Download Slide ()

Survey
yes no Was this document useful for you?
   Thank you for your participation!

* Your assessment is very important for improving the workof artificial intelligence, which forms the content of this project

Document related concepts

Non-coding DNA wikipedia , lookup

Promoter (genetics) wikipedia , lookup

Transcriptional regulation wikipedia , lookup

RNA-Seq wikipedia , lookup

Community fingerprinting wikipedia , lookup

Gene desert wikipedia , lookup

Point mutation wikipedia , lookup

Endogenous retrovirus wikipedia , lookup

List of types of proteins wikipedia , lookup

Gene therapy of the human retina wikipedia , lookup

Clinical neurochemistry wikipedia , lookup

Molecular evolution wikipedia , lookup

Personalized medicine wikipedia , lookup

Silencer (genetics) wikipedia , lookup

Gene therapy wikipedia , lookup

Vectors in gene therapy wikipedia , lookup

Gene regulatory network wikipedia , lookup

X-inactivation wikipedia , lookup

Artificial gene synthesis wikipedia , lookup

Transcript
Proposed genetic rearrangement of chromosome 11 in a subset of sporadic parathyroid adenomas. An inversion of DNA sequence near the centromere
of chromosome 11 places the 5′-regulatory region of the PTH gene (also on chromosome 11) adjacent to the PRAD1 gene, whose product is involved in
cell cycle control. This places the PRAD1 gene under the control of PTH regulatory sequences, which would be predicted to be highly active in parathyroid
cells. (Redrawn, with permission, from Arnold A. Molecular genetics of parathyroid gland neoplasia. J Clin Endocrinol Metab. 1993;77:1109.)
Source: Disorders of the Parathyroids & Calcium & Phosphorus Metabolism, Pathophysiology of Disease: An Introduction to Clinical Medicine,
7e
Citation: Hammer GD, McPhee SJ. Pathophysiology of Disease: An Introduction to Clinical Medicine, 7e; 2013 Available at:
http://mhmedical.com/ Accessed: August 03, 2017
Copyright © 2017 McGraw-Hill Education. All rights reserved