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Hamilton CME Paediatric Respiratory Emergencies Spring 2008 Paediatrics Stages of development: Newborn / Neonate: Birth to 29 days Infancy: 1 month to 1 year Toddler: 1 – 3 years Pre-school: 3 - 5 years School child: 5 - 15 years Adolescent: 15 - 19 years Respiratory System: The respiratory system matures as the child gets older. Newborns are usually nose breathers. This facilitates breathing while suckling. Age: Range of normal / min. Rapid / min. Newborn: 30-50 >60 Infancy: 20-30 >50 Toddler: 20-30 >40 Given a Competent Primary Survey Hands off approach. When examining a child, perform the most critical assessment you need to do before the child starts to cry. Take some history, visualize the child and decide which assessment you need to do first to confirm or rule out your suspicion. Listen to MOM! ( ‘my baby doesn’t quite seem right’ ) – A good mother will often make a better diagnosis than a poor Doctor ( or Paramedic ). Respiratory System Infection: – Can cause a relative arterial hypoxemia. – Predisposed to disease because of size & structure. – Small airways, poor muscle development, can’t clear mucous well during infections. Respiratory System Respiratory Distress: Lower airway: – short trachea, bifurcation at 45o. – Airways close more easily. – Incomplete lung development until 8 years old. Chest wall: – Muscles tire more easily. – Highly compliant, makes rib cage inefficient in producing an increase in lung volume & allows for distortion under stress retractions. – Large stomach & liver encroach on respiratory effort. Pneumonia Introduction – Pneumonia is defined pathologically as an inflammation of lower tract lung tissue. (1) Pneumonia Pneumonia Pathophysiology – Defense mechanisms • Macrophages • Antibodies • Lymphatic drainage Pneumonia Pathophysiology – anatomic defenses may be d/t preceding viral infection of upper respiratory tract. Pneumonia Pathophysiology – Acute inflammatory response • • • • Exudative fluid Fibrin deposition Leukocytes Macrophages Pneumonia Clinical Features – Fever can increase an infant's respiratory rate by 10 breaths/min for each degree centigrade of elevation. (1) Pneumonia Clinical Features – Adventitious breath sounds – WOB – Grunting respirations – Abdominal distention Signs of Respiratory Trouble: Facial Signs Colour ( lips and circumoral ) Nasal flaring Neck Tracheal tugging Supraclavicular Retractions Chest Lower Sternal Retraction Intercostal and/or subcostal indrawing Pneumonia Typical – – – – – Acute onset High-grade fever Pleuritic chest pain Productive cough Bacterial pathogen Atypical – – – – Gradual onset Low-grade fever Non-productive cough Viral pathogen Pneumonia Clinical Features – Infants frequently lack the classic symptoms and present with a variety of nonspecific findings. (1) Pneumonia Clinical Features – More severe pneumonia is associated with deterioration of the patient's mental status, the use of accessory muscles, and the presence of retractions, nasal flaring, splinting, and cyanosis. (1) Asthma Asthma Pathophysiology – Classifications • Extrinsic (IgE-mediated) • Intrinsic (infection-induced) • Mixed Asthma Pathophysiology – Two-stage process 1. 2. Bronchoconstriction (early) Mucosal edema & plugging (late) Asthma Pathophysiology – Bronchospasm, mucosal edema, and mucous plugging cause variable and reversible airflow obstruction with subsequent air trapping and impaired oxygen exchange.(2) Asthma Pathophysiology – Inadequate alveolar ventilation • Carbon dioxide retention • Respiratory acidosis • Respiratory failure Asthma Pathophysiology – The child with asthma is at higher risk of respiratory failure d/t: • • • • compliance of rib cage Immature diaphragm Lung tissue lacks elastic recoil Airway walls are relatively thicker Asthma Evaluation – Treatment with inhaled β2-agonists should not be withheld while the initial evaluation is in progress. (2) Asthma Evaluation – “silent” or “quiet” wheezer • Prolonged expiratory phase • Extreme air trapping Asthma Evaluation – Tripod positioning – Nasal flaring – Polyphonic wheezes – Cyanosis – Insensible fluid losses – Pulsus paradoxus & JVD Asthma Evaluation – History • • • • • Precipitating factors