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... “…careful inspection of some thirty records where… contractions were induced by direct stimulation [of the ventricle] showed without fail that [the] early phase of slower contraction is prolonged and that the tension develops more gradually. When artificial stimuli are given… at rates corresponding ...
... “…careful inspection of some thirty records where… contractions were induced by direct stimulation [of the ventricle] showed without fail that [the] early phase of slower contraction is prolonged and that the tension develops more gradually. When artificial stimuli are given… at rates corresponding ...
Zool 352 Lecture 33
... • 1. shift activation of L channels to more negative voltages, causing threshold to be reached sooner; increase current carried by L channels. • 2. Increase If, causing hypolarization at beginning of prepotential to be smaller. ...
... • 1. shift activation of L channels to more negative voltages, causing threshold to be reached sooner; increase current carried by L channels. • 2. Increase If, causing hypolarization at beginning of prepotential to be smaller. ...
Electrocardiogram (EKG, ECG) - Dr. Ray Winstead`s Front Page
... As the heart undergoes depolarization and repolarization, the electrical currents that are generated spread not only within the heart, but also throughout the body. This electrical activity generated by the heart can be measured by an array of electrodes placed on the body surface. The recorded trac ...
... As the heart undergoes depolarization and repolarization, the electrical currents that are generated spread not only within the heart, but also throughout the body. This electrical activity generated by the heart can be measured by an array of electrodes placed on the body surface. The recorded trac ...
Chapter 20 - FacultyWeb
... 1. The AV node slows the heart to an average between its own rate and that of the SA node. 2. Parasympathetic innervation slows the HR. 3. The AV node takes over causing the HR to be 40–60 bpm. 4. None of these is correct. ...
... 1. The AV node slows the heart to an average between its own rate and that of the SA node. 2. Parasympathetic innervation slows the HR. 3. The AV node takes over causing the HR to be 40–60 bpm. 4. None of these is correct. ...
Heart Failure Clinical Process Guideline Deborah Ayers, RN, MSN Quality Improvement Nurse
... symptoms Structural disorder, past or current HF symptoms End-stage disease, requiring specialized treatment ...
... symptoms Structural disorder, past or current HF symptoms End-stage disease, requiring specialized treatment ...
Exploring new concepts in the management of heart failure with
... Despite having preserved systolic function at rest, these patients display reduced cardiac functional reserve and morbidity/mortality rates similar to values observed in HF patients with reduced systolic function (HFrEF). Although HFpEF has been clinically recognized for over 30 years, our understan ...
... Despite having preserved systolic function at rest, these patients display reduced cardiac functional reserve and morbidity/mortality rates similar to values observed in HF patients with reduced systolic function (HFrEF). Although HFpEF has been clinically recognized for over 30 years, our understan ...
Preventing SCA Fact sheet
... ACE inhibitor was captopril (Capoten). Now there are 11 ACE inhibitors and they are used for other heart conditions as well. Statins are a relatively new group of drugs used to lower blood cholesterol levels. The best known statins are simvastatin (Zoco$ and atorvastatin (Lipitor). A high cholestero ...
... ACE inhibitor was captopril (Capoten). Now there are 11 ACE inhibitors and they are used for other heart conditions as well. Statins are a relatively new group of drugs used to lower blood cholesterol levels. The best known statins are simvastatin (Zoco$ and atorvastatin (Lipitor). A high cholestero ...
YABC - Anatomy 2
... from the time it enters the right atrium of the heart until it enters the left atrium. What is the circuit called? ...
... from the time it enters the right atrium of the heart until it enters the left atrium. What is the circuit called? ...
Valorisation - Maastricht University Research Portal
... The arrhythmia goes hand in hand with an important increase in mortality and morbidity; AF is associated with a doubled death rate (independent of other mortality predictors), an increased stroke risk (approximately every ęfth stroke is due to AF), frequent hospitalizations, a reduced quality of lif ...
... The arrhythmia goes hand in hand with an important increase in mortality and morbidity; AF is associated with a doubled death rate (independent of other mortality predictors), an increased stroke risk (approximately every ęfth stroke is due to AF), frequent hospitalizations, a reduced quality of lif ...
File
... o Located in the right atrium and is the Pacemaker of the heart o Specialized muscle cells o Automatically generate electrical impulses Electrical impulses are generated through polarization and depolarization o “speedy” route: SA NODE AV NODE bundle of His Purkinje fibers Commonly known as the ...
