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Immune System Concept Maps
Immune System Concept Maps

... 2. IMMUNE RESPONSE, PRIMARY IMMUNE RESPONSE, PATHOGEN, ANTIGEN, TCELL, B-CELL, ANTIBODY, PLASMA CELLS, MEMORY B-CELLS, ANTIBODIES, PHAGOCYTES, KILLER T-CELL (CYTOTOXIC T-CELL), HELPER T-CELL, SECONDARY IMMUNE RESPONSE, SUPPRESSOR T-CELL (10 points) ...
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Powerpoint version

... Combining non-specific and adaptive immune response Bacterial infection: At first: phagocytes, histamine release, inflammatory response ...
Name of Student:
Name of Student:

... them on MCH II by antigen presenting cells. Fibroblasts are non-professional antigen presenting cells that express specific MCH class II. Although not ubiquitously expressed, they can be induced to express IDO on stimulation with interferon-gamma (IFN-γ). We hypothesized that Treg cells upregulated ...
Mediators of Immediate Hypersensitivity
Mediators of Immediate Hypersensitivity

... ANCA, Antineutrophil cytoplasmic antibodies; TSH, thyroid-stimulating hormone. Complement mediated damage. Complement activation on cells also leads to the formation of the membrane attack complex, which disrupts membrane integrity by “drilling holes” through the lipid bilayer, thereby causing osmot ...
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... O Lamprey and Hagfish have a distinct lymphocyte derived molecule. O These molecules are believed to bind pathogenic antigens in a similar way to antibodies. ...
Distinguished Visitor Programme
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... of germline-encoded receptors to recognize conserved molecular patterns that occur on the surfaces of a broad range of pathogens. The B and T lymphocytes of the specific immune response use complex gene rearrangement machinery to generate a wide diversity of antigen receptors capable of recognizing ...
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... – Exposure of normally inaccessible self-antigens sometimes induces an immune attack against these antigens – Normal self-antigens may be modified by factors such as drugs, environmental chemicals, viruses, or genetic mutations so that they are no longer recognized and tolerated by the immune system ...
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BiTE® ANTIBODIES: Designed to Bridge T Cells

... and graft versus tumor effect6–9 2. Adoptive Cell Therapy: Adoptive cell therapy utilizes autologous antitumor activity of cells such as tumor-infiltrating lymphocytes, to treat cancer10 3. Immunovirus: Using a modified virus that has the potential to induce tumor cell lysis through replication with ...
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... • once activated, CD8+ will leave the lymph node and home towards the site of infection and conduct its cytotoxic activity towards infected cells via release the cytotoxins perforin, granzymes, and granulysin • Through the action of perforin, granzymes enter the cytoplasm of the target cell and thei ...
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... 1. Where do lymphocytes develop? Bone marrow 2. Where do T cells mature? Thymus 3. Where do B cells mature? Bone marrow 4. Where does the word ‘humoral’ originate from and what does it mean? Humor = latin for body fluid, circulate in body fluids. 5. Which type of cell does HIV attack? Helper T cells ...
Autoimmune Disease - Harvard Life Science Outreach Program
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... Helper T cells recognize non self antigens and stimulate B cells to produce antibodies  B cells release antibodies which bind to non self antigens present on infected cells  B cells complete their maturation upon binding to non self antigens and destroying infected cells ...
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... b. diverse gene families (3 genes encode MHC I, 3 genes encode MHC II α chain and at least 3 genes encode MHC II β chain) c. expression of MHC is co-dominant (all genes of a given class (i.e., class I or class II) are expressed simultaneously on a single cell) 5. Exam II material commences from here ...
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... Growth factors play a wide-ranging role in a variety of cellular processes. As the name suggests, they often help regulate the growth and expansion of specific types of cells. That has obvious implications for cancer, which was an early target for drugs designed to inhibit the effects of growth fact ...
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Cancer immunotherapy



Cancer immunotherapy (immuno-oncology) is the use of the immune system to treat cancer. Immunotherapies fall into three main groups: cellular, antibody and cytokine. They exploit the fact that cancer cells often have subtly different molecules on their surface that can be detected by the immune system. These molecules, known as cancer antigens, are most commonly proteins, but also include molecules such as carbohydrates. Immunotherapy is used to provoke the immune system into attacking the tumor cells by using these antigens as targets.Antibody therapies are the most successful immunotherapy, treating a wide range of cancers. Antibodies are proteins produced by the immune system that bind to a target antigen on the cell surface. In normal physiology the immune system uses them to fight pathogens. Each antibody is specific to one or a few proteins. Those that bind to cancer antigens are used to treat cancer. Cell surface receptors are common targets for antibody therapies and include the CD20, CD274, and CD279. Once bound to a cancer antigen, antibodies can induce antibody-dependent cell-mediated cytotoxicity, activate the complement system, or prevent a receptor from interacting with its ligand, all of which can lead to cell death. Multiple antibodies are approved to treat cancer, including Alemtuzumab, Ipilimumab, Nivolumab, Ofatumumab, and Rituximab.Cellular therapies, also known as cancer vaccines, usually involve the removal of immune cells from the blood or from a tumor. Immune cells specific for the tumor are activated, cultured and returned to the patient where the immune cells attack the cancer. Cell types that can be used in this way are natural killer cells, lymphokine-activated killer cells, cytotoxic T cells and dendritic cells. The only cell-based therapy approved in the US is Dendreon's Provenge, for the treatment of prostate cancer.Interleukin-2 and interferon-α are examples of cytokines, proteins that regulate and coordinate the behaviour of the immune system. They have the ability to enhance anti-tumor activity and thus can be used as cancer treatments. Interferon-α is used in the treatment of hairy-cell leukaemia, AIDS-related Kaposi's sarcoma, follicular lymphoma, chronic myeloid leukaemia and malignant melanoma. Interleukin-2 is used in the treatment of malignant melanoma and renal cell carcinoma.
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