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Steven Ma, D.O. Post Graduate Fellow in Neuromuscular Medicine Larkin Community Hospital September 24, 2016 Overview: o Basic Knee Anatomy o Types of Knee Pain o General Approach o Special Tests o Basic Workup o Treatment Options o Commonly Seen Dysfunctions Case Presentation: “Mrs. Smith” presents to the office with the chief complaint of a severe 8/10 left lateral knee pain. The pain started one month ago when she was visiting home in New York. The pain was a gradual onset, aggravated by going up and down flights of stairs, and ambulating. She noted that there was associated decreased mobility, lack of strength, and a constant “cracking of the knees.” The patient also stated that the pain resulted with her having difficulty walking, and getting out of bed had become an increasingly hard task for her. The patient was seeing a Chiropractor for adjustments, which provided temporary relief. Massage, Heat, and Therapeutic Ultrasound Treatments have also helped temporarily. The patient was also seeing an Orthopedic Surgeon, who scheduled her for a Left Knee Arthroscopy for a Lateral Meniscus Tear seen on MRI. The patient came to our office for evaluation and for a second opinion before proceeding with the surgery. Basic Anatomy: o Largest Joint in the Body (Tibiofemoral Joint) o Modified Hinge Joint: Flexion and Extension o o with Slight Internal and External Rotation Compound Joint: Three Parts of Articulation o o o 1. Between Femur and Tibia (Tibiofemoral Joint) 2. Between Femur and Patella (Patellofemoral Joint) 3. Between Tibia and Fibula (Tibiofibular Joint) Basic Anatomy: o Tibiofemoral Joint: o 2 “C” Shaped Menisci: Medial + Lateral o Act as Shock Absorbers and Aid in Nutrition and Lubrication o 4 Ligaments: o Anterior Cruciate Ligament (ACL) o Posterior Femur to Anterior Tibia: Prevents Anterior Translation of Tibia on Femur o Posterior Cruciate Ligament (PCL) o Anterior Femur to Posterior Tibia: Prevents Posterior Translation of Tibia on Femur o Medial Collateral Ligament (MCL) o Femur to Tibia: Stabilizes the Knee Medially o Lateral Collateral Ligament (LCL) o Femur to Fibula: Stabilizes the Knee Laterally Basic Anatomy: o Tibiofemoral Joint: o Medial Meniscus o Lateral Meniscus o Anterior Cruciate Ligament (ACL) o Posterior Cruciate Ligament (PCL) o Medial Collateral Ligament (MCL) o Lateral Collateral Ligament (LCL) Basic Anatomy: o Patellofemoral Joint: o Space between the Patella and the Patellar Groove of the Femur Basic Anatomy: o Tibiofibular Joint: o The Fibular Head Glides Anteriorly with Pronation of the Foot. o Pronation = Dorsiflexion, Eversion, and ABduction of Foot o The Fibular Head Glides Posteriorly with Supination of the Foot. o Supination = Plantarflexion, Inversion, and ADDuction of Foot Biomechanics: o o o Function: Flexibility for Walking Stability while Standing Biomechanics: o Range of Motion: o Flexion: 135-155 Degrees o Extension: 0-10 Degrees o Internal/External Rotation: Up to 10 Degrees Biomechanics: o o o Between Each Joint is Cartilage and Connective Tissue. Joint Capsule: The Knee Joint is contained inside a membrane, bathed in Synovial Fluid. Together, they allow for Smooth, Friction-Free Motion. Biomechanics: o Cartilage Have No Nerve Endings. o So there should be no pain with Regular Physiology. o Bone Has Nerve Endings. Pain is caused when these nerve endings are exposed due to a breakdown or disruption of the normal Cartilage tissue. Initial Evaluation: o Must Determine: o Acute vs. Chronic o Traumatic vs. Overuse vs. Pathologic Disease o Do We Treat or Do We Refer??? Types of Knee Pain: o Acute Knee