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LESSON ONE AMINISTRATION COMPONENTS OF FITNESS AEROBIC FITNESS VO2 MAX ENERGY SYSTEMS ONSET OF BLOOD LACTIC ACID (OBLA) COMPONENTS OF FITNESS CARDIORESPIRATORY ENDURANCE MUSCULAR STRENGTH MUSCULAR ENDURANCE BODY COMPOSITION FLEXIBILITY AEROBIC ACTIVITY USES LARGE MUSCLE GROUPS RHYTHMIC CONTINUOUS/INTERVAL RAISES HR TO TRAINING LEVEL GREATER THAN 20 MINUTES OXYGEN CONSUMPTION VO2 MAX: THE MAX AMOUNT OF O2 THAT THE BODY IS ABLE TO UTILIZE PER MINUTE OF PHYSICAL ACTIVITY VO2 MAX IS CONSIDERED THE SINGLE, BEST INDICATOR OF A PERSONS LEVEL OF AEROBIC FITNESS VO2 MAX OCCURS WHERE A FURTHER INCREASE IN WORK IS NOT ACCOMPANIED BY AN ADDITIONAL INCREASE IN O2 CONSUMPTION STROKE VOLUME sv VOLUME OF BLOOD PUMPED PER BEAT CARDIAC OUTPUT VOLUME OF BLOOD PUMPED PER MINUTE CO = HR X SV FICK PRINCIPLE THE AMOUNT OF OXYGEN USED IS A FUNCTION OF HOW MUCH BLOOD IS SENT TO MUSCLE AND HOW MUCH O2 IS IN THE BLOOD (A-V) O2 DIFFERENCE FICK EQUATION: VO2 = CARDIAC OUTPUT X (A-V) 02 DIFFERENCE GAS TRANSPORT AND EXCHANGE DIFFUSION OXYGEN HEMOGLOBIN CARBON DIOXIDE CARBON MONOXIDE CHANGES IN OXYGEN TRANSPORT CAPACITY CAUSES: LOSS OF BLOOD LACK OF OXYGEN IN AIR BLOCKADE ANEMIA OF Hb SOURCES OF ENERGY CARBOHYDRATE FAT PROTEIN ATP ADENOSINE TRIPHOSPHATE ENERGY SYSTEMS PHOSPHAGEN (ATP + CP) ANAEROBIC GLYCOLYSIS AEROBIC RESPIRATION –AEROBIC GLYCOLYSIS –BETA OXIDATION ATP + CP CREATINE PHOSPHATE (CP): HIGH ENERGY COMPOUND RAPID ENERGY PRODUCTION OCCURS WITHOUT OXYGEN IMMEDIATE ENERGY RESERVE 30 SECONDS HIGH INTENSITY ANAEROBIC GLYCOLYSIS SPLITTING OF GLUCOSE WITHOUT OXYGEN BRIDGES THE GAP BEGINNING OF EXERCISE PRODUCES HIGH LACTIC ACID CONCENTRATIONS 2-3 MINUTES OF INTENSE EXERCISE AEROBIC GLYCOLYSIS BEGINS WITH GLUCOSE FORMS PYRUVIC ACID IN THE PRESENCE OF O2 PYRUVIC ACID IS METABOLIZED INTO CO2 AND H2O KREBS CYCLE - CHEMICAL BREAKDOWN OF PYRUVIC ACID BETA OXIDATION FAT METABOLISM REQUIRES OXYGEN LONG DURATION EXERCISE LOWER INTENSITY SUBSTRATE UTILIZATION DEPENDENT ON 3 FACTORS: INTENSITY/DURATION STATE DIET OF TRAINING O.B.L.A LACTIC ACID ACCUMULATION EXPRESSED AS A PERCENTAGE OF MAXIMAL O2 UPTAKE NORMALLY OCCURS BETWEEN 55% AND 65% OF VO2 MAX OCCURS AT 80% VO2 MAX IN HIGHLY TRAINED ATHLETES REMOVAL OF LACTIC ACID OXIDATION TO CO2 AND H2O LOSS IN URINE AND SWEAT CONVERSION TO GLUCOSE OR GYCLOGEN OBLA “FACTS” INTENSITY DEPENDENT CAN BE TRAINED CAN BE POSTPONED LESSON TWO HEART RATE RESPONSE HEART RATE MONITORS MAX HEART RATE/RECOVERY RUN HEART RATE