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Nursing Care of the Child with Gastrointestinal Disorders Ann Hearn RNC, MSN Fall 2009 Cleft Lip and Cleft Palate Failure of maxillary and median nasal processes to fuse during embryonic development Unilateral, bilateral, midline p h o t Treatment Surgical repair done ASAP Rule of 10 > 10#, 10 weeks, 10 HGB Multidisciplinary team Pre-op Goals • Prevent aspiration • Maintain nutrition • Provide emotional support to family Prevent Aspiration / Maintain Nutrition Breast feed – small cleft lip Bottle feed – special feeding devises – Special nipples – Enlarge cross cut hole Bubble frequently Hold upright ESSR Provide Emotional Support Assist with accepting of defect Teach proper feeding Point out positive attributes Encourage participation in care Explain surgical procedure Pre-op Teaching Remind parents that defect is operableshow photographs of corrected clefts Introduce cup, spoon feeding devices Explain elbow restraints Explain Logan Bow Post-Op Prevent trauma to suture line – Reduce pain & infection Cleanse suture lines as ordered Facilitate breathing Maintain nutrition Referral to appropriate team members Esophageal Atresia Failure of the esophagus to totally differentiate during uterine development. Assessment Findings Respiratory difficulties Drooling Coughing, choking, cyanosis Gastric distention - if fistula present Hx of ??? during pregnancy? – Polyhydramnios gastrointestinal obstruction fetus unable to swallow Management Early diagnosis – Ultrasound – Radiopaque catheter inserted in the esophagus to illuminate defect on X-ray Surgical repair – Thoracotomy and anastomosis Pre-Op Nursing Priority Maintain airway Prevent aspiration pneumonia Keep NPO- administer IV fluids – Elevate HOB 30 degrees – Suction PRN – Prophylactic antibiotics Post-Op Maintain nutrition – TPN – Gastrostomy Maintain airway – Prevent aspiration Monitor weigh, growth and development achievements Complications – GERS – Stricture formation Teaching Plan: Gastrostomy Tube Equipment Procedure Psychosocial needs Medication administration Stoma care Problem solving Gastroesophagial Reflux Disease (GERD) The cardiac sphincter and lower portion of the esophagus are weak, allowing regurgitation of gastric contents back into the esophagus. Assessment findings: Infant Regurgitation almost immediately after each feeding when the infant is laid down Excessive crying, irritability Failure to thrive (FTH) Complications: – aspiration pneumonia – apnea Assessment findings: Child Heartburn Abdominal pain Cough, recurrent pneumonia Dysphagia Diagnosis Assess pH of • < 7.0 esophageal • acidic secretions Baruim Swallow • Visualization of esophageal abnormalities Management & Nursing Care Nutritional needs – Small frequent feedings – Frequent burping Positioning – Prone flat or head elevated after feedings (not for sleep) Medications CPR instruction for parents/caregivers Surgery Nissen fundoplication Post Op Nursing Care Feedings Burping (bubbling) Positioning Airway Medications Medications H2 Histamine receptor antagonists – suppress gastric acid secretions – Zantac and Pepcid Proton-pump inhibitors – reduce gastric acid production – Prevacid and Prilosec Gastric emptying - increases – Reglan Antacids – neutralize gastric acidity – Gaviscon **be sure to study nursing implications and side effects Pyloric Stenosis Results when the circular area of the muscle surrounding the pylorus hypertrophies & obstructs gastric emptying. – Incidence: 3 in 1000 births – Possible genetic predisposition Pyloric Stenosis Narrowing of the pyloric spincter Delayed emptying of the stomach Assessment Vomiting - projectile Constant hunger and fussiness Distended upper abdomen Hypertrophied pylorus – olive shaped mass Visible peristaltic waves Diagnosis History and Physical Laboratory values X-ray or Ultrasound Surgery Fred Ramstedt procedurePyloromyotomy via laparoscopy Pre-op Hydration and electrolyte balance Weigh daily & I and O NG tube Support of parents Management and Nursing Care: Post-Op NPO until bowel function – Progressive feeds: Feeding begins with clear liquids containing glucose and electrolytes. Regime example: 8 hours NPO, 10cc sterile water feed X 2. Increase to 15cc X 2, progressing to ½ strength formula, then full strength formula. Observe and record the infant’s response to feeding. Position with head elevated Assess surgical site for infection - Antibiotics Analgesia Patient teaching Critical Thinking A 4 week old infant with a history of vomiting after feeding has been hospitalized with a tentative diagnosis of pyloric stenosis. Which of these actions is priority for the nurse? a. Begin an intravenous infusion b. Measure abdominal circumference c. Orient family to unit d. Weigh infant Gastroschisis & Omphalocele Abdominal Wall Defects Gastroschisis Herniation of abdominal viscera outside the abdominal cavity through a defect in the abdominal wall to the side of the