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Rulebased Assignment of Myocardial Sheet Orientation
Rulebased Assignment of Myocardial Sheet Orientation

... tricles. There are typically four myocytes across the thickness of a layer. One axis of the sheets is built by the myocardial fibre orientation. The other axis is measured relative to the local outer-wall normal in planes built by the principal axis of the heart and a vector radial to it. Near the e ...
Anesthesia-Related Cardiac Arrest in Children with Heart Disease
Anesthesia-Related Cardiac Arrest in Children with Heart Disease

... inability to wean from cardiopulmonary bypass were categorized as not related to anesthesia when there were no anesthesia-related problems in the prebypass period; such cases (n ⫽ 74) were not included in the data analysis. Heart disease lesions were grouped based on underlying pathophysiology into ...
With right → left shunt
With right → left shunt

... Abnormal blood flow (as indicated by the shaded blue arrow) is from the right atrium and right ventricle through an atrial septal defect to the left side of the heart. Blood can reach the pulmonary arteries only through a patent ductus arteriosus. ...
What is a heart murmur? - Willows Vets
What is a heart murmur? - Willows Vets

... If a heart valve is abnormally narrowed, usually due to a congenital defect (eg. Aortic Stenosis or Pulmonic Stenosis) then blood flow pumped out through the narrowed valve is pinched, resulting in an abnormal squirt of flow, ie. a murmur. This can be likened to putting a thumb over the end of a hos ...
Conducting Pathways of the Human Heart
Conducting Pathways of the Human Heart

... poikilothermic animals had an analogous counterpart in mammals. This was accomplished by W. His, Jr. (fig. 2) in 1893 (7, 8, 9), and almost simultaneously by the independently working A. F. Stanley Kent (16). Predictably, the ensuing years were occupied in successful search and survey of alternate p ...
July - Congenital Cardiology Today
July - Congenital Cardiology Today

... morphologic tricuspid valve, ventricular septal defect, morphologic left ventricular (pulmonary) outflow tract obstruction and complete heart block.8-10 Extensive study of the atrioventricular conduction system in corrected transposition with situs solitus patients by several investigators11-16 dete ...
cardiologycmc.in
cardiologycmc.in

... Duration of the "hangout interval" is a measure of impedance in the pulmonary artery system. In the highly compliant (low-resistance, highcapacitance) pulmonary vascular bed, the hangout interval may vary from 30 to 120 msec, contributing significantly to the duration of right ventricular ejection. ...
The Correlation between Left and Right Ventricular Ejection
The Correlation between Left and Right Ventricular Ejection

... Ventricular Ejection Fractions in Patients with Ischemic Heart Disease, Documented by Cardiac Magnetic Resonance Imaging. J Cardiothorac Med. 2016; 4(1):407-410. ...
Percentage of Left Ventricular
Percentage of Left Ventricular

... between the left ventricular stroke work loss and the aortic valve area in cm2 and cm2/m2 was - 0.79 and - 0.80, respectively (fig. 2). With five exceptions (cases 5, 6, 9, 10, and 47), patients who had aortic valve areas of 1.0 cm2 or less had greater than 30% left ventricular stroke work loss, and ...
A tracing of the electrical activity of the heart is called a/an . 6. What
A tracing of the electrical activity of the heart is called a/an . 6. What

... C) end-systolic volume D) end-diastolic volume E) ejection fraction Answer: C Learning Outcome: 20-3 Bloom's Taxonomy: Comprehension 92) What occurs at "A" on the graph? A) semilunar valve opens B) semilunar valve closes C) AV valve opens D) AV valve closes E) end systolic volume Answer: A Learning ...
EFFECT OF EXTERNALLY APPLIED LOAD ON THE WORK DONE
EFFECT OF EXTERNALLY APPLIED LOAD ON THE WORK DONE

... on a skeletal muscle since it begins to be effective only after the onset of contraction. the analogy between the cardiac and skeletal muscle, in this respect, is quite a close one. Any increase in load, whether by pre- or after-loading, produces an immediate faD the amplitude of contraction of the ...
through a rational characterization of left ventricular phenotypes
through a rational characterization of left ventricular phenotypes

... largely irreversible and is associated with significant increases in morbidity and mortality. There are still areas of debate here. Thus, although wall thickness normalized, a proportion of ‘5 years deconditioned athletes’ who exhibited previous physiological increased left ventricle (LV) size prese ...
athology 6020 - Year 2005  Paul Urie, M.D., Ph.D. Dec.
athology 6020 - Year 2005 Paul Urie, M.D., Ph.D. Dec.

