Survey
* Your assessment is very important for improving the workof artificial intelligence, which forms the content of this project
* Your assessment is very important for improving the workof artificial intelligence, which forms the content of this project
Coronary Artery Disease: A Time for Dietary Intervention HILARY SHONE QUEEN OF THE VALLEY MEDICAL CENTER MARCH 25, 2015 Outline • Introduction/Patient Profile • Medical/Surgical Data • Summary/Overview of Disease State(s) • Admission Nutrition Assessment • Chronology of Medical and Nutrition Treatments • Discharge Plan • Summary Introduction/Patient Profile PG is a 59 year old Caucasian male, works in furniture restoration He is married, lives with his wife and they have one son Non-smoker, chronic alcoholic until December 26, 2014 Reason for admission: Chest pain (determined to be unstable angina) Admitting diagnosis: Atrial fibrillation with rapid ventricular response • Discovered first in ER • Likely the result of preceding cardiac events • Other forms of cardiovascular disease discovered soon after in hospital course Introduction/Patient Profile Primary Medical Diagnosis: Coronary Artery Disease Coronary Artery Disease (CAD): • Narrowing or blockage of the arteries that deliver blood and oxygen to the heart • Caused by atherosclerosis • Reduces blood and oxygen flow to myocardium = ischemia • AKA Ischemic Heart Disease (IHD), Coronary Heart Disease (CHD), Atherosclerotic Heart Disease (AHD) • Ischemic Heart Disease more broad than Coronary Artery Disease Medical & Surgical Data Past Medical History History of hypertension Heavy alcohol consumption, recently discontinued Atrial fibrillation ‘apparently’ for 3 years; intermittent chest pain for years Surgical History Varicose vein stripping Family Medical History Father deceased at age 35 ‘secondary to angina’ Brother has a history of atrial fibrillation Son experienced a CVA at age 12, unidentified etiology Hospitalization was pt’s first documented cardiovascular event Admission Physical Data General: Pleasant, alert male, currently in no acute distress Vital signs: Blood pressure initially 192/148 w/heart rates ranging between 115 and 143, respirations 19, afebrile Cardiovascular: Tachycardic and irregular Abdomen: Positive bowel sounds. Soft, nontender Extremities: No clubbing, cyanosis or edema No outpatient medications Normal Cardiovascular Function Forms a closed loop of blood vessels Heart acts as two pumps Right atrium and ventricle pump through pulmonary circulation for O2 Left atrium and ventricle pump O2rich blood through systemic circulation Cardiac muscle has unique electrical properties Normal Cardiovascular Function Electrical Properties of the Heart: Myocardial cells generate spontaneous electrical activity at SA node Atria contract Electric current spreads to AV node Current spreads to ventricles, causing contraction Measured on electrocardiogram (ECG/EKG) Pathophysiology of CAD Atherosclerosis (AS): Root cause of CAD and stroke Development of atherosclerotic plaque in the vascular wall that will occlude the lumen of the vessel, creating ischemic conditions Begins as fatty, fibrous growth and with time may calcify Atherosclerotic plaque can result in partial or complete stenosis of the blood vessel If severe, may cause an infarct myocardial infarction Severe CAD can lead to Congestive Heart Failure Non-Modifiable Risk Factors for AS: Modifiable Risk Factors for AS: Family history Obesity Atherogenic diet Ethnicity Dyslipidemia Cigarette smoking Age Hypertension Impaired fasting glucose Sex Physical Inactivity Pathophysiology of CAD Stable vs unstable angina Angina is primary symptom of CAD Collateral artery compensation Dislodged clot-thrombus