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CHRONIC SPECIFIC BONE INFECTION DR.MARWAN ZAMZAMI,ABOS CHRONIC SPECIFIC BONE INFECTION TUBERCULOSIS BRUCELLOSIS FUNGAL SYPHILIS TUBERCULOSIS THE BASIC MICROSCOPIC LESION; THE TUBERCLE FIRST DISCOVERED BY THE FRENCH PHYSICIAN LAENNEC (1781-1826) WHO DIED AT THE AGE OF 45 BY TB TUBERCULOSIS ESTIMATED 30 MILLION TB PATIENTS WORLD WIDE. 1 -3 % (300 000 – 1000 000) HAVE SKELETAL INVOLVEMENT TUBERCULOSIS OF BONES AND JOINTS TB Bacilli lived in symbiosis with mankind since time immemorial. Recorded in ancient Egyptian mummies Still common in developing countries REDUCED INCIDENCE OF TB DUE TO: IMPROVED LIVING STANDARDS; SANITATION, HYGIENE, NUTRITION B.C.G. VACCINE (80% PROTECTION) TUBERCULOSIS BACILLI MYCOBACTERIUM TUBERCULOSIS BOVINE UNPASTEURISED MILK HUMAN MORE COMMON OTHERS LESS COMMON TUBERCULOSIS GROUPS AT RISK NON AFFLUENT COUNTRIES OVER GROWING MALNUTRITION, POOR YOUNGER AGE AFFLUENT COUNTRIES IMMUNE DEFICIENT STEROIDS ANTICA OLDER AGE TUBERCULOSIS FACTORS FAVORING LOCALISATION BLOOD SUPPLY AND STAGNATION LOCAL TRAUMA; HAEMATOMA? LOCAL STEROIDS ? TB PATHOLOGY Secondary to other primary TB lesions (Pulm., GL, Renal, LN) Route of spread: HAEMATOGENOUS **** DIRECT (much less) * bone to joint * soft tissue to bone THE PRIMARY LESION QUIESCENT ACTIVE: (Apparent, Latent) TB PATHOLOGY INFLAMMATION HYPEREMIA - OSTEOPENIA TB FOLLICLES (TUBERCLE): LYPHOCYTE – MONOCYTES ENDOTHELIAL CELLS LANGHANS GIANT CELLS COALESCE CASEATION LATER GRANULATION TISSUE BONE DISTRUCTION SINUSES TB Follicle TB PATHOLOGY (JOINTS) SYNOVIAL SWELLING GRANULATION TISSUE PERIPHERAL ARTICULAR DESTRUCTION NO PROTEOLYTIC ENZYMES CENTRAL ARTICULAR WEIGHTBEARING AREA PRESERVED RICE BODIES FIBRIN & ARTICULAR CARTILAGE INCREASED BLOOD SUPPLY OSTEOPENIA CLINICAL PICTURE AGE INSIDIOUS ONSET MONO ARTICULAR OTHER LESIONS FAMILY HISTORY – CONTACT GROUPS AT RISK SYMPTOMS & SIGNS CONSTITUTIONAL LOW GRADE FEVER ANOREXIA WEIGHT LOSS NIGHT SWEATING TACHYCARDIA ANEMIA SYMPTOMS & SIGNS LOCAL Symptoms : PAIN NIGHT CRIES SWELLING STIFFNESS ULTERED FUNCTION Signs : WASTING SYNOVIAL SWELLING TENDERNESS WARM STIFFNESS LIMPING INVESTIGATIONS LEUCOPENIA – LYMPHOCYTOSIS ANEMIA RAISED ESR MANTOUX POSITIVE NOT IN: MILIARY TB / RECENTLY VACCINATED/ ON STEROIDS / REDUCED IMMUNITY / FEVER RADIOLOGY CHEST X-RAY : ALL PATIENTS JOINTS: PHEMISTER’S TRIAD: 1. PERIARTIC. OSTEOPENIA 2. REDUCED JOINT SPACE 3. PERIPH. OSSEOUS EROSIONS BONES: 