Prescription medications Hospitalizations Intubations Tracheostomies Asthma Evaluation – History • Neonatal - prematurity, BPD, NICU? • Adolescents - substance abuse? • All ages - aspiration / choking? Asthma Treatment – β2-Receptors are widely distributed on bronchial smooth muscle and airway epithelial cells. (2) Asthma Treatment – Salbutamol can be concurrently administered to an intubated patient via MDI and ETT spacer device or a patient assisted with BVM and spacer device. Asthma Asthma Asthma Treatment – Most children presenting in status asthmaticus will be dehydrated because of increased insensible losses. (2) Asthma Complications – Respiratory failure – Atelectasis – Pneumomediastinum – Pneumothorax Bronchiolitis Introduction – A clinical syndrome of wheezing, chest retractions, and tachypnea in children younger than age 2 years. (2) Bronchiolitis Epidemiology – October thru May – Peak age of incidence is 2 months Bronchiolitis Pathophysiology – Respiratory syncytial virus (RSV) causes 50 to 70 percent of clinically significant bronchiolitis. (2) Bronchiolitis Pathophysiology – Mucous plugging • Necrosis of respiratory epthelium • Destruction of ciliated epithelial cells – Submucosal edema Bronchiolitis Clinical Features – 911 may be called because of wheezing, increased respiratory symptoms, nasal congestion, and difficulty feeding. (2) Bronchiolitis Clinical Features – RSV-related apnea • Infants at highest risk are younger than 6 weeks old and have a history of prematurity, apnea of prematurity, and low O2 saturation. (2) Bronchiolitis Treatment – Keep patient & environment calm – Oxygen therapy PRN – Fluid therapy PRN Bronchiolitis Treatment – A trial of bronchodilator therapy, is an optional and reasonable treatment and can be aborted if the child fails to show a response. (2) Bronchiolitis Treatment – Epinephrine is an effective treatment for the wheezing of bronchiolitis. (2) Stridor Introduction – Stridor is due to Venturi effects created by somewhat linear airflow through a semicollapsible tube, the airway. (3) Stridor Introduction – Supraglottic – Subglottic – Trachea – Primary bronchi Stridor Introduction – Expiratory stridor, or wheeze, is common in distal airways, since intrathoracic pressure may become much greater than atmospheric pressure during expiration. (3) Stridor Introduction – Patients with marked variation in the pattern of stridor should be considered to have a foreign body in the airway until proven otherwise. (3) Epiglottitis Clinical Features – Since the introduction of the Haemophilus influenzae vaccine, the incidence and demographics of this disease have changed remarkably. (3) Epiglottitis Clinical Features – Abrupt onset – High-grade fever – Sore throat – Stridor – Dysphagia +/- drooling Epiglottitis Treatment – DO NOT attempt to visualize the airway unless respiratory failure/arrest is imminent. Epiglotittis Swollen, horseshoe-shaped epiglottis of a child with epiglottitis Same child with ETT in place. Epiglotittis Normal Paediatric trachea Epiglottitis Treatment – Should the child develop respiratory fatigue or if airway obstruction or apnea occurs before the airway has been secured, bag-valve-mask ventilation can be effective. (3) Croup Introduction – aka laryngotracheobronchitis – Peak 1-2 y.o. – Late fall thru early winter – child age = effect of airway edema Croup Radiograph of patient with Croup. Croup Clinical Features – Insidious onset – Barking cough – Stridor – S&S @ night Croup Treatment – Croup is an upper airway infection made worse by agitating the child. – Do not attempt to examine the throat. Croup Treatment – Do not attempt to initiate an IV unless it is required for essential medications or fluid resuscitation. Croup Treatment – Nebulized epinephrine decreases airway edema by vasoconstriction of the boggy mucosal vessels. (3) References 1. Emergency Medicine: A Comprehensive Study Guide - 6th Ed • VIRAL & BACTERIAL PNEUMONIA IN CHILDREN - Kathleen Brown, Willie Gilford, Jr. 2. Emergency Medicine: A Comprehensive Study Guide - 6th Ed • PEDIATRIC ASTHMA AND BRONCHIOLITIS - Maybelle Kou, Thom Mayer 3. Emergency Medicine: A Comprehensive Study Guide - 6th Ed • UPPER RESPIRATORY EMERGENCIES - Randolph Cordle