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Reinforcement & Drug Effects
Reinforcement & Drug Effects

...  DA agonist  euphoria  cocaine, amphetamine  Heroin  opioids   DA in NA  via Ventral Tegmental Area (VTA)  most addictive drugs  DA antagonists  Blocks learning  Also blocks responding for food ~ ...
TOXIDROMES
TOXIDROMES

...  Tachycardia and hypertension ...
Nonsteroidal Anti-Inflammatory Drugs (NSAIDs)
Nonsteroidal Anti-Inflammatory Drugs (NSAIDs)

...  The occasional use of an abusable drug is distinct from repeated use and the emergence of chronic drug addiction.  The cellular and molecular mechanisms that mediate the transition from occasional controlled drug use to addiction are only just beginning to be understood. ...
biochem mcq - Pass the FracP
biochem mcq - Pass the FracP

... The oxidative-modification hypothesis of atherosclerosis proposes that atherogenesis is initiated by oxidation of the lipids in low-density lipoprotein (LDL), supported by the epidemiological evidence of an inverse relation between increased intake of antioxidant vitamins such as vitamin E arid vita ...
Effects of Three Reversal Agents on Detomidine- Induced
Effects of Three Reversal Agents on Detomidine- Induced

... Detomidinea gel is well absorbed and elicits many of the same cardiac and behavioral changes observed with the parental formulations. Antagonism of central nervous system and cardiovascular effects by ␣2 antagonists after parenteral administration of detomidine have been well characterized. The goal ...
Pharmacokinetics
Pharmacokinetics

... Effectively, the drug has been metabolized before it ever reaches the systemic circulation and/or its target organ ...
SPANBEC (GLIMEPIRIDE 1/2/3 gm) GENERIC NAME : Glimepride
SPANBEC (GLIMEPIRIDE 1/2/3 gm) GENERIC NAME : Glimepride

...  It stimulates the pancreas beta cells to make more insulin and the insulin is secreted into the blood. Insulin causes sugar to leave the blood and enter cells throughout the body. It has also been shown to have pleiotropic effects. It makes the peripheral tissues more sensitive to insulin. Thus, i ...
Pharmacology MCQs
Pharmacology MCQs

... a. blocks actions of both endogenous & exogenous sympathomimetic amines. b. alkylates the receptor by forming a covalent bond. c. blocks both the alpha and beta receptors. d. acts only on receptors in the CNS. 40. Non-catecholamine sympathomimetics are: a. ephedrine. b. dopamine. c. phenylephrine. ...
IOW Dementia MUR Steve Bleakley Nov 2010
IOW Dementia MUR Steve Bleakley Nov 2010

... 6-7% of hospital admissions related to adverse drug reactions Drug interactions account for 1-2% 0.13% are fatal (US data) Around 70% considered avoidable Estimated NHS costs £500million ...
Understanding Drugs and Medicines
Understanding Drugs and Medicines

... • Information processing in the brain takes place at the connections between neurons, called synapses. ...
5HT1F- and 5HT7-Receptor Agonists for the Treatment of Migraines
5HT1F- and 5HT7-Receptor Agonists for the Treatment of Migraines

... reserpine -induced attacks in reports by Kimball [3] and Anthony [4]. The subsequent discovery of sumatriptan almost 20 year ago [5] fuelled the research in migraines but also in the field of serotonin and its receptors. Although 5-HT exerts its effects via 14 receptor subtypes at least [6], there i ...
Autonomic Nervous System
Autonomic Nervous System

... CNS stimulation followed by depression Decreased cardiovascular response to parasympathetic (vagal) stimulation that slows heart rate. Bronchodilation and decrease respiratory secretions Antispasmodic effects in GI system Change in intra-ocular pressure in patients with glaucoma ...
felix may 2nd year neuroscience Neuroreceptor characterisation by
felix may 2nd year neuroscience Neuroreceptor characterisation by

... Binding of QNB can be seen to reach a baseline value where no additional atropine can displace it (fig.1). This is considered to be ‘undisplaceable’ radioligand that is bound to sites other than mAch receptors, or even just absorbed in the filter paper. Subtracting the lowest value for QNB binding ...
Non-selective CNS depressants ©2010 Mark Tuttle Non
Non-selective CNS depressants ©2010 Mark Tuttle Non