Pain: o <6 weeks o Usually Traumatic o Chronic Knee Pain o >6 weeks o Overuse o Other Acute Knee Pain: o o o o o o o o o Medial/Lateral Collateral Ligament Tear ACL Tear Meniscus Tear Patellar Dislocation Patellar Tendon Tear Intra-Articular Fracture PCL Tear Quadriceps Tendon Tear Knee Dislocation Chronic Overuse Knee Pain: o o o o o Bursitis Capsulitis Synovitis Tendonitis Chondromalacia Patellae (Patellofemoral Pain Syndrome) Other Causes: o o o o o o Osteoarthritis Rheumatoid Arthritis Gout Osgood-Schlatter’s Disease Hip Dysfunction Ankle Dysfunction Evaluating the Knee: o History o Mechanism of Injury Usually Suggests Diagnosis o Where, What, When, How??? o Key Symptoms: Pop, Lock, & Drop o Trauma or Overuse Evaluating the Knee: o Physical Exam (Always Perform Bilaterally!) o Inspection o Palpation o Range of motion o Strength o Special Tests Evaluating the Knee: o Inspection: o Gait: Limp, Pain, Guarding, Favoring One Side o Anatomy: Position of Hips, Feet, Genu Varus/Valgus o Deformities? o Swelling? Evaluating the Knee: o Palpation: o Temperature: Inflammation? o Pulses: Intact? o Pain: Location! Location!! Location!!! o Defects: Ruptured Quadriceps Tendons? Evaluating the Knee: o Evaluate the Hip AND Ankle!!! o Hips: FABER’s (Patrick’s) Test, ASIS Compression, etc. o Ankle: Anterior/Posterior Glide, Supination/Pronation Where is the Pain? o Medial Joint Line: Medial Collateral Ligament o Lateral Joint Line: Lateral Collateral Ligament o o Lateral Patellar Edge, or When Pressing Patella Down Firmly: Chondromalacia Posterior Fossa: Baker’s Cyst Where is the Pain? o At the Tibial Tuberosity: Osgood-Schlatter’s Disease o Superior to Tibial Tubercle: Patellar Tendinitis (“Jumper’s Knee”) o Medial to Tibial Tuberosity: Pes Anserine Bursitis/Tendonitis Range of Motion: o Flexion o Extension o Perform Bilaterally! o Acute Limitation may be due to Injury of Somatic Dysfunction Range of Motion: o Chronic Limitation in both Flexion and Extension: THINK CAPSULITIS!!! ACL Tear Special Testing: Meniscus Tear Collateral Ligament Tear http://orthoinfo.aaos.org/topic.cfm?topic=a00549 ACL Injury: o History: Mechanism of Injury o o o Sudden Change in Direction Landing After A Jump Lateral Blow to a Partially Flexed Knee ACL Injury: o Key Symptoms o o “Pop” Sound at Time of Injury Slipping Out of Place http://a2.espncdn.com/combiner/i?img=%2Fphoto%2F2015%2F0822%2Fusatsi_8760978_r3417_1296x729_16%2D9.jpg&w=570 ACL Special Tests: o Lachman Test o Anterior Drawer Test PCL Injury: o o o It is stronger than the ACL and is injured less often. They are often subtle and more difficult to evaluate than the other ligament injuries. Usually occur along with injuries to other structures in the knee. o o Causes: A direct blow to the front of a bent knee (such as hitting the dashboard in a car accident or a football player falling on a bent knee. Symptoms: the knee feeling unstable, like it may "give out." PCL Special Testing: Meniscus Tear: o History: o An Acute Forceful Injury Involving Twisting of the Knee while the Foot is Planted. Meniscus Tear: o Key Symptoms: o Complaining of popping, locking, catching, and the knee "giving out" Meniscus Tests: o Thessaly Test: o Patient and Examiner face each other, holding hands for support. o Patient stands on one leg with their knee flexed to 20 degrees. o Patient rotates the knee and body while maintaining knee flexion, (which internally and externally rotates the knee, while loading the meniscus.) o Reproduction of the Catching, Clicking, or Pain is a Positive Test. o Apley’s Compression Test: o o Compression of the Tibia onto the Knee Joint while Externally Rotating. Production