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Ventricular Arrhythmias in Doberman Pinschers
Ventricular Arrhythmias in Doberman Pinschers

... treat. The same drug that causes proarrhythmia in one person may not in another person. Another important finding of all of these studies, in people, is that no drug used to treat ventricular tachycardia has been shown to prevent sudden death, even if it improves symptoms of syncope or fainting and ...
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Chapter 7 Body Systems - Silver Cross Emergency Medical Services
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Perioperative Management of DORV Case
Perioperative Management of DORV Case

... ultrasound guidance. Because these patients may be undergoing a redo sternotomy and/or complex repair with multiple suture lines, it is important to have venous access which will allow for rapid transfusion of blood products. Maintenance of anesthesia is with a balanced technique consisting of fenta ...
Congenital cardiac defect in a pygmy goat (Capra hircus)
Congenital cardiac defect in a pygmy goat (Capra hircus)

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Biomarkers in cardiac diseases
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... relaxation rates, and central venous pressure. Additionally, there were no significant differences found in electrocardiograms from fumonisin-treated pigs. Discussion The major findings of these studies were that fumonisin decreases the cardiac contractility of pigs (reduced dP/dtmax) without effect ...
Congestive Cardiac Failure Pathophysiology Precipitating Causes
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Slide () - AccessAnesthesiology
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46. Anatomy of the heart
46. Anatomy of the heart

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PowerPoint Presentation - No Slide Title
PowerPoint Presentation - No Slide Title

... a. A-V valve closes - partially extends into atrium b. papillary muscles contract pulling on chordae tendineae reducing valve extension back into atria. c. A-V valve remains closed as atria refills - pressure increases d. ventricle pressure increases opening semi-lunar valves e. Blood leaves ventric ...
Congestive Heart Failure - Missouri Alliance for Home Care
Congestive Heart Failure - Missouri Alliance for Home Care

... May feel tired for up to 6 weeks while titrating to final dose Takes 3 full months of therapy to begin seeing the positive i i benefits b fi Using a combination beta/alpha blocker may decrease the negative effects early in therapy by decreasing afterload (Coreg) Inform patients that this is a longlo ...
14-2
14-2

... • Blood pressure in aorta is 120mm Hg • Blood pressure in pulmonary trunk is 30mm Hg • Differences in ventricle wall thickness allows heart to push the same amount of blood with more force from the left ventricle • The volume of blood ejected from each ventricle is 70ml (stroke volume) • Why do both ...
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Hypertrophic cardiomyopathy



Hypertrophic cardiomyopathy (HCM) is a primary disease of the myocardium (the muscle of the heart) in which a portion of the myocardium is hypertrophied (thickened) without any obvious cause, creating functional impairment of the cardiac muscle. It is a leading cause of sudden cardiac death in young athletes.The occurrence of hypertrophic cardiomyopathy is a significant cause of sudden unexpected cardiac death in any age group and as a cause of disabling cardiac symptoms. Younger people are likely to have a more severe form of hypertrophic cardiomyopathy.HCM is frequently asymptomatic until sudden cardiac death, and for this reason some suggest routinely screening certain populations for this disease.A cardiomyopathy is a disease that affects the muscle of the heart. With HCM, the myocytes (cardiac contractile cells) in the heart increase in size, which results in the thickening of the heart muscle. In addition, the normal alignment of muscle cells is disrupted, a phenomenon known as myocardial disarray. HCM also causes disruptions of the electrical functions of the heart. HCM is most commonly due to a mutation in one of nine sarcomeric genes that results in a mutated protein in the sarcomere, the primary component of the myocyte (the muscle cell of the heart). These are predominantly single-point missense mutations in the genes for beta-myosin heavy chain (MHC), myosin-binding protein C, cardiac troponinT, or tropomyosin. These mutations cause myofibril and myocyte structural abnormalities and possible deficiencies in force generation. Not to be confused with dilated cardiomyopathy or any other cardiomyopathy.While most literature so far focuses on European, American, and Japanese populations, HCM appears in all ethnic groups. The prevalence of HCM is about 0.2% to 0.5% of the general population.
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