Majority of MI result of blood clots and atherosclerotic debris Acute Coronary Syndrome Coronary Artery Disease Definition: Narrowing or blockage of the arteries that deliver blood and oxygen to the heart (atherosclerosis) Creates ischemic conditions, can lead to myocardial infarction Diagnosis: Personal/family medical history, risk factors and physical exam EKG Stress test Chest x ray Blood test Cardiac catheterization/coronary angiogram Etiologies Presence of 5 major risk factors: Smoked for 20 years, quit a few years ago Hypertension Low HDL cholesterol (< 40 mg/dL) Family history of premature CAD Age (men > 45 years) Strong genetic predisposition Diet history: Had poor diet in younger years, high fat and salt Alcoholic for most of life, quit day after Christmas Physical inactivity Only recently began exercising regularly Clinical Manifestations Admit diagnosis: Atrial fibrillation w/rapid ventricular response Discovered on EKG, then uncovered presence of other cardiac events Intermittent chest pain for years upon exertion Recent episode occurred while pt doing paperwork (at rest) with no trigger Severe intensity EKG showed ST depression, indicating myocardial injury Troponin I level 0.82, normal level below 0.04 ng/mL (QVMC), 0.5 ng/dL (literature) Diagnostic of myocardial infarction, ruled in while in ER Exact chronology unclear Unstable angina was very brief warning of MI Afib w/RVR may have immediately followed MI Cardiac muscle near area of infarct can become irritated, causing arrhythmias Clinical Manifestations Determined to have Acute Coronary Syndrome: Acute on chronic of Coronary Artery Disease Change in atherosclerotic plaque causing unstable angina Plaque expanded, eroded or ruptured Thrombus has moved or broken loose Coronary artery suddenly tightened in vasospasm Unstable angina can be brief or last to cause sufficient ischemia leading to MI Based on findings, pt eligible for cardiac catheterization Found to have obstruction in left main (coronary artery) and circumflex Stenosis of left anterior descending (LAD) Surgical candidate for urgent revascularization Coronary Artery Bypass Graft (CABG) Coronary Artery Bypass Graft (CABG) Goal: Restore perfusion of oxygen and nutrient-rich blood to the cardiac muscle Using grafted artery or vein to bypass blocked coronary artery(ies) Indications: Classification by American College of Cardiology & American Heart Association Level of evidence rating usefulness and efficacy of procedure (Class I, II, IIa, IIb, III) Class I for: Left main stenosis Stenosis of proximal LAD and circumflex 3 vessel disease NSTE-ACS and unstable hemodynamics Studies have shown 33% reduction in risk of all cause mortality after 5 years of CABG placement as compared to percutaneous coronary intervention (PCI) CABG PG had CABG x 2 Left radial artery grafted onto left marginal artery Internal mammary artery grafted onto LAD distal to obstruction Customary Nutrition Interventions Post MI period in CAD: No caffeine Low cholesterol, Therapeutic Lifestyle Changes guidelines (< 200 mg/day) Restrict sodium if HTN present Therapeutic Lifestyle Changes (ATP III): < 7% of daily total energy from saturated and trans fat 25-35% daily total energy from fat w/unsaturated, including omega-3 FA’s, in place of saturated fat 50-60% daily total energy from carbohydrate, approx. 