Survey
* Your assessment is very important for improving the workof artificial intelligence, which forms the content of this project
* Your assessment is very important for improving the workof artificial intelligence, which forms the content of this project
Schmerber v. California wikipedia , lookup
Blood transfusion wikipedia , lookup
Blood donation wikipedia , lookup
Autotransfusion wikipedia , lookup
Jehovah's Witnesses and blood transfusions wikipedia , lookup
Hemolytic-uremic syndrome wikipedia , lookup
Men who have sex with men blood donor controversy wikipedia , lookup
Plateletpheresis wikipedia , lookup
Hemorheology wikipedia , lookup
ABO blood group system wikipedia , lookup
Chapter 14: Blood Part I Matching: 1) 2) 3) 4) 5) 6) 7) 8) 9) 10) 11) Blood and Blood Tests F C D J H B I E K A G Read the Diagram 1) 2 2) 1 3) 5 4) 2, 3 and 4 5) 2 6) 5 7) 2 8) 5 9) 2 and 4 10) 1 Matching: 1) 2) 3) 4) 5) 6) 7) 8) 9) 10) 11) 12) 13) 14) 15) Red Blood Cells, White Blood Cells and Platelets WBC RBC RBC P RBC RBC WBC WBC RBC RBC P WBC WBC WBC WBC 16) 17) 18) 19) 20) 21) 22) 23) 24) RBC P RBC P WBC WBC RBC RBC RBC Tell a Story: Polly Cythemia and Her Oxygen An elderly patient with emphysema was chronically hypoxic and developed an elevated hematocrit. Why, you ask? In response to the low tissue levels of oxygen, the kidneys secrete the hormone, erythropoietin. This hormone travels via the blood to the bone marrow, where it stimulates the formation of red blood cells. The increases amount of hemoglobin delivers more oxygen to the tissues. Chronic hypoxia, therefore, causes a secondary polycythemia and an increase in hematocrit. There are other clinical conditions that involve erythropoietin. Patients in renal failure develop anemia (a deficiency of red blood cells) because of the lack of erythropoietin. Patients receiving cancer chemotherapy receive Epoetin (an erythropoietin-like drug) because of a chemotherapy-induced myelosupression and anemia. Finally, athletes who train “at altitude” become mildly hypoxic, and therefore secrete erythropoietin; in response they too develop a secondary polycythemia. Labeling: Breakdown of the Red Blood Cell See the figure on page 260 of your textbook Matching: 1) 2) 3) 4) 5) 6) 7) 8) 9) 10) 11) 12) 13) 14) Hemostasis E A I D B F K L C H M G N J Labeling: Hemostasis See figure on page 264 of your textbook Tell a Story: Got a Clot Mr. Flea Bitis was admitted to the hospital with a diagnosis of deep vein thrombosis (DVT), a life-threatening condition associated with clot formation in the veins of the legs. He was given the drug heparin by intravenous (IV) infusion. This anticoagulant is an antithrombin agent. The drug-included removal of thrombin prevents the conversion of fibrinogen to fibrin threads (the clot). On the following day he was also given coumadin, an oral anticoagulant. This anticoagulant works by preventing the hepatic utilization of vitamin K in the synthesis of protothrombin. The goal of oral anticoagulant therapy is to cause hypoprothrombinemia; confirmed by prolonged prothrombin time, a lab test used to monitor therapy. The intravenous anticoagulant was discontinued within the first week; however, the oral anticoagulant was continued for several months until the thrombotic condition was resolved. Matching: 1) 2) 3) 4) 5) 6) 7) 8) 9) 10) 11) 12) 13) Blood Types C D D C C D D G E F H A B Tell a Story: The Yellow Baby! Ms. Billie Rubin (blood type A-) gave birth to Baby girl (type A+), a second daughter. Within 4 hours after birth, the baby appeared jaundiced; the jaundice intensified over the next 12 hours. What happened? Ms. B has type A (-) blood and antiRH antibodies, having been sensitized from the first pregnancy. Buring the second pregnancy, the maternal antibodies slipped across the placenta into the baby and attacked the Rh antigens of the baby’s red blood cells, thereby causing agglutination and hemolysis. The ruptured red blood cells release bilirubin causing hyperbilirubinemia and jaundice. This condition is called erythroblastosis fetalis. Part II Multiple Choice 1) C 2) B 3) A 4) D 5) 6) 7) 8) 9) 10) 11) 12) 13) 14) 15) 16) 17) 18) B C B D B A D C B C A D B C Case Study 1) C 2) A 3) C 4) C 5) C Part III Critical Thinking Questions 1) D 2) C 3) C