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NEUROMUSCULARSKELETAL TEST #1 SUMMER 2006 ALL NEUROLOGICAL DEFICITS ARE THE Reduced or absence of conduction RESULT OF THE TERMS “RADICULAR, DERMATOME OR Neurological lesion & segmental deficit MYOTOME” HAVE REFERENCE TO ANISOCORIA IN A BRIGHTLY LIT ROOM Mydriasis WILL LEAD THE EXAMINER TO CONSIDER A PLEXOPATHY WILL AFFECT THE Distribution of only the anterior primary rami WHICH DOES NOT NECESSARILY HAVE REFERENCE TO A NEUROLOGICAL DEFICIT Pain (paresthesiae, Tinel sign, myotome has reference to a neurological deficit) Loss of both direct and consensual light reflexes on the left Right lateralization on the Weber LOSS OF VE FUNCTION OF THE LEFT CN III WILL RESULT IN INCREASED BONE CONDUCTION ON THE RINNE TEST OF THE RIGHT EAR SHOULD ALSO DEMONSTRATE WHAT IS THE RELATIONSHIP BETWEEN Their innervating neurons enter and exit the THE DERMATOME, MYOTOME AND REFLEX same IVF THE FACET CAPSULE IS INNERVATED BY A branch of the posterior primary ramus A TOTAL LESION OF CN II OF THE RIGHT Absence of the left consensual light reflex EYE WILL DEMONSTRATE WHICH IS UNLIKELY TO OCCUR IN A Ataxia RADICULAR LESION Occur in radicular lesion: 1. Hypoesthesia 2. segmental paresthesiae 3. hypotonia WHAT IS THE SEGMENTAL The neurons are bound together in the same RELATIONSHIP OF THE NEURONS IN A IVF MIXED SPINAL NERVE WHICH STATEMENT IS FALSE Mechanoreceptors of the skin are considered segmental True statements: 1. Nociceptors of the skin are considered segmental 2. The A-delta fibers are segmentally arranged in the sensory cortex 3. Mechanoreceptors are larger and faster conducting than nociceptors LOSS OF THE ALPHA MOTOR NEURON WILL MANIFEST AS Weakness NEUROMUSCULARSKELETAL TEST #1 THE DESCENDING INHIBITORY PATHWAY WILL CAUSE THE RELEASE OF ____ INTO THE ____ INTERRUPTION OF THE THALAMOCORTICAL FIBERS COULD RESULT IN A RIGHT HEMISECTION OF THE CORD AT T1 WILL NOT RESULT IN Endorphins/dorsal horn Agraphesthesia Left + Babinski Will result in: 1. Right spastic paralysis 2. Right flaccid paralysis 3. Right akinesthesia A POSITIVE ROMBERG IS EVIDENCE THAT A LESION EXIST IN THE WHICH FIBER TYPES INNERVATE THE DISC A LESION OF THE LEFT MOTOR CORTEX WOULD LIKELY DEMONSTRATE HYPERTONICITYASSOCIATED WITH AN UPPER MOTOR NEURON LESION IS THE RESULT OF LEFT HEMISECTION OF THE CORD WILL RESULT IN ____ BELOW THE LESION SEGMENTAL SENSORY AND MOTOR DEFICITS DEFINE A VISCERAL AFFERENTS DO NOT ALLOW FOR Cord Mechanoreceptors Nociceptors Autonomics Right positive Babinski Loss of inhibition from the extrapyramidal Ipsilateral spasticity and hyperreflexia Contralateral loss of pain perception Radiculopathy Somatotopic discrimination Do allow for: 1. Pain perception 2. Establishment of a reflex 3. Excitation of the autonomics VISCERAL EFFERENTS TO THE PUPIL ORIGINATE FROM THE THE PRESENCE OF A TINEL SIGN PROVIDES EVIDENCE THAT THE LESION IS WHICH IS UNLIKELY FOLLOWING A LESION OF A PERIPHERAL NERVE Likely to occur: 1. Areas of anesthesia 2. Absent or depressed DTR 3. Positive Tinel sign Edinger-Westphal nucleus Lateral horn Neurological Myotome