The Blood - SVSD SharePoint Web Site
... production of erythrocytes. Normally we have small amounts of erythropoietin in our blood circulation at all times and RBC’s are constantly being produced. The liver produces some of this hormone but most of it is made in the kidneys When blood levels and oxygen decline for any reason the kidn ...
... production of erythrocytes. Normally we have small amounts of erythropoietin in our blood circulation at all times and RBC’s are constantly being produced. The liver produces some of this hormone but most of it is made in the kidneys When blood levels and oxygen decline for any reason the kidn ...
Chapter 11 Quiz Show - Madison County Schools
... smooth muscle cells, and endothelial cells to make necessary repairs in damaged vessels? ...
... smooth muscle cells, and endothelial cells to make necessary repairs in damaged vessels? ...
Histology Lymphoid system General Concepts Functions Provides
... 1) Capsule of connective tissue surrounds the node and sends short trabeculae into the node. Reticular connective tissue forms the stroma for the remainder of the node. 2) Outer zone. Filled primarily with lymphoid nodules composed of B lymphocytes. 3) Inner zone (paracortex or deep cortex). Filled ...
... 1) Capsule of connective tissue surrounds the node and sends short trabeculae into the node. Reticular connective tissue forms the stroma for the remainder of the node. 2) Outer zone. Filled primarily with lymphoid nodules composed of B lymphocytes. 3) Inner zone (paracortex or deep cortex). Filled ...
Systemic_Lupus_Erythematosus
... – 10-year survival rates ranging from 75-85% – > 90% of patients surviving more than 5 years • Early deaths: – Infection – Active disease – Thrombosis – Renal failure • Late complications: – Atherosclerosis – Osteoporosis – Avascular bone necrosis – Dementia ...
... – 10-year survival rates ranging from 75-85% – > 90% of patients surviving more than 5 years • Early deaths: – Infection – Active disease – Thrombosis – Renal failure • Late complications: – Atherosclerosis – Osteoporosis – Avascular bone necrosis – Dementia ...
New Title - cloudfront.net
... elliptocytosis. This sometimes causes mild anemia but usually requires no treatment. People with sickle-shaped red blood cells have sickle cell disease, another inherited disorder and the most serious of the three disorders. Sickle cell disease can cause severe anemia, organ damage, deformities, and ...
... elliptocytosis. This sometimes causes mild anemia but usually requires no treatment. People with sickle-shaped red blood cells have sickle cell disease, another inherited disorder and the most serious of the three disorders. Sickle cell disease can cause severe anemia, organ damage, deformities, and ...
BLOOD PHYSIOLOGY
... A hypertonic extracellular solution is one in which the water concentration is less outside the cell than inside; water leaves the cell; cell volume decreases. An isotonic extracellular solution is one in which the water concentration is the same inside and outside the cell; no water movement; cell ...
... A hypertonic extracellular solution is one in which the water concentration is less outside the cell than inside; water leaves the cell; cell volume decreases. An isotonic extracellular solution is one in which the water concentration is the same inside and outside the cell; no water movement; cell ...
Dental Disease in Dogs - PetCare Veterinary Hospital
... called plaque or biofilm. Some of this plaque is removed naturally by the dog's tongue and chewing habits. If allowed to remain on the tooth surface, the plaque thickens and becomes mineralized. Mineralized plaque forms tartar and as the tartar thickens further it becomes calculus. The tartar accumu ...
... called plaque or biofilm. Some of this plaque is removed naturally by the dog's tongue and chewing habits. If allowed to remain on the tooth surface, the plaque thickens and becomes mineralized. Mineralized plaque forms tartar and as the tartar thickens further it becomes calculus. The tartar accumu ...
New Title - AIS IGCSE Science
... elliptocytosis. This sometimes causes mild anemia but usually requires no treatment. People with sickle-shaped red blood cells have sickle cell disease, another inherited disorder and the most serious of the three disorders. Sickle cell disease can cause severe anemia, organ damage, deformities, and ...
... elliptocytosis. This sometimes causes mild anemia but usually requires no treatment. People with sickle-shaped red blood cells have sickle cell disease, another inherited disorder and the most serious of the three disorders. Sickle cell disease can cause severe anemia, organ damage, deformities, and ...
Managing patients with rare blood groups
... Anti-Inb; red cells were typed as In(a+b-) Might have been immunized during pregnancies (two) Sustained antibody level may presumably be due to a constant stimulus through her underlying cancer as CD44 antigen is known to be a tumor antigen, and that Ina/Inb are on CD44 molecules. > 2000 blood units ...
... Anti-Inb; red cells were typed as In(a+b-) Might have been immunized during pregnancies (two) Sustained antibody level may presumably be due to a constant stimulus through her underlying cancer as CD44 antigen is known to be a tumor antigen, and that Ina/Inb are on CD44 molecules. > 2000 blood units ...
B antigen
... • Anti-D can also be stimulated by pregnancy with an Rh(D) positive baby – Sensitisation can be prevented by the use of anti-D ...
... • Anti-D can also be stimulated by pregnancy with an Rh(D) positive baby – Sensitisation can be prevented by the use of anti-D ...
