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Critical Care Nursing Fourth Class/ Nursing College Dr.Huda B.Hassan Critical Care Nursing (1) Nurse as Patient Advocate Evolution of Critical Care • Beginnings with polio units, recovery room, and coronary care units • Patient outcomes improved Critical Care in 21st Century Professional Organizations American Association of Critical-Care Nurses (AACN) – AACN’s Vision Create a healthcare system driven by patient’s and family’s needs in which critical care nurses make their optimum contributions Critical Care Certification Standards of Practice (Medina, 2000) Trends and Issues • SBAR communication strategy The Critical Care Experience (2) • Critically ill patients at high risk for actual or potential life-threatening health problems • Critically ill patients cared for in a variety of settings, not just the critical care unit Critical Care Environment Healthy Work Environment What strategies can be used to enhance communication? Communication Strategies • Critically Ill Patient responses vary according to: Patients’ Recall About Critical Care: Psychosocial Support What are strategies to achieve these goals? Perceptions of Caring Practices: Critical care patients need to feel safe • Nursing interventions: Quality of Life After Critical Care • from the unit Geriatric Concerns: • • Diminished ability to adapt or cope to stressors of critical illness At greater risk for negative outcomes Family Members: Calgary Family Assessment Family Needs Family Interventions Comfort and Sedation (3) • Pain occurs from a variety of causes • • Pain – Unpleasant sensory and emotional experience associated with actual or potential tissue damage – It is what the patient says it is Anxiety • Influences anxiety Physiology Positive Effects of Pain/Anxiety • • • Increase performance levels Removes one from potential harm Fight-or-flight response Negative Effects of Pain/Anxiety: 5-Step Assessment of Pain Subjective Assessment Tools Subjective PQRST: Chest pain characteristics • P = The Face, Legs, Activity, Cry, Consolability scale or FLACC scale is a measurement used to assess pain for children between the ages of 2 months and 7 years or individuals that are unable to communicate their pain. The scale is scored in a range of 0–10 with 0 representing no pain. The scale has five criteria, which are each assigned a score of 0, 1 or 2. • • Sedation Assessment—Tools Sedation medication is given to reduce symptoms; dose is adjusted based on tools or scales – Richmond Agitation-Sedation Scale (RASS) – Ramsey Sedation Scale (Ramsey) – Sedation-Agitation Scale (SAS) Interobserver agreement in assessment using various scales is important Score +4 +3 +2 +1 0 -1 Term Combative Very agitated Agitated Restless Alert and calm Drowsy -2 Light sedation -3 -4 Moderate sedation Deep sedation -5 Unarousable Richmond Agitation Sedation Scale (RASS) * Description Overtly combative, violent, immediate danger to staff Pulls or removes tube(s) or catheter(s); aggressive Frequent non-purposeful movement, fights ventilator Anxious but movements not aggressive vigorous Not fully alert, but has sustained awakening (eyeopening/eye contact) to voice (>10 seconds) Briefly awakens with eye contact to voice (<10 seconds) Movement or eye opening to voice (but no eye contact) No response to voice, but movement or eye opening to physical stimulation No response to voice or physical stimulation Verbal Stimulation Verbal Stimulation Verbal Stimulation Physical Stimulation Physical Stimulation Sedation Assessment—Technology – Delirium • • Acutely changing mental status • Indications Nursing Care for Neuromuscular Blockad: Preventing Pain is More Effective Than Treating Pain” Management Challenges Nutritional Support(4) • • All critically ill patients are assumed to be at nutritional risk Nutritional support is an important part of overall care plan Anatomy and Physiology Review Nutritional Assessment: • Objective data in ICU patient • Subjective data in ICU patient Nutritional Therapy Goal: Nutrition Care Plan: • Delivery of nutrients to GI tract Enteral Formulas Guidelines for Enteral Feeding: Drug-Nutrient Interactions: • Check medications for compatibility with enteral feeding Nursing Care: • Monitoring Complications: Prevent Complications: Monitoring and Evaluating: Dysrhythmia Interpretation and Management(5, 6) • Dysrhythmia interpretation is a fundamental skill of critical care nurses • Crucial for early nursing and medical intervention Basic Electrophysiology: Cardiac Cycle: Cardiac Action Potential: Muscular Contraction: • Depolarization leads to contraction • Repolarization leads to resting and filling of ventricles from atria • ECG is evidence of electrical activity, not contraction Cardiac Conduction Pathway: • SA node 12-Lead ECG: • Leads None of the 12 leads record activity over the posterior of the heart Cardiac Monitoring: Analyzing Cardiac Rhythms: • Graph paper – Used to standardize tracings – Vertical boxes measure voltage/amplitude – Horizontal boxes measure time Measuring: Rate: • Waveform Configuration and Location Interval • Rhythm Interpretation: Basic Dysrhythmias: – Normal Sinus Rhythm: Normal Sinus Rhythm • Sinus Tachycardia: Assess for symptoms of low cardiac output Sinus Tachycardia Sinus Bradycardia: Sinus Bradycardia Sinus Dysrhythmia: Sinus Dysrhythmia Sinus Arrest or Exit Block: • Sinus Arrest or Exit Block: Atrial Dysrhythmias: Causes Premature Atrial Contractions: Premature Atrial Contractions • Ventilators Assistance (7,8) Essential nursing interventions Respiratory Anatomy and Physiology: Physiology of Breathing: Gas Exchange: Regulation of Breathing: Terminology: Work of Breathing (WOB): Compliance: Resistance: Lung Volumes and Capacities: • • Assess baseline function Monitor responses Volumes and Capacities Selected Measures: Assessment: Health History: Physical Examination: Palpation: Percussion: • Auscultation: • Assess breath sounds, presence of adventitious lung sounds, voice sounds Oxygenation: – ) Terms: • Hypoxemia: decreased oxygenation of arterial blood • Hypoxia: decreased oxygenation at tissue level • PaO2 and SaO2 Relationship: pH: • Concentration of hydrogen ions (H+) PaCO2: HCO3—Bicarbonate: – Buffer System: Bicarbonate Buffer System Respiratory Buffer System Buffer Systems Interpretation of ABGs: Oxygenation: • Pulse oximetry (SpO2) Assessment of Ventilation Oxygen Administration: Oxygen Delivery Devices: Airway Management: : Endotracheal Tube Insertion of an endotracheal tube through the mouth or nose – Which is preferred and why? • Used to: Verify Placement: • Nasotracheal Intubation: Tracheostomy: Tracheostomy: • Endotracheal Suctioning: Indicators for Ventilation: Positive Pressure Ventilation: • Movement of gases into lungs through positive pressure • Opposite of normal respirations Ventilator Settings: Shock, Sepsis(9,10) • Shock is a clinical syndrome • Normal Anatomy and Physiology: Pathophysiology: Stages of Shock— Stage I: Initiation • Hypoperfusion: inadequate delivery or extraction of oxygen • No obvious clinical signs • Early, reversible Stage II: Compensatory: • Sustained reduction in tissue perfusion • Initiation of compensatory mechanisms – Neural: baroreceptors and chemoreceptors – Endocrine: ACTH and ADH – Chemical • Low oxygen tension • Hyperventilation and respiratory alkalosis Stage III: Progressive: • Failure compensatory mechanisms • Profound CV cardiovascular effects – Increased hypoperfusion – Vasoconstriction • Extremity ischemia • Cellular hypoxia • Lactic acid production • Failure Na+/K+ pump • Increased capillary hydrostatic pressure • Intravascular fluid shifts – Interstitial edema – Decreased circulating intravascular volume • Decreased coronary perfusion – MDF released – Decreased myocardial contractility Stage IV: Refractory: • Prolonged inadequate tissue perfusion – Unresponsive to therapy – Contributes to multiple organ dysfunction and death Systemic Inflammatory Response Syndrome (SIRS): Shock Assessment: • Central nervous system • Cardiovascular system • Pulmonary system • Renal system • Gastrointestinal (GI) system – Slowing intestinal activity • Decreased bowel sounds, distention, nausea, and constipation • Hepatic • Hematologic • Integumentary Laboratory Values: Management: Management: – Administer supplemental oxygen – Life-sustaining therapies/end of life Mechanical Management: Specific Classifications of Shock and Management— Hypovolemic Shock: Cardiogenic Shock: • Arterial vasodilators Obstructive Shock: – Relief of compression or obstruction —Distributive Shock: Distributive Shock—Anaphylactic: • Management • Distributive Shock—Septic