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Headache/Vertigo in the ED Nicholas Cascone, PA-C Headache in the ED 4% of all ED visits are due to headache – 4% of these headache visits have serious or secondary pathology Objectives of evaluation: – Appropriately select patients for emergency investigation when critical secondary causes are present – Provide effective treatment for primary and benign secondary headaches Evaluation of Headache in ED History – Pattern – worst ever, first severe, steady worsening, differences from prior headaches – Onset – sudden headaches that begin during exertion – up to 25% of such HA are SAH – Associated symptoms – dizziness, nausea, confusion, LOC, fever, neck pain/stiffness, visual changes, seizure Evaluation of Headache in ED History (cont’d.) – Medical history – trauma, previous lumbar puncture, use of nitrates, MAOIs, exposure to toxics (e.g., CO) – Family history – migraines, SAH run in families Evaluation of Headache in ED Physical examination – Temperature, blood pressure – Palpate sinuses, temporal artery, temporomandibular joint – Eye exam for acute glaucoma, fundoscopy for signs of hypertension, papilledema – Thorough neurological exam Labs – CT scan, lumbar puncture if indicated Killer Headaches Subarachnoid hemorrhage – More common in women – Severe, constant occipitonuchal HA, “worst in my life” – Often presents suddenly, with vomiting and alteration of consciousness – History may indicate activities which raise blood pressure (e.g., intercourse, defecation, coughing) Killer Headaches Subarachnoid hemorrhage (cont’d) – Dx: plain CT 93% sensitivity within 24 h of onset – If CT nondiagnostic, LP Xanthochromia on spectrophotometry nearly 100% sensitive Naked-eye detection only 50% sensitive – Tx: angiogram and surgery consult, nimodipine, prohylactic phenytoin, antiemetics, decrease BP if elevated Killer Headaches Meningitis – Occipitonuchal headache with fever, meningeal signs, altered consciousness – Dx: immediate LP in pts without neurological signs, normal LOC and no papilledema If LP delayed and bacterial meningitis suspected, initiate empiric antibiotic tx Killer Headaches Subdural hematoma – History of remote trauma with headache – High risk patients: Anticoagulation Chronic alcoholics Elderly patients – If plain CT nondiagnostic, contrast CT or MRI – Tx: surgery consult Killer Headaches Brain tumor – Headache may be unilateral/bilateral, intermittent/continuous – Classic presentation is headache with vomiting, worse upon arising – Reliable pt with no neuro findings and no papilledema can follow-up as outpatient within 24 hours Other Headaches Secondary headaches – – – – – – Temporal arteritis Acute glaucoma Hypertensive headache Sinusitis Post-LP Drug-related/toxic Primary headaches – Migraine – Cluster – Tension Vertigo Sensation of movement where none exists Peripheral causes vs. central causes – peripheral causes usually benign, central causes can be urgent – Peripheral vertigo: sudden onset, intense, paroxysmal, aggravated by position/movement, associated with nausea or hearing loss/tinnitus, horizontal nystagmus, fatiguable, CNS signs absent – Central vertigo: any onset, ill-defined, constant, variable association with position/movement/nausea, not associated with hearing loss/tinnitus, vertical nystagmus, not fatiguable, CNS signs usually present Vertigo – causes Peripheral causes – BPPV, Ménière’s disease, labrynthitis, ototoxicity, head injury Central causes – Cerebellar stroke, VBI, MS, migraine, epilepsy, neoplasm General causes – Anemia, EtOH intoxication, hypoglycemia, renal failure, thyroid disease Vertigo - evaluation History – Description of sensation – vertigo, syncope/nearsyncope, disequilibrium – Onset – Associated symptoms Peripheral associated with nausea/vomiting, tinnitus/hearing loss, photophobia Central associated with diplopia, dysarthria, visual abnormalities Headache suggests migraine or space-occupying lesion Head trauma, medications Vertigo - evaluation Physical exam – Ear: otoscopy, hearing exam, Webber/Rinne – Eye: nystagmus, EOMs – Heart rate, rhythm, murmurs – Cranial nerves – Cerebellar testing – Proprioception/vibration – Test patients with near-syncope for orthostasis Vertigo – evaluation Dix-Hallpike position testing: pt seated upright, head turned 45° to right, swiftly reclined with head tilted backward additional 45°; repeated with head turned to left – Warn pt that test may produce vertigo – Positive test indicated by nystagmus; positive side is side with lesion – Contraindicated in patients with carotid bruits, cervical spondylosis Vertigo – evaluation Labs – Depend on suspected etiology Labrynthitis: CBC, blood culture Head injury: CT for bleeding Near-syncope: ECG, cardiac monitoring, CBC for anemia Electrolytes, glucose, kidney function, thyroid Vertigo – symptomatic treatment Pharmacotherapy – Scopolamine – Antihistamines – diphenhydramine (Benadryl®), meclizine (Antivert®) – Neuroleptics – metoclopramide (Reglan®), promethazine (Phenergan®) – Benzodiazepines for anxiety – diazepam (Valium®), clonazepam (Klonopin®)