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SHORT-TERM EEG-VIDEO MONITORING REPORT EPILEPSY LABORATORY CLINICAL NEUROPHYSIOLOGY LABORATORIES DEPARTMENT OF PEDIATRICS UNIVERSITY OF MICHIGAN Patient: @NAME@ Age: @AGEPEDS@ MRN: @MRN@ Date: @ED@ Referring physician attending: @ORDPROV@ Introduction: The patient is a @AGEPEDS@ {boy/girl:304001889} with a history of ***. This short-term video-EEG monitoring study was performed in order to diagnose the etiology of the patient's clinical events. The patient {IS/IS NOT:27046} on neuroactive medications. This recording was obtained using the standard 21-channel 10-20 electrode placement. Single lead EKG monitoring and infraorbital electrodes were included. A number of standard bipolar and referential montages were used. Current Medications: @NAME@ @CMEDPBRAND@ Baseline EEG Recording: The baseline recording was obtained during {wakefulness_drowsiness_sleep_free:304014064}. In the alert state, the posterior background rhythm was a symmetric, well-modulated, *** Hz, *** uV rhythm which reacted symmetrically to eye opening and had a normal frequency-amplitude gradient with an age-appropriate mixture of frequencies. There were no focal or epileptiform abnormalities and no clinical or electrographic seizures occurred during the baseline. Hyperventilation, performed with {desc; poor/fair/good/excellent:19665} effort, produced physiological slowing. Stepwise photic stimulation at 2-30 Hz evoked symmetric posterior driving responses. No abnormalities were activated by hyperventilation or by photic stimulation. During drowsiness there were bursts of diffuse slowing and waxing and waning of the posterior dominant rhythm. During stage II sleep symmetric V waves, K complexes, and sleep spindles were seen. The EKG channel revealed no abnormalities. Interictal EEG Samples: During wakefulness, the EEG was as described above. During drowsiness the background rhythm waxed and waned and there were periods of slowing. Interictal EEG samples during stage II sleep demonstrated symmetric V waves, K complexes, and sleep spindles. There was appropriate diffuse delta activity during slow wave sleep. No abnormalities were activated by sleep. The EKG channel revealed no abnormalities. Ictal EEG Recording / Push-Button Events: During this period the patient had no events or seizures. Summary of EEG and Behavior: The interictal EEG was {EEG abnormal/normal interpretation:304014062}. *** of the patient’s typical events were recorded. *** EEG change occurred during the patient’s clinical event(s). Clinical Correlation: This *** hour short-term EEG recording was a {EEG abnormal/normal interpretation:304014062} awake and asleep EEG. ***No patterns with a specific correlation with seizures were present, and no focal abnormalities were found. The patient’s typical episodes were recorded and were consistent with ***. **This EEG was abnormal due to the following: 1. *** 2. *** 3. *** ***The disorganized background, with diffuse *** Hz slowing was consistent with a mild/mild to moderate/moderate/moderate to severe/severe encephalopathy. ***The continuous focal slowing over *** was consistent with an underlying structural lesion or a post-ictal state. ***The intermittent focal slowing over *** suggested underlying neuronal dysfunction. The focal sharps/spikes/spike and wave complexes over *** conferred an increased risk of focal seizures arising from this region. ***The generalized sharps/spikes/spike and wave complexes conferred an increased risk of seizures with a generalized onset. ***The multifocal sharps/spikes/spike and wave complexes conferred an increased risk of focal and generalized seizures. ***There were *** seizures recorded, clinically manifesting as ***, and with the electrographic appearance of ***. ***There were *** events recorded, and there was no abnormal EEG change with these events. These were not seizures. ***Overall, this EEG is improved/similar/worse compared to a previous EEG recorded mm/dd/yyyy, because of ***. ***In view of the clinical picture, a recording including sleep/with reactivity testing repeated/ with longer duration to capture spells, etc. could be useful. A normal recording does not exclude the possibility of epilepsy, in this clinical setting, repeat recording could be useful. @MECREDENTIALNOREFRESH@ Epilepsy Fellow This EEG was reviewed with epilepsy attending {ldattending:28131}. Please note this is a preliminary report until signed by the attending. Clinical Diagnosis Code: {lddiagnosiscodesICD9-10:28354}