet may also want to do some blood tests (particularly to check that their kidneys are working properly) before starting your pet on treatment. ...
Arrhythmogenic Right Ventricular Dysplasia / Cardiomyopathy : A
Arrhythmogenic Right Ventricular Dysplasia / Cardiomyopathy : A

... cause of sudden death in individuals under 30 years of age, and has been found in up to 20% of cases of sudden death in young people 2,5,7,8. There is a familial predilection of the disease, which is typically inherited as an autosomal dominant trait with variable penetrance and incomplete expressio ...
Ch 11 The Heart
Ch 11 The Heart

... in line with middle of clavicle ...
Cardiac Resynchronization Therapy in Asymptomatic and Mildly
Cardiac Resynchronization Therapy in Asymptomatic and Mildly

... results of this late analysis, CRT therapy’s beneficial effect on morbidity and mortality rates did not differ at all among the aforementioned groups. This led the authors to question the validity of symptom severity (as expressed by NYHA class) as a predictor in recognizing CRT candidates.13 Other ...
Cardiac Output
Cardiac Output

... •Cardiac output is the product of heart rate and stroke volume (0 = HR X SV) The pacemaker of the heart is the SA node. SA node activity is modified by the parasympathetic nervous system (slows HR) and the sympathetic nervous system (increases HR). Heart rate increases at the beginning of exercise d ...
The Valvular Involvement of Lupus: Congestive Heart Failure Can
The Valvular Involvement of Lupus: Congestive Heart Failure Can

... (30 mg daily) for the vegetations, which were suspected caused by Libman-Sacks endocarditis, and ACE inhibitor and diuretic for symptoms of acute heart failure. No antibiotics were prescribed, since there was no toxic sign or evidence of infection clinically. Cardiac catherization on 17th February 2 ...
Role of nuclear imaging in cardiac amyloidosis
Role of nuclear imaging in cardiac amyloidosis

... Background: Amyloidosis refers to large group of disorders caused by extracellular deposition of insoluble abnormal fibrils of misfolded proteins, which can alter tissue structure and impair function of multiple organs, including the heart. Cardiac amyloidosis is often misdiagnosed. Histological ana ...
Mannequin - CHFpatients.com
Mannequin - CHFpatients.com

... and final ventricular volume. Three outcomes are possible: the resulting ventricle is too small, too large or the correct size. Making the ventricle too small is the worst scenario, because it will lead to immediate pulmonary hypertension (Dor, Seminars in Thoracic and Cardiovascular Surgery, Vol. 1 ...
Fish oil supplementation and the risk of ventricular tachycardia and
Fish oil supplementation and the risk of ventricular tachycardia and

... omega-3 PUFAs in patients with a history of ventricular tachycardia (VT) or ventricular fibrillation (VF) and an implantable cardioverter defibrillator. Three trials involving 1148 patients were included in this review. The population was homogenous. Results of the three studies showed no significan ...
Prevention of sudden cardiac death in
Prevention of sudden cardiac death in

... Hypertrophic cardiomyopathy (HCM) is a genetic disorder of cardiac muscle with a prevalence of 1 in 500 of the general population.w1 In most adults, HCM is inherited as an autosomal dominant trait caused by mutations in genes encoding cardiac sarcomere proteins.1 The disease is clinically defined by ...
Susan F. Saleeb, Wing Yi V. Li, Shira Z. Warren... ; originally published online October 10, 2011;
Susan F. Saleeb, Wing Yi V. Li, Shira Z. Warren... ; originally published online October 10, 2011;

... absenteeism, as well as patient, parent, and physician anxiety.1–4 As opposed to adults, for whom CP often signals a cardiac problem, multiple studies have demonstrated that the most common causes of CP among children are benign idiopathic, musculoskeletal, gastrointestinal, pulmonary, or psychogeni ...
Left ventricular endocardial longitudinal and transverse - AJP
Left ventricular endocardial longitudinal and transverse - AJP

... valve is open during the first three-quarters of IVC. It closed at the beginning of the last quarter, confirming data presented previously by our group (14, 15) (Fig. 2B). Given that motion must be preceded by an acceleration phase and that acceleration in fluids is driven by pressure gradients, it ...
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Hypertrophic cardiomyopathy



Hypertrophic cardiomyopathy (HCM) is a primary disease of the myocardium (the muscle of the heart) in which a portion of the myocardium is hypertrophied (thickened) without any obvious cause, creating functional impairment of the cardiac muscle. It is a leading cause of sudden cardiac death in young athletes.The occurrence of hypertrophic cardiomyopathy is a significant cause of sudden unexpected cardiac death in any age group and as a cause of disabling cardiac symptoms. Younger people are likely to have a more severe form of hypertrophic cardiomyopathy.HCM is frequently asymptomatic until sudden cardiac death, and for this reason some suggest routinely screening certain populations for this disease.A cardiomyopathy is a disease that affects the muscle of the heart. With HCM, the myocytes (cardiac contractile cells) in the heart increase in size, which results in the thickening of the heart muscle. In addition, the normal alignment of muscle cells is disrupted, a phenomenon known as myocardial disarray. HCM also causes disruptions of the electrical functions of the heart. HCM is most commonly due to a mutation in one of nine sarcomeric genes that results in a mutated protein in the sarcomere, the primary component of the myocyte (the muscle cell of the heart). These are predominantly single-point missense mutations in the genes for beta-myosin heavy chain (MHC), myosin-binding protein C, cardiac troponinT, or tropomyosin. These mutations cause myofibril and myocyte structural abnormalities and possible deficiencies in force generation. Not to be confused with dilated cardiomyopathy or any other cardiomyopathy.While most literature so far focuses on European, American, and Japanese populations, HCM appears in all ethnic groups. The prevalence of HCM is about 0.2% to 0.5% of the general population.
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