RESPONSE HEART RATE AND OXYGEN UPTAKE HAVE A LINEAR RELATIONSHIP HEART RATE IS CONSIDERED A GOOD WAY TO ESTIMATE EXERCISE INTENSITY. DETERMINING TRAINING HEART RATE ESTIMATE MAXIMUM HR (MHR) 220 - AGE = MHR DETERMINE HEART RATE RESERVE HRR = MHR - RESTING HEART RATE TRAINING HEART RATE (THR) (____% X HRR) + RHR = THR RATE OF PERCEIVED EXERTION 6 7 VERY VERY LIGHT 8 9 VERY LIGHT 10 11 LIGHT 12 13 SOMEWHAT HEAVY 14 15 HEAVY 16 17 VERY HEAVY 18 19 VERY VERY HEAVY 20 LESSON THREE FACTORS AFFECTING AEROBIC EXERCISE SHORT TERM RESPONSES LONG TERM ADAPTATIONS FACTORS AFFECTING AEROBIC EXERCISE MODE OF EXERCISE HEREDITY POTENTIAL FOR FITNESS GENDER AGE BODY COMP0SITION LEVEL OF ACTIVITY EXERCISE PRESCRIPTION FITT PRINCIPLE FREQUENCY 3 - 5 TIMES PER WEEK INTENSITY 60%-90% OF MHR 12-14 RPE SCALE 50%-80% VO2 MAX TIME 20-30 MINUTES TYPE LARGE MUSCLE GROUPS RHYTHMIC CONTINUOUS SHORT TERM RESPONSES TO AEROBIC EXERCISE INCREASE IN PULMONARY VENTILATION (RESPIRATION) INCREASE IN HEART RATE INCREASE IN STROKE VOLUME INCREASE IN CARDIAC OUTPUT INCREASE IN MUSCLE TEMPERATURE INCREASE IN BLOOD FLOW TO HEART LONG TERM ADAPTATIONS TO AEROBIC EXERCISE DECREASE IN RHR LOWER HR AT A GIVEN WORKLOAD DECREASE IN HR RECOVERY TIME INCREASE NUMBER AND SIZE OF FUNCTIONAL CAPILLARIES INCREASE IN CARDIAC OUTPUT INCREASE IN STROKE VOLUME INCREASE IN THE NUMBER AND SIZE OF MITACHONDRIA INCREASE IN THE ABILITY TO MOBILIZE AND UTILIZE FAT DECREASE IN BLOOD LIPIDS DECREASE IN RESTING BLOOD PRESSURE LONG TERM ADAPTATIONS TO AEROBIC EXERCISE MUSCULOSKELATAL BENEFITS INCREASED MUSCULAR STRENGTH INCREASED CAPILLARY DENSITY IN MUSCLE STRENGTHENING OF TENDONS, LIGAMENTS, AND JOINTS COUNTERACTS OSTEOPOROSIS MISCELLANEOUS BENEFITS ENHANCED TOLERANCE TO HEAT RELEASE OF ENDORPHINES “RUNNERS HIGH” LESSON FOUR AEROBIC FITNESS ASSESSMENT EXERCISE PRESCRIPTION AEROBIC FITNESS ASSESSMENT VO2 MAX: THE MAX AMOUNT OF O2 THAT THE BODY IS ABLE TO UTILIZE PER MINUTE OF PHYSICAL ACTIVITY VO2 MAX IS CONSIDERED THE SINGLE, BEST INDICATOR OF A PERSONS LEVEL OF AEROBIC FITNESS VO2 MAX OCCURS WHERE A FURTHER INCREASE IN WORK IS NOT ACCOMPANIED BY AN ADDITIONAL INCREASE IN O2 CONSUMPTION EXPRESSING VO2 ABSOLUTE VO2/VO2 MAX: L/min RELATIVE VO2/VO2 MAX: ml O2/Kg/min ASSESSING VO2 MAX MAXIMAL TEST: REQUIRES A PERSON TO EXERCISE TO EXHAUSTION SUBMAXIMAL TEST:MEASURES AN INDIVIDUALS RESPONSE AT SUBMAXIMAL EXERCISE. VO2 MAX IS PREDICTED FROM EXERCISE HEART RATE FOR THE GIVEN SUBMAXIMAL EXERCISE ASSESSING VO2 MAX LAB FIELD MAX SUBMAX BENEFITS