weakness NEUROMUSCULARSKELETAL TEST #1 WHEN SENSORY DEFICITS INDICATE A Both anterior and posterior primary rami will DERMATOMAL PATTERN be involved LOSS OF THE SE FIBER WILL REDUCE OR DTR ELIMINATE Superficial reflex Muscle tone A PATIENT WHO DEMONSTRATES Cord AKINESTHESIA BUT MAINTAINS SENSITIVITY TO PAIN LIKELY HAS A LESION OF THE WHICH TRACT CARRIES PREDOMINATELY Spinoreticulothalamic “C” FIBER PAIN AND INTERACTS WITH THE LIMBIC AND ACTIVATING CENTERS AN EXTRAPYRAMIDAL LESION Will not demonstrate a +Babinski IF A SPASM WERE THE RESULT OF AN UPPER MOTOR NEURON LESION WEAKNESS, HYPOTONIA AND HYPOREFLEXIA WOULD MANIFEST WITH A LESION OF GLUTAMATE RELEASE INTO THE SYNAPTIC CLEFT BETWEEN THE FIRST AND SECOND ORDER NEURONS The tone would be increased The DTR would be hyperactive The peripheral nerve Sensitizes the secondary neuron SPRING 2006 WHICH IS NOT CONSIDERED TO BE A NEUROLOGICAL DEFICIT A LESION OF SOMATOSENSORY AREA I IS DEMONSTRATED BY THE APPEARANCE OF WHICH LESION SITES WOULD RESULT IN SEGMENTAL DEFICITS RADICULAR LESIONS WOULD BE EXPECTED TO COMPROMISE A LESION OF THE DORSAL COLUMN WILL CAUSE WEAKNESS, LOSS OF TONE AND HYPOREFLEXIA WOULD MANIFEST WITH A LESION OF IF A SPASM WERE THE RESULT OF AN UPPER MOTOR NEURON LESION All of the following are examples of neurological deficits: Akinesthesia Paresthesiae Dermatome pain Agnosia The anterior ramus and plexus Both the anterior and primary rami Ipsilateral loss of position sense The peripheral nerve The DTR would be hyperactive NEUROMUSCULARSKELETAL TEST #1 THE PRESENCE OF TINEL SIGN PROVIDES Neurological EVIDENCE THAT THE LESION IS WHICH OF THE FOLLOWING DEFINES A One alpha motor neuron and all muscle fibers MOTOR UNIT innervated by it TENS UNITS ARE THEORIZED TO WORK Increasing the release of GABA into the EFFECTIVELY BY dorsal horn WHICH COULD NOT OCCUR WITH A Akinesthesia RADICULAR LESION Occur with a radicular lesion: 1. Hypoesthesia 2. Hypotonia 3. weakness THE PERIAQUEDUCTAL GRAY AREA SOMATOTOPIC DISCRIMINATION IS GENERALLY NOT POSSIBLE FOR THE GLUTAMATE RELEASED INTO THE SYNAPTIC CLEFT IN THE DORSAL HORN LOSS OF THE ANNULOSPIRAL WILL RESULT IN ALL OF THE FOLLOWING EXCEPT Is the initial site for the descending inhibitory pathway C fibers Sensitizes the secondary neuron Weakness Results in the following: 1. Hypotonia 2. Hyporeflexia 3. Preservation of kinesthesia THE PROPER USE OF THE TERM “DERMATOME” IS RESTRICTED TO DESCRIBING A PATIENT WHO DISPLAYS A +ROMBERG SIGN WILL ALSO DEMONSTRATE VISCERAL NOCICEPTION REACHES THE HIGHER CENTERS VIA THE DYSDIADOCHOKINESIA MAY RESULT FROM A LESION OF THE WHEN VISUAL INPUT SIGNIFICANTLY REDUCES THE ATAXIA EXTRAPYRAMIDAL LESION WILL DEMONSTRATE THE FACET JOINT IS ____ AND INNERVATED BY ____ THE DISC IS INNERVATED BY WHICH IS APPROPRIATELY DESCRIBED AS NONSEGMENTAL Neurological deficits Ataxia Spinoreticulothalamic tract Cerebellum Cord The lesion is likely in the tract Hypertonicity Synovial/both nociceptors and mechanoreceptors Both nociceptors and mechanoreceptors Plexopathy Peripheral neuropathy Parietal lobe lesion NEUROMUSCULARSKELETAL TEST #1 THE FACET IS