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The History of Cardiovascular Magnetic Resonance
The History of Cardiovascular Magnetic Resonance

... wall, a widely recognized and highly respected cardiac cell biologist, was recruited to set up a laboratory at the Peter Bent Brigham Hospital in the late 1970s. The collaboration started with the assessment of Thallium-201, the radionuclide myocardial perfusion imaging agent. Ionic thallium chlorid ...
Ivabradine and outcomes in chronic heart failure (SHIFT)
Ivabradine and outcomes in chronic heart failure (SHIFT)

... and left-ventricular systolic dysfunction. The study was undertaken in 677 centres in 37 countries. Eligible patients were men or women aged 18 years and older who were in sinus rhythm and had a resting heart rate of 70 bpm or higher, as measured on 12-lead electrocardiography (ECG) after at least 5 ...
Renal Damage after Myocardial Infarction Is Prevented by Renin-Angiotensin-Aldosterone-System Intervention
Renal Damage after Myocardial Infarction Is Prevented by Renin-Angiotensin-Aldosterone-System Intervention

... between cardiovascular and renal disease. Impaired renal function is detrimental for the heart (renocardiac interaction) both in clinical (1– 8) and in experimental (9,10) settings. Impaired cardiac function is detrimental to the kidney (cardiorenal interaction), which is less well characterized and ...
Diagnosis and Management of Rheumatic Heart
Diagnosis and Management of Rheumatic Heart

autonomous regulation of circulation in children after
autonomous regulation of circulation in children after

... Baroreflex sensitivity, Anthracycline, Leukaemia, Blood pressure INTRODUCTION ...
Activity Overview continued
Activity Overview continued

... When the ventricles contract, blood is squeezed much like water in an eyedropper bulb. Blood pushes against the heart walls and valves with increased pressure. Eventually, blood moves through the path of least resistance, out the arteries. In the case of pulmonary circulation, oxygen-poor blood mov ...
Cardiac Embryology and Molecular Mechanisms of Congenital
Cardiac Embryology and Molecular Mechanisms of Congenital

... In the past, it was assumed that the heart tube contained all elements necessary to form the final 4-chambered heart. New research shows that the initial heart tube, formed by cells from the first heart field, ultimately ends up forming only a small part of the 4-chambered heart. While cells from th ...
Imaging pitfalls, normal anatomy, and anatomical variants that can
Imaging pitfalls, normal anatomy, and anatomical variants that can

... The interatrial septum is thin and often not well visualized on imaging studies. Normal fat deposition within the septum measures between 0.9–9.9 mm in thickness (18). Due to advanced age or obesity, increased fat deposition occurs and makes the septum easier to visualize. The most common abnormalit ...
Partial anomalous pulmonary venous return associated with
Partial anomalous pulmonary venous return associated with

... sinus venous atrial septal defects, may be present in up to 80% of cases. In our case, there was no cardiac anomaly but vascular anomalies of the aorta. The right and left common carotid arteries had a single origin from the arcus aorta, and then the left vertebral artery, left subclavian artery, an ...
Do you know?
Do you know?

... from the heart. As though the blood as trying to flow toward the heart and to accumulate just below the block because it simply couldn’t flow away from the heart. This was true of all veins. In the arteries, the blood bulged on the heart side of any block he put in, as though it were trying to flow ...
Prenatal Diagnosis of Congenital Heart Disease: Where Are We Now?
Prenatal Diagnosis of Congenital Heart Disease: Where Are We Now?

... atrium. In the human fetus, more than one-fifth of combined ventricular output is directed to the lungs and eventually drained back to the heart through the pulmonary veins [49,50]. At least one pulmonary vein should always be seen entering the left atrium. The ventricular chambers also appear simil ...
Medic One/Emergency Medical Services
Medic One/Emergency Medical Services

... We recommend that you contact a doctor to have your blood pressure checked again as soon as possible. We may call you in a week or two to find out how you are doing. ...
EISENMENGER SYNDROME
EISENMENGER SYNDROME

... Use of oral anticoagulant treatment in Eisenmenger’s syndrome is ...
Original Article
Original Article

Which Type of Right Ventricular Pressure Overload Is Worse? An
Which Type of Right Ventricular Pressure Overload Is Worse? An

... of pressure overload compared to normal individuals and also to find out whether there are any differences between the different pathologies that cause pressure loading on this chamber. The most important tissue Doppler imaging characteristics of PAH should be strain, systolic and diastolic SR (S, E ...
EISENMENGER SYNDROME
EISENMENGER SYNDROME

... Use of oral anticoagulant treatment in Eisenmenger’s syndrome is ...
The Cardiovascular System - Florida International University
The Cardiovascular System - Florida International University

... associated with changes in pressure and blood volume in the heart  The pressure differences cause opening and closing of heart valves that allow one-way blood flow through heart  Changes in pressure and blood volume correspond with electrical events on the EKG ...
Right heart catheterisation: how to avoid the most common mistakes
Right heart catheterisation: how to avoid the most common mistakes