OF TESTING ASSESSES STRENGTHS AND WEAKNESSES PROVIDES BASELINE DATA PROVIDES FEEDBACK PROVIDES HEALTH ASSESSMENT PROVIDES UNDERSTANDING TESTING WILL NOT PREDICT FUTURE “GOLD MEDALISTS” SIMULATE ACTUAL PHYSICAL DEMANDS OF SOME SPORTS 2 MILE RUN TEST MEN: VO2MAX = 99.7 - (3.35 X TIME) WOMEN: VO2MAX = 72.9 - (1.77 X TIME) PRINCIPLES OF EXERCISE OVERLOAD PROGRESSION RECOVERY SPECIFICITY REGULARITY VARIETY BALANCE 3 PHASES OF AN AEROBIC EXERCISE PROGRAM PREPARATORY PHASE USUALLY LASTS 4-6 WEEKS CONDITIONING PHASE USUALLY LASTS 12-20 WEEKS INTENSITY 70 - 80 HRR DURATION/FREQUENCY INCREASED BY 10% PER WEEK MAINTENANCE PHASE BEGINS 6 MONTHS AFTER STARTING PROGRAM CONTINUES FOR A LIFETIME PHYSIOLOGICAL EFFECTS OF A WARM UP INCREASE IN BODY TEMPERATURE INCREASE IN BLOOD FLOW TO MUSCLES AND HEART DECREASED MUSCLE CONTRACTION AND REFLEX TIME DECREASES CHEMICAL REACTION TIME PHYSIOLOGICAL EFFECTS OF A COOL DOWN DECREASED LACTIC ACID LEVELS PREVENTS BLOOD POOLING STRETCHING WARM MUSCLES IMPROVES FLEXIBILITY DECREASED CHANCES OF CARDIAC IRREGULARITIES EXERCISE ADHERENCE FUN CROSS-TRAINING REGULARITY PROPER EQUIPMENT TRAINING PARTNER SET GOALS AVOID OVERTRAINING KEEP A LOG ASSESSMENTS MONITOR HEALTH LESSON FIVE FATIGUE DELAYED ONSET OF MUSCLE SORENESS (DOMS) CONTRAINDICATIONS ENVIRONMENTAL CONSIDERATIONS STADIUM STAIR CLIMBING MECHANISMS OF FATIGUE POSSIBLE CAUSES: DECREASED ATP SUPPLY INCREASE IN LACTIC ACID DEHYDRATION MOTIVATION HYPERTHERMIA DEPLETION OF GLYCOGEN ELECTROLYTE IMBALANCE POSSIBLE CAUSES OF MUSCLE SORENESS ACUTE: LACK OF BLOOD FLOW (O2) AND GENERAL FATIGUE OF MUSCLES DELAYED ONSET OF MUSCLE SORENESS (D.O.M.S.): 12 HOURS AFTER EXERCISE MAY LAST 2-4 DAYS TEARS TO MUSCLE AND CONNECTIVE TISSUE INCREASE FLUID RETENTION MUSCLE SPASMS NOT A SPRAIN OR INJURY PREVENTION: WARM UP, START SLOW, COOL DOWN PROPERLY CONTRAINDICATIONS OF EXERCISE RECENT HEART ATTACK UNSTABLE ANGINA PECTORIS ABNORMAL HEART ACTIVITY PAIN IN CHEST, ARMS FOLLOWING ACTIVITY DIZZINESS, LIGHT HEADINESS LACK OF COORDINATION, CONFUSION, COLD SWEATING ILLNESS, PARTICULARLY VIRAL INFECTIONS SYMPTOMS OF OVERTRAINING MUSCLE SORENESS HEADACHES, SORE THROAT, MILD COLD IRRITABILITY INSOMNIA LACK OF INTEREST IN DAILY ACTIVITIES LOSS OF APPETITE SUDDEN DROP IN WEIGHT CONSTIPATION OR DIARRHEA SKIN ERUPTIONS ABNORMALLY HIGH RHR RISK FACTORS AGE HEREDITY GENDER SMOKING OBESITY LACK OF EXERCISE HIGH CHOLESTEROL HIGH BLOOD PRESSURE ABNORMAL EKG STRESS DIABETES EXERCISING IN THE COLD DRESS IN LAYERS AVOID PROFUSE SWEATING