
Infectious Transmission of Human T-cell Lymphotropic Virus Type I1
... weeks PI) and persistent (as late as 24 weeks PI) antiHTLV antibody response and demonstration of viral antigens or amplifiable proviral sequences in tissues of inoculated rabbits. Similar to findings in HTLV-infected h ~ m a n s , ' the ~ . ~earliest and most intense serologic reactivity occurred a ...
... weeks PI) and persistent (as late as 24 weeks PI) antiHTLV antibody response and demonstration of viral antigens or amplifiable proviral sequences in tissues of inoculated rabbits. Similar to findings in HTLV-infected h ~ m a n s , ' the ~ . ~earliest and most intense serologic reactivity occurred a ...
Pathogenesis of porcine reproductive and respiratory syndrome virus
... The structure and composition of the virion are reviewed elsewhere [7]. The nucleocapsid (N) protein forms a polymer surrounding the viral genome. The surface of the virion is dominated by glycoprotein (GP) 5 disulfide linked to the matrix (M) protein. Minor surface glycoproteins form a trimer comp ...
... The structure and composition of the virion are reviewed elsewhere [7]. The nucleocapsid (N) protein forms a polymer surrounding the viral genome. The surface of the virion is dominated by glycoprotein (GP) 5 disulfide linked to the matrix (M) protein. Minor surface glycoproteins form a trimer comp ...
PPT
... Paracrine signaling. The signaling molecules released by one cell act on neighboring target cells (neurotransmitters). Autocrine signaling. Cells respond to signaling molecules that they themselves produce (response of the immune system to foreign antigens and cancer cells). ...
... Paracrine signaling. The signaling molecules released by one cell act on neighboring target cells (neurotransmitters). Autocrine signaling. Cells respond to signaling molecules that they themselves produce (response of the immune system to foreign antigens and cancer cells). ...
Chapter 19 Notes
... Every time the host divides, it copies the phage DNA and passes the copies to daughter cells. ○ The viruses thus propagate without killing the host cells on which they depend. ○ A single infected cell can quickly give rise to a large population of bacteria carrying the virus in prophage form. ...
... Every time the host divides, it copies the phage DNA and passes the copies to daughter cells. ○ The viruses thus propagate without killing the host cells on which they depend. ○ A single infected cell can quickly give rise to a large population of bacteria carrying the virus in prophage form. ...
Chapter 19 lecture outline
... Every time the host divides, it copies the phage DNA and passes the copies to daughter cells. ○ The viruses thus propagate without killing the host cells on which they depend. ○ A single infected cell can quickly give rise to a large population of bacteria carrying the virus in prophage form. ...
... Every time the host divides, it copies the phage DNA and passes the copies to daughter cells. ○ The viruses thus propagate without killing the host cells on which they depend. ○ A single infected cell can quickly give rise to a large population of bacteria carrying the virus in prophage form. ...
Interferon

Interferons (IFNs) are a group of signaling proteins made and released by host cells in response to the presence of several pathogens, such as viruses, bacteria, parasites, and also tumor cells. In a typical scenario, a virus-infected cell will release interferons causing nearby cells to heighten their anti-viral defenses.IFNs belong to the large class of proteins known as cytokines, molecules used for communication between cells to trigger the protective defenses of the immune system that help eradicate pathogens. Interferons are named for their ability to ""interfere"" with viral replication by protecting cells from virus infections. IFNs also have various other functions: they activate immune cells, such as natural killer cells and macrophages; they increase host defenses by up-regulating antigen presentation by virtue of increasing the expression of major histocompatibility complex (MHC) antigens. Certain symptoms of infections, such as fever, muscle pain and ""flu-like symptoms"", are also caused by the production of IFNs and other cytokines.More than twenty distinct IFN genes and proteins have been identified in animals, including humans. They are typically divided among three classes: Type I IFN, Type II IFN, and Type III IFN. IFNs belonging to all three classes are important for fighting viral infections and for the regulation of the immune system.