... persons annually. (Also includes other cardiac disorders (10-20%): congenital abnormalities, aortic stenosis, MVP, myocarditis, cardiomyopathies, pulmonary hypertension, conduction defects) Death is due to ventricular electrical instability (arrhythmia). ...
Ch 14: Cardiovascular Physiology
Ch 14: Cardiovascular Physiology

... There are 4 sets of valves 1- Tricusped valve (between right atrium & right ventricle). 2- Mitral valve (between left atrium & left ...
Heart Dysrhythmias Cheat Sheet Arrhythmias Description Causes
Heart Dysrhythmias Cheat Sheet Arrhythmias Description Causes

... with normal beats; focus from one or more sites. Ominous when clustered, multifocal, with R wave on T pattern. Ventricular rate 140 to 220 bpm, regular or irregular. ...
Heart
Heart

... Location and Surface of the Heart: The heart is situated in the middle mediastinum and is divided into right and left sides by an obliquely placed, longitudinal septum. Each side consists of an atrium, which receives blood from the pulmonary veins, and a ventricle, which propels the blood into the a ...
Heart Dysrhythmias Cheat Sheet
Heart Dysrhythmias Cheat Sheet

... with normal beats; focus from one or more sites. Ominous when clustered, multifocal, with R wave on T pattern. Ventricular rate 140 to 220 bpm, regular or irregular. ...
Surgical Procedures - The Children`s Heart Foundation
Surgical Procedures - The Children`s Heart Foundation

... The most common aortic and pulmonic stenosis is caused by congenital fusion of one or more moving valve leaflets. Repair is done using cardiopulmonary bypass and by cutting the fused leaflets apart. Occasionally, however, the seat of the valve, the annulus, is small, a condition called hypoplastic. ...
- Austin Publishing Group
- Austin Publishing Group

... evaluated through echocardiography and magnetic resonance), as well as histopathological (if endomyorcardial biopsy is chosen), electrical (by electrocardiography, exercise testing and Holter monitor) and genetic/familial [1]. Frequently, the risk of sudden cardiac death is evaluated by electrophysi ...
Contractile cells
Contractile cells

... • 1. atrial excitation and contraction should be complete before ventricular excitation and contraction • opening and closing of valves within the heart depend upon pressure – which is generated by muscle contraction • so simultaneous contractions of atria and ventricles would lead to permanent clos ...
Jake - wendyblount.com
Jake - wendyblount.com

... In clinic test to distinguish cardiac from respiratory dyspnea Validated in dogs JACVIM January 2008 <210 pmol/L – more likely respiratory disease >210 pmol/L – more likely cardiac disease Falsely elevated by increased creatinine Helpful in distinguishing cardiac from respiratory dyspnea when creati ...
External Cardiac Defibrillators for the Prevention of Sudden Death
External Cardiac Defibrillators for the Prevention of Sudden Death

... artery disease. The automatic implantable cardioverter defibrillator (AICD) has proven effective in reducing mortality for survivors of SCA and for patients with documented malignant ventricular arrhythmias. The use of AICDs has been potentially broadened by studies reporting a reduction in mortalit ...
aed awareness - Alex Dishman`s Portfolio
aed awareness - Alex Dishman`s Portfolio

... • Sports facilities around the country have added them • American Heritage added 3 AEDs since the accident and the athletic trainer is required to carry one at all times ...
Homogeneity out of heterogeneity.
Homogeneity out of heterogeneity.

... turbulence. This remarkable adaptation of form to function becomes apparent when disease alters the patterns of blood flow through the heart. For example, a narrowed valve or a defect in the interventricular septum creates turbulence that dissipates the energy of cardiac contraction and is readily d ...
Sample chapter – 7: Assessment of cardiac output and peripheral
Sample chapter – 7: Assessment of cardiac output and peripheral

... a. the pulse pressure is calculated as diastolic pressure plus one-third of systolic pressure. b. the pulse pressure serves as an index of the left ventricle stroke volume. c. the pulse pressure increases in direct proportion to arterial compliance. d. it is necessary to measure the heart rate too. ...
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Hypertrophic cardiomyopathy



Hypertrophic cardiomyopathy (HCM) is a primary disease of the myocardium (the muscle of the heart) in which a portion of the myocardium is hypertrophied (thickened) without any obvious cause, creating functional impairment of the cardiac muscle. It is a leading cause of sudden cardiac death in young athletes.The occurrence of hypertrophic cardiomyopathy is a significant cause of sudden unexpected cardiac death in any age group and as a cause of disabling cardiac symptoms. Younger people are likely to have a more severe form of hypertrophic cardiomyopathy.HCM is frequently asymptomatic until sudden cardiac death, and for this reason some suggest routinely screening certain populations for this disease.A cardiomyopathy is a disease that affects the muscle of the heart. With HCM, the myocytes (cardiac contractile cells) in the heart increase in size, which results in the thickening of the heart muscle. In addition, the normal alignment of muscle cells is disrupted, a phenomenon known as myocardial disarray. HCM also causes disruptions of the electrical functions of the heart. HCM is most commonly due to a mutation in one of nine sarcomeric genes that results in a mutated protein in the sarcomere, the primary component of the myocyte (the muscle cell of the heart). These are predominantly single-point missense mutations in the genes for beta-myosin heavy chain (MHC), myosin-binding protein C, cardiac troponinT, or tropomyosin. These mutations cause myofibril and myocyte structural abnormalities and possible deficiencies in force generation. Not to be confused with dilated cardiomyopathy or any other cardiomyopathy.While most literature so far focuses on European, American, and Japanese populations, HCM appears in all ethnic groups. The prevalence of HCM is about 0.2% to 0.5% of the general population.
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