... Abuse: Used in ways that are not medically approved Tolerance: Escalation of dose to maintain effect Dependence: Drug now required for homeostasis - Barbiturates - Metabolic: induction of hepatic enzymes - Withdrawal syndrome - Ethanol o Hypnotic dose does this o Hyperexcitability of entire CNS - Ot ...
Advancements in the Use of iPS Cell
Advancements in the Use of iPS Cell

29.2 Neurons
29.2 Neurons

... – dendrites receive impulses – axon carries impulses ...
Detection of multiple nuclear receptor–coregulator interactions in a single
Detection of multiple nuclear receptor–coregulator interactions in a single

... different NRs on 155 known NR coregulator proteins harboring either LXXLL (in coactivators) or LXXXIXXXL (in corepressors) motifs. Each of the NRs shows a different and specific modulation by its natural/synthetic ligand (figure 2). ...
Snímka 1 - TOP Recommended Websites
Snímka 1 - TOP Recommended Websites

... • that produce wakefullness and arousal and stimulate behavior, • their current clinical use is limited to treatment of specific sleep disorders such as narcolepsy, and certain childhood behavioral problems such as attention deficit disorder, • more importantly, the psychomotor stimulants are a clas ...
How Do Drugs Work?
How Do Drugs Work?

... aspirin, gently block less-critical proteins for a few hours. With the use of these drugs, we can make changes inside our own cells, such as the blocking of pain signals. Many structures of drugs that bind to proteins have been determined by scientists. These atomic structures allow us to see how dr ...
Why is it important to consider the route of
Why is it important to consider the route of

... Cell Membranes: This barrier is permeable to many drug molecules but not to others, depending on their lipid solubility. Small pores, 8 angstroms, permit small molecules such as alcohol and water to pass through. Walls of Capillaries: Pores between the cells are larger than most drug molecules, allo ...
Screening for Hormone-Like Therapeutic Compounds.pdf
Screening for Hormone-Like Therapeutic Compounds.pdf

... linked to a broad spectrum of diseases, including breast, endometrial and prostate cancer, leukemia, cardiovascular diseases, osteoporosis and inflammations. Therefore the discovery of novel compounds with the ability to modulate these targets could lead to the development of valuable therapeutics a ...
Slide 1
Slide 1

... impairment or distress as manifested by one (or more) of the following, within a 12-month period:  Recurrent substance use resulting in failure to fulfill major role ...
Module 2: Drug and Alcohol Awareness
Module 2: Drug and Alcohol Awareness

... down” period causes fatigue, and depressed mood. Heart attacks, hgh blood pressure, stroke, and kidney damage ...
Antidepressant Drugs
Antidepressant Drugs

... overdose Don’t carry abuse potential, Development of dependence is less ...
PARKINSON DISEASE UPDATE
PARKINSON DISEASE UPDATE

... Fahn, S., Przedborski, S. Parkinsonism in Merritt’s Neurology. Eleventh Edition. Chapter 115. 2005. Blumenfeld, H. Basal Ganglia in Neuroanatomy Through Clinical Cases. Chapter 16. 2002. Hauser, R., Pahwah, R. Current Treatment Challenges and Emerging Therapies in Parkinsons Disease Suppl. To Neurol ...
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Neuropharmacology

Neuropharmacology is the study of how drugs affect cellular function in the nervous system, and the neural mechanisms through which they influence behavior. There are two main branches of neuropharmacology: behavioral and molecular. Behavioral neuropharmacology focuses on the study of how drugs affect human behavior (neuropsychopharmacology), including the study of how drug dependence and addiction affect the human brain. Molecular neuropharmacology involves the study of neurons and their neurochemical interactions, with the overall goal of developing drugs that have beneficial effects on neurological function. Both of these fields are closely connected, since both are concerned with the interactions of neurotransmitters, neuropeptides, neurohormones, neuromodulators, enzymes, second messengers, co-transporters, ion channels, and receptor proteins in the central and peripheral nervous systems. Studying these interactions, researchers are developing drugs to treat many different neurological disorders, including pain, neurodegenerative diseases such as Parkinson's disease and Alzheimer's disease, psychological disorders, addiction, and many others.
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