Blood physiology
... - problems occur if the 2nd baby is also Rh+ - antibodies from mother´s blood enter blood of the fetus through the placenta, attach to baby´s Er - agglutination and hemolysis of Er of the fetus ...
... - problems occur if the 2nd baby is also Rh+ - antibodies from mother´s blood enter blood of the fetus through the placenta, attach to baby´s Er - agglutination and hemolysis of Er of the fetus ...
Cord Blood Leucocyte Expression of Functionally Significant
... hypothesize that reduced IL-2Rb may curtail early autocrine IL-2 activation of immune responses in the newborn. This hypothesis was supported by the observation that an increased proportion of stimulated T cells from newborns produced IL-2 at 4 h poststimulation, but at 24 h the proportion was lower ...
... hypothesize that reduced IL-2Rb may curtail early autocrine IL-2 activation of immune responses in the newborn. This hypothesis was supported by the observation that an increased proportion of stimulated T cells from newborns produced IL-2 at 4 h poststimulation, but at 24 h the proportion was lower ...
Massive haemoptysis - e-SAFE
... Pulmonary AVMs are developmental abnormalities causing persistent communication between arteries and veins within the lung therefore bypassing the pulmonary capillary bed. 95% derive their blood supply from branches of the pulmonary artery but others have a systemic supply from the aorta, subclavian ...
... Pulmonary AVMs are developmental abnormalities causing persistent communication between arteries and veins within the lung therefore bypassing the pulmonary capillary bed. 95% derive their blood supply from branches of the pulmonary artery but others have a systemic supply from the aorta, subclavian ...
Resolution
... • Acidification of phagosomes by H+ ion pumps • Macrophages make lysozyme and various other hydrolytic enzymes that do not require oxygen • Defensins – Cysteine-rich cationic peptides containing 29-35 aa residues – Tend to cause bacterial permeability and osmotic lysis ...
... • Acidification of phagosomes by H+ ion pumps • Macrophages make lysozyme and various other hydrolytic enzymes that do not require oxygen • Defensins – Cysteine-rich cationic peptides containing 29-35 aa residues – Tend to cause bacterial permeability and osmotic lysis ...
INFLAMMATION and YOUR HEALTH
... Duration: Few days Outcome: Resolution, abscess, or chronic inflammation ...
... Duration: Few days Outcome: Resolution, abscess, or chronic inflammation ...
Stress as a Risk Factor for Cardiovascular Events - e
... Almost eight decades passed after the first definition of the term “stress”. In 1936 Hans Selye defined this term as “the nonspecific response of the body to any demand for change” which was later referred to as “general adaptation syndrome”. Simply put, this model suggests that all stimuli are “str ...
... Almost eight decades passed after the first definition of the term “stress”. In 1936 Hans Selye defined this term as “the nonspecific response of the body to any demand for change” which was later referred to as “general adaptation syndrome”. Simply put, this model suggests that all stimuli are “str ...
I. BACTERIA Percent Shift from Gram Positive (facultative) to Gram
... “Porphyromonas gingivalis, one of the major causative agents of periodontal diseases, produces large amounts of arginine- and lysine-specific cysteine proteinases in cell-associated and secretory forms, which are now referred to as Arg-gingipain (abbreviated Rgp or Arg-X) and Lys-gingipain (abbrevi ...
... “Porphyromonas gingivalis, one of the major causative agents of periodontal diseases, produces large amounts of arginine- and lysine-specific cysteine proteinases in cell-associated and secretory forms, which are now referred to as Arg-gingipain (abbreviated Rgp or Arg-X) and Lys-gingipain (abbrevi ...
Diskrepansi Sistem Golongan Darah ABO
... •The differences in human blood are due to the presence or absence of certain protein molecules called antigens and antibodies. •The antigens are located on the surface of the RBCs and the antibodies are in the blood plasma. •Individuals have different types and combinations of these molecules. •The ...
... •The differences in human blood are due to the presence or absence of certain protein molecules called antigens and antibodies. •The antigens are located on the surface of the RBCs and the antibodies are in the blood plasma. •Individuals have different types and combinations of these molecules. •The ...
The Blood Group Systems
... •The differences in human blood are due to the presence or absence of certain protein molecules called antigens and antibodies. •The antigens are located on the surface of the RBCs and the antibodies are in the blood plasma. •Individuals have different types and combinations of these molecules. •The ...
... •The differences in human blood are due to the presence or absence of certain protein molecules called antigens and antibodies. •The antigens are located on the surface of the RBCs and the antibodies are in the blood plasma. •Individuals have different types and combinations of these molecules. •The ...
Immunology and Blood Groups
... down by the body's natural processes, their number diminishes and protection is slowly lost. For example, antibodies from a mother can cross the placenta and enter her foetus. In this way they provide protection for the baby until its own immune system is fully functional. Passive immunity may also ...
... down by the body's natural processes, their number diminishes and protection is slowly lost. For example, antibodies from a mother can cross the placenta and enter her foetus. In this way they provide protection for the baby until its own immune system is fully functional. Passive immunity may also ...