... common mistakes and a major confounding factor during RHC. Zero level setting must be meticulous because it represents the starting point for all pressure measurements. In fact, each pressure measured during RHC is a difference between the pressure at the chosen zero level and the pressure in the ch ...
Adverse Effects of Combination Angiotensin II Receptor Blockers
Adverse Effects of Combination Angiotensin II Receptor Blockers

... searched for eligible RCTs that included 500 or more subjects, had a follow-up of 3 months or longer, and reported adverse effects. We used a random effects model to calculate the relative risk (RR) and 95% confidence interval (CI) for the following outcome measures: medication discontinuations beca ...
Anatomy Heart and Cardiovascular 2015
Anatomy Heart and Cardiovascular 2015

... Pathway of Blood Through the Heart • Equal volumes of blood pumped to pulmonary and systemic circuits • Pulmonary circuit short, low-pressure circulation • Systemic circuit long, high-friction circulation • Anatomy of ventricles reflects differences – Left ventricle walls 3X thicker than right • Pu ...
Right ventricular function in patients with pulmonary hypertension
Right ventricular function in patients with pulmonary hypertension

... (MPI) has been proposed6 – 8 for the evaluation of combined systolic and diastolic ventricular function. This index that can be measured by both pulsed-wave Doppler and tissue Doppler imaging (TDI), has been proved to correlate well with invasive procedures such as catheterization,9 to reflect disea ...
Functional assessment of the fetal heart: a review
Functional assessment of the fetal heart: a review

... ventricle, at first passively and later actively, because of atrial depolarization and contraction (the so-called ‘atrial kick’). The terms ‘active’ and ‘passive’ of course refer to the macroscopic appearance, although both processes are active at the molecular level. The atrial contribution to CO b ...
Changes in left ventricular filling dynamics with treadmill exercise in
Changes in left ventricular filling dynamics with treadmill exercise in

... ORIGINAL PAPERS, CLINICAL J Clin Basic Cardiol 1999; 2: 89 ...
AICD and Pacemaker Update
AICD and Pacemaker Update

... than the sinus node. •Also occurs when transmission from the SA node to A-V node is blocked (A-V block). •During sudden onset of A-V block, sinus node impulses do not get through, and next fastest area of discharge becomes pacemaker of heart beat. •Delay in pickup of the heart beat is called “Stokes ...
Pharmacology and the Nursing Process, 4th ed. Lilley/Harrington
Pharmacology and the Nursing Process, 4th ed. Lilley/Harrington

... The heart is unable to pump blood in sufficient amounts from the ventricles to meet the body’s metabolic needs Symptoms depend on the cardiac area affected ...
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Myocardial infarction



Myocardial infarction (MI) or acute myocardial infarction (AMI), commonly known as a heart attack, occurs when blood flow stops to a part of the heart causing damage to the heart muscle. The most common symptom is chest pain or discomfort which may travel into the shoulder, arm, back, neck, or jaw. Often it is in the center or left side of the chest and lasts for more than a few minutes. The discomfort may occasionally feel like heartburn. Other symptoms may include shortness of breath, nausea, feeling faint, a cold sweat, or feeling tired. About 30% of people have atypical symptoms, with women more likely than men to present atypically. Among those over 75 years old, about 5% have had an MI with little or no history of symptoms. An MI may cause heart failure, an irregular heartbeat, or cardiac arrest.Most MIs occur due to coronary artery disease. Risk factors include high blood pressure, smoking, diabetes, lack of exercise, obesity, high blood cholesterol, poor diet, and excessive alcohol intake, among others. The mechanism of an MI often involves the rupture of an atherosclerotic plaque, leading to complete blockage of a coronary artery. MIs are less commonly caused by coronary artery spasms, which may be due to cocaine, significant emotional stress, and extreme cold, among others. A number of tests are useful to help with diagnosis, including electrocardiograms (ECGs), blood tests, and coronary angiography. An ECG may confirm an ST elevation MI if ST elevation is present. Commonly used blood tests include troponin and less often creatine kinase MB.Aspirin is an appropriate immediate treatment for a suspected MI. Nitroglycerin or opioids may be used to help with chest pain; however, they do not improve overall outcomes. Supplemental oxygen should be used in those with low oxygen levels or shortness of breath. In ST elevation MIs treatments which attempt to restore blood flow to the heart are typically recommended and include angioplasty, where the arteries are pushed open, or thrombolysis, where the blockage is removed using medications. People who have a non-ST elevation myocardial infarction (NSTEMI) are often managed with the blood thinner heparin, with the additional use angioplasty in those at high risk. In people with blockages of multiple coronary arteries and diabetes, bypass surgery (CABG) may be recommended rather than angioplasty. After an MI, lifestyle modifications, along with long term treatment with aspirin, beta blockers, and statins, are typically recommended.Worldwide, more than 3 million people have ST elevation MIs and 4 million have NSTEMIs each year. STEMIs occur about twice as often in men as women. About one million people have an MI each year in the United States. In the developed world the risk of death in those who have had an STEMI is about 10%. Rates of MI for a given age have decreased globally between 1990 and 2010.
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