15% daily total energy from protein Additionally include: 25-35 g daily fiber intake, at least half from soluble fiber Plant stanols/sterols (2 g per day) as an option Adequate energy intake to maintain desirable body weight or prevent weight gain Moderate exercise to expend at least 200 kcal per day If indicated, patients w/HTN may have sodium restriction of 1500-2300 mg/day Admission Nutrition Assessment Diet History Usual dietary intake: Breakfast: Bowl of oatmeal (prepared w/water), blueberries, walnuts and maple syrup Lunch: 3 different pieces of fruits and/or vegetables, several pieces of cheese and salami or other meat, yogurt and a handful of nuts Dinner: Chicken and mashed potatoes, vegetable sides, occasionally has red meat, shares steak w/wife Fluid choices: water and non-fat milk States a recent cut back on salt and red meat, ‘started eating healthier a few years ago’ Wife does all of meal preparation and cooking States almost all cooking is done w/olive oil, butter is used ‘when the food calls for it but not often’ Admission Nutrition Assessment Allergies: NKFA No previous MNT, doctor ‘has been on me for years’ regarding weight and alcohol consumption Has not been to a doctor in over 5 years Wife has attempted intervention for EtOH, unsuccessful Hospitalization and need for surgery was ‘wake up call’ Diet Order: Cardiac AHA Heart Healthy Anthropometrics: Height: 6’2” (188 cm) BMI: 29.4 Weight: 104 kg (240 lbs) Weight history: has been ‘about this weight for years’, small fluctuations UBW: ~220 lbs, ‘224 at Christmas, 210 when I’m hiking’ ABW: 92 kg (202 lbs) IBW: 86.4 kg (190 lbs) %IBW: 121% IBW Estimated Needs & Current Intake Kcal: (MSJ x 1.1-1.3) = 2125-2511 kcal Protein: 125-146 grams (1.2-1.4 g/kg body weight) Fluids: 2125-2511 mL (1 mL/kcal) Evaluation of intake at QVMC: Adequate, pt consuming 100% of all meals Adequacy of diet order: Approx. 115% average intake of diet needed to meet estimated needs, elevated energy requirements d/t elevated body weight; diet order adequate to meet needs. Evaluation of intake prior to admission* 2237 kcals consumed on average day – 97% of EER Breakfast: 480 kcals Lunch: 1200 kcals Dinner: 557 kcals 109 grams protein consumed on average day – 81% of estimated needs Approx. 30 fl oz (900 mLs) – 39% of estimated fluid requirements *using USDA Super Tracker Admission Nutrition Assessment 2/11/15 Day prior to CABG placement Medical treatments: Stabilized in ER w/meds Referred to cardiology and cardiothoracic surgeon Recommended cardiac catheterization/coronary angiogram Determined a candidate for CABG Treatment goals: Revascularize the heart Restore cardiac blood perfusion Diagnostic tests: EKG (MI) Cardiac catheterization/coronary angiogram (CAD, need for CABG) Blood Pressure 240 High: 214/81 4 hours after surgery 220 Low: 92/69 Blood Pressure (mmHg) 200 180 Average: 146/66 160 Most monitored on 2/12 140 120 100 80 60 40 2/9/15 Time 2/16/15 Lab Results 2/9/15 2/9/15 2/10/15 2/11/15 0.61 0.97 0.65 50 Troponin I 0.82 2 hours after surgery 45 *Released after death of cardiomyocytes 40 35 gm/dL % 30 25 Hemoglobin Lipid Panel 20 Hematocrit Triglycerides 151 15 Cholesterol 137 10 LDL Cholesterol 79 5 HDL Cholesterol 28 0 2/9/15 Time Includes POC Hgb (12.5-16.3) & Hct (36.7-47.1) Checked q 2 hours after surgery Checked 1x/day after 2/15/15 Albumin (2/10/15): 5.2 Nutrition-related Medications Pt was on variety of medications w/varying degrees of nutrition implications Majority were a one time frequency on operative days, insufficient time for effect of potential nutrition implications Two medications prescribed several times for several days: Medication Uses Potential Nutrition Implications When prescribed Metoprolol tartrate (Lopressor) Antihypertensive, antiangina, CHF treatment, MI treatment Recommended decrease dietary Na & kcal, decrease in BP w/possible hypotension, avoid natural licorice (↑[cortisol], ↑Na reabsorption, water retention, K excretion and BP) On admission, for 2 days btwn angiogram and CABG*, day after CABG until discharge* Avoid salt subs, caution w/ K supplement, ↑ serum K HCTZ: ↓ serum Na, Cl, K, ↑ glc (urinary excretion) For 2 days btwn angiogram and CABG, day after CABG until discharge Cardioselective BetaBlocker Lisinopril (Zestril) ACE inhibitor, antihypertensive, acute MI adjunct *highest doses Initial Diagnosis and Interventions Nutrition Diagnosis: Overweight R/T excessive energy intake prior to admission AEB BMI 29.5 Nutrition Interventions Diet Order: Cardiac AHA Heart Healthy Diet Counseling & Education: Educated patient on the Therapeutic Lifestyle Changes diet Discussed lifestyle habits that in combination w/genetics may have led to CAD Advised that surgery is first part of solution, lifestyle changes are necessary second Will need to monitor more closely which foods are consumed and portion size Will need to start incorporating regular physical activity, 30 minutes, 5x/week once medically cleared (pt/wife admitted low) Initial Goals and Monitoring Primary goals and objectives for MNT: Gain an understanding of TLC diet and its importance to medical condition Recognize dietary and lifestyle factors that likely contributed to development of CAD Openness to change/modification Nutrition Monitoring and Evaluation: Continue to provide cardiac diet-related nutrition education Monitor PO intake and weight trends Assess patient knowledge of information presented at next visit Next education planned POD #1 Follow-Up Nutrition Education 2/13/15 POD #1 s/p CABG placement *In ICU, pt’s wife and son present Medical treatments: CABG placed on 2/12 in AM Intubated for 6 hours on ICU on 2/12 Treatment goals: Restore cardiac blood perfusion Achieve hemodynamic stability Stable recovery Follow-Up Nutrition Education Diet Order: Cardiac AHA Heart Healthy Intake: 100% average meal intake Nutrition Interventions: Provided and went over CABG nutrition therapy (TLC diet) guidelines handout w/wife Types and sources of lipids Limiting cholesterol in diet (egg example) Choosing leaner proteins Tending toward a more plant-based diet Including fiber, especially soluble, in diet Asked/confirmed typical preparation methods at home Frequency of eating out/choices when eating out Wife asked questions about specific foods (salami, olive oil, proteins) Follow Up Nutrition Education Nutrition Monitoring and Evaluation: Important to have pt’s wife understand nutrition education Meal preparation, dietary and lifestyle change support, emotional support Pt hearing information again, will provide continuing education on POD #4, continue to assess knowledge and change readiness Continue to monitor nutrition parameters Laboratory values Weight trends (awareness of fluid gains) Adequacy of intake Changes in medical conditions and/or medical treatments Nutrition Assessment #2 2/16/15 POD #4, day of discharge No new medical treatments Treatment goal: discharge patient Nutrition Diagnosis: Food and nutrition-related knowledge deficit R/T new diagnosis of acute MI, atrial fib, coronary artery disease AEB need for CABG placement and lack of prior cardiac diet education Nutrition Interventions Diet Order: Cardiac AHA Heart Healthy Diet Nutrition Assessment #2 Date 2/9 Blood Glucose Levels Weight (kg) Likely fluid gain, not true weight gain 102 150 140 103 2/11 104.2 2/12 104.9 2/14 109.9 2/15 109.1 90 2/16 109 80 Reference Ranges Received many IV medications and fluids Blood Glucose (mg/dL) 2/10 130 120 110 100 Time 2/9/15 2/12 2/12 2/12 2/12 2/13 2/14 2/15 Na (POC) 136-145 140 140 140 135 133 131 134 K (POC) 3.5-5.1 4.5 4.6 4.4 4.1 4.2 4.2 4.0 4.64-5.28 4.7 4.4 (L) X X Ionized Ca2+ 4.4 (L) 4.2 (L) 4.3 (L) 2/16/15 Monitoring Na for blood pressure, hypertension Monitoring K for hyperkalemia, cardiac arrest potential Ionized calcium: major surgery, low levels