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Diagnosis of arrhythmogenic right ventricular
Diagnosis of arrhythmogenic right ventricular

... predispose them to perforation, a risk that becomes even greater be possible to avoid further diagnostic studies that can be both when considering that multiple ICD lead replacements may invasive and expensive. For example, right ventricular biopsy be necessary, given the relatively young age of ons ...
PATHOLOGY OF THE CARDIOVASCULAR SYSTEM
PATHOLOGY OF THE CARDIOVASCULAR SYSTEM

... Cardiac muscle has a greater liability because of its continual activity and dependence on aerobic glycolysis. The papillary muscles of the left ventricle seem to be most sensitive believed to be due to the vascular supply of the heart. Myocardial cells are considered to be a permanent cell populati ...
Atrial Septal Defect
Atrial Septal Defect

... • The atrial septum is thin, particularly in the region of the fossa ovalis. • From an apical view, the septum is parallel to the ultrasound beam, resulting in dropout. • Evaluate surrounding structures including PVs, the SVC, IVC, and CS. ...
Regulation of stroke volume
Regulation of stroke volume

... the left ventricle ejects a larger volume of blood with each systole than the right ventricle. the intrinsic rate of the heart's pacemaker is 100 beats/min. cardiac output increases with increased heart rate. stroke volume increases with increased venous return. ...
Papillary muscle rupture following myocardial
Papillary muscle rupture following myocardial

... and a transient loss of consciousness. A diagnosis of acute myocardial infarction was made. The electrocardiogram (ECG) revealed atrial fibrillation and changes of acute inferolateral myocardial infarction (Fig. la). The patient recovered and was discharged after six weeks. No cardiac murmurs were h ...
Low-Flow, Low-Gradient Aortic Stenosis With Preserved
Low-Flow, Low-Gradient Aortic Stenosis With Preserved

... function and outcome even in this subgroup and therefore might be considered following current guidelines (IIb recommendation) (1,5). The controversial new entity of “paradoxical low-flow, low-gradient severe AS.” Since the group of Pibarot and Dusmenil (6) published for the first time the new entit ...
Historical Perspectives of Cardiac Electrophysiology
Historical Perspectives of Cardiac Electrophysiology

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- Journal of the Saudi Heart Association
- Journal of the Saudi Heart Association

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International Cardiovascular Research Journal
International Cardiovascular Research Journal

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... • Find the extent of a coronary artery blockage • Provide a prognosis for patients who have suffered a myocardial infarct • Evaluate the effectiveness of cardiac procedures • Ascertain the cause of chest pain. Ideally, exercise is used to stress the heart as this is the natural way to increase myoca ...
Document
Document

... Foramen Primum ...
The Systolic Murmur—Benign or Serious?
The Systolic Murmur—Benign or Serious?

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Cardiac Anatomy for Radiology
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RIGHT ATRIAL PRESSURE AS A MEASURE OF VENTRICULAR

... Due to their close apposition to the heart, the chestwall, the lungs and the pericardium constrain the heart, limit diastolic filling, and thus limit cardiac output. Experimentally, ventricular constraint can be quantified by measuring pericardial pressure using a balloon transducer but, as yet, no ...
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Mechanical Properties of the Heart Contractility Cardiac Cycle

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Contractile function of myocardium and pumping function
Contractile function of myocardium and pumping function

... the pressure decreased. These events become evident as the first moderately negative wave. Thereafter the atrial pressure increases in direct proportion to the amount of blood flowed into the atrii which is recorded as the third positive wave. At the onset of diastole the AV valves open and the blood ...
Double Outlet Right Ventricle (DORV)
Double Outlet Right Ventricle (DORV)

... muscle. Hypertrophic cardiomyopathy (also known as idiopathic hypertrophic subaortic stenosis, or IHSS, or as asymmetric septal hypertrophy or ASH) can occur in families. With these conditions, the left ventricular muscle, especially the septal wall separating the right and the left ventricles, beco ...
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The Evolution of ICD Therapy:

... The degree that the pacing system “sees” or senses signals, controlled by the sensitivity setting which is graduated in millivolts (mV) Sensitivity (mV) 5 (mV) ...
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When to Start Treatment in the Asymptomatic Patient with LQT? Is

... So in LQT patients with a greater risk of sudden cardiac death than WPW, variable expressivity, increased heterogeneity, maybe more parental and familial anxiety regarding the condition- why would I not treat everyone? ...
Should Moderate or Greater Mitral Regurgitation Be Repaired in All
Should Moderate or Greater Mitral Regurgitation Be Repaired in All

... Without question, any heart failure patient for whom mitral repair is being considered for functional MR should be optimally medically managed with ␤-blockers and angiotensin-converting enzyme inhibitors. Optimal medical management is the cornerstone of modern heart failure therapy. In addition to p ...
06. Heart failure
06. Heart failure

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Role of serum cardiac troponin T in the diagnosis of - Heart
Role of serum cardiac troponin T in the diagnosis of - Heart

... with carditis had heart failure, which probably indicates only a slight cardiac involvement, and may also explain the low serum concentrations of cTnT. Our results are in agreement with the study of Williams and colleagues,6 who did not observe elevation of troponin values in patients with carditis. ...
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Hypertrophic cardiomyopathy



Hypertrophic cardiomyopathy (HCM) is a primary disease of the myocardium (the muscle of the heart) in which a portion of the myocardium is hypertrophied (thickened) without any obvious cause, creating functional impairment of the cardiac muscle. It is a leading cause of sudden cardiac death in young athletes.The occurrence of hypertrophic cardiomyopathy is a significant cause of sudden unexpected cardiac death in any age group and as a cause of disabling cardiac symptoms. Younger people are likely to have a more severe form of hypertrophic cardiomyopathy.HCM is frequently asymptomatic until sudden cardiac death, and for this reason some suggest routinely screening certain populations for this disease.A cardiomyopathy is a disease that affects the muscle of the heart. With HCM, the myocytes (cardiac contractile cells) in the heart increase in size, which results in the thickening of the heart muscle. In addition, the normal alignment of muscle cells is disrupted, a phenomenon known as myocardial disarray. HCM also causes disruptions of the electrical functions of the heart. HCM is most commonly due to a mutation in one of nine sarcomeric genes that results in a mutated protein in the sarcomere, the primary component of the myocyte (the muscle cell of the heart). These are predominantly single-point missense mutations in the genes for beta-myosin heavy chain (MHC), myosin-binding protein C, cardiac troponinT, or tropomyosin. These mutations cause myofibril and myocyte structural abnormalities and possible deficiencies in force generation. Not to be confused with dilated cardiomyopathy or any other cardiomyopathy.While most literature so far focuses on European, American, and Japanese populations, HCM appears in all ethnic groups. The prevalence of HCM is about 0.2% to 0.5% of the general population.
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