umbilicus. Content not covered. Treatment and Nursing Care Pre-operatively – focus is on protection of the contents / sac. Cover with warm, sterile, saline-soaked dressings over the defect. May choose to replace the gut to the abdomen gradually over several weeks. May place silo or silastic material over gut until it returns to the abdomen. Surgery used to close defect. Gastroschisis Assessment- noted on ultrasound and obvious at birth Treatment - surgical repair in stages Nursing care: – monitor thermoregulation and loss of fluids – assess for ileus – maintain parenteral feeding – provide support to the parents Omphalocele Herniation of abdominal contents through the umbilical cord. Contents are covered by a translucent sac. Omphalocele Assessment- ultrasound and at birth Treatment - surgical repair in stages Nursing care- same as for Gastroschisis Intussuception Invagination of a section of the intestine, into the distal bowel that causes bowel obstruction. Results in inflamed bowel & bleeding – Leading to necrosis & perforation Intussuception Most commonly seen in infants 3-12 months Bowel “telescopes” within itself Intussuception: Clinical Manifestations Intermittent then constant pain Vomiting Abdominal distention Currant jelly-like stools Diarrhea Dehydration Serious complications: Ischemia, perforation & shock Volvulus A twisting of the bowel that leads to a bowel obstruction. Clinical Manifestations and Assessment Pain Vomiting (fecal material) Abdominal distention Stools Dehydration Serious complication: shock Diagnosis X-ray Abdominal ultrasound Therapeutic Intervention Hydrostatic reduction Laparoscopic Surgery Post-op Nursing Care: NPO- NG tube, IV Assess – V/S, pain Monitor stools Re-introduce food Appendicitis Inflammation of the lumen of the appendix at the end of the cecum which becomes quickly obstructed causing edema, necrosis and pain. Clinical Manifestations Abdominal pain – McBurney’s point Silent Abdomen Anorexia & nausea Diarrhea Elevated temperature IF PERFORATED: – Sudden pain relief – Fever – Dehydration Diagnosis History and Physical Ultrasound X-Ray Laboratory values – increased WBC 15,000 – 20,000 Management and Nursing Care: Pre-Op NPO IV Comfort measures Antibiotics Thermal therapy Elimination Patient education What is the most common symptom indicating that the appendix may have ruptured? Hirschsprung’s Disease Congenital disorder: absence of ganglia (nerve cells) in lower colon leading to an obstruction. Assessment Failure to pass meconium Vomiting Bowel assessment Breath Older child Diagnosis History & Physical Barium enema (X-ray) Rectal biopsy- absence of ganglionic cells in bowel mucosa Management Surgical intervention – Colostomy – Resection Nursing Care: Pre-op – Cleanse bowel – NPO – Patient/parent teaching Post-op – – – – NPO VS (no rectal temperatures) Assessment Patient/parent teaching Diarrhea/Gastroenteritis Severe A disturbance of the intestinal tract that alters motility and absorption, and accelerates the excretion of intestinal contents. Most infectious diarrheas in this country are caused by Rotovirus but could be C. difficele Clinical Manifestations Increase in peristalsis Large volume stools Increase in frequency of stools Nausea, vomiting, cramps Metabolic Acidosis: – Increased heart & resp. rate, decreased B/P, arrhythmias – Cold, clammy skin – Changes in CNS – stupor, lethergy Diagnosis Blood gases Stool O & P Stool culture Complications Dehydration Little fluid volume reserve in children Metabolic Acidosis Increase HR & RR, Decrease BP, Arrhythmias Hypovolemic Shock Priority Nursing Interventions Treat underlying cause Restore fluid & electrolyte balance Daily weights I&O Assess for dehydration Isolation protocol Monitor electrolytes/metabolic acidosis Skin care Oral Rehydration Critical Thinking Why is there an increase in incidence of diarrhea in lower socio-economic groups? Why is there and increase in young children? Celiac Disease The inability to digest gliadin which is a by-product of gluten breakdown. This results in the accumulation of the amino acid glutamine which is toxic to the mucosal cells in the intestines. Damage to the villi impairs the ability of the small intestines to absorb nutrients Signs and Symptoms The child with celiac disease commonly demonstrates failure to grow and wasting of extremities. The abdomen can appear large due to intestinal distension and malnutrition Complications: Hypocalcemia, osteomalacia, osteoporosis, depression. Celiac Disease Assessment - Growth pattern, GI pattern Failure to Thrive Treatment - Dietary restrictions Nursing Care - monitor for dehydration, encourage compliance with dietary restrictions, provide support groups for patient and caregiver Diagnostic Findings Measurement of fat content Duodenal or Jejunal biopsy Elevated IGA antibodies Treatment and Nursing Care Teach parents DIETARY REGULATIONS: NO ! Gluten Free Diet Wheat Rye Barley Disease specific support groups The End