of free Ca2+ can cause tachy- or bradycardia, muscle spasms, even coma Key player in cardiac contractility Nutrition-related medications at discharge Lopressor and Lisinopril also prescribed at discharge Medication Uses Potential nutrition implications When prescribed Docusate sodium (Colace) Stool softener, laxative High fiber w/1500-2000 mL fluid to prevent constipation, altered int abs of water & electrolytes After CABG through discharge, at discharge as outpt Aspirin To prevent CVA or MI, platelet aggregation inhibitor N/V, dyspepsia, black tarry stools, limit caffeine, limit foods that affect coagulation, anorexia After CABG through discharge, at discharge as outpt Acetominophen Analgesic, antipyretic Avoid alcohol (hepatotoxicity), ↑liver function enzymes At discharge as outpt Atorvastatin calcium (Lipitor) Antihyperlipidemic, Caution w/grapefruit/related citrus, ↓risk of cardio events ↓serum chol, TG, LDL, VLDL, ↑ HDL and ↓prog. of athero. At discharge as outpt Amiodarone HCl (Pacerone) Antiarrhythmic Avoid grapefruit/related citrus, anorexia, N/V, constipation At discharge as outpt Pantoprazole sodium (Protonix) Anti-GERD May ↓abs of Fe, B12, ↓gastric acid secretion, ↑gastric pH, diarrhea After CABG through discharge, at discharge as outpt Nutrition Assessment #2 Counseling & Education: Reinforced 4 key components of TLC diet: Most applicable to patient Limiting saturated fat intake Increasing unsaturated fatty acids Increasing intake of dietary fiber, especially soluble Limiting sodium Explained rationale of each component in relation to cardiac condition Provided handout on sources of soluble dietary fiber Offered to go over a general meal plan, pt responded “I think I get the food changes in the new lifestyle” Discussed potential obstacles to implementing changes/equipped for success at home “I don’t know, I just know I don’t ever want go through this again” Nutrition Assessment #2 Memorable quotes from final education: o “I know how important the salt reduction is” o “I didn’t put brown sugar in my oatmeal this morning because I knew I didn’t need it” o “I got to live 59 years with an extravagant lifestyle (food/alcohol), now I don’t get to do that” o “I realize that just because a food is good quality doesn’t mean it’s appropriate for me” o “Thank you for your help” o “I think I can do this” Discharge Plan Hospital protocol for MD to refer a post-CABG pt to a Cardiac Rehabilitation Program: Several months in length Monitored moderate physical activity Monitored respiratory and cardiac function while exercising Nutrition counseling (TLC diet, weight management as needed) Discharge report: Adhere to a sodium restricted, low cholesterol, low fat diet No fluid restriction Check weight daily (especially rapid/significant gains) Adhere to all prescribed medications Gradually begin physical activity, no fatigue Watch for certain signs and symptoms indicating cardiac dysfunction Follow up as outpt w/cardiologist in 1-2 weeks, w/surgeon in 4 weeks Gave point of contact to wife for any questions Summary Suspected major contributors to CAD: Genetics HTN Dietary choices and lack of physical activity Medical and dietary intervention to remedy an acute on chronic cardiac event Coronary angiogram CABG placement Pt given extensive education on dietary management of CAD and post-CABG Equipped w/tools for success Hope lesson was learned and pt has good prognosis Effectiveness of MNT reflected in quotes, pt and wife’s attitude toward nutrition counseling Increasingly tailored to pt’s education readiness, needs and learning lifestyle • Approx. 