AVOID WIND CHILL FACTOR DRINK PLENTY OF WATER ____________________________ HYPOTHERMIA FROSTBITE MAJOR FORMS OF HEAT ILLNESS HEAT CRAMPS: MUSCLE PAIN AND SPASM HEAT EXHAUSTION: WEAK, RAPID PULSE, LOW BLOOD PRESSURE, HEADACHE, AND DIZZINESS HEAT STROKE: CEASATION OF SWEATING, DRY, HOT SKIN AND VERY HIGH BODY TEMPERATURE - THE MOST SERIOUS AND COMPLEX HEAT PROBLEM FACTORS TO MODIFY HEAT TOLERANCE GRADUALLY ACCLIMATIZE DRINK PLENTY OF WATER BEFORE DURING AND AFTER ALLOW FOR EVAPORATION LIMITED IN HUMID WEATHER REPLACE ELECTOROLYTES LOST THROUGH SWEATING AVOID EXERCISE DURING HOTTEST TIME OF DAY EFFECTS OF DEHYDRATION REDUCED CIRCULATORY CAPACITY REDUCED MAX CARDIAC OUTPUT ALTERED AUTONOMIC NERVOUS CONTROL OF SWEAT GLANDS REDUCED WORK CAPACITY FLUID REPLACEMENT WITH CARBOHYDRATE INCREASE IN BLOOD GLUCOSE INCREASE IN GLUCOSE UTILIZATION BY MUSCLE INCREASE IN ENDURANCE BENEFITS OF FLUID REPACEMENT IMPROVED REGULATION OF BODY TEMPERATURE INCREASED SWEAT RATE DECREASED BODY CORE TEMP INCREASED PLASMA VOLUME INCREASED STROKE VOLUME INCREASED PERFORMANCE LESSON SIX RUNNING BASICS DIFFERENT RUNNING MODALITIES/TRAINING METHODS INTERVAL RUNNING RUNNING MODALITIES AND TRAINING METHODS LONG SLOW DISTANCE FARTLEK “SPEED PLAY” FAST, CONTINUOUS INDIAN RUNS INTERVALS INTERVAL TRAINING DEFINITION: SERIES OF REPEATED BOUTS OF EXERCISE ALTERNATED WITH PERIODS OF RELIEF VARIABLES (TO EMPHASIZE A PARTICULAR ENERGY SYSTEM) RATE AND DISTANCE OF WORK (BASED ON PERFORMANCE TIME) NUMBER OF REPETITIONS RELIEF TIME BETWEEN INTERVALS FREQUENCY OF TRAINING PER WEEK BENEFITS OF INTERVAL TRAINING ALLOWS FOR QUALITY, HIGH INTENSITY WORK APPLIES PRINCIPLE OF SPECIFICITY CAN STRESS ALL ENERGY SYSTEMS TRAINS NERVOUS SYSTEM FORMULA FOR DETERMINING INTERVAL PACE (4 -8 X 400M) MILE TIME = 8:00 MINUTE 400 METER = 2:00 MINUTE SUBTRACT 1-4 SECONDS : = 1:56-1:59 LESSON 14 NUTRITION BASICS SUBSTRATE CARBOHYDRATES FATS PROTEINS NUTRITION PROTEIN: 12% OF DAILY CALORIC INTAKE FAT: NOT TO EXCEED 30% OF DAILY CALORIC INTAKE CARBOHYDRATE: AT LEAST 50%-60%, CONSIDER 70%-75% OF DAILY CALORIC INTAKE MAJOR SOURCE OF ENERGY SPARES BREAKDOWN OF PROTEIN METABOLIC PRIMER FOR FAT IMPORTANT FOR ATHLETIC PERFORMANCE RELEASED WITHIN MUSCLE 3 TIMES AS FAST AS FAT GENERAL GUIDELINES FOR A HEALTHFUL DIET EAT A VARIETY OF FOODS AVOID FATS AND CHOLESTEROL EAT FOODS HIGH IN STARCH AND FIBER AVOID EXCESSIVE SUGAR AND SODIUM MAINTAIN ADEQUATE CALCIUM INTAKE MAINTAIN RECOMMENDED BODY WEIGHT USE ALCOHOL IN MODERATION DRINK PLENTY OF WATER