A. Anemia caused by decreased production of red blood cells
... and intracellular degradation. In this way iron stuck in macrophages and can not be used for Hb synthesis. ...
... and intracellular degradation. In this way iron stuck in macrophages and can not be used for Hb synthesis. ...
2 - JPC
... Ohio, and Mississippi River valleys.6 It is soil-borne and prefers nitrogen-rich organic matter such as bird and bat excrement.3 The disease is non-contagious and affects humans as well as a wide variety of animals.7 Infection is usually subclinical without signs or lesions, and can result in a late ...
... Ohio, and Mississippi River valleys.6 It is soil-borne and prefers nitrogen-rich organic matter such as bird and bat excrement.3 The disease is non-contagious and affects humans as well as a wide variety of animals.7 Infection is usually subclinical without signs or lesions, and can result in a late ...
Title of Presentation
... drainage (IJ, VRT and or AZG VV) alter cerebral venous flow patterns and pressure. These alterations cause: • Increased expression of endothelial adhesion molecules, chemokines, cytokines, and prothrombotic factors. Increased vsmc injury response and generation of oxygen-derived free radicals Adhere ...
... drainage (IJ, VRT and or AZG VV) alter cerebral venous flow patterns and pressure. These alterations cause: • Increased expression of endothelial adhesion molecules, chemokines, cytokines, and prothrombotic factors. Increased vsmc injury response and generation of oxygen-derived free radicals Adhere ...
Atherosclerosis
Atherosclerosis (also known as arteriosclerotic vascular disease or ASVD) is a specific form of arteriosclerosis in which an artery wall thickens as a result of invasion and accumulation of white blood cells (WBCs) (foam cell) and proliferation of intimal smooth muscle cell creating a fibrofatty plaque.The accumulation of the WBCs is termed ""fatty streaks"" early on because of the appearance being similar to that of marbled steak. These accumulations contain both living, active WBCs (producing inflammation) and remnants of dead cells, including cholesterol and triglycerides. The remnants eventually include calcium and other crystallized materials within the outermost and oldest plaque. The ""fatty streaks"" reduce the elasticity of the artery walls. However, they do not affect blood flow for decades because the artery muscular wall enlarges at the locations of plaque. The wall stiffening may eventually increase pulse pressure; widened pulse pressure is one possible result of advanced disease within the major arteries.Atherosclerosis is therefore a syndrome affecting arterial blood vessels due to a chronic inflammatory response of WBCs in the walls of arteries. This is promoted by low-density lipoproteins (LDL, plasma proteins that carry cholesterol and triglycerides) without adequate removal of fats and cholesterol from the macrophages by functional high-density lipoproteins (HDL). It is commonly referred to as a ""hardening"" or furring of the arteries. It is caused by the formation of multiple atheromatous plaques within the arteries.The plaque is divided into three distinct components: The atheroma (""lump of gruel"", from Greek ἀθήρα (athera), meaning ""gruel""), which is the nodular accumulation of a soft, flaky, yellowish material at the center of large plaques, composed of macrophages nearest the lumen of the artery Underlying areas of cholesterol crystals Calcification at the outer base of older or more advanced lesions.Atherosclerosis is a chronic disease that remains asymptomatic for decades. Atherosclerotic lesions, or atherosclerotic plaques, are separated into two broad categories: Stable and unstable (also called vulnerable). The pathobiology of atherosclerotic lesions is very complicated, but generally, stable atherosclerotic plaques, which tend to be asymptomatic, are rich in extracellular matrix and smooth muscle cells. On the other hand, unstable plaques are rich in macrophages and foam cells, and the extracellular matrix separating the lesion from the arterial lumen (also known as the fibrous cap) is usually weak and prone to rupture. Ruptures of the fibrous cap expose thrombogenic material, such as collagen, to the circulation and eventually induce thrombus formation in the lumen. Upon formation, intraluminal thrombi can occlude arteries outright (e.g., coronary occlusion), but more often they detach, move into the circulation, and eventually occlude smaller downstream branches causing thromboembolism. Apart from thromboembolism, chronically expanding atherosclerotic lesions can cause complete closure of the lumen. Chronically expanding lesions are often asymptomatic until lumen stenosis is so severe (usually over 80%) that blood supply to downstream tissue(s) is insufficient, resulting in ischemia.These complications of advanced atherosclerosis are chronic, slowly progressive and cumulative. Most commonly, soft plaque suddenly ruptures (see vulnerable plaque), causing the formation of a thrombus that will rapidly slow or stop blood flow, leading to death of the tissues fed by the artery in approximately five minutes. This catastrophic event is called an infarction. One of the most common recognized scenarios is called coronary thrombosis of a coronary artery, causing myocardial infarction (a heart attack). The same process in an artery to the brain is commonly called stroke. Another common scenario in very advanced disease is claudication from insufficient blood supply to the legs. Atherosclerosis affects the entire artery tree, but mostly larger, high-pressure vessels such as the coronary, renal, femoral, cerebral, and carotid arteries. These are termed ""clinically silent"" because the person having the infarction does not notice the problem and does not seek medical help, or when they do, physicians do not recognize what has happened.