1. DESTRUCTION 2. SEQUESTRATION 3. ABSCESS FORMATION RADIOLOGY BONE SCAN: MONO – ARTICULAR CF: RHEUMATOID ARTHRITIS CALLIUM SCAN INFECTION CT SCAN MORPHOLOGY MRI MORPHOLOGY DIAGNOSTIC ASPIRATION: AFB POSITIVE HISTOLOGICAL CULTURE TB SPINE SURGICAL PATHOLOGY FIRST THREE DECADES THORACO-LUMBAR CENTRAL SPINE SPARKS POSTERIOR ELEMENTS SPREADEDS UP/DOWN ANT./POST. LONG. LIGS. LESIONS COALESCE – COLLAPSE KYPHUS FORMATION TB SPINE SURGICAL PATHOLOGY PARA VERTEBRAL ABSCESS CERVICAL : RETROPHARALYGEAL THORACIC : P.V. & ALONG RIBS LUMBAR : PSOAS ABSCESS POSTERIOR:LUMBAR TRIANGLE ANTERIOR: ILIAC FOSSA BELOW ING. LIG. NEUROLOGICAL COMPLICATION MORE IN THORACIC (NARROWEST CANAL) TB SPINE CLINICAL FEATURES GENERAL: INSIDIOUS ONSET CONSTITUTIONAL LOCAL: PAIN – FIRST INDICATION LOCAL – REFERRED STIFFNESS – SPASM WEAKNESS – NEUROLOGICAL SIGNS OF TB SPINE MUSCLE SPASM KHPHUS – GIBBOUS TENDERNESS STIFFNESS PARA VERTEBRAL ABSCESS NEUROLOGICAL – WEAKNESS PARAPLEGIA TB SPINE RADIOLOGICAL FEATURES DISC NOT INVOLVED PRIMARILY NARROWING OF DISC SPACE BONE DESTRUCTION USUALLY TWO ADJACENT VERTEBRAE MAY SHOW SKIP LESIONS PARA VERTEBRAL ABSCESS KHYPUS CT/MYELOGRAM/MRI IN PARAPLEGIA PARAPLEGIA IN TB SPINE IN 10-30% OF TB SPINE MORE IN THORACIC REGION PRESSURE ON CORD ANTERO LATERAL MOTOR EARLIER THAN SENSORY SIGNS: UPPER MOTOR NEURON MAY START BY CORD SHOCK REMARKABLE ABILITY TO RECOVER PARAPLEGIA IN TB SPINE CAUSED BY EXTRADURAL PRESSURE GRANULATION TISSUE PRESSURE OF ABSCESS & CASEATON SEQUESTRUM PATHOLOGICAL FRACTURE/DISLOC. SEVERE KYPHUS INFLAMMATION: TOXIC EDEMA VASCULAR MANAGEMENT OF TB SPINE USUALLY CONSERVATIVE GENERAL SPECIFIC REST IMMOBILISE CHEMOTHERAPY SURGICAL DIAGNOSE ASPIRATION DRAIN ABSCESS DEBRIDE DECOMPRESS ANTERIOR ANTERO-LATERAL STABILISE FUSION MOST CASES OF TB SPINE RESPOND VERY WELL TO CONSERVATIVE TREATMENT INCLUDING THOSE WITH PARAPLEGIA THE NEED FOR SURGICAL DECOMPRESSION OF THE CORD IS LIMITED BRUCELLOSIS MILK AND MILK PRODUCTS BACK PAIN AND STIFFNESS MUSCLE SPASM FEVER – MILD SACRO-ILIAC JOINT LESS DESTRUCTIVE OF TB BRUCELLA TITRE ANTIBIOTICS e.g. SEPTRIN - OXYTETRACYCLINE SYPHILIS SPIROCHETE TREPONEMA PALLIDUM CONGENITAL SYPHILIS – COMMONEST CHRONIC OSTEOCHONDRITIS PERIOSTEITIS OSTEITIS TIBIA LESABRE TIBIA FUNGAL INFECTION CHRONIC – VERY LOW GRADE FEET – FARMERS – THORNS Madura Foot SLOW DESTRUCTION SINUSES – GRANULES SECONDARY BACTERIAL INFECTION RESISTANT TO CHEMOTHERAPY NEEDS SURGICAL DEBRIDEMENT IF ADVANCED MAY NEED AMPUTATION