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FACIAL NERVE TRAUMA
FACIAL NERVE TRAUMA

... Grafting and Nerve Transfer Overview Approach is based on availability of ...
Neurons
Neurons

... and postsynaptic membranes are in close apposition. When the action potential invades the axon terminals, it (1) causes voltagegated Ca2+ channels to open, which (2) triggers vesicles to bind to the presynaptic membrane. Then, (3) neurotransmitter is released into the synaptic cleft by exocytosis an ...
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Nissl substance and cellular structures involved in the intraneuronal
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... cells capture an extracellular material by means of endocytosis. In MRN, however, the vesicular transport between perikarion and the neuronal membrane is restricted by the fibrillar envelope surrounding the cell body. Rather few vesicles are observed within this fibrillar layer. Vesicles approach th ...
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... intercellular space with filamentous material, which is contiguous with submembrane cytoskeletal elements. • A receptor system highly expressed in this stage of development: astrostatin (neuronal glycoprotein). • Earlier studies: the function of radial glia have stemmed from another neurological mou ...
Autonomic Nervous System
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Acutouch Therapy
Acutouch Therapy

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Stimulus Response Time Lab
Stimulus Response Time Lab

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Chapter 12: Spinal Cord And Spinal Nerves
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NMSI - 4 Central Nervous System

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Case Study 29 - University of Pittsburgh
Case Study 29 - University of Pittsburgh

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Sense Organs - human anatomy
Sense Organs - human anatomy

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Endocrine and nervous system
Endocrine and nervous system

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Ch 48-49 Reading Guide
Ch 48-49 Reading Guide

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hydroxytryptamine-containing neurons in the snail Effect of

... ganglia of untreated controls, but if the treatment with nerve growth factor was stopped the enzyme activities of both the operated and control ganglia returned within 6 weeks to the same values as in animals who had received no treatment with nerve growth factor. Thus nerve growth factor was unable ...
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Neuroregeneration

Neuroregeneration refers to the regrowth or repair of nervous tissues, cells or cell products. Such mechanisms may include generation of new neurons, glia, axons, myelin, or synapses. Neuroregeneration differs between the peripheral nervous system (PNS) and the central nervous system (CNS) by the functional mechanisms and especially the extent and speed. When an axon is damaged, the distal segment undergoes Wallerian degeneration, losing its myelin sheath. The proximal segment can either die by apoptosis or undergo the chromatolytic reaction, which is an attempt at repair. In the CNS, synaptic stripping occurs as glial foot processes invade the dead synapse.Nervous system injuries affect over 90,000 people every year. It is estimated that spinal cord injuries alone affect 10,000 each year. As a result of this high incidence of neurological injuries, nerve regeneration and repair, a subfield of neural tissue engineering, is becoming a rapidly growing field dedicated to the discovery of new ways to recover nerve functionality after injury. The nervous system is divided into two parts: the central nervous system, which consists of the brain and spinal cord, and the peripheral nervous system, which consists of cranial and spinal nerves along with their associated ganglia. While the peripheral nervous system has an intrinsic ability for repair and regeneration, the central nervous system is, for the most part, incapable of self-repair and regeneration. There is currently no treatment for recovering human nerve function after injury to the central nervous system. In addition, multiple attempts at nerve re-growth across the PNS-CNS transition have not been successful. There is simply not enough knowledge about regeneration in the central nervous system. In addition, although the peripheral nervous system has the capability for regeneration, much research still needs to be done to optimize the environment for maximum regrowth potential. Neuroregeneration is important clinically, as it is part of the pathogenesis of many diseases, including multiple sclerosis.
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