... o Located in the right atrium and is the Pacemaker of the heart o Specialized muscle cells o Automatically generate electrical impulses Electrical impulses are generated through polarization and depolarization o “speedy” route: SA NODE AV NODE bundle of His Purkinje fibers Commonly known as the ...
Postgraduate Research Presentation
... Antzelevitch C. T peak-Tend interval as an index of transmural dispersion of repolarization. Eur ...
... Antzelevitch C. T peak-Tend interval as an index of transmural dispersion of repolarization. Eur ...
Electrical System Of The Heart
... 2. The SA node sends out an electrical stimulus that causes the atria to contract. When this stimulus reaches the AV node, it is passed through the bundle of His and the Purkinje fibres. The stimulus causes the ventricles to contract, starting from the apex and then upward, which forces blood toward ...
... 2. The SA node sends out an electrical stimulus that causes the atria to contract. When this stimulus reaches the AV node, it is passed through the bundle of His and the Purkinje fibres. The stimulus causes the ventricles to contract, starting from the apex and then upward, which forces blood toward ...
Cardio II
... the T-tubule that lets calcium in when stimulated by an action potential d. Intracellular Ca++ concentration - It is the rise in intracellular Ca (From the SR) that causes muscle cell contraction e. Sarcoplasmic reticulum - Store of intracellular calcium f. The SERCA transporter - ATP pump to get Ca ...
... the T-tubule that lets calcium in when stimulated by an action potential d. Intracellular Ca++ concentration - It is the rise in intracellular Ca (From the SR) that causes muscle cell contraction e. Sarcoplasmic reticulum - Store of intracellular calcium f. The SERCA transporter - ATP pump to get Ca ...
Evaluation and Management of Heart Failure in Patients with
... (despite treatment with an ACEI and a b-blocker) with HFrEF and left ventricular ejection fraction (LVEF) ≤35%, to reduce mortality and HF hospitalization.12,13 Caution should be exercised when MRAs are used in patients with impaired renal function and in those with serum potassium levels ≥5.0 mmol/ ...
... (despite treatment with an ACEI and a b-blocker) with HFrEF and left ventricular ejection fraction (LVEF) ≤35%, to reduce mortality and HF hospitalization.12,13 Caution should be exercised when MRAs are used in patients with impaired renal function and in those with serum potassium levels ≥5.0 mmol/ ...
A1987K475500001
... oxygen, and drugs can be precisely controlled, and the cells receive these compounds from their own capillary beds. This preparation has proven to be a highly useful tool for studies on alterations2 of mechanical function of the heart, the relation between mechanical function and metabolism,3’4 and ...
... oxygen, and drugs can be precisely controlled, and the cells receive these compounds from their own capillary beds. This preparation has proven to be a highly useful tool for studies on alterations2 of mechanical function of the heart, the relation between mechanical function and metabolism,3’4 and ...
Myocardial infarction
... • Identify people at risk • Know pathophysiology of MI • Know the sign & symptom • Learn the way that diagnoses MI • Identify treatment that given to person who has MI ...
... • Identify people at risk • Know pathophysiology of MI • Know the sign & symptom • Learn the way that diagnoses MI • Identify treatment that given to person who has MI ...
Supplemental Content
... infarction, ICD implantation should be considered in the patient with cardiomyopathy if : LVEF is < 35% despite maximal medical therapy at least 40 days following myocardial infarction. Recurrent MI and cardiac rupture are common causes of death during 40 days post MI and not be prevented by ICD ...
... infarction, ICD implantation should be considered in the patient with cardiomyopathy if : LVEF is < 35% despite maximal medical therapy at least 40 days following myocardial infarction. Recurrent MI and cardiac rupture are common causes of death during 40 days post MI and not be prevented by ICD ...
Electrical Conduction PPT
... conduction pathway (normal rate 20 – 40 BPM) spread electrical impulses to all of the muscle tissue in the ventricles ventricles then contract http://www.nhlbi.nih.gov /health/healthtopics/topics/hhw/electric al.html ...
... conduction pathway (normal rate 20 – 40 BPM) spread electrical impulses to all of the muscle tissue in the ventricles ventricles then contract http://www.nhlbi.nih.gov /health/healthtopics/topics/hhw/electric al.html ...
Complex Heart Failure – Inpatient and Outpatient Management
... Effect of ARB vs placebo derived from CHARM-Alternative trial Effect of ACE inhibitor vs placebo derived from SOLVD-Treatment trial Effect of LCZ696 vs ACE inhibitor derived from PARADIGM-HF trial ...
