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Termination and Inhibition of Recurrent Tachyeardias
Termination and Inhibition of Recurrent Tachyeardias

... tachycardias were relatively slow, with rates less than 160 beats per minute. At rates below this level, single paced beats are usually able to enter re-entrant conduction pathways thus terminating the tachycardia.10 At faster rates, a refractory period entrance block may develop around the re-entra ...
ECG Notes
ECG Notes

... caused by disease below the AV node, ie. the bundle of His may be associated with anterior AMI ventricular rates can be quite slow, with dyspnoea, syncope & fatigue frequently progresses to a higher level of block & requires pacing pacing does not alter 60-70% mortality rate ∝ native disease ...
Role of Structural Barriers in the Mechanism of Alternans
Role of Structural Barriers in the Mechanism of Alternans

... produced by a 10x2 mm epicardial laser-lesion. In controls, APD alternated in phase at all ventricular sites above a critical heart rate, i.e. concordant alternans. Also, above a faster critical heart rate threshold, APD alternated with opposite phase between sites, i.e. discordant alternans. In con ...
NIH Public Access - IUPUI ScholarWorks
NIH Public Access - IUPUI ScholarWorks

... We successfully developed a model of ES in these normal ventricles. We defined ES as multiple (≥3) consecutive episodes of SVF recurrences after initial successful defibrillation. In control, no SVF was observed with 5 attempts of the induced VF-defibrillation episodes. During isoproterenol infusion ...
Cardiovascular response to physical exercise in adult
Cardiovascular response to physical exercise in adult

... been reported. Systemic right ventricular failure, atrial arrhythmias, and baffle obstruction are risk factors for late death of patients after atrial correction of TGA.10–13 Furthermore, right ventricular dysfunction contributes to the occurrence of atrial arrhythmias in these patients,10 12 stress ...
Atrial Fibrillation : Present Treatment Protocols by Drugs and Interventions Abstract
Atrial Fibrillation : Present Treatment Protocols by Drugs and Interventions Abstract

... An issue for concern about calcium channel blockers and beta blockers when used for initial ventricular rate control is their cardio-depressive effects, particularly in patients with heart failure. However, as a common practice – though not appropriately supported by data, one should feel comfortabl ...
Malignant Mitral Valve Prolapse - ORBi
Malignant Mitral Valve Prolapse - ORBi

... identify features related with stretch-induced scar/fibrosis and arrhythmic MVP, Perazzolo Marra et al,11 in this issue of Circulation: Cardiovascular Imaging, performed a morphological assessment of the mitral valve with a particular focus on mitral annular disjunction (MAD). They studied 2 distinc ...
Editd anti arrhythmic - Presentation Transcript
Editd anti arrhythmic - Presentation Transcript

... Approved only in serious ventricular arrhythmias Broad spectrum of action on the Very effective Na+ channel blocker but low affinity for activated channels Markedly lengthens AP by blocking also K+ channels Weak Ca++ channel blocker Noncompetetive inhibitor of beta adrenoceptors Powerful inhibitor o ...
Association between left ventricular hypertrophy
Association between left ventricular hypertrophy

... LVM in hypertensive patients.(17,46) These researchers ...
Pacing Lead Perforation on the Right Side of the Heart
Pacing Lead Perforation on the Right Side of the Heart

... walls (rather than septal fixation) may endanger the LAD with the helix of an active-fixation lead and cause acute myocardial infarction. The paced ECG may show a right bundle branch pattern if the lead paces the left ventricle usually from the pericardial space (Figure 1). The chest X ray may show ...
Left ventricular long-axis diastolic function is
Left ventricular long-axis diastolic function is

... In order to determine left ventricular global and regional myocardial functional reserve in endurance-trained and strength-trained athletes, and to identify predictors of exercise capacity, we studied 18 endurance-trained and 11 strength-trained athletes with left ventricular hypertrophy (172p27 and ...
Ventricular Structure and Function in Hypertensive Participants With
Ventricular Structure and Function in Hypertensive Participants With

... dyspnea) and physical signs (edema, pulmonary rales, gallop rhythm, displaced LV apical impulse) or by supporting clinical findings, such as those from chest radiography. Diagnosis of heart failure was confirmed also if, in addition to having a previous physician diagnosis, the participant was recei ...
Hypertensive heart disease. A complex syndrome or a hypertensive
Hypertensive heart disease. A complex syndrome or a hypertensive

... increase in fibroblastic activity that occurs in hypertensive heart disease may be an important factor in pathological rather than physiological left ventricular hypertrophy. Cardiac myocytes of the left ventricle are enlarged in hypertensive heart disease[46] and fibrosis is another feature of the ...
AHEART July 46/1 - AJP
AHEART July 46/1 - AJP

... delays the onset and slows down the rate of relaxation, whereas an increase in afterload late in systole abbreviates contraction time and increases the rate of relaxation (5). In isolated heart studies, interventions designed to increase afterload immediately after aortic valve opening increased the ...
Surgery for Atrial Fibrillation - ANNALS Academy of Medicine
Surgery for Atrial Fibrillation - ANNALS Academy of Medicine

