Septal to Posterior Wall Motion Delay Fails to Predict
... dyssynchrony parameters and CRT benefit in any category. Reproducibility of SPWMD measurements. There was poor inter-observer precision of SPWMD (observer 1 with the primary observer ⫽ 141 ms, n ⫽ 37; observer 2 with the primary observer ⫽ 194 ms, n ⫽ 47). For example, the within-subjects standard d ...
... dyssynchrony parameters and CRT benefit in any category. Reproducibility of SPWMD measurements. There was poor inter-observer precision of SPWMD (observer 1 with the primary observer ⫽ 141 ms, n ⫽ 37; observer 2 with the primary observer ⫽ 194 ms, n ⫽ 47). For example, the within-subjects standard d ...
Rx for Success - Atrial and Ventricular Septal Defects(052)
... two atria and the two ventricles. Congenital holes in this septum allow blood to flow (or shunt) between the right and left sides of the heart. This abnormal flow of blood causes heart enlargement and failure. If left unchecked, it can lead to permanent heart and lung damage. An atrial septal defect ...
... two atria and the two ventricles. Congenital holes in this septum allow blood to flow (or shunt) between the right and left sides of the heart. This abnormal flow of blood causes heart enlargement and failure. If left unchecked, it can lead to permanent heart and lung damage. An atrial septal defect ...
BASIC CARDIAC DEFECTS - Ohio Association of Physician Assistants
... Incidence: 0.1% to 0.3% of population Gender ratio: male to female 2:1 Pre-excitation ...
... Incidence: 0.1% to 0.3% of population Gender ratio: male to female 2:1 Pre-excitation ...
Glidepath - Syncope
... were randomized trials, observational studies, cohort studies or case series of >10 patients. In addition, footnotes 2,5, and 7 (below) are based on these papers. Linzer M, Yang EH, Estes M 3rd, Wang, P, et al. Diagnosing Syncope Part 1: Value of history, physical examination, and electrocardiograph ...
... were randomized trials, observational studies, cohort studies or case series of >10 patients. In addition, footnotes 2,5, and 7 (below) are based on these papers. Linzer M, Yang EH, Estes M 3rd, Wang, P, et al. Diagnosing Syncope Part 1: Value of history, physical examination, and electrocardiograph ...
PhD THESIS
... fall in systolic function is not related to the increased myocardial afterload (endsystolic stress), which is in fact reduced when LV mass is inappropriate, while it is substantially associated with a depressed wall mechanics indicating reduced myocardial contractility. These findings substantially ...
... fall in systolic function is not related to the increased myocardial afterload (endsystolic stress), which is in fact reduced when LV mass is inappropriate, while it is substantially associated with a depressed wall mechanics indicating reduced myocardial contractility. These findings substantially ...
Valvular Heart Disease - Nursing PowerPoint Presentations
... occurrence of rheumatic fever to onset of symptoms. • Once symptoms develop, there is a further 10 years before symptoms become disabling. • Once significant limiting symptoms occur, the 10year survival rate is 5-15%. • When there is severe pulmonary hypertension, mean survival falls to < 3 years. • ...
... occurrence of rheumatic fever to onset of symptoms. • Once symptoms develop, there is a further 10 years before symptoms become disabling. • Once significant limiting symptoms occur, the 10year survival rate is 5-15%. • When there is severe pulmonary hypertension, mean survival falls to < 3 years. • ...
Effects of the Purkinje system and cardiac geometry on
... amelioration of functional capacity, inverse remodeling, and reduction of morbidity and mortality.1,5,13 Despite these benefits, clinical trials have demonstrated that around one third of patients do not respond favorably to CRT using standard clinical selection criteria. Sub-optimal resynchronizatio ...
... amelioration of functional capacity, inverse remodeling, and reduction of morbidity and mortality.1,5,13 Despite these benefits, clinical trials have demonstrated that around one third of patients do not respond favorably to CRT using standard clinical selection criteria. Sub-optimal resynchronizatio ...