of pain is a Positive Test. Meniscus Tests: o McMurray Test: o External Rotation with a Valgus Stress: Medial Meniscus o Internal rotation with a Varus Stress: Lateral Meniscus o Slowly Extend The Knee o Production of a “Click” Sound is a Positive Test Collateral Ligament Injury: o History: o Trauma involving Twisting of the Leg or a Direct Blow leading to a Varus or Valgus Displacement. http://bleacherreport.com/articles/1322157-2012-fantasy-football-injuries-10-players-owners-should-be-worried-about Collateral Ligament Injury: o Varus and Valgus Stress Test: Collateral Ligament Injury: o Apley’s Distraction Test: o Stabilizethe patient’s leg with your knee on the posterior thigh. o Distract the Tibia, and Externally and Internally Rotate the leg. o Excessive Motion on Rotation is a Positive Test. For ACL, Meniscus, & Collateral Ligament Injuries: o Acute Treatment: PRICES o Protection o Rest o Ice o Compression o Elevate o Support (Crutches) For ACL, Meniscus, & Collateral Ligament Injuries: o Imaging: o X-Ray: 3 Views- AP, Lateral, and Sunrise Views o MRI For ACL, Meniscus, & Collateral Ligament Injuries: o o o Based on the findings, formulate a Plan of Care. Treat and manage what you are comfortable doing. Refer to a Specialist if the Severity of Injury is Warranted. Capsulitis: o o o Painful Disorder that results from the Chronic Inflammation, Scarring, Thickening and Shrinkage of the Capsule that Surrounds the Involved Joint. Severely Restricted, with Progressive Loss of Both Active and Passive Range of Motion. For the Knee: A Remarkably Decrease in both Flexion AND Extension. Pes Anserine Tendonitis/Bursitis o Pes Anserine-The conjoining of the three tendons of the Sartorius, Gracilis and Semitendosus Muscles. o Presents as Chronic Pain and Weakness Inferomedial to the Knee Joint. o Commonly caused by Overuse in Athletes. o Often coexists with other Knee Disorders. Chondromalacia (Patellofemoral Syndrome) o o Patellofemoral Syndrome: Retropatellar or Peripatellar Pain resulting from Physical and Biochemical Changes in the Patellofemoral Joint. Chondromalacia: When there is Fraying and Damage to the underlying Patellar Cartilage. Chondromalacia (Patellofemoral Syndrome): o o o Presents as Anterior Knee pain that typically occurs with activity, and often worsens when they are walking down steps or hills. It can also be triggered by prolonged sitting. Usually due to Weakness of the Medial Quadriceps, Tight Iliotibial Bands, Tight Hamstrings, Weakness of the Hip ADDuctors, and/or Tight Calf Muscles. Chondromalacia (Patellofemoral Syndrome): o Patellar Grind Test: A Positive Test indicates Patellofemoral Syndrome. Chondromalacia (Patellofemoral Syndrome): For Overuse Injuries: o Treatment: o Rest o Ice o NSAIDs, Anti-inflammatory Medications o OMT o Stretches o Exercises o Therapeutic Ultrasonography o Injections Injection Therapies: o o Corticosteroid Injections: can offer fast-acting relief of inflamed muscles, joints, tendons, and bursa. Prolotherapy: also known as Regenerative Injection Therapy, involves the injection of substances into degenerated or injured areas to stimulate healing. o Commonly used solutions include Hyperosmolar Dextrose, Glycerine, Lidocaine, and Homeopathic Solutions such as TRAUMEEL. Injection Therapies: o Platelet-Rich Plasma: A concentration of Platelet cells extracted from blood, containing several different Growth Factors and Cytokines that can stimulate healing. Injection Therapies: Somatic Dysfunctions: Anterior Tibial Dysfunction (on the Femur): o o o Symptoms: Knee Discomfort, Inability to Comfortably Extend the Knee. Inspection: Restricted Posterior