1 in 18 Americans 18 y/o and older has CAD (CDC 2013) • Chronic development, strongest risk factor is age • Annually, 935,000 Americans have a heart attack (2/3 are first time) • Heart disease is leading cause of death (1 in every 4) • 30% reduction in mortality since 1950’s, due to medical intervention such as CABG • Early intervention is key References Nelms, M., Sucher, K., Lacey, K, and Roth, S. (2011) Nutrition Therapy & Pathophysiology. 2nd ed. Belmont: Wadsworth Cengage Learning. Crowe, J.C. and Pronsky, Z.M. (2012). Food-Medication Interactions (17th ed.). Birchrunville, PA: Food-Medication Interactions. http://www.medicinenet.com/hemoglobin/page2.htm http://medical-dictionary.thefreedictionary.com/hemodynamic+instability https://www.nutritioncaremanual.org/topic.cfm?ncm_category_id=1&lv1=5803&lv2=5806&ncm_toc_id=5807&ncm_he ading=Nutrition%20Care https://www.virginiamason.org/WhatareNormalBloodGlucoseLevels http://emedicine.medscape.com/article/766479-overview http://www.ncbi.nlm.nih.gov/pubmed/17627191 http://www.healthline.com/health/calcium-ionized#Preparation4 http://www.mayoclinic.org/diseases-conditions/atrial-fibrillation/multimedia/img-20096412 http://www.mayoclinic.org/diseases-conditions/atrial-fibrillation/basics/causes/con-20027014 http://www.mayoclinic.org/diseases-conditions/coronary-artery-disease/basics/symptoms/con-2003203 References http://biology-forums.com/index.php?action=gallery;sa=view;id=9172 http://www.nhlbi.nih.gov/health/health-topics/topics/cad/diagnosis http://www.nhlbi.nih.gov/health/health-topics/topics/echo/ http://www.nhlbi.nih.gov/health/health-topics/topics/ca/ http://www.mayoclinic.org/diseases-conditions/coronary-artery-disease/basics/tests-diagnosis/con-20032038 http://www.nhlbi.nih.gov/health/health-topics/topics/cad/diagnosis http://www.nlm.nih.gov/medlineplus/ency/article/007452.htm http://www.nhlbi.nih.gov/health/health-topics/topics/cabg https://www.supertracker.usda.gov/Nutrientsreport.aspx http://www.nlm.nih.gov/medlineplus/ency/article/003646.htm http://en.ecgpedia.org/wiki/File:Changing_ST.svg https://www.atrainceu.com/course-module/1711374-102_coronary-artery-disease-cad-module-05 References http://www.ehealthconnection.com/regions/mhp/healthimages.asp?src=100190&typeid=3 http://www.middleeasthealthmag.com/cgi-bin/index.cgi?http://www.middleeasthealthmag.com/jan2011/feature3.htm http://emedicine.medscape.com/article/1893992-overview http://emj.bmj.com/content/19/2/129.full http://www.ncbi.nlm.nih.gov/pubmed/17627191 http://www.healthline.com/health/calcium-ionized#Preparation4 http://www.mayoclinic.org/diseases-conditions/atrial-fibrillation/multimedia/img-20096412 http://www.mayoclinic.org/diseases-conditions/atrial-fibrillation/basics/causes/con-20027014 http://www.mayoclinic.org/diseases-conditions/coronary-artery-disease/basics/symptoms/con-20032038 http://en.ecgpedia.org/wiki/File:Changing_ST.svg https://www.atrainceu.com/course-module/1711374-102_coronary-artery-disease-cad-module-05 http://www.ehealthconnection.com/regions/mhp/healthimages.asp?src=100190&typeid=3 References http://www.middleeasthealthmag.com/cgibin/index.cgi?http://www.middleeasthealthmag.com/jan2011/feature3.htm http://emedicine.medscape.com/article/1893992-overview http://emj.bmj.com/content/19/2/129.full http://www.hopkinsmedicine.org/healthlibrary/conditions/cardiovascular_diseases/vital_signs_body_temperatur e_pulse_rate_respiration_rate_blood_pressure_85,P00866/ http://www.vhn.ca/patient_menu.php http://www.nhlbi.nih.gov/health-pro/guidelines/current/cardiovascular-health-pediatric-guidelines/full-reportchapter-2 https://vpn.lib.ucdavis.edu/science/article/pii/,DanaInfo=www.sciencedirect.com+S1388198111001570 http://www.ncbi.nlm.nih.gov/gene/335 http://ghr.nlm.nih.gov/gene/ABCA1 http://ghr.nlm.nih.gov/gene/LCAT http://en.wikipedia.org/wiki/Proband