INNERVATED FROM THE ____ PRIMARY RAMUS AND THE DISC FROM THE ____ PRIMARY RAMUS WHICH OF THE FOLLOWING IS A “MIXED” NERVE THE MOTOR EXAM INVOLVES CAREFUL ANALYSIS OF A LESION OF THE CAUDA EQUINA WOULD NOT RESULT IN Posterior/anterior Peripheral nerve Strength and tone Myotome weakness and hyporeflexia Would result in: 1. Ataxia 2. Diminished pain perception 3. Flexor plantar response SPASTIC WEAKNESS OF THE LEG WITH A +BABINSKI AND NONSEGMENTAL LOSS OF TACTILE PERCEPTION. LESION SITE? ALL REFLEXES REQUIRE Cord An afferent and efferent branch A LOWER MOTOR NEURON LESION WOULD NOT INCLUDE Muscular dystrophy Would include: 1. Polio 2. Myasthenia gravis MOTOR UNIT DECENTRALIZATION MAY RESULT IN WHICH WOULD RESULT FROM A LESION OF THE LEMNISCAL PATHWAY ALPHA MOTOR NEURONS A SMALL, WELL LOCALIZED AREA OF ANESTHESIA TO BOTH PIN PRICK AND 2POINT DISCRIMINATION IS BEST EXPLAINABLE BY A LESION OF THE Fibrillation potentials Diminished proprioception Receive their strongest excitation from the spindle Peripheral nerve NO DATE WHICH IS NOT EXPECTED TO PRODUCE FLACCID WEAKNESS Expected to produce flaccid weakness: 1. CN III nucleus lesion 2. Hemissection of the cord 3. Cauda equina syndrome Dorsal column lesion NEUROMUSCULARSKELETAL TEST #1 LOSS OF A SENSORY ROOT COULD MANIFEST THE LEFT PUPIL WILL NOT DILATE IN A DIMLY LIT ROOM. LESION SITE? ANISOCORIA COULD NOT RESULT FROM Dermatome hypoesthesia Left sympathetic A unilateral loss of CN II Could result from: 1. A unilateral loss of the sympathetic 2. A unilateral loss of CN III 3. A unilateral loss of VE fibers to the pupil THE SYMPTOMOTOLOGY ASSOCIATED WITH AN IVF LESION SHOULD BE A LEMNISCAL TRACT LESION WOULD DEMONSTRATE THE PRESENCE OF PARESTHESIAE CONFIRMS THAT THE LESION IS FLACCID WEAKNESS CONFInD TO A NONSEGMENTAL GROUP OF MUSCLES OF ONE EXTREMITY IS LIKELY AN EXTRAPYRAMIDAL LESION Segmental Radicular Neurological +Romberg sign loss of discriminated proprioception Neurologic A peripheral neuropathy Will not produce a +Babinski sign Consists of: 1. Will present a LMN deficit 2. Will produce spastic paralysis 3. Is not considered to be an UMN lesion WHICH NEUROTRANSMITTER BEHAVES MOST LIKE GABA MYDRIASIS IS THE RESULT OF LOSS OF THE CN III NUCLEUS A +ROMBERG INDICATES THAT THE LESION IS WHICH IS AN EXAMPLE OF A CONDUCTION DEFICIT WHICH RESPRESENTS A VE LESION CAUDA EQUINA SYNDROME WHEN THE RIGHT PUPIL CONSTRICTS DIRECTLY BUT NOT CONSENUALLY Enkaphalin Decreased parasympathetic tone Will have no effect on the papillary light reflex Within the tract Tinel Sign Hypoesthesia Paresthesia Miosis Mydriasis Will produce nonsegmental hypotonicity and hyporeflexia The left pupil will not constrict directly NEUROMUSCULARSKELETAL TEST #1 A RADICULOPATHY COULD NOT EXPLAIN THE PRESENCE OF Agraphesthesia Could explain the presence of: 1. A Tinel’s sign 2. Segmental hypotonicity 3. Myotome hypotonicity DERMATOME HYPERESTHESIA Is impossible “C” NOCICEPTION IS GENERALLY CARRIED Spinoreticulothalamic OVER WHICH TRACT TRUE “AGNOSIA” CAN ONLY OCCUR WITH Sensory cortex A LESION OF THE INCREASED ATAXIA WITH EYES CLOSED Cordially, cortex IS