Cardiac Physiology
... 4. snap shut 5. smaller opening increases flow velocity 6. subject to mechanical abrasion v. abnormalities 1. murmur – abnormal heart sound, usu resulting from left valve defect (can be right) 2. stenosis - failure of valves to fully open (narrowing) 3. insufficiency/regurgitant - failure to seal up ...
... 4. snap shut 5. smaller opening increases flow velocity 6. subject to mechanical abrasion v. abnormalities 1. murmur – abnormal heart sound, usu resulting from left valve defect (can be right) 2. stenosis - failure of valves to fully open (narrowing) 3. insufficiency/regurgitant - failure to seal up ...
Hemodynamic Measurements of Left Ventricle Function Post
... to select for analysis only those samples of IVC occlusion that were performed by similar technique including method of occlusion and vena cava location of preload reduction. Additionally, parameter such as ESPVR, EDPVR, time varying elastance (Emax), PRSW, dP/dtmax vs. EDV are all declining post-MI ...
... to select for analysis only those samples of IVC occlusion that were performed by similar technique including method of occlusion and vena cava location of preload reduction. Additionally, parameter such as ESPVR, EDPVR, time varying elastance (Emax), PRSW, dP/dtmax vs. EDV are all declining post-MI ...
Sparman, Manchineel Poisoning Bradyarrhythmia.qxp
... was placed on the treadmill and reached Stage III; she was unable to reach 85% of her maximum predicted heart rate. She achieved 10.1 METS on the treadmill. The test was terminated secondary to fatigue and completion of protocol. There were no arrhythmias or electrocardiogram changes noted. ...
... was placed on the treadmill and reached Stage III; she was unable to reach 85% of her maximum predicted heart rate. She achieved 10.1 METS on the treadmill. The test was terminated secondary to fatigue and completion of protocol. There were no arrhythmias or electrocardiogram changes noted. ...
IABP
... 1980 – Development of percutaneous catheter introduction Moulopoulos et al, Am Heart J 1962; Goldberg et al, NEJM 1999; Antman et al, Circulation 2008 Buckley et al, Circ 1973; Housman et al, JAMA 1973; Bergman et al, A. Th. Surg 1980; Subramanian et al, Circ 1980 ...
... 1980 – Development of percutaneous catheter introduction Moulopoulos et al, Am Heart J 1962; Goldberg et al, NEJM 1999; Antman et al, Circulation 2008 Buckley et al, Circ 1973; Housman et al, JAMA 1973; Bergman et al, A. Th. Surg 1980; Subramanian et al, Circ 1980 ...
Clarifying the Physiological Processes behind Foucault Cardiogram
... considered to be a mechanism of the FouCG origination. The FouCG waveform, recorded with a single-coil sensor positioned with its centre slightly below the apexbeat, is promisingly similar [5] to ventricular volume curves typically found in medical books. To clarify the physiological processes behin ...
... considered to be a mechanism of the FouCG origination. The FouCG waveform, recorded with a single-coil sensor positioned with its centre slightly below the apexbeat, is promisingly similar [5] to ventricular volume curves typically found in medical books. To clarify the physiological processes behin ...
File
... Ventricular ectopic are usually incapable of exciting the bundle of His and retrograde conduction to the atria therefore dose not occur (therefore the atrial rate remain unaltered). Furthermore, ventricular ectopic beats do not interrupt the regular discharge of the SA node, whereas atrial pre-matur ...
... Ventricular ectopic are usually incapable of exciting the bundle of His and retrograde conduction to the atria therefore dose not occur (therefore the atrial rate remain unaltered). Furthermore, ventricular ectopic beats do not interrupt the regular discharge of the SA node, whereas atrial pre-matur ...
Left Ventricular Response to Isometric Exercise
... reflex is initiated by a metabolite activation of receptors, reflex efferents have no direct path to the denervated heart. Any increases in heart rate and contractility are mediated by circulating catecholamines, while a-receptor-mediated increases in peripheral resistance still respond to the neura ...
