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Abstract:
Abstract:

... high prevalence in rural areas. Suicide is second leading cause of death among young people and with the decline in maternal death rates; it could soon become the leading cause of death among young women in India. Ingestion of agrochemical compounds is the principle mode for suicide. Of the 1.87000 ...
Chapter 6 Toxic Metals and Elements
Chapter 6 Toxic Metals and Elements

... • Contact with high concentrations of toxic metals is most likely to occur in an occupational setting, e.g., among persons who work with metals. • Lower-level exposures may result from contact with the ambient environment (e.g., children may ingest toxic metals present in paint). ...
Abstract:
Abstract:

... case of a 55 yrs old farmer has also been reported, who developed significant toxicity by accidental inhalational exposure during handling of multiple OP pesticides. [10]. Acute poisoning is a medical emergency, which poses a major threat to life. Its type, associated morbidity and mortality varies ...
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Upon completion of Chapter 7, you should be able to

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File - chemistryattweed

... the reaction of excess acid with iron(II) sulfide and hydrochloric acid, calculate the mass of FeS reacted. Answer the above example by following these steps: 1. Write a balanced chemical chemical equation for the reaction of Iron(ll) sulfide with hydrochloric acid. 2. Identify the stoichiometric ra ...
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... three broad categories: bacteria living in symbiotic relationships with certain plants, free anaerobic bacteria, and algae. Crops such as alfalfa and beans are often planted in order to remedy nitrogen-depletion in soils. Nitrogen fixing bacteria employ an enzyme, known as nitrogenase, to do the ene ...
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nitrogen cycle

... 6.The process of converting nitrogen is called fixation. 7.There are specialized bacteria whose function it is to fix nitrogen, converting it, so that it can be used by plants. 8.There are still other bacteria who do the reverse. That is, they return nitrogen to is gaseous form. 9. After nitrogen i ...
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... - caused by deficiencies of urea cycle enzymes - severity depends on proximity of defect to point of entry of ammonia in its processing to urea - CPS-1 defects or ornithine transcarbamoylase defects  severe hyperammonemia; these two defects can be distinguished by evaluating appearance of pyrimidin ...
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Nitrogen dioxide poisoning



Nitrogen dioxide poisoning is the illness resulting from the toxic effect of Nitrogen (II) oxide. It usually occurs after the inhalation of the gas beyond the threshold limit value.Nitrogen (II) oxide is reddish-brown with very a sharp, harsh smell at high concentrations. It is colourless and odourless at lower concentration but yet harmful. Nitrogen dioxide poisoning depends on the duration, frequency and intensity of exposure.Nitrogen (II) oxide is an irritant of the mucous membrane linked with other air pollutant that causes pulmonary diseases such as OLD, asthma, Chronic obstructive pulmonary disease and sometimes Acute exacerbation of COPD and in fatal cases, deaths.Its poor solubility in water enhances its passage and its ability to pass through the moist oral mucosa of the respiratory tract.Like most toxic gases, the dose inhaled determines the toxicity on the respiratory tract. Occupational exposures constitute the highest risk of toxicity and domestic exposure is uncommon. Prolonged exposure to low concentration of the gas may have lethal effects, as can short-term exposure to high concentrations like Chlorine gas poisoning. It is one of the major air pollutant capable of causing severe heath hazards such as Coronary artery disease as well as Stroke.Nitrogen (II) oxide is often released into the environment as a byproduct of fuel combustion but rarely released by Spontaneous combustion. Known sources of Nitrogen gas poisoning includes automobile exhaust, Power stations, The toxicity may also results from non-combustible sources such as the one released from anaerobic fermentation of food grains and Anaerobic digestion of Biodegradable waste.The WHO developed a global recommendation limiting exposures less than 20 part per billion for chronic exposure and value less 100ppb for one hour for acute exposure, using Nitrogen (II)oxide as a marker for other pollutant from fuel combustions. The standardss also based on the concentration of Nitrogen (II) oxide that show a significant and profound effects on the function of the pulmonary of asthmatic patients.Historically, some states in the U.S including Chicago and L.A have high levels of Nitrogen (II) oxide but the EPA set a standard values less than 100 ppb for one hour exposure and less than 53 ppb for chronic exposure.
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