OFTEN ASSOCIATED WITH A LESION OF THE WHEN DISCRIMINATED PAIN IS Within the cord PRESERVED BUT CONSCIOUS PROPRIOCEPTION IS ABSENT, THE LESION IS AN UMN LESION RESULTS IN Statement is true, explanation is false SPASTICITY BECAUSE OF THE INCREASING AMOUNT OF AFFERENTATION FROM THE ANULOSPIRAL WHICH CAN ONLY RESULT FROM A +Romberg NEUROPATHY WHAT OCCURS AT THE ACTUAL LEVEL OF Ipsilateral LMN lesion THE CORD HEMISECTION THE SINUVERTEBRAL (RECURRENT) NERVE Proprioceptive, nociceptive and sympathetic CONTAINS fibers THE DESCENDING INHIBITORY PATHWAY Will inhibit A-delta, C fiber and tract pain (DIP) FROM THE PAG AREA conduction HYPERALGESIA IS THE RESULT OF A non-neurological lesion WHEN ONLY THE DORSAL ROOT IS LESIONED SOMATOTOPIC DISCRIMINATION REFERS TO COMPRESSION OF THE MIXED NERVE AT THE IVF WHICH COMBINATION IS MOST LIKELY Least likely combination: 1. Hypontonicity and a +Babinski 2. Paresthesiae and hyperesthesiae 3. Agnosia and dermatome deficits The DTR may be reduced or diminished “A” delta fibers Will reduce sensation in the painful dermatome Hyperreflexia and a + Babinski NEUROMUSCULARSKELETAL TEST #1 WHAT IS THE RELATIONSHIP BETWEEN THE FOLLOWING? AGRAPHESTHESIA, PARESTHESIA, HYPERREFLEXIA, MIOSIS, AND A +BABINSKI SIGN THE MOST POWERFUL EXCITATION FORCE ACTION ON THE LMN IS THE They all represent neurological deficits Anulospiral SUMMER 2002 THE SYMPTOMATOLOGY ASSOCIATED WITH AN IVF LESION SHOULD BE ANISOCORIA COULD NOT RESULT FROM A NEUROPATHY INVOLVING LOSS OF A SENSORY ROOT MAY MANIFEST WHICH SHOULD NOT PRODUCE FLACCID WEAKNESS A LEMNISCAL TRACT LESION WILL CAUSE A LESION AFFECTING SOMATOSENSORY AREA 1 SHOULD DEMONSTRATE THE RIGHT EYE WILL NOT DILATE IN A DIMLY LIT ROOM. POSSIBLE LESION SITE A LESION OF THE INTERNAL CAPSULE WOULD RESULT IN CEREBELLAR LOSS WILL PRODUCE Radicular Neurological Segmental Unilateral loss of CN II Reduction of the DTR Lesion of the dorsal columns Loss of discriminated proprioceptors Discriminatory nociceptive deficits Sympathetic atonia Facial and extremity paralysis Dysmetria LOSS OF A PROTION OF THE ABDOMINAL REFLEX THE PRESENCE OF PARESTHESIAE CONFIRMS THAT THE LESION IS A GRADED POTENTIAL Is likely when a +Babinski is also present WHICH COULD NOT BE CLASSIFIED AS A REFLEX stereoagnosia Neurological May be either excitatory or inhibitory Classified as a reflex: 1. A cough 2. Papillary constriction in bright light 3. Reciprocal inhibition A CONDITION CLASSIFIED AS EXTRAPYRAMIDAL GABA Should not demonstrate a +Babinski Provides CNS inhibitory potentials NEUROMUSCULARSKELETAL TEST #1 THE DTR Is monosynaptic Is of peripheral origin Is a primitive proprioceptive response Segmental in presentation A UNIQUE AND EXCLUSIVE INDICATOR OF A RADICULOPAHTY IS THAT ALL SYMPTOMS AND SIGNS ARE A CORTICOBULBAR LESION Is considered to be upper motor neuron PARESTHESIAE Are perceived distal to the site of irritation LOSS OF VE FIBERS OF CN III WILL PRESERVED DILATION OF THE PUPIL AND VOLUNTARY EYE MOVEMENT MYDRIASIS IS THE RSULT OF SYMPATHETIC ATONIA A +ROMBERG MEANS THAT THE ATAXIA IS IMPROVED WITH EYES OPEND AND HAS RESULTED FROM A TRACT