... reflex is initiated by a metabolite activation of receptors, reflex efferents have no direct path to the denervated heart. Any increases in heart rate and contractility are mediated by circulating catecholamines, while a-receptor-mediated increases in peripheral resistance still respond to the neura ...
ardiac output, heart rate, stroke volume and systolic blood pressure is
... occurring tachycardia and the higher mean arterial blood pressure, all result in an augmentation of the cardiac work. Thus, long-term athletic training primarily causes changes of the left ventricular morphology represented by large diastolic cavity dimension and high values of wall thickness. These ...
... occurring tachycardia and the higher mean arterial blood pressure, all result in an augmentation of the cardiac work. Thus, long-term athletic training primarily causes changes of the left ventricular morphology represented by large diastolic cavity dimension and high values of wall thickness. These ...
A criss-cross heart
... no cyanosis. Echocardiography demonstrated crisscross morphology as well as situs solitus levocardia, congenitally corrected transposition of great arteries (ccTGA) with right anterior aorta, straddling mitral valve, large inlet ventricular septal defect (VSD), atrial septal defect (ASD) and valvula ...
... no cyanosis. Echocardiography demonstrated crisscross morphology as well as situs solitus levocardia, congenitally corrected transposition of great arteries (ccTGA) with right anterior aorta, straddling mitral valve, large inlet ventricular septal defect (VSD), atrial septal defect (ASD) and valvula ...
Chapter 19
... e. tachycardia - HIGHER than normal resting heart rate (over 100); may lead to fibrillation f. bradycardia - LOWER than normal resting heart rate (below 60); parasympathetic drug side effects; physical conditioning; sign of pathology in non-healthy patient ...
... e. tachycardia - HIGHER than normal resting heart rate (over 100); may lead to fibrillation f. bradycardia - LOWER than normal resting heart rate (below 60); parasympathetic drug side effects; physical conditioning; sign of pathology in non-healthy patient ...
11 Heart - bloodhounds Incorporated
... Hold the AV valves during diastole Hold the right and left ventricles together Transmit the electrical impulse form the atria to the ventricles Contract when the ventricles contract ...
... Hold the AV valves during diastole Hold the right and left ventricles together Transmit the electrical impulse form the atria to the ventricles Contract when the ventricles contract ...
Cardiac Auscultation The Ohio State University
... auscultatory exam is expedient and cost effective. When completed by an experienced clinician, auscultation carries a high predictive value for identification of many—though not all—serious heart diseases. Definitive diagnosis may be possible by auscultation, as when classic murmurs of patent ductus ...
... auscultatory exam is expedient and cost effective. When completed by an experienced clinician, auscultation carries a high predictive value for identification of many—though not all—serious heart diseases. Definitive diagnosis may be possible by auscultation, as when classic murmurs of patent ductus ...
Response of the fetal heart to changes in load
... ensues? Responses to volume and pressure loading in infancy and childhood include the re-expression and redistribution of substances seen usually only during cardiac modelling,13 and suggest a deregulation of cytoskeletal and stress protein expression under conditions of varied haemodynamic imbalanc ...
... ensues? Responses to volume and pressure loading in infancy and childhood include the re-expression and redistribution of substances seen usually only during cardiac modelling,13 and suggest a deregulation of cytoskeletal and stress protein expression under conditions of varied haemodynamic imbalanc ...
VitalSim - Laerdal Medical
... Vital signs for manikins and skill trainers The VitalSim Vital Signs Simulator is the heart of Laerdal s VitalSim product line. When connected to VitalSim enabled manikins and skills trainers, VitalSim simulates ECGs, heart sounds, fetal heart tones, breath sounds, bowel sounds, blood pressure and p ...
... Vital signs for manikins and skill trainers The VitalSim Vital Signs Simulator is the heart of Laerdal s VitalSim product line. When connected to VitalSim enabled manikins and skills trainers, VitalSim simulates ECGs, heart sounds, fetal heart tones, breath sounds, bowel sounds, blood pressure and p ...