... primary local generator, such as a single small re-entry circuit or ectopic focus.30,31 In human studies, up to 94% of patients with paroxsymal atrial fibrillation have been shown to have their arrhythmia originate from ectopic foci in the pulmonary veins.32 Indeed, catheter ablation of pulmonary ve ...
Arrhythmias
Arrhythmias

... Cardiac Electrophysiology ...
fibrillation
fibrillation

... concentrations. None was taking medication other than digoxin. For comparison we studied eight men (mean age 53 3 (39-62) years; weight 77 1 (65-90) kg) with ischaemic heart disease in sinus rhythm. The diagnosis of ischaemic heart disease was based upon previous myocardial infarction. None had clin ...
Impact of Longitudinal Myocardial Deformation on the Prognosis of
Impact of Longitudinal Myocardial Deformation on the Prognosis of

... Background—Longitudinal myocardial deformation indexes appear superior to left ventricular ejection fraction (LVEF) in assessing myocardial contractility. However, few studies have addressed the prognostic value of longitudinal motion markers (velocity, strain, and strain rate) in predicting outcome ...
acoustic cardiography
acoustic cardiography

... Therefore, both techniques facilitate the identification of heart sounds, permit accurate measurements of timing relationships (with respect to the ECG and to other heart sounds) and display the duration, relative intensity and contour of murmurs. Both also permit the non-invasive evaluation of left ...
Heart Failure With Normal Left Ventricular Ejection Fraction
Heart Failure With Normal Left Ventricular Ejection Fraction

... applied (12). Compared with patients with HF and reduced LVEF, individuals with HFNEF are typically older and more likely to be women, together with a higher likelihood of hypertension (prevalence up to 88% [13]), obesity (prevalence of body mass index ⬎30 kg/m2 typically approximately 40% [11,13]), ...
PDF
PDF

... Peripartum cardiomyopathy (PPCM) is a disorder in which initial left ventricular systolic dysfunction and symptoms of heart failure occur between the late stages of pregnancy and the early postpartum period. It is common in some countries and rare in others. The causes and pathogenesis are poorly un ...
Arrhythmic Mitral Valve Prolapse and Sudden Cardiac Death
Arrhythmic Mitral Valve Prolapse and Sudden Cardiac Death

... females, median age 32 years, range 19-40) with MVP due to myxomatous valve disease were identified. They represent 7% of all SCD cases and 13% of women who died suddenly, being the first structural cause in the latter group. Main clinical and pathologic data are reported in Table 1. SCD occurred mo ...
Myocardial infarction
Myocardial infarction

... skeletal muscle, but the MB fraction is much more specific for cardiac muscle: about 15 to 40% of CK in cardiac muscle is MB, while less than 2% in skeletal muscle is MB. The BB fraction (found in brain, bowel, and bladder) is not routinely measured. Thus, CK-MB is a very good marker for acute myoca ...
Light-induced termination of spiral wave arrhythmias by optogenetic
Light-induced termination of spiral wave arrhythmias by optogenetic

... painful and shockless method of cardioversion is warranted. Electrical cardioversion relies on synchronous depolarization of large areas of the atrial myocardium, in order to terminate the reentrant conduction underlying fibrillation.5 In theory, shockfree cardioversion might be elicited by genetica ...
Taquicardia de QRS largo em paciente com extrema
Taquicardia de QRS largo em paciente com extrema

... should always treat the patient for VT. While this teaching is generally true, the use of traditional “VT medications,” such as lidocaine, procainamide or amiodarone, for any of the mimics noted above can actually be deadly. Recall that lidocaine and procainamide are Class I antiarrhythmics — sodium ...
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Ventricular fibrillation



Ventricular fibrillation (V-fib or VF) is a condition in which there is uncoordinated contraction of the cardiac muscle of the ventricles in the heart, making them quiver rather than contract properly. Ventricular fibrillation is the most commonly identified arrhythmia in cardiac arrest patients. While there is some activity, the lay person is usually unable to detect it by palpating (feeling) the major pulse points of the carotid and femoral arteries. Such an arrhythmia is only confirmed by electrocardiography. Ventricular fibrillation is a medical emergency that requires prompt Advanced Life Support interventions. If this arrhythmia continues for more than a few seconds, it will likely degenerate further into asystole (""flatline""). This condition results in cardiogenic shock and cessation of effective blood circulation. As a consequence, sudden cardiac death (SCD) will result in a matter of minutes. If the patient is not revived after a sufficient period (within roughly 5 minutes at room temperature), the patient could sustain irreversible brain damage and possibly become brain-dead, due to the effects of cerebral hypoxia. On the other hand, death often occurs if sinus rhythm is not restored within 90 seconds of the onset of VF, especially if it has degenerated further into asystole.
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