LOSS THE SOMATOSENSORY AREA II IS CAPABLE OF “C” FIBER DISCRIMINATION CAUDA EQUINA SYNDROME IS OFTEN ACCOMPANIED WITH A +BABINSKI True False True False False FALL 2004 WHEN THE PAG FIBERS ARE STIMULATED A nociceptive afferentation is reduced WHICH OF THE FOLLOWING REUSLTS FROM A NEUROLOGICAL DEFICIT MAKING NO DISTINCTION BETWEEN PERCEPTION AND DISCRIMINATION, THE PRESENCE OF AGRAPHESTHESIA AND ASTEREOGNOSIA WOULD RULE OUT A THEORECTICALLY, A DERMATOME COULD BECOME ANESTHETIC TO AN AREA OF REFERRED PAIN AND PARESTHESIA IS EXPECTED TO DEMONSTRATE WHICH COULD PRESENT SEGMENTALLY Hyperreflexia INCREASED ATAXIA WHEN EYES ARE CLOSED IS A FINDING WHICH WOLD BE ATTRIBUTABLE TO Brown-Sequard syndrome Radiculopathy Pin prick Deficit paresthesia NEUROMUSCULARSKELETAL TEST #1 SOMATOTOPIC DISCRIMINATION IS NOT A FUNCTION OF ____ FIBERS EXTRAPYRAMIDAL LOSS IS COMMONLY ASSOCIATED WITH SEGMENTAL FLACCIDITY WOULD, IN ALL LIKELIHOOD, BE FOUND IN CONJUNCTION WITH DAMAGE TO THE ASCENDING TRACT COULD NOT EXPLAIN VA Hyperreactivity of the DTR Hypertonicity of skeletal muscle Dermatomal deficits Horner’s syndrome Explained the following: 1. +Romberg’s sign 2. ataxia of the lower extremity 3. loss of the superficial reflex DERMATOMES PRIMARILY HAVE REFERENCE TO WHICH IS AN EXAMPLE OF A CONDUCTION DEFICIT NONSEGMENTAL SENSORY DEFICITS THROUGHOUT A HYPOTONIC AND WEAK EXTREMITY ARE BEST EXPLAINED BY WHICH OF THE FOLLOWING DEMONSTRATES THE MOST PRIMITIVE LEVELS OF MECHANOREPTION A LESION OF THE VENTRAL ROOT NEURONS SHOULD NOT RESULT IN A-delta fiber endings Tinel sign Paresthesia Agnosia A Cortical lesion DTR Paresthesia Results in: 1. VE deficits 2. Hypotonia 3. hyporeflexia WHEN THE RIGHT PUPIL CONSTRICTS DIRECTLY BUT NOT CONSENUALLY CAUDA EQUINA SYNDROME The left pupil will not constrict directly ATAXIA OF CEREBELLAR ORIGIN Will produce nonsegmental hypotonicity and hyporeflexia Is not correctible by visual input WHICH IS A VA-VE REFLEX Papillary WHICH STATEMENT IS TRUE Only a VE lesion can cause miosis False statements: 1. Miosis can only occur due to a lesion of CN II 2. Miosis can only occur due to an upper motor neuron lesion THE FACET IS INNERVATED BY Sinuvertebral nerve NEUROMUSCULARSKELETAL TEST #1 WEAKNESS, HYPERTONICITY, HYPERREFLEXIA, AND A +BABINSKI OF THE RIGHT LEG, ASSOCIATED WITH RIGHT MIOSIS COULD BEST BE EXPLAINED BY A LESION OF THE LOCALIZATION OF PAIN ON STIMULATION OF THE LESION SITE IS INDICATIVE OF THE COMBINATION OF UPPER AND LOWER MOTOR NEURON LESION PRESENTATION IS UNIQUE TO ____ LESION SPASTICITY DUE TO A NEUROLOGICAL LESION RESULTS FROM ANISOCORIA SHOULD NOT RESULT FROM Right thoracic spine A somatic lesion Cord A decreased inhibition to the alpha motor neuron A unilateral lesion of CN II Anisocoria should result from: 1. A unilateral lesion of CN III 2. A unilateral sympathetic loss of the eyE WHICH OF THE FOLLOWING DOES NOT INVOLVE A TRACT DTR The following involve a tract: 1. Superficial reflex 2. Babinski sign 3. agnosia THE PATIENT SUFFERING FROM AN EXTRAPYRAMIDAL LESION A CONDITION DIAGNOSED AS “LEFT MYDRIASIS” SHOULD ALSO DEMONSTRATE THE MOTOR EXAMINATION