Spring with Loss of Anterior Motion. Palpation: Prominence of the Tibial Tuberosity. Anterior Tibial Dysfunction (on the Femur): Dysfunction: Tibia “Stuck” forward. Anterior Tibial Dysfunction (on the Femur): Treatment Posterior Tibial Displacement (on the Femur): o o Symptoms: Knee Discomfort, Inability to Comfortably Flex the Knee. Inspection: Restricted Posterior Spring with Loss of Anterior Motion. Posterior Tibial Displacement (on the Femur): Treatment Posterior Tibial Displacement (on the Femur): Treatment Anterior Fibular Head: o Symptoms: o Lateral leg Pain and Soreness. o Tenderness over the Proximal Fibula. o Inspection: o Increased Anterior Glide of the Proximal Fibular Head, with Resisted Posterior Springing. o Distal Fibula may be Posterior. o Talus Externally Rotated, causing the foot to Evert and Dorsiflex. o History: o Commonly follows a Medial Ankle Sprain or Forced Dorsiflexion of the Ankle. Anterior Fibular Head: Posterior Fibular Head: o Symptoms: o Lateral leg Pain and Soreness. o Tenderness at the Fibular Head. o Persistent Ankle Pain Beyond that of a Normal Ankle Injury. o Inspection: o Increased Posterior Glide of the Proximal Fibular Head, with Restricted Anterior Glide. o Proximal Fibular Head Prominent Posteriorly. o Talus Internally Rotated, causing the foot to Invert and Plantarflex. o History: o Commonly follows an Inversion Ankle Sprain. Posterior Fibular Head Posterior Fibular Head: Treatment So What Happened to “Mrs. Smith???” Case Presentation: “Mrs. Smith” was diagnosed with Left Posterior Fibular Head and Bilateral Pes Anserine Bursitis. Her First Treatment Session consisted of Osteopathic Manipulative Treatment to Correct all Somatic Dysfunction, including her Posterior Fibular Head. Case Presentation: Just after the one treatment session, “Mrs. Smith’s” pain went from an 8/10 to a 3/10. “Mrs. Smith” pain improved so dramatically that she called the Orthopedic Surgeon the very next day to cancel the Left Knee Arthroscopy. Case Presentation: Subsequent treatment sessions consisted of Prolotherapy with Homeopathic Solutions to the Pes Anserine Bursas and more Osteopathic Manipulation. Her pain continued to improve. After her last treatment session, “Mrs. Smith” informed us that she was planning a trip back to New York, something that she did not think that she was going to do ever again out of fear of having to walk up and down stairs. In Conclusion: o o o A good history of the Mechanism of Action can give a good indication of the Injury. Perform a good physical exam, using Special Testing to confirm your diagnosis. Formulate a Plan of Care. In Conclusion: o o Osteopathic Medicine can make a difference!!! There is a lot that we, as Osteopaths, can do to help our patients. “To find health should be the object of the doctor. Anyone can find disease.” – A.T. Still MD, DO Resources: o o o o o o o Kinetic Anatomy, Third Edition. Robert S. Behnke. 2012. Human Kinetics Inc. Atlas of Osteopathic Techniques, 2nd Edition. Alexander S. Nicholas, D.O., FAAO. Evan A. Nicholas, D.O. 2012. Lippincott Williams and Wilkins. OMT Review, Third Edition. Robert G. Savarese, D.O. 1998. Bates' Guide to Physical Examination and History Taking, 9th Edition. Lynn S. Bickley. 2007. Lippincott Williams and Wilkins. Clinically Oriented Anatomy, Fifth Edition. Keith L. Moore. Arthur F. Dalley. 2005. Lippincott Williams and Wilkins. Netter's Clinical Anatomy, 2nd Edition. John T. Hansen, PhD. 2009. Saunders. Musculoskeletal Medicine, 3rd Edition. Jeffrey Gross, MD. Joseph Fetto, MD. Elaine Rosen, PT, DHSc, OCS. 2009. Wiley-Blackwell. 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