... Effect of ARB vs placebo derived from CHARM-Alternative trial Effect of ACE inhibitor vs placebo derived from SOLVD-Treatment trial Effect of LCZ696 vs ACE inhibitor derived from PARADIGM-HF trial ...
Cardiovascular toxicity Cardiac Structure The cardiovascular system
... time required for all ventricular cells to be activated ( i.e the intraventricular conduction time). The QT interval reflects the duration of the ventricular action potential. Cardiac Output Normal cardiac output at rest is approximately 5 L/min in an average adult human, and that value may increas ...
... time required for all ventricular cells to be activated ( i.e the intraventricular conduction time). The QT interval reflects the duration of the ventricular action potential. Cardiac Output Normal cardiac output at rest is approximately 5 L/min in an average adult human, and that value may increas ...
Health - SynCardia
... then they are a candidate for an implant.” Traditionally, there have been two types of electrical implants. “Pacemakers are meant for patients who have a slow heartbeat, a dysfunction of the body’s own pacemaker,” notes Bhatia. The small device is implanted below the collarbone and connected to the ...
... then they are a candidate for an implant.” Traditionally, there have been two types of electrical implants. “Pacemakers are meant for patients who have a slow heartbeat, a dysfunction of the body’s own pacemaker,” notes Bhatia. The small device is implanted below the collarbone and connected to the ...
Apical Ballooning Syndrome: An Important Differential Diagnosis of
... ABS, and all 4 criteria must be present. The Table illustrates a modified version of the criteria that incorporates contemporary knowledge about the syndrome. 4 The diagnosis of ABS is most likely to be made at institutions where primary percutaneous coronary intervention and an early invasive strat ...
... ABS, and all 4 criteria must be present. The Table illustrates a modified version of the criteria that incorporates contemporary knowledge about the syndrome. 4 The diagnosis of ABS is most likely to be made at institutions where primary percutaneous coronary intervention and an early invasive strat ...
VT in abnormal heart DR SANMATH
... Dependent on achievement of critical conduction delay in HPS following VES. At longer coupling intervals, retrograde conduction occurs through RB. At shorter coupling intervals, retrograde block occurs in RB. Retrograde conduction occurs via LB causing long V2-H2 interval. Further shortening of ...
... Dependent on achievement of critical conduction delay in HPS following VES. At longer coupling intervals, retrograde conduction occurs through RB. At shorter coupling intervals, retrograde block occurs in RB. Retrograde conduction occurs via LB causing long V2-H2 interval. Further shortening of ...
Statin therapy - Gastaldi Congressi
... Atorvastatin but not rosuvastatin restores ANS balance in non-ischemic DCM ...
... Atorvastatin but not rosuvastatin restores ANS balance in non-ischemic DCM ...
Cardiac contractility modulation
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Cardiac contractility modulation (CCM) is a treatment for patients with moderate to severe left ventricular systolic heart failure (NYHA class II–IV). The short- and long-term use of this therapy enhances both the strength of ventricular contraction and the heart’s pumping capacity. The CCM mechanism is based on stimulation of the cardiac muscle by non-excitatory electrical signals (NES). CCM treatment is delivered by a pacemaker-like device that applies the NES, adjusted to and synchronized with the electrical action in the cardiac cycle.In CCM therapy, electrical stimulation is applied to the cardiac muscle during the absolute refractory period. In this phase of the cardiac cycle, electrical signals cannot trigger new cardiac muscle contractions, hence this type of stimulation is known as a non-excitatory stimulation. However, the electrical CCM signals increase the influx of calcium ions into the cardiac muscle cells (cardiomyocytes). In contrast to other electrical stimulation treatments for heart failure, such as pacemaker therapy or implantable cardioverter defibrillators (ICD), CCM does not affect the cardiac rhythm directly. Rather, the aim is to enhance the heart’s natural contraction (the native cardiac contractility) sustainably over long periods of time. Furthermore, unlike most interventions that increase cardiac contractility, CCM is not associated with an unfavorable increase in oxygen demand by the heart (measured in terms of Myocardial Oxygen Consumption or MVO2). This may be explained by the beneficial effect CCM has in improving cardiac efficiency. A meta-analysis in 2014 and an overview of device-based treatment options in heart failure in 2013 concluded that CCM treatment is safe, that it is generally beneficial to patients and that CCM treatment increases the exercise tolerance (ET) and quality of life (QoL) of patients. Furthermore, preliminary long-term survival data shows that CCM is associated with lower long-term mortality in heart failure patients when compared with expected rates among similar patients not treated with CCM.