Assessment of the pressure–volume relationship of the single
... contraction, ventricular emptying, isovolumic relaxation and ventricular-filling phases. Although all four phases are present in the PA loop, the timing of the phases is considerably different than in the mammalian ventricle. In the mammalian four-chambered heart, approximately 33% of the cardiac cy ...
... contraction, ventricular emptying, isovolumic relaxation and ventricular-filling phases. Although all four phases are present in the PA loop, the timing of the phases is considerably different than in the mammalian ventricle. In the mammalian four-chambered heart, approximately 33% of the cardiac cy ...
PDA - Jantung Sehat
... • The factors that determine the nature of clinical features are the same as in VSD and ASD i.e the size of the defect, the presence of PH, the development of Eisenmenger’s syndrome • A patent PDA is more likely in babies born at high altitude, probably due to low atmospheric oxygen concentration; i ...
... • The factors that determine the nature of clinical features are the same as in VSD and ASD i.e the size of the defect, the presence of PH, the development of Eisenmenger’s syndrome • A patent PDA is more likely in babies born at high altitude, probably due to low atmospheric oxygen concentration; i ...
Cardiac Muscle and Mechanics I
... -left ventricle depolarizes later, THUS shift in axis of depolarization to the left Left Axis Deviation is due to: 1. left ventricular hypertrophy 2. left ventricular bundle block He didn’t go over them, but he said to be familiar with the structures from the first slide in this lecture, also found ...
... -left ventricle depolarizes later, THUS shift in axis of depolarization to the left Left Axis Deviation is due to: 1. left ventricular hypertrophy 2. left ventricular bundle block He didn’t go over them, but he said to be familiar with the structures from the first slide in this lecture, also found ...
Overview of cardiac adverse drug reactions reported in
... patients), use of cardiac medication like ACE inhibitors, ß-Blockers or CalciumBlockers (5) and in four reports no information was present. One patient suffered from a non fatal cardiac arrest two years of after start of rosiglitazone. The other cases concerned fluid retention without further cardi ...
... patients), use of cardiac medication like ACE inhibitors, ß-Blockers or CalciumBlockers (5) and in four reports no information was present. One patient suffered from a non fatal cardiac arrest two years of after start of rosiglitazone. The other cases concerned fluid retention without further cardi ...
Hypertrophic cardiomyopathy
Hypertrophic cardiomyopathy (HCM) is a primary disease of the myocardium (the muscle of the heart) in which a portion of the myocardium is hypertrophied (thickened) without any obvious cause, creating functional impairment of the cardiac muscle. It is a leading cause of sudden cardiac death in young athletes.The occurrence of hypertrophic cardiomyopathy is a significant cause of sudden unexpected cardiac death in any age group and as a cause of disabling cardiac symptoms. Younger people are likely to have a more severe form of hypertrophic cardiomyopathy.HCM is frequently asymptomatic until sudden cardiac death, and for this reason some suggest routinely screening certain populations for this disease.A cardiomyopathy is a disease that affects the muscle of the heart. With HCM, the myocytes (cardiac contractile cells) in the heart increase in size, which results in the thickening of the heart muscle. In addition, the normal alignment of muscle cells is disrupted, a phenomenon known as myocardial disarray. HCM also causes disruptions of the electrical functions of the heart. HCM is most commonly due to a mutation in one of nine sarcomeric genes that results in a mutated protein in the sarcomere, the primary component of the myocyte (the muscle cell of the heart). These are predominantly single-point missense mutations in the genes for beta-myosin heavy chain (MHC), myosin-binding protein C, cardiac troponinT, or tropomyosin. These mutations cause myofibril and myocyte structural abnormalities and possible deficiencies in force generation. Not to be confused with dilated cardiomyopathy or any other cardiomyopathy.While most literature so far focuses on European, American, and Japanese populations, HCM appears in all ethnic groups. The prevalence of HCM is about 0.2% to 0.5% of the general population.