CONSISTS OF EVALUATION OF THE ONLY DIRECT INFLUENCE ON THE ALPHA MOTOR NEURON IS FROM THE WHEN THE NEUROLOGICAL SYMPTOMS ARE REPRODUCIBLE BY THE PATIENT, THE LESION IS LIKELY THE ONLY DEFICIT WHICH IS CONTRALATERAL AND DISTAL TO THE SIDE OF CORD HEMISECTION Experiences hypertonicity Intact right consensual light reflex Absent left direct light reflex Absent left consensual light reflex Strength and tone SUMMER 2000 Spindle In the PNS Nociception NEUROMUSCULARSKELETAL TEST #1 A “LESION” IS BEST DEFINED AS Hypofunction WHICH IS NOT ASSOCIATED WITH A PYRAMIDAL LESION Ataxia Associated with a pyramidal lesion: 1. Apraxia 2. Extensor plantar reflex A LESION OF THE ANNULOSPIRAL FIBER FROM THE SPINDLE WOULD RESULT IN HORNER’S SYNDROME IS A LESION OF THE ____ FIBERS LEFT MIOSIS IS LIKELY TO ALSO ACCOMPANY WHICH IS NOT CONSIDERED AN EXTRAPYRAMIDAL TRACT Hypotonicity Hyporeflexia VE Left ptosis Corticospinal Considered an extrapyramidal tract: 1. rubrospinal 2. tectospinal 3. vestibulospinal RECIPROCAL INHIBITION TO THE ALPHA Antagonistic muscle MOTORO NEURO ORIGINATES FROM THE WHICH OF THE FOLLOWING WILL OCCUR Loss of pain and temperature sensation in the WITH LEFT HEMISECTION OF THE CORD right leg AT T8 FOLLOWING SPINAL SHOCK WHICH WILL RESULT IN THE Pyramidal lesion MANIFESTATION OF A +BABINSKI SOMATOTOPIC DISCRIMINATION IS A Post-central gyrus FUNCTION OF THE LOSS OF THE RIGHT CONSENUAL REFLEX Left CN II COULD OCCUR WITH A LESION OF THE Right CN II Edinger-Westphal nucleus MYDRIASIS IS BEST EXPLAINED AS Parasympathetic atonia THE CN III CARRIES VE and SE fibers only LOSS OF VE FIBERS TO THE EYE COULD MANIFEST AS HORNER’S SYNDROME IS BEST DEFINED AS WHICH OF THE FOLLOWING IS A MONOSYNAPTIC REFLEX AN IVF LESION MUS Miosis Mydriasis Sympathetic atonia SEGMENTAL NEUROPATHIES ARE THE RESULT OF LESIONS OF THE TINEL SIGN INDICATES THAT THE LESIOIN IS Annulospiral to alpha motor neuron Involve both primary rami Nerve root Neurological NEUROMUSCULARSKELETAL TEST #1 WHICH TRACT IS MOST LIKE THE PYRAMIDAL WHEN VISUAL INPUT DOES NOT IMPROVE THE ATAXIA SEGMENTAL MUSCLE DEFICITS Rubrospinal The lesion is likely within the cerebellum Are located within the myotome THE GRASP REFLEX Occurs from the same lesion that results in a +Babinski A SOMATOVISCERAL REFLEX COULD ALSO SA-VE BE WRITTEN THE ONLY ANATOMICAL SITE WHERE The cord SENSORY MODALITIES ARE SEPARATED IS A +ROMBERG TEST INDICATES THAT The lesion is in the cord SUPERFICIAL REFLEXES ARE DIMINISHED IN LESIONS OF THE THE LEMNISCAL SYSTEM CARRIES SOMATOVISCERAL, VISCEROSOMATIC, ETC…REFLEXES ALL RESULT FROM WHEN THE RIGHT PUPIL DEMONSTRATES AN INTACT CONSENUAL BUT AN ABSENT DIRECT LIGHT REFLEX PARESTHESIAE RESULT FROM A NEUROLOGICAL IRRITATION THE DEEP TENDON AND GTO REFLEXES ARE BOTH SOMATOSOMATIC THE DORSAL HORN RECEIVES ONLY SA INFORMATION AKINESTHESIA IS AN EXAMPLE OF ATAXIA DILATED PUPILS IN A WELL-LIT ROOM GENERALLY INDICATE SYMPATHETICOTONIA Cortex Cord Peripheral nerve Conscious proprioception Neurological irritation The lesion is the right CN II False Statement True Statement False Statement False Statement True Statement