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Ch 35 PowerPoint - Damien Rutkoski
Ch 35 PowerPoint - Damien Rutkoski

... Mimic natural chemicals in the brain such as Endorphins, which normally help to overcome sensations of pain. If the user attempts to stop taking these drugs, the body cannot produce enough of the natural endorphins that are needed to prevent the user from the uncontrollable pain and sickness that ac ...
Muscular Disorders and Diseases
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... A few hours after a person or animal dies, the joints of the body stiffen and become locked in place. This stiffening is called rigor mortis. Depending on temperature and other conditions, rigor mortis lasts approximately 72 hours. The phenomenon is caused by the skeletal muscles partially contracti ...
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Cognitive neuroscience lecture

... Sodium ions are concentrated on the outside of the axon membrane. Potassium ions are concentrated on the inside of the axon membrane. Ion channels are closed. The inside of the axon membrane is more negative that is the outside. ...
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Lecture #13 – Animal Nervous Systems

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Axon - Cloudfront.net

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Simplified view of how a neuron sends a signal

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Understanding the Transmission of Nerve Impulses
Understanding the Transmission of Nerve Impulses

... called ions. The important ions in the nervous system are sodium and  potassium  (both  have  1  positive  charge,  +),  calcium  (has  2  positive  charges,  ++)  and  chloride  (has  a  negative  charge,  ‐).  There  are  also  some  negatively  charged  protein  molecules.  It  is  also  importan ...
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SECTION 3 - THE NERVOUS SYSTEM AND SENSORY

... into the neuron cell cytoplasm – with ion gates closed. During the production of an action potential, the pumps remain “on” while the sodium channels open first allowing the initial entry of sodium ions into the neuron (depolarization), followed immediately by the potassium channels opening with pot ...
section 3 - the nervous system and sensory physiology
section 3 - the nervous system and sensory physiology

... into the neuron cell cytoplasm – with ion gates closed. During the production of an action potential, the pumps remain “on” while the sodium channels open first allowing the initial entry of sodium ions into the neuron (depolarization), followed immediately by the potassium channels opening with pot ...
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End-plate potential



End plate potentials (EPPs) are the depolarizations of skeletal muscle fibers caused by neurotransmitters binding to the postsynaptic membrane in the neuromuscular junction. They are called ""end plates"" because the postsynaptic terminals of muscle fibers have a large, saucer-like appearance. When an action potential reaches the axon terminal of a motor neuron, vesicles carrying neurotransmitters (mostly acetylcholine) are exocytosed and the contents are released into the neuromuscular junction. These neurotransmitters bind to receptors on the postsynaptic membrane and lead to its depolarization. In the absence of an action potential, acetylcholine vesicles spontaneously leak into the neuromuscular junction and cause very small depolarizations in the postsynaptic membrane. This small response (~0.5mV) is called a miniature end plate potential (MEPP) and is generated by one acetylcholine-containing vesicle. It represents the smallest possible depolarization which can be induced in a muscle.
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