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Mechanical Injuries Of Brain and Meniges ๐ 1 Traumatic Lesions ๐ 2 Alterations ๐ 1 Traumatic Lesions Extracerebral lesions Intracerebral lesions ๐ 1 Traumatic Lesions Close injury Open injury Extracerebral Lesions Epidural bleeding Subdural bleeding Subaracnoid bleeding Intraventricular bleeding Intracerebral Lesions Contusions Lacerations (or Wounds) ๐ 2 Alterations Circulatory disorder Necroses and hemorrhages Post-traumatic hydrocephalus Secondary infections Fat and air embolism Epidural Bleeding Epidural Bleeding Epidural / Extradural Hemorrhage / Hematoma Causes Skull fracture Separation of dura and skull bone Tear of a dural artery ,its branches and/or occasionally of a vein Most common site: lateral convexity of a cerebral hemisphere Location: it almost always at the site of a skull fracture Uncommon occur in the elderly Children: skull deformation with separation of the dura from the bone without skull fracture Acute hematoma: artery bleeding Delayed hematoma: venous bleeding, transient arterial spasm Progression of the bleeding Space occupying hematoma Increase intracranial pressure Confusion Alteration of consciousness Pupillary dilatation: on the hematoma side Central respiratory failure If venous bleeding ,or transient arterial spasm: Lucid interval Consciousness (may be) ,no signs of confusion: occipital poles and/or cerebellum Chronic Epidural Hematoma The hematoma spontaneously shrinks and becomes encapsulated by fibrous connective tissue. Subdural Bleeding Subdural bleeding Trauma Rupture of aneurysm Arteriovenous malformation Vein: - Tearing of one or - Several bridging vein - Insignificant trauma (sometime): abnormally located blood vessels Artery: - particularly in branches of the middle cerebral artery - severe cortical contusions and bleeding into subarachnoid space: (usually) tears of arachnoid membrane Artery: - More frequently on the side opposite the impact - (May) without brain contusions or significant subarachnoid hemorrhage Time of onset Acute: within 12 to 24 hr. Subacute: from 24 hr. to 7 d. Chronic: more than 7 d. Most Location: over the convexities and the lateral aspects of the cerebral hemisphere Often: extend over the base of frontal and temporal lobes Occasionally: between the hemisphere In skull intact: occur as often as with skull fracture Rare in the posterior cranial fossa , around the brain stem and cerebellum Chronic Subdural Hematoma Enlargement if untreat Isotonicity Local presence of fibrinolytic enzymes: bleeding tendency Subaracnoid Bleeding Subaracnoid bleeding Trauma / Nontrauma Extension of intraventricular hemorrhage Moderately severe blow to the face or forehead Sudden ,usually severe hyperextension of the head , as from a fall onto the forehead Subarachnoid over the brain stem and basal cisterns = hydrocephalus Forgetfulness , confusion , psychotic state Spasticity of the lower extremities Intraventricular bleeding Intraventricular bleeding Most often arterial in origin Trauma Non-trauma: such as rupture AVM or Aneurysms Intracerebral Lesions Contusions Lacerations (Wounds) Contusions Contusion hemorrhage Contusion necrosis Contusion tear Intracerebral Hematoma In the deeper portions of contusions More frequent in the frontal and /or temporal lobes Location: white matter > grey matter Intracerebral Hematoma Secondary rupture into the ventricular system and/or the subarachnoid space usually does not occur. Lacerations Stab wounds Gunshot wounds Gunshot wounds Shearing forces within brain tissue Expansile cavitation Distant contusions (hemorrhages) Classification of Contusions According to causative mechanism Depending on site and direction of impact : Coup , Intermediary coup , Contrecoup Independent of site and direction of impact : Fracture contusion , Gliding contusion , Herniation contusion Axonal injury Shearing forces due to blunt head injuries Focal , diffuse Early ,the areas: little or no change on gross examination of the white matter Older lesions: slightly gray pallor ๐ 2 Alterations ๐ 2 Alterations Circulatory disorder Necroses and hemorrhages Post-traumatic hydrocephalus Secondary infections Fat and air embolism Circulatory disorder Swelling of the brain: edema and cell necrosis Usually reversible Perifocal: surrounding a 1๐ brain lesion Generalize: a primary lesion , shock Other rare causes Obstruction of the superior sagittal sinus Traumatic thrombus or obstruction in internal carotid artery Necroses/Hemorrhages Vascular compression Shearing lesions Necroses/Hemorrhages Many lesion are large: such as midbrain and pons If rapidly progressing space occupying lesion: secondary lesion may appear within 30 mins. After injury Hemorrhage : sometimes small or absent Hydrocephalus Traumatic or Non-traumatic cause White matter loss following a shearing lesion and degeneration of myelinated axons Distension of ventricles by elevated pressure of the CSF Secondary infections Meningitis Intracerebral abscesses Meningitis An infected open injury caused by a foreign body A fracture in the wall of one of the cranial sinuses associated with a tear in the dura and arachnoid Intracerebral abscesses In the vicinity of the primary lesion Complication: rupture into the underlying ventricle (Pyocephalus) Fat and air embolism Primary or Secondary lesions Fat embolism: fractures , stab wound at neck Air embolism: stab wound at neck , a skull fracture lacerating a paranasal dural sinus