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Nucleosome Assembly Protein 1 - MSOE Center for BioMolecular
Nucleosome Assembly Protein 1 - MSOE Center for BioMolecular

... aminoacyl tRNA synthetase (AARS) family, a set of structurally heterogeneous enzymes, specific for each amino acid. AARS are potential drug targets as they are essential for survival and are structurally different between species. AARS also functions as an immunosuppressant, blocking interleukin 8 r ...
Immune Response 101
Immune Response 101

... a. Intracellular microbe invades. b. Activation of innate immunity may include: interferon response, inflammation, phagocytes (to eat up dead cells) c. Dendritic cell takes up microbe through phagocytosis, chews it up in the phagolysosome, and displays some of the microbial protein antigens on MHC c ...
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Nonspecific and Specific Immunity
Nonspecific and Specific Immunity

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... Protein must be cooked or breaks down in the stomach from stomach acid ...
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...  Discuss how viruses cause disease ...
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Your Pre-Build Model should have been impounded the morning of

... How does this technique enable the Next-Generation sequencing technology used to sequence Nic Volker’s DNA? (2 pts) The hairy beads generated by the emulsion PCR are passed over a slide containing a regular array of picoliter-sized wells, each one large enough to accommodate one (but not two) hairy ...
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Microbes and diseases: what to study-1

... • Spore-like: dormant and resistant • Infectious: form that moves between cells – Reticulate body: 0.6-1.5 µm, metabolically active, reproduce inside host cells ...
Lecture 15 – Effector Functions of Humoral Immunity:
Lecture 15 – Effector Functions of Humoral Immunity:

T Cell Immunology for the Clinician
T Cell Immunology for the Clinician

... tremendous TCR-binding site diversity results from this process—the large number of possible recombinations of V, D, and J segments provide combinatorial diversity, and additional diversity is conferred by imprecision in the DNA-joining reaction.2,3,7 The maturing T cells can be divided into 2 group ...
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CTL - Molecular Immunology

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... The term ‘secretor’ or ‘non-secretor’ refers to the ability of an individual to secrete ABO blood group antigens in bodily fluids such as saliva, sweat, tears, serum and the gastrointestinal mucus secretions. The function of ABO antigens remain an enigma, however it has recently been suggested that ...
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IMMUNOLOGY

... General feature of immunodeficiency diseases The principal consequence of immunodeficiency is an increased susceptibility to infection. Patients with immunodeficiencies are also susceptible to certain types of cancer. Paradoxically, certain immunodeficiencies are associated with an increased incide ...
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Immunology MCQ exam 1. All of the following are true with respect

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factor v leiden mutation

... many receptors on NK cells. Class II MHC molecules: HLA DR, DP, DQ Present on the surface of antigen-presenting cells as: Dendritic cells, macrophages- monocytes, B-cells, and tissue specific cells. Protect against extracellular pathogens. Major ligand for: TCR on CD-4+ T- helper cells. ...
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12Immuniz

... Conditional lethal mutants. Temperature-sensitive mutants in influenza A and RSV have been made by mutation with 5-fluorouracil and then selected for temperature sensitivity. In the case of influenza, the temperature-sensitive gene can be reassorted in the laboratory to yield a virus strain with the ...
Acquired Haemolytic Anaemias
Acquired Haemolytic Anaemias

...  DAT +ve and haemolysis may ensue  Occurs after 7-10 days of treatment  Ceases few days to 2 weeks after drug stopped Neoantigen type  Formerly known as immune complex / innocent bystander  Old theory suggested drug formed immune complex with anti-drug antibody  attached non-specifically to re ...
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Article UCSD Etomoxir in MS - Meta-IQ
Article UCSD Etomoxir in MS - Meta-IQ

... use substrates such as glucose (glycolysis) or fatty acids (beta-oxidation) to produce ATP6. Beta-oxidation of fatty acids occurs in the mitochondria which necessitates the importation of fatty acyl groups from the cytosol through the outer and inner mitochondrial membranes7. However, the mitochondr ...
CellsNoTP
CellsNoTP

... What happens if they aren’t replaced correctly? If they are deficient in number? If they are produced in excess to what is needed? If they are not structurally or functionally normal? ...
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Molecular mimicry

Molecular mimicry is defined as the theoretical possibility that sequence similarities between foreign and self-peptides are sufficient to result in the cross-activation of autoreactive T or B cells by pathogen-derived peptides. Despite the promiscuity of several peptide sequences which can be both foreign and self in nature, a single antibody or TCR (T cell receptor) can be activated by even a few crucial residues which stresses the importance of structural homology in the theory of molecular mimicry. Upon the activation of B or T cells, it is believed that these ""peptide mimic"" specific T or B cells can cross-react with self-epitopes, thus leading to tissue pathology (autoimmunity). Molecular mimicry is a phenomenon that has been just recently discovered as one of several ways in which autoimmunity can be evoked. A molecular mimicking event is, however, more than an epiphenomenon despite its low statistical probability of occurring and these events have serious implications in the onset of many human autoimmune disorders. In the past decade the study of autoimmunity, the failure to recognize self antigens as ""self,"" has grown immensely. Autoimmunity is a result of a loss of immunological tolerance, the ability for an individual to discriminate between self and non-self. Growth in the field of autoimmunity has resulted in more and more frequent diagnosis of autoimmune diseases. Consequently, recent data show that autoimmune diseases affect approximately 1 in 31 people within the general population. Growth has also led to a greater characterization of what autoimmunity is and how it can be studied and treated. With an increased amount of research, there has been tremendous growth in the study of the several different ways in which autoimmunity can occur, one of which is molecular mimicry. The mechanism by which pathogens have evolved, or obtained by chance, similar amino acid sequences or the homologous three-dimensional